ライフサイエンスコーパス: MEG

1 e imaging (fMRI) and magnetoencephalography (MEG).

2 g memory task during magnetoencephalography (MEG).

3 itude, measured with magnetoencephalography (MEG).

4 nd adolescents using magnetoencephalography (MEG).

5 dball paradigm using magnetoencephalography (MEG).

6 eural activity using magnetoencephalography (MEG).

7 subjects (HC) using magnetoencephalography (MEG).

8 ore fully exploit the unique capabilities of MEG.

9 have on designing sensor arrays for wearable MEG.

10 on-invasive recording techniques such as EEG/MEG.

11 ngs from 6 areas of macaque cortex and human MEG.

12 searches, or for expanding established small MEGS.

13 act MEGS, particularly for highly imbalanced MEGS.

14 primary human MKs and MK cells lines, DAMI, Meg-01 and MO7e express TrkA, the primary receptor for N

15 We used the human megakaryoblastic cell line MEG-01 as an in vitro model for human megakaryocytes and

16 nduced a sustained increase in [Ca(2+)] i in Meg-01 cells and enhanced the frequency of repetitive Ca

17 We conclude that platelets and Meg-01 cells express the MS cation channel Piezo1, which

18 WDR1 knockdown (KD) in MEG-01 cells increased adhesion and spreading in both th

19 Following the virus infection, the MEG-01 cells showed a marked reduction in the surface ex

20 acological studies in human megakaryoblastic MEG-01 cells showed that DREAM is important for A23187-i

21 ), and found expression levels of GLP-1Rs in MEG-01 cells to be higher than those in the human lung b

22 We have studied DENV infection of MEG-01 cells to understand its effect on megakaryopoiesi

23 ection caused an enhanced Notch signaling in MEG-01 cells where the virus envelope protein was shown

24 e observed that DENV could infect only naive MEG-01 cells, and differentiated cells were refractory t

25 gonist exenatide elicited a cAMP response in MEG-01 cells, and exenatide significantly inhibited thro

26 ut larger than PLPs produced from unmodified MEG-01 cells, and had significantly increased adhesion i

27 However, DENV-infected MEG-01 cells, when induced for differentiation with PMA,

28 lotting, respectively, in both platelets and Meg-01 cells.

29 MEG-01 is the human megakaryoblastic leukemia cell line

30 tress on Ca(2+) entry in human platelets and Meg-01 megakaryocytic cells loaded with Fluo-3 was exami

31 Stimulation of MEG-01 with thrombin reduced levels of WDR1 transcripts

32 ) mRNA from a human megakaryocyte cell line (MEG-01), and found expression levels of GLP-1Rs in MEG-0

33 y lower in lo-Meg (46%, P = 0.034) and in si-Meg (23%, P < 0.001) than each control.

34 In C. elegans, MEG-3 and MEG-4 function redundantly to assemble germ gr

35 r findings suggest that the formation of the MEG-3 concentration gradient and the segregation of P gr

36 MEG-3 in the anterior cytoplasm, leading to MEG-3 enrichment in the posterior.

37 In vivo, MEG-3 forms a posterior-rich concentration gradient that

38 EX-5 is necessary and sufficient to suppress MEG-3 granule formation in vivo, and suppresses RNA-indu

39 xcess)-5 and MEX-6 suppress the retention of MEG-3 in the anterior cytoplasm, leading to MEG-3 enrich

40 MEG-3 is an intrinsically disordered protein that binds

41 In the primordial germ cells of meg-3 meg-4 mutants, rde-11 and sid-1 transcripts disper

42 We report that a mutant (meg-3 meg-4) that does not assemble P granules in primor

43 ormation in vivo, and suppresses RNA-induced MEG-3 phase separation in vitro.

44 -3's access to RNA, thus locally suppressing MEG-3 phase separation to drive P granule asymmetry.

45 in conjunction with PLK-1 kinase to suppress MEG-3 retention in the anterior.

46 el-like assemblies of the disordered protein MEG-3 that associate with liquid PGL-3 droplets in the e

47 MEG-3 traps mRNAs into non-dynamic condensates in vitro

48 findings suggest that MEX-5 interferes with MEG-3's access to RNA, thus locally suppressing MEG-3 ph

49 granules are two parallel manifestations of MEG-3's response to upstream polarity cues.

50 ced phase separation of the granule scaffold MEG-3.

