ライフサイエンスコーパス: MEG

1 dball paradigm using magnetoencephalography (MEG).

2 g memory task during magnetoencephalography (MEG).

3 emporal precision of magnetoencephalography (MEG).

4 itude, measured with magnetoencephalography (MEG).

5 eously measured in the magnetoencephalogram (MEG).

6 nd adolescents using magnetoencephalography (MEG).

7 s question in human subjects while recording MEG.

8 ell as a deeper fundamental understanding of MEG.

9 er processing speeds both inside and outside MEG.

10 lectrophysiological methods, such as EEG and MEG.

11 the core pathway to layer 4 recorded in the MEG.

12 bserved in recordings of human resting-state MEG.

13 enous malformation who had undergone IAP and MEG.

14 on-invasive recording techniques such as EEG/MEG.

15 act MEGS, particularly for highly imbalanced MEGS.

16 searches, or for expanding established small MEGS.

17 We used the human megakaryoblastic cell line MEG-01 as an in vitro model for human megakaryocytes and

18 forms a disulfide bond with beta-actin when MEG-01 cells adhere via the alphaIIbbeta3 integrin to fi

19 nduced a sustained increase in [Ca(2+)] i in Meg-01 cells and enhanced the frequency of repetitive Ca

20 Using Meg-01 cells and mouse megakaryocytes, we found that NFk

21 We labeled total RNA of MEG-01 cells by incorporation of 5-ethynyluridine (EU) a

22 We conclude that platelets and Meg-01 cells express the MS cation channel Piezo1, which

23 WDR1 knockdown (KD) in MEG-01 cells increased adhesion and spreading in both th

24 hat both proteins colocalized when spreading MEG-01 cells on fibronectin.

25 acological studies in human megakaryoblastic MEG-01 cells showed that DREAM is important for A23187-i

26 ), and found expression levels of GLP-1Rs in MEG-01 cells to be higher than those in the human lung b

27 Transfection of MEG-01 cells with (S422D)SGK1 significantly increased ph

28 Treatment of (S422D)SGK1-transfected MEG-01 cells with the IkappaB kinase inhibitor BMS-34554

29 gonist exenatide elicited a cAMP response in MEG-01 cells, and exenatide significantly inhibited thro

30 ut larger than PLPs produced from unmodified MEG-01 cells, and had significantly increased adhesion i

31 lotting, respectively, in both platelets and Meg-01 cells.

32 ocal microscopy to determine that platelets, MEG-01 megakaryoblastic cells, and bone marrow megakaryo

33 tress on Ca(2+) entry in human platelets and Meg-01 megakaryocytic cells loaded with Fluo-3 was exami

34 derived from the megakaryoblastic cell line MEG-01 stimulate proliferation of HepG2 cells.

35 Stimulation of MEG-01 with thrombin reduced levels of WDR1 transcripts

36 establish a human megakaryocytic cell line (MEG-01) as a model system for the study of dense granule

37 ) mRNA from a human megakaryocyte cell line (MEG-01), and found expression levels of GLP-1Rs in MEG-0

38 In human megakaryoblastic cells (MEG-01), transfection with constitutively active (S422D)

39 We demonstrate that MEG-1 and MEG-3 are substrates of the kinase MBK-2/DYRK

40 These results support the identification of MEG-14 as a classic intrinsically disordered protein, an

41 s analyses suggest the soluble domain of the MEG-14 protein will be largely disordered, and using syn

42 for (17)O-excessVSMOW-SLAP is 22 +/- 11 per meg (1sigma).

43 de a (17)O-excessVSMOW-SLAP of 23 +/- 10 per meg (1sigma); an average of previously reported values f

44 We demonstrate that MEG-1 and MEG-3 are substrates of the kinase MBK-2/DYRK and the ph

45 In vivo, MEG-3 forms a posterior-rich concentration gradient that

46 EX-5 is necessary and sufficient to suppress MEG-3 granule formation in vivo, and suppresses RNA-indu

47 MEG-3 is an intrinsically disordered protein that binds

48 opy on live embryos, we show that GFP-tagged MEG-3 localizes to a dynamic domain that surrounds and p

49 ormation in vivo, and suppresses RNA-induced MEG-3 phase separation in vitro.

50 -3's access to RNA, thus locally suppressing MEG-3 phase separation to drive P granule asymmetry.

