ライフサイエンスコーパス: MLR

1 MLR and skin grafting confirmed donor-specific tolerance

2 MLR stimulation below the threshold for overt movement s

3 MLR was performed with IVIG (0.2-5 mg/mL), RAPA (0.02-40

4 MLR-Tagging software package is publicly available at ht

5 MLRs are essential for the regulation of the physiology

6 MLRs are plasmalemmal microdomains enriched in sphingoli

7 1 resulted in increased expression of Cav-1, MLRs, and MLR-localization of Cav-1 and tropomyosin-rela

8 At day 120, MLR demonstrated unresponsiveness to the host and donor

9 most influential sensory predictor of the 28 MLR models was flavour, while the aroma compound most in

10 g CD4+CD25 high FOXP3+ cell development; (4) MLR-generated CD4+CD25 high FOXP3+ cells added as third

11 cantly reduced allostimulatory activity in a MLR using naive CD4(+) T cells, and inhibited tetanus to

12 naive T cells taken from cord blood using a MLR assay.

13 ed suppressor cells functioned early in allo-MLR because expression of activation antigens and accumu

14 gamma and IL-17 in response to alloantigens (MLR), anti-CD3, and the glycolipid alpha-galactosyl cera

15 d cytolytic activity following an allogeneic MLR in vitro, without increasing autologous MLR activity

16 pon T cell stimulation and during allogeneic MLR.

17 of LC to induce CD4(+) T cells in allogeneic MLR, and thus resulting in a decreased ability of LC to

18 uppressed T cell proliferation in allogeneic MLR.

19 lls in both primary and secondary allogeneic MLR and showed tolerogenic potential.

20 We found mDC function in the allogeneic MLR directly corresponded to the magnitude of skin DTH r

21 ly inactive as stimulators of the allogeneic MLR, in contrast to potent stimulation by noninfected DC

22 oduction of IL-6 and TNF-alpha in allogeneic MLRs, impairing the activation of central and effector m

23 ines do not activate T cells in an allogenic MLR; however, they express low levels of MHC class I mol

24 suppressed T-cell proliferation in allogenic MLR in a dose-dependent manner.

25 ly lower proliferative response in allogenic MLR than the B cells from untreated patients.

26 capacity of AG-stimulated DCs is altered, an MLR with naive Th cells was performed.

27 N-gamma and inhibited T-cell responses in an MLR culture.

28 In an MLR system using porcine PBMCs, MSC-induced IL-10(+)IFN-

29 interferon secretion by naive T cells in an MLR.

30 d the production of regulatory T cells in an MLR.

31 pact of neuron-targeted overexpression of an MLR scaffold protein, caveolin-1 (Cav-1) (via a synapsin

32 e derived from two different Cre (Le-Cre and MLR-10) transgenic mice in which lens-specific Cre expre

33 ELISA and MLR assays showed that TGF-beta2 was involved in mediati

34 n general, PLS and PCA outperform linear and MLR models.

35 ss in standard cell-mediated lympholysis and MLR assays.

36 However, the effect of MLR and MLR-associated proteins on neuronal aging is unknown.

37 in increased expression of Cav-1, MLRs, and MLR-localization of Cav-1 and tropomyosin-related kinase

38 ytokines in the sera of graft recipients and MLR culture supernatants was tested using ELISA.

39 MLR in vitro, without increasing autologous MLR activity.

40 tion, and T cell proliferation in autologous MLRs.

41 locking were tested in vitro by (autologous) MLR.

42 vastatin treatment of stimulated pAEC before MLR reduced proliferation of human PBMC (P<0.05) and CD4

43 Atorvastatin treatment of human PBMC before MLR reduced their response to stimulated WT (P<0.05) and

44 ish the significance of the relation between MLR and HIV-1 infectivity.

45 ersistent responsiveness to donor antigen by MLR.

46 onclude that supraspinal commands (caused by MLR stimulation) select one of the numerous forms of ope

47 in vitro donor-specific unresponsiveness by MLR and CML and did not demonstrate anti-donor antibody

48 timulatory activity was assessed in vitro by MLR and CTL assays and in vivo by the influence on B10 c

49 FACS analysis of mouse T cell MLR revealed increased dual TCR T cells among alloreacti

50 xcitatory synaptic inputs into patch-clamped MLR cells, accompanied by activity in reticulospinal cel

51 tions were performed between ABO-compatible, MLR-mismatched NHPs.

52 This was similar to the third component MLR Tregs generated entirely in vitro.

53 under many typical water quality conditions, MLR- and BLM-based criteria are quite comparable.

54 By contrast, MLR stimulation evoked well coordinated stepping movemen

55 3(+) mismatched irradiated PBMCs in a 7-10 d MLR.