51 by condensation with the disordered protein MEG-3.

52 ~10 generations in wild-type descendants of meg-3/4 ancestors.

53 meg-3/4 animals produce aberrant siRNAs that are propaga

54 re transgenerationally disconnected from the meg-3/4 genotype.

55 Here, we show that meg-3/4 mutant animals exhibit defects in RNA interferen

56 Surprisingly, we find that the retention of MEG-3::Halo in the posterior cytoplasm surrounding P gra

57 reaction/diffusion mechanisms that result in MEG-3::Halo segregation.

58 In C. elegans, MEG-3 and MEG-4 function redundantly to assemble germ granules in

59 In the primordial germ cells of meg-3 meg-4 mutants, rde-11 and sid-1 transcripts disperse in

60 We report that a mutant (meg-3 meg-4) that does not assemble P granules in primordial g

61 G (maternal-effect germline defective)-3 and MEG-4, which are enriched in P granules and in the poste

62 ) biosynthesis was significantly lower in lo-Meg (46%, P = 0.034) and in si-Meg (23%, P < 0.001) than

63 spanning spatial scales from single units to MEG - a valuable framework for relating human and animal

64 ect current stimulation (HD-tDCS) can reduce MEG abnormalities and transiently improve language perfo

65 muli during the same magnetoencephalography (MEG) acquisition.

66 the coupling between the time courses of the MEG activity and attended speech stream, multitalker bac

67 We trained pattern classifiers on the MEG activity elicited by direct presentation of the visu

68 Here, we recorded magnetoencephalographic (MEG) activity in participants (N = 24) who viewed images

69 tations of both in magneto-encephalographic (MEG) activity.

70 ne such technology, Optically Pumped MEG (OP-MEG) allows for a scalp mounted system that provides mea

71 The MEG analysis demonstrated that phonological training inc

72 The MEG analysis showed that the PD patients had a significa

73 led a Watson-Crick-like pairing between O(6)-MeG and 2"-deoxythymidine-5"-[(alpha, beta)-imido]tripho

74 d image, we investigated this question using MEG and 7 Tesla fMRI in humans.

75 In both MEG and behavioral measures, task processing was optimal

76 we leveraged the high temporal resolution of MEG and combined it with a scene imagination task.

77 imits of highest temporal resolution for EEG/MEG and ECoG were shown as ~200 ms and ~10 ms and are no

78 Here we recorded simultaneous MEG and EEG (total of 328 sensors) in 9 human subjects (

79 In two studies, MEG and EEG activity was measured as human participants

80 o maximizing the sharing and exploitation of MEG and EEG data, and we also discuss how this 'living'

81 The MEG and EEG datasets revealed converging evidence that t

82 istributed cortical activity can explain the MEG and EEG patterns generated by deep sources.

83 1 Hz), other neuroimaging modalities such as MEG and EEG suggest that much faster timescales may be e

84 Thus, MEG and EEG were dominated by early visual and ventral s

85 ntered on midline regions of the DMN in both MEG and fMRI, boosting confidence in a possible pathophy

86 s of typical functional connectivity in both MEG and fMRI.

87 tion, in addition to its known importance in Meg and G/M maturation.

88 Both MEG and iEEG data show that decreases in alpha/beta powe

89 Using tools combining MEG and intracranial EEG with brain connectivity analyse

90 alyses of high-gamma activity recorded using MEG and intracranial EEG.

91 Remarkably, one-third of MEGs and nearly one-half of PEGs were associated with gr

92 Our data further reveal that MEGs and PEGs differ in their targeting by 24-nucleotide

93 Our data indicate that both MEGs and PEGs in rice regulate nutrient metabolism and e

94 t mechanisms establishing DNA methylation at MEGs and PEGs.

95 Most MEGs and some PEGs were expressed specifically in the en

96 enting the spatiotemporal resolution of 'EEG/MEG' and 'ECoG' were incorrect.

97 specific DNA lesion O(6)-methylguanine (O(6)MeG) and characterized by arrest of cells in the G(2)-M

98 nd for small interfering RNA for megalin (si-Meg) and control (si-Ctr).

99 r such practices in magnetoencephalographic (MEG) and electroencephalographic (EEG) research, recentl

100 We used magnetoencephalography (MEG) and electroencephalography (EEG), in combination wi

101 noninvasively, using magnetoencephalography (MEG) and electroencephalography (EEG).