51 findings suggest that MEX-5 interferes with MEG-3's access to RNA, thus locally suppressing MEG-3 ph

52 inted noncoding RNAs (ncRNAs), such as Rian, Meg-3, and Mirg, which are implicated in hepatocarcinoge

53 ced phase separation of the granule scaffold MEG-3.

54 Magnetoencephalography (MEG), a non-invasive technique for characterizing brain

55 We trained pattern classifiers on the MEG activity elicited by direct presentation of the visu

56 ould underpin well documented changes in EEG/MEG activity indicating the existence of a mirror neuron

57 The MEG analysis demonstrated that phonological training inc

58 MEG analysis was completed using a synthetic aperture ma

59 led a Watson-Crick-like pairing between O(6)-MeG and 2"-deoxythymidine-5"-[(alpha, beta)-imido]tripho

60 In both MEG and behavioral measures, task processing was optimal

61 as a central target in the response to O(6)-MeG and demonstrate that p50 is required for S(N)1-methy

62 istributed cortical activity can explain the MEG and EEG patterns generated by deep sources.

63 used parametric multiobject tracking tasks, MEG and EEG recordings, and data-driven source-space ana

64 has been largely obtained with sensor-level MEG and EEG recordings, which yield only limited anatomi

65 taneous magneto- and electroencephalography (MEG and EEG) data while subjects observed threshold-leve

66 res of neural activity, including fMRI, DTI, MEG and EEG.

67 At the group level, MEG and fMRI data showed spatial convergence in the left

68 We examined the spatial concordance between MEG and fMRI results in 16 adolescents performing a thre

69 ntered on midline regions of the DMN in both MEG and fMRI, boosting confidence in a possible pathophy

70 s of typical functional connectivity in both MEG and fMRI.

71 tion, in addition to its known importance in Meg and G/M maturation.

72 l and spatial pattern similarity analysis in MEG and intracranial EEG in a context-match paradigm.

73 Using tools combining MEG and intracranial EEG with brain connectivity analyse

74 alyses of high-gamma activity recorded using MEG and intracranial EEG.

75 w timescales using both source-reconstructed MEG and intracranial stereotactical electroencephalograp

76 Both MEG and SEEG revealed avalanche dynamics that were chara

77 Remarkably, one-third of MEGs and nearly one-half of PEGs were associated with gr

78 Our data further reveal that MEGs and PEGs differ in their targeting by 24-nucleotide

79 Our data indicate that both MEGs and PEGs in rice regulate nutrient metabolism and e

80 t mechanisms establishing DNA methylation at MEGs and PEGs.

81 Most MEGs and some PEGs were expressed specifically in the en

82 Using magnetoencephalography (MEG) and a tactile temporal discrimination task in human

83 noninvasively, using magnetoencephalography (MEG) and electroencephalography (EEG).

84 re we acquired human magnetoencephalography (MEG) and functional magnetic resonance imaging (fMRI) re

85 question using human magnetoencephalography (MEG) and multivariate analyses of instantaneous activity

86 ological relevance, O(6)-methylguanine (O(6)-MeG) and O(6)-carboxymethylguanine (O(6)-CMG), were char

87 elopment of primary cultured megakaryocytes (MEG) and primary erythroblasts (ERY) from murine fetal l

88 e identified 162 maternally expressed genes (MEGs) and 95 paternally expressed genes (PEGs), which we

89 oximately 14% of maternally expressed genes (MEGs) and approximately 29% of paternally expressed gene

90 cephalography (EEG), magnetoencephalography (MEG), and functional magnetic resonance imaging (fMRI) m

91 , (2) schizophrenia, (3) 40 Hz, (4) EEG, (5) MEG, and (6) steady state response.

92 nes suggested that 10-DAP endosperm-specific MEGs are involved in nutrient uptake and allocation and

93 c networks and demonstrates the potential of MEG as a tool for understanding the mechanisms that unde

94 The efficacy of magnetoencephalography (MEG) as an alternative to invasive methods for investiga

95 L1, TAL1, FLI1, ERG, RUNX1, LMO2) binding to MEG-associated cis-regulatory modules (CRMs) in multipot

96 MEG-based imaging holds potential as a noninvasive bioma

97 a novel method for group-level analysis with MEG beamformer images that utilizes the peak locations w

98 robust method for group-level analysis with MEG beamformer images.

