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1 MEN 1 is an autosomal dominant familial neoplasia syndro
2 es with multiple endocrine neoplasia type 1 (MEN 1), with the ultimate aim of early tumor detection a
3 having multiple endocrine neoplasia, type 1 (MEN-1) (high increased risk), and 123 not having MEN-1 (
4 se with multiple endocrine neoplasia type 1 (MEN-1) and ZES, there was a significant relationship bet
5 of the multiple endocrine neoplasia type 1 (MEN-1) gene was studied in gastric carcinoids of patient
8 at 11q13 loci was detected in 15 of 20 (75%) MEN-1-ZES carcinoids, and each ECL-cell carcinoid with L
11 retrospectively reviewed for 21 consecutive MEN 1 patients undergoing pancreatic resection for NETs
17 ar invasion (P < 0.001), and the presence of MEN-1 (P = 0.035) were prognostically significant advers
18 ll carcinoid is an independent tumor type of MEN-1 that shares a common developmental mechanism (via
20 icular attention to the stomach of high risk MEN-1 patients, were correlated with the gastric biopsy
22 1 of 6 A-CAG carcinoids displayed LOH at the MEN-1 gene locus, and none of the 9 intestinal and recta
23 lopmental mechanism (via inactivation of the MEN-1 gene) with enteropancreatic and parathyroid MEN-1
24 et cell tumors arise in association with the MEN-1 syndrome, the majority of these neoplasms are spor
27 udied in gastric carcinoids of patients with MEN-1 and chronic atrophic type A gastritis (A-CAG), as
29 FSG (P <.001) in patients with sporadic ZES; MEN-1 patients lived longer than sporadic ZES patients (
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