102 question using human magnetoencephalography (MEG) and multivariate analyses of instantaneous activity

103 ological relevance, O(6)-methylguanine (O(6)-MeG) and O(6)-carboxymethylguanine (O(6)-CMG), were char

104 e identified 162 maternally expressed genes (MEGs) and 95 paternally expressed genes (PEGs), which we

105 oximately 14% of maternally expressed genes (MEGs) and approximately 29% of paternally expressed gene

106 , (2) schizophrenia, (3) 40 Hz, (4) EEG, (5) MEG, and (6) steady state response.

107 halography (EEG) and magnetoencephalography (MEG) are unique in their ability to provide neurophysiol

108 ing gamma power from magnetoencephalography (MEG) as a proxy measure for homeostatic balance in 35 un

109 neural activity with magnetoencephalography (MEG) before and while participants briefly viewed an amb

110 stimulated megalin expression 2.1-fold in lo-Meg cells (P < 0.01).

111 h lower megalin (lo-Meg, P < 0.001) or in si-Meg cells (P < 0.05).

112 We used magnetoencephalography (MEG) combined with continuous theta burst stimulation (c

113 Combining human magnetoencephalography (MEG), computational modeling, and laminar recordings in

114 r breast cancer, we identified a significant MEGS consisting of TP53 and four infrequently mutated ge

115 chanistic explanation of these effects using MEG data acquired from healthy human volunteers (N = 13,

116 We recorded whole-head MEG data during a multiple-object tracking paradigm, in

117 To uncover this, we combined MRI and MEG data from 17 male and 11 female participants, invest

118 thm to simulated and experimentally-recorded MEG data from auditory experiments in the cocktail party

119 representations and directed connectivity in MEG data obtained while human participants listened to s

120 To this end, resting-state MEG data of 22 healthy adults was analysed.

121 I to validate and extend the prediction from MEG data of a right auditory cortex contribution to the

122 The MEG data presented here provide novel insights about the

123 Application to MEG data reveals improvements over existing TRF estimati

124 t empirical evidence based on behavioral and MEG data that global information encoded at high levels

125 elated beamformer source models for auditory MEG data under typical experimental conditions: monaural

126 al dynamics of these processes by collecting MEG data while human participants listened to spoken wor

127 ial spatiotemporal patterns in resting-state MEG data, and large-scale waves in human electrocorticog

128 ovide evidence from multivariate analysis of MEG data, behavioral data, and computational modelling,

129 In our MEG data, spontaneous beta activity from somatosensory a

130 ng multivariate classification of full-brain MEG data, we first probed where the brain represents aco

131 resent in human RS activity in both SEEG and MEG data.

132 nds, and we recorded magnetoencephalography (MEG) data as participants adapted to the delay.

133 tory processing from magnetoencephalography (MEG) data in a cocktail party setting.

134 tern analysis on time-resolved neuroimaging (MEG) data to examine how object-color knowledge affects

135 variate decoding of magneto-encephalography (MEG) data to track the neural representation of within-s

136 Magnetoencephalography (MEG) data was acquired from healthy subjects who were cu

137 source-reconstructed magnetoencephalography (MEG) data.

138 active site configurations with either O(6)-MeG:dC or O(6)-MeG:dT bound compared with the correspond

139 MEG decoding results revealed that scene-based facilitat

140 This primer introduces the basic concepts of MEG, discusses its strengths and limitations in comparis

141 While O(6)-methylguanine (O(6)-MeG)-DNA methyltransferase (MGMT) promoter methylation s

142 Around the transcription start sites of endo-MEGs, DNA methylation and H3K4me3 specifically marked pa

143 nfigurations with either O(6)-MeG:dC or O(6)-MeG:dT bound compared with the corresponding situations

144 We measured fMRI and MEG during symmetric finger tapping, in which fast tappi

145 mporal resolution of magnetoencephalography (MEG), during a rapid serial visual presentation (RSVP) t

146 Abnormality in Meg/E or erythroid progenitors could potentially be cons

147 and creates stress-resistant preleukemic Pre-Meg/E progenitors predisposed to malignant transformatio

148 utonomously causes expansion of abnormal Pre-Meg/E progenitors with compromised erythroid specificati

149 henotypic pre-megakaryocyte/erythrocyte (Pre-Meg/E) progenitor population.