99 th cortical neuronal avalanches and LRTCs in MEG but not SEEG.

100 mosensitive DNA lesion, O6-methylguanine (O6-MeG), by O6-methylguanine-DNA-methyltransferase (E.C. 2.

101 selectivity for incorporation opposite O(6)-MeG compared to unmodified purines.

102 Combining human magnetoencephalography (MEG), computational modeling, and laminar recordings in

103 r breast cancer, we identified a significant MEGS consisting of TP53 and four infrequently mutated ge

104 Using MEG constrained by individual cortical anatomy obtained

105 chanistic explanation of these effects using MEG data acquired from healthy human volunteers (N = 13,

106 The MEG data are analyzed using a unique combination of beam

107 ctional connectivity patterns extracted from MEG data in 14 subjects with schizophrenia and 14 health

108 ly this approach to beamformer reconstructed MEG data in source space.

109 representations and directed connectivity in MEG data obtained while human participants listened to s

110 To this end, resting-state MEG data of 22 healthy adults was analysed.

111 I to validate and extend the prediction from MEG data of a right auditory cortex contribution to the

112 MEG data revealed that 7-mo-old infants activate auditor

113 MEG data showed that the amplitude of MBS reduction corr

114 t empirical evidence based on behavioral and MEG data that global information encoded at high levels

115 elated beamformer source models for auditory MEG data under typical experimental conditions: monaural

116 All MEG data was imaged in the time-frequency domain using b

117 MEG data were analyzed in the 25-150 Hz frequency range

118 al dynamics of these processes by collecting MEG data while human participants listened to spoken wor

119 In our MEG data, spontaneous beta activity from somatosensory a

120 a region of interest beamformer analysis of MEG data, we compare the 4 Hz component of the ASSR to t

121 nds, and we recorded magnetoencephalography (MEG) data as participants adapted to the delay.

122 ecoding", methods to magnetoencephalography (MEG) data has allowed researchers to characterize, in hi

123 variate decoding of magneto-encephalography (MEG) data to track the neural representation of within-s

124 Magnetoencephalography (MEG) data was acquired from healthy subjects who were cu

125 Magneto-encephalographic (MEG) data were recorded from 13 adult human participants

126 hnique for analysing Magnetoencephalography (MEG) data.

127 active site configurations with either O(6)-MeG:dC or O(6)-MeG:dT bound compared with the correspond

128 through activation space, as measured using MEG decoding methods, correlates with reaction times for

129 MEG decoding results revealed that scene-based facilitat

130 Around the transcription start sites of endo-MEGs, DNA methylation and H3K4me3 specifically marked pa

131 nfigurations with either O(6)-MeG:dC or O(6)-MeG:dT bound compared with the corresponding situations

132 agnostic imaging techniques such as FDG PET, MEG, DTI and intra-cranial EEG are widely used to establ

133 sources detected by magnetoencephalography (MEG) during identical language tasks.

134 Abnormality in Meg/E or erythroid progenitors could potentially be cons

135 and creates stress-resistant preleukemic Pre-Meg/E progenitors predisposed to malignant transformatio

136 utonomously causes expansion of abnormal Pre-Meg/E progenitors with compromised erythroid specificati

137 henotypic pre-megakaryocyte/erythrocyte (Pre-Meg/E) progenitor population.

138 Here, we used high-density MEG-EEG recordings in combination with individual MRI im

139 tamine's effects have emerged, there are few MEG/EEG studies examining the acute subanesthetic effect

140 omatosensory evoked magnetoencephalographic (MEG) elicited by air puff stimulation of right index fin

141 The use of MEG enabled us to obtain the first characterization of t

142 The preleukemic Pre-Meg/Es display dysregulated erythroid and megakaryocytic

143 Furthermore, these abnormal preleukemic Pre-Meg/Es have enhanced stress resistance and are prone to

144 Spatio-temporal MEG estimates to RW and PN are consistent with the highl

145 s in functional connectivity between groups, MEG eyes-closed recordings from 30 sMCI and 19 pMCI subj

146 124 healthy human subjects and two different MEG facilities using different sensor technologies.

147 index finger and recorded using 306-channel MEG from 21 healthy subjects.