150 This multi-dimensional aspect of the MEG/EEG based connectivity increases the challenges of t

151 ultivariate analysis methods to a multimodal MEG/EEG dataset.

152 esent two robust and widely used methods for MEG/EEG functional connectivity estimation.

153 Many MEG/EEG studies address this complexity by using a hypot

154 rators is a limitation that constrains using MEG/EEG to reveal novel principles of information proces

155 Magneto- and electro-encephalography (MEG/EEG) non-invasively record human brain activity with

156 d clinicians interpret the neural origins of MEG/EEG.

157 al origins of electrical currents generating MEG/EEG.

158 The use of MEG enabled us to obtain the first characterization of t

159 The preleukemic Pre-Meg/Es display dysregulated erythroid and megakaryocytic

160 Furthermore, these abnormal preleukemic Pre-Meg/Es have enhanced stress resistance and are prone to

161 Next, we designed a magnetoencephalography (MEG) experiment to measure the neuromagnetic activity ev

162 We train classifiers to distinguish MEG field patterns during presentation of two probabilis

163 Strikingly, MEG, fMRI and MRI showed additive effects supporting dis

164 Model-driven MEG-fMRI data fusion revealed the spatiotemporal evoluti

165 cal workup including magnetoencephalography (MEG) followed by resective surgery after determination o

166 ation (C(max)) was associated with increased MEG gamma power.

167 ng-state whole-brain magnetoencephalography (MEG) gamma power 6-9 h post-infusion.

168 The sensor fabricated with C6-MEG has a specificity factor ( approximately 13.5) subst

169 While OP-MEG has the potential to provide high information conten

170 o investigate this phenomenon in humans (EEG/MEG), however, are inherently limited by their spatial r

171 -resolution magnetoencephalographic imaging (MEG-I) to define with millisecond precision the underlyi

172 (BOLD) measures, and magnetoencephalography (MEG), implemented during resting state conditions, revea

173 ybridization probe system for detecting O(6)-MeG in a sequence-specific manner on the basis of colori

174 ccessfully measure in-gene abundance of O(6)-MeG in mixtures with competing unmodified DNA.

175 MEG in passive listeners and those actively detecting ap

176 multiple previously unreported non-pairwise MEGS in multiple cancer types.

177 dynamics recorded by magnetoencephalography (MEG) in human subjects performing a threshold-level visu

178 n by capitalising on magnetoencephalography (MEG) in humans who made choices in a risky foraging task

179 e imaging (fMRI) and magnetoencephalography (MEG) in the same group of subjects, we analyzed resting-

180 f the boxes representing 'fMRI/PET' and 'EEG/MEG' incorrectly showed the highest spatial resolution l

181 Disruption of two MEGs increased the amount of small starch granules and r

182 MEG indicated that the texture-invariant scene layout re

183 allele-specific epigenetic features as endo-MEGs, indicating similar mechanisms for the regulation o

184 3, Plxnc1, Pcdh7, and Selp) that support HSC-Meg interactions with the BM niche.

185 eories in a parallel magnetoencephalography (MEG)-intracranial electroencephalography (iEEG) study in

186 O(6)-Methylguanine (O(6)-MeG) is a highly mutagenic DNA adduct that forms in huma

187 O(6)-Methyl-2'-deoxyguanosine (O(6)-MeG) is a ubiquitous DNA lesion, formed not only by xeno

188 Magnetoencephalography (MEG) is an invaluable tool to study the dynamics and con

189 is study, the methoxy-terminated diluent (C6-MEG) is the most effective in alleviating both nonspecif

190 a to insert both dC and dT opposite the O(6)-MeG lesion with similar efficiencies.

191 An HSC/MPP subpopulation expressing Meg markers was also increased in Runx1-deficient mice.

192 P granule segregation depends on MEG (maternal-effect germline defective)-3 and MEG-4, wh

193 We combine magnetoencephalographic (MEG) measures of neural processing speed with magnetic r

194 opose a multimodal model to robustly combine MEG, MRI and fMRI for prediction.