148 ed using noninvasive magnetoencephalography (MEG) from 124 healthy human subjects and two different M

149 similarity analyses applied to the data from MEG gradiometers uncovered a pronounced decrease in vari

150 The sensor fabricated with C6-MEG has a specificity factor ( approximately 13.5) subst

151 Although MEG holds great promise, it has proven challenging to im

152 o investigate this phenomenon in humans (EEG/MEG), however, are inherently limited by their spatial r

153 ere, we use magnetoencephalographic imaging (MEG-I) in human speakers to demonstrate that efference c

154 ity of MEG imaging, and to determine whether MEG imaging can become a viable alternative to the intra

155 ts demonstrate excellent correlation between MEG imaging findings and the IAP for language lateraliza

156 determine the sensitivity and specificity of MEG imaging, and to determine whether MEG imaging can be

157 ateralization using magnetoencephalographic (MEG) imaging, to determine the sensitivity and specifici

158 (BOLD) measures, and magnetoencephalography (MEG), implemented during resting state conditions, revea

159 ybridization probe system for detecting O(6)-MeG in a sequence-specific manner on the basis of colori

160 ccessfully measure in-gene abundance of O(6)-MeG in mixtures with competing unmodified DNA.

161 MEG in passive listeners and those actively detecting ap

162 We recorded 275 channel MEG in two experiments (n = 25 human males) examining th

163 the infertile interspecific hybrids matched MEGs in fertile intraspecific hybrids.

164 multiple previously unreported non-pairwise MEGS in multiple cancer types.

165 a oscillations using magnetoencephalography (MEG) in children undergoing CRT to test whether gamma ch

166 dynamics recorded by magnetoencephalography (MEG) in human subjects performing a threshold-level visu

167 nderstanding of multiple exciton generation (MEG) in organic materials has been restricted by the lim

168 inglet fission (a molecular manifestation of MEG) in pentacene.

169 e imaging (fMRI) and magnetoencephalography (MEG) in the same group of subjects, we analyzed resting-

170 Disruption of two MEGs increased the amount of small starch granules and r

171 udgments, whereas the reverse was true for a MEG index of familiarity.

172 An MEG index of the process of recollection was larger for

173 allele-specific epigenetic features as endo-MEGs, indicating similar mechanisms for the regulation o

174 3, Plxnc1, Pcdh7, and Selp) that support HSC-Meg interactions with the BM niche.

175 Unlike other neuroimaging methods, MEG is a direct measure of neural activity that is not t

176 MEG is useful to detect early synaptic dysfunction assoc

177 O(6)-Methylguanine (O(6)-MeG) is a highly mutagenic DNA adduct that forms in huma

178 O(6)-methylguanine (O(6)-MeG) is a miscoding DNA lesion arising from the alkylati

179 O(6)-Methyl-2'-deoxyguanosine (O(6)-MeG) is a ubiquitous DNA lesion, formed not only by xeno

180 activity measured by magnetoencephalography (MEG) is near critical and organizes as neuronal avalanch

181 is study, the methoxy-terminated diluent (C6-MEG) is the most effective in alleviating both nonspecif

182 a to insert both dC and dT opposite the O(6)-MeG lesion with similar efficiencies.

183 pathway induced by O(6)-methylguanine (O(6)-MeG) lesions is poorly understood.

184 In addition to its role in MEG lineage priming, GATA2 plays an extensive role in la

185 veal important new insights into how ERY and MEG lineages arise from a common bipotential progenitor

186 semantic association between 300 and 500 ms; MEG localized the differential neural response within th

187 In bipotent pre-MegEs, loss of Dicer up-regulated transcription factors

188 An HSC/MPP subpopulation expressing Meg markers was also increased in Runx1-deficient mice.

189 The MEG (maternal-effect germline defective) proteins are ge

190 We obtained magnetoencephalographic (MEG) measurements from subjects performing a working mem

191 However, fMRI diverged from early EEG/MEG measures in revealing semantic enhancement effects w

192 s experiment, where magnetoencephalographic (MEG) measures of neural activity were acquired in the te

193 We combine magnetoencephalographic (MEG) measures of neural processing speed with magnetic r

194 re analyzed with an anatomically-constrained MEG method.

195 oth behavioural and magnetoencephalographic (MEG) metrics associated with responses to pure-tone soun

196 Our graph filtration over MEG network revealed these inter-regional time-delayed c

197 enes (FLT3, IDH2, NRAS, KIT, and TP53) and a MEGS (NPM1, TP53, and RUNX1) whose mutation status was s

198 counting for the mutagenic potential of O(6)-MeG observed in vivo.

199 The micro-exon genes (MEG) of Schistosoma mansoni, a parasite responsible for

200 p protagonists using magnetoencephalography (MEG), one-on-one positive and conflictual interactions w

201 Conversely, both the structures with O(6)- MeG opposite dCTP and dC display sheared configuration o

202 of spontaneous activity, using the LFP, EEG, MEG or fMRI suggest that the default state of the cortex

203 activity measured by magnetoencephalography (MEG) or electroencephalography (EEG).

204 nown MGMT substrate O(6)-methylguanine (O(6)-MeG) or O(6)-CMG effectively inactivate MGMT in vitro (I

205 forced Tek expression in HSCs/MPPs increases Meg output.

206 50-fold higher catalytic efficiency for O(6)-MeG over guanine in the template.

207 During MEG, participants fixated on a centrally-presented cross

208 and the capability of identifying the exact MEGS, particularly for highly imbalanced MEGS.

209 ing feature sets based on variable-dimension MEG power spectra in the primary visual cortex and fusif

210 MEG power spectra revealed a rich set of significant osc

211 Traditionally, this MEG process has been observed as an intermolecular proce

212 ), an efficient multiple exciton generation (MEG) process in organic semiconductors, is one promising

213 th a prominent skewing toward megakaryocyte (Meg) progenitors.

214 Phosphorylation of the MEGs promotes granule disassembly and dephosphorylation

215 we tested this neural resonance theory using MEG recordings as female and male individuals listened t

216 Using MEG recordings in humans maintaining steady isometric mu

217 Using noninvasive human MEG recordings in subjects performing a visuospatial att

218 ormer-reconstructed source time courses from MEG recordings of 52 human subjects during the baseline

219 MEG recordings of brain activity were taken before and a

220 e demonstrate such an interaction in EEG and MEG recordings of task-free human brain activity.

221 Here we used concurrent EEG and MEG recordings to determine how sensory information and

222 ing key falsifiable predictions of NRT using MEG recordings, we demonstrate the emergence of neural o

223 ectome Project, and magnetoencephalographic (MEG) recordings from a subset, the heritability of conne

224 acquired whole-head magnetoencephalographic (MEG) recordings from human subjects performing a variabl

225 ing intracranial and magnetoencephalography (MEG) recordings, we show that saccades are locked to the

226 lographic (EEG) and magnetoencephalographic (MEG) recordings.

227 Multiple exciton generation (MEG) refers to the creation of two or more electron-hole

228 ry in a scene could be reliably decoded from MEG response patterns as early as 160 ms, despite substa

229 es and primary visual cortex (V1), and later MEG responses and inferior temporal (IT) cortex.

230 ontent-specific correspondence between early MEG responses and primary visual cortex (V1), and later

231 s from single-trial magnetoencephalographic (MEG) responses.

232 Our MEG results show that, during articulation, coherent osc

233 MEG revealed that, during phase-2 initial conditioning,

234 ltivariate pattern classification applied to MEG revealed the time course of object processing: where

235 We used MEG (RRID: NIFINV:nlx_inv_090918) to investigate languag

236 The MEG sensor data were source localized using a time-frequ

237 e categories within a scene was decoded from MEG sensor patterns by training linear classifiers on di

238 We identified large deflections at single MEG sensors and combined them into spatiotemporal cascad

239 esponses obtained by magnetoencephalography (MEG) shows that for maskers without the underlying acous

240 ce was accompanied by significant changes in MEG signal power, and a DCM assay disclosed related chan

241 ncephalographic/magnetoencephalographic (EEG/MEG) signal is generated primarily by the summation of t

242 e for neuronal system identification [6], to MEG signals from prefrontal cortex, we demonstrate that

243 , we studied in detail the interplay between MEG signals from the primary sensorimotor (SM1) cortex a

244 Finally, we correlated human MEG signals to single-unit responses in monkey IT.

245 Whole-head MEG signals were analyzed with an anatomically-constrain

246 res for spontaneous magnetoencephalographic (MEG) signals from humans during altered states of consci

247 ber probabilistic diffusion tractography and MEG source analysis of conditioning-test (C-T) median ne

248 onse complex presents a challenging case for MEG source-modelling, because symmetrical, phase-locked

249 switching that mediates further induction of MEG-specific genes following lineage commitment.