195 Our graph filtration over MEG network revealed these inter-regional time-delayed c

196 ole of alpha synchrony in attention, we used MEG neurofeedback to train subjects to manipulate the ra

197 enes (FLT3, IDH2, NRAS, KIT, and TP53) and a MEGS (NPM1, TP53, and RUNX1) whose mutation status was s

198 p protagonists using magnetoencephalography (MEG), one-on-one positive and conflictual interactions w

199 One such technology, Optically Pumped MEG (OP-MEG) allows for a scalp mounted system that prov

200 Conversely, both the structures with O(6)- MeG opposite dCTP and dC display sheared configuration o

201 of spontaneous activity, using the LFP, EEG, MEG or fMRI suggest that the default state of the cortex

202 forced Tek expression in HSCs/MPPs increases Meg output.

203 50-fold higher catalytic efficiency for O(6)-MeG over guanine in the template.

204 )D(3) did so in hMSCs with lower megalin (lo-Meg, P < 0.001) or in si-Meg cells (P < 0.05).

205 and the capability of identifying the exact MEGS, particularly for highly imbalanced MEGS.

206 We classified the MEG patterns for each press in the sequence and examined

207 Traditionally, this MEG process has been observed as an intermolecular proce

208 ), an efficient multiple exciton generation (MEG) process in organic semiconductors, is one promising

209 th a prominent skewing toward megakaryocyte (Meg) progenitors.

210 Using magnetoencephalographic (MEG) recording combined with event-related desynchronisa

211 ers while conducting magnetoencephalography (MEG) recording.

212 we tested this neural resonance theory using MEG recordings as female and male individuals listened t

213 In resting-state MEG recordings from healthy participants (N = 187) using

214 Using MEG recordings in humans maintaining steady isometric mu

215 Using noninvasive human MEG recordings in subjects performing a visuospatial att

216 ormer-reconstructed source time courses from MEG recordings of 52 human subjects during the baseline

217 e demonstrate such an interaction in EEG and MEG recordings of task-free human brain activity.

218 Eye movement and MEG recordings revealed how participants represent choic

219 MEG recordings showed that hippocampus and prefrontal co

220 During MEG recordings, subjects attended to four different 5 mi

221 ing key falsifiable predictions of NRT using MEG recordings, we demonstrate the emergence of neural o

222 rained temporal scale using eye tracking and MEG recordings.

223 ectome Project, and magnetoencephalographic (MEG) recordings from a subset, the heritability of conne

224 To allow wearable magnetoencephalography (MEG) recordings to be made on unconstrained subjects the

225 ing intracranial and magnetoencephalography (MEG) recordings, we show that saccades are locked to the

226 lographic (EEG) and magnetoencephalographic (MEG) recordings.

227 ry in a scene could be reliably decoded from MEG response patterns as early as 160 ms, despite substa

228 Using human magnetoencephalography (MEG) responses to a 2-talker mixture, we show evidence f

229 MEG results show that dissonant dyads evoke a pitch onse

230 We used MEG (RRID: NIFINV:nlx_inv_090918) to investigate languag

231 eproduced them in a magneto-encephalography (MEG) scanner.

232 e categories within a scene was decoded from MEG sensor patterns by training linear classifiers on di

233 quency activity recorded over frontotemporal MEG sensors plays a critical role in choice option integ

234 esponses obtained by magnetoencephalography (MEG) shows that for maskers without the underlying acous

235 s modulated the direction represented in the MEG signal as early as 150 ms after visual stimulus onse

236 reflection of the predicted direction in the MEG signal.

237 We use OPMs to record human cerebellar MEG signals elicited by air-puff stimulation to the eye.

238 , we studied in detail the interplay between MEG signals from the primary sensorimotor (SM1) cortex a

239 y significant coherence was observed between MEG signals originating from the auditory system and the

240 res for spontaneous magnetoencephalographic (MEG) signals from humans during altered states of consci

241 Pathological MEG slowing in these patients was correlated with aphasi

242 onse complex presents a challenging case for MEG source-modelling, because symmetrical, phase-locked

243 e we used a novel approach in human fMRI and MEG studies to reveal supra-additive scene-object intera

244 trode and sources-space responses in EEG and MEG studies.