250 dentified a robust, genome-wide mechanism of MEG-specific lineage priming by a previously described s

251 e we used a novel approach in human fMRI and MEG studies to reveal supra-additive scene-object intera

252 urate estimates of laterality across various MEG studies.

253 trode and sources-space responses in EEG and MEG studies.

254 from brainstem nuclei, a recent study using MEG suggested that there is also a right-lateralized con

255 with a whole-cortex magnetoencephalography (MEG) system using a word recognition paradigm optimized

256 NA mismatch substrates containing G:T or O(6)meG:T mismatches, the purification of recombinant native

257 d we test the hypothesis by recording, using MEG, the neural responses of human subjects listening to

258 lectromagnetic responses recorded by EEG and MEG to an auditory paradigm in which we factorially cros

259 Here we use MEG to investigate changes in oscillatory power while he

260 A study uses MEG to provide the spatial as well as the temporal resol

261 Here we used MEG to record cortical oscillations in the context of ad

262 ilarity analysis, we combined human fMRI and MEG to show content-specific correspondence between earl

263 We use psychophysics and MEG to test how sensitivity to input statistics facilita

264 Recently, studies comparing MEG to the gold standard Wada procedure have found incon

265 We used magnetoencephalography (MEG) to assess plasticity of human auditory cortex induc

266 s et al. (2016) uses magnetoencephalography (MEG) to characterize the hierarchical organization of hu

267 Here, we used magnetoencephalography (MEG) to investigate neural oscillations in a task that s

268 This work used magnetoencephalography (MEG) to investigate the degree of neural overlap between

269 -invasive whole-head Magnetoencephalography (MEG) to look at theta oscillations and Functional Magnet

270 rticipants underwent magnetoencephalography (MEG) to measure neuronal activity directly and functiona

271 We used magnetoencephalography (MEG) to measure participants' brain activity during task

272 We use magnetoencephalography (MEG) to monitor brain oscillations in 22 participants du

273 eech processing with magnetoencephalography (MEG) to unravel the principles of speech segmentation an

274 d development into mature cells, and HSC and Meg transcription signatures.

275 analysis confirmed that somatosensory evoked MEG was mainly generated from the contralateral primary

276 MEG was measured from nine healthy subjects as they view

277 MEG was recorded during an auditory verb generation task

278 Here, MEG was used to investigate neuronal activity while subj

279 of memory encoding, magnetoencephalography (MEG) was analyzed over multi-regional network of negativ

280 Resting-state magnetoencephalography (MEG) was used to assess whether functional connectivity

281 High-density magnetoencephalography (MEG) was utilized to evaluate the basic neurophysiology

282 Here, using MEG we directly investigated how these visual signals in

283 Using magnetoencephalography (MEG), we demonstrate that stimulus-evoked activity in hu

284 First, using magneto-encephalography (MEG), we found a shift in the stimulus-evoked time cours

285 wo experiments using magnetoencephalography (MEG), we investigated motor brain activation, as well as

286 To identify mutually exclusive gene sets (MEGS), we developed a powerful and flexible analytic fra

287 ing a multivariate approach to both fMRI and MEG, we characterize the functional neuroanatomy and neu

288 EEG and MEG were recorded while participants reported the spatia

289 tructures of both dC and dT paired with O(6)-MeG were solved in both insertion and extension modes.

290 rm-specific maternally expressed genes (endo-MEGs) were associated with maternally preferred H3K4me3

291 The majority of maternally expressed genes (MEGs) were shared among all three F1 interspecific cross

292 early 80% of the maternally expressed genes (MEGs) were specific to 10 DAP and were primarily attribu

293 We recorded MEG while 24 human subjects (12 females) listened to rad

294 rain activity using magnetoencenphalography (MEG) while human participants were exerting physical eff

295 Here, we recorded magnetoencephalography (MEG) while human subjects performed a novel non-spatial

296 We recorded whole-head MEG, while participants viewed a visual grating stimulus

297 For acute myeloid leukemia, we identified a MEGS with five genes (FLT3, IDH2, NRAS, KIT, and TP53) a

298 Magnetoencephalography (MEG) with an established index of auditory perception, t

299 e, we combined human magnetoencephalography (MEG) with multivariate decoding techniques to probe the

300 ofuranose and their use for quantifying O(6)-MeG within mutational hotspots of the human KRAS gene.