245 During MEG, subjects participated in a visual picture-word matc

246 from brainstem nuclei, a recent study using MEG suggested that there is also a right-lateralized con

247 Here we demonstrate a lifespan-compliant MEG system, showing recordings of high fidelity data in

248 gnetometer-based magnetoencephalographic (OP-MEG) system for the measurement of human cerebellar acti

249 merged promising new Magnetoencephalography (MEG) systems in which the sensors can be placed close to

250 Here, we used EEG and MEG techniques to show that the brain is able to use the

251 We conclude that OP-MEG technology offers a promising way to advance the und

252 neration of wearable magnetoencephalography (MEG) technology with the potential to revolutionise elec

253 Here we show using magnetoencephalography (MEG) that tactile stimulation produces occipital cortex

254 lectromagnetic responses recorded by EEG and MEG to an auditory paradigm in which we factorially cros

255 ed word reading task was administered inside MEG to assess tDCS-induced neurophysiological changes in

256 the Montreal cognitive assessment (MoCA) and MEG to evaluate differences in cerebral activation patte

257 we leveraged the high temporal resolution of MEG to investigate the construction of novel mental imag

258 We use psychophysics and MEG to test how sensitivity to input statistics facilita

259 It remains challenging to relate EEG and MEG to underlying circuit processes and comparable exper

260 s et al. (2016) uses magnetoencephalography (MEG) to characterize the hierarchical organization of hu

261 Here, we used magnetoencephalography (MEG) to investigate neural oscillations in a task that s

262 This work used magnetoencephalography (MEG) to investigate the degree of neural overlap between

263 We used magnetoencephalography (MEG) to investigate the frequency-specific cortical trac

264 rrent study, we used magnetoencephalography (MEG) to investigate the primary somatosensory responses

265 rticipants underwent magnetoencephalography (MEG) to measure neuronal activity directly and functiona

266 We used magnetoencephalography (MEG) to measure participants' brain activity during task

267 Here, we used magnetoencephalography (MEG) to measure the time course of neural responses to f

268 We use magnetoencephalography (MEG) to show that disruption of network dynamics may be

269 is question by using magnetoencephalography (MEG) to study human subjects while they performed a deci

270 nventional cryogenic magnetoencephalography (MEG) to the study of cerebellar functions is highly limi

271 d development into mature cells, and HSC and Meg transcription signatures.

272 A new generation of MEG using optically pumped magnetometers (OPMs) that can

273 Moreover, the contribution of MEG was best explained by cortical power spectra between

274 analysis confirmed that somatosensory evoked MEG was mainly generated from the contralateral primary

275 Here, MEG was used to investigate neuronal activity while subj

276 of memory encoding, magnetoencephalography (MEG) was analyzed over multi-regional network of negativ

277 Magnetoencephalography (MEG) was recorded during a picture naming task to provid

278 narrative while their magnetoencephalogram (MEG) was recorded.

279 Here, using MEG we directly investigated how these visual signals in

280 Using magnetoencephalography (MEG), we demonstrate that stimulus-evoked activity in hu

281 First, using magneto-encephalography (MEG), we found a shift in the stimulus-evoked time cours

282 Using high-precision magnetoencephalography (MEG), we show that during the classical event-related de

283 Using magnetoencephalography (MEG), we show that the reduced ability to suppress memor

284 Using magnetoencephalography (MEG), we show that this stress-induced memory control de

285 To identify mutually exclusive gene sets (MEGS), we developed a powerful and flexible analytic fra

286 ing a multivariate approach to both fMRI and MEG, we characterize the functional neuroanatomy and neu

287 tructures of both dC and dT paired with O(6)-MeG were solved in both insertion and extension modes.

288 nt sensitivity is maximized when the MR-ARFI MEGs were maximally aligned with the FUS propagation dir

289 that the MR-ARFI motion-encoding gradients (MEGs) were aligned with a single-element FUS transducer

290 rm-specific maternally expressed genes (endo-MEGs) were associated with maternally preferred H3K4me3

291 The majority of maternally expressed genes (MEGs) were shared among all three F1 interspecific cross

292 We recorded MEG while 24 human subjects (12 females) listened to rad

293 behavior and recording neural activity using MEG while observers (male and female) were acquiring and

294 Here, we recorded magnetoencephalography (MEG) while human subjects performed a novel non-spatial

295 der occlusion, using magnetoencephalography (MEG), while participants were presented with images of o

296 For acute myeloid leukemia, we identified a MEGS with five genes (FLT3, IDH2, NRAS, KIT, and TP53) a

297 Magnetoencephalography (MEG) with an established index of auditory perception, t

298 ere we combine human magnetoencephalography (MEG) with behavioural and neural modelling to identify a

299 e, we combined human magnetoencephalography (MEG) with multivariate decoding techniques to probe the

300 ofuranose and their use for quantifying O(6)-MeG within mutational hotspots of the human KRAS gene.