56 CD4CD45RA/CD45RO T cells generated in 9-day MLR were cocultured with anti-CD3 and autologous antigen

57 CTLA-4.FasL inhibits 1 degrees MLR and induces specific hyporesponsiveness in 2 degrees

58 ces specific hyporesponsiveness in 2 degrees MLR, with both effects only partially reversible with ex

59 lutamatergic transmission largely diminished MLR cell responses and locomotion.

60 ed a subset of neurons located in the dorsal MLR that send direct inputs to neurons in the respirator

61 L-2, but not neutralization of IL-10, during MLR ameliorated the reduced allostimulatory capacity of

62 as the agonistic antibody to caCD28 enhanced MLR.

63 Interestingly, M. leprae-exposed MLR cells secreted increased Th2 cytokines as well as si

64 (PRAGs) were considered in the final fitted MLR model, which explained 96.8% of the variability obse

65 MAE=7.6 for ANN to r2=0.61 and MAE=16.2 for MLR.

66 These cells can be assayed for MLR inhibitory and Treg recruitment functions, so as to

67 However, no evidence for MLR inhibition was seen, and most animals eventually dev

68 uppression was 3- to 10-fold more active for MLRs induced by the original alloantigens than for third

69 regs were more potent suppressors of a fresh MLR by CD25- CD4+ T cells.

70 added as third component modulators in fresh MLRs significantly enhanced newly developed Tregs in the

71 Foxp3(+) nTreg can be directly isolated from MLR cultures with high purity.

72 ult and aged hippocampus enhances functional MLRs with corresponding roles in cell signaling and prot

73 Skin grafting, MLR, and flow cytometry revealed that tolerance is neith

74 oxp3(+) nTreg proliferation in vitro in 72-h MLRs.

75 t AG reduced T cell proliferation in a human MLR.

76 4(+) T cell proliferation in mouse and human MLRs.

77 ferating cells, autologous and HLA identical MLRs generated the highest FOXP3+:FOXP3- cell ratios; (3

78 In MLR, T cells did not proliferate in response to M. lepra

79 cytokines, both within the allograft and in MLR supernatant of recipient lymphocytes cultured with W

80 of CD14, and had low stimulatory capacity in MLR.

81 f Ag processing, triggering naive T cells in MLR, and presenting Ags to specific T cell clones throug

82 the percentage of CD4(+)Foxp3(+) T cells in MLR, which is dependent upon expression of B7-H1.

83 een cells were recognized by bm12 T cells in MLR.

84 anced proliferation of allogeneic T cells in MLR.

85 n ELISPOT, 3) neutralization of MIG/CXCL9 in MLR reduced T lymphocyte proliferation, 4) IFN-gamma-ind

86 ion, purified CD4+ CD127- cells generated in MLR in the presence of MPA and added as third component

87 , and TNF-alpha production by lymphocytes in MLR culture supernatants.

88 ve effects of ICOS blockade were observed in MLR using peripheral blood mononuclear cells from dog le

89 ve effects of ICOS blockade were observed in MLR when anti-ICOS was combined with suboptimal concentr

90 ll proliferation and IFN-gamma production in MLR.

91 y the inability of T cells to proliferate in MLR.

92 to promote allogenic T cell proliferation in MLR in vitro.

93 yet did not restore T cell proliferation in MLR.

94 in mice and to suppress INF-gamma release in MLR.

95 onstrated donor-specific unresponsiveness in MLR assays, development of peripheral CD45RAhigh/CD4 dou

96 Treg cells blocked cytokine accumulation in MLRs, with a less robust inhibition of chemokine product

97 antigen, a superagonist anti-CD28 Ab, and in MLRs.

98 roliferative responses of RAGE-/- T cells in MLRs and in WT T cells cultured with TTP488.

99 d the number of IFN-gamma-producing cells in MLRs by ELISPOT.

100 Corroborated in MLRs, greater proliferation levels and robust IFN-gamma

101 CR3 ligands were added to CD4 lymphocytes in MLRs, and the proliferative responses were measured.

102 UFU enhanced HLA-DR expression and increased MLR.

103 emented strains were less potent at inducing MLR.

104 third components allospecifically inhibited MLR proliferation and recruited additional CFSE-labeled

105 INTERPRETATION: MLR-HFS can improve disordered locomotor function in a r

106 arboxyfluorescein succinimidyl ester-labeled MLR responders.

107 Each group of (non-CFSE labeled) MLR-generated cells were then added as third components

108 s) than two DR-mismatched MLRs, 2 DR-matched MLRs generated more than twofold higher percentages when

109 regs compared with addition of TAC- or media-MLR-generated Tregs.

110 t and remediate leaks could further minimize MLR.

111 lustrating how careful siting would minimize MLR in heavily used sedimentary basins.

112 e proliferation in a MHC class II-mismatched MLR, 2) MIG/CXCL9 also increased the number of IFN-gamma

113 leukocyte antigen DR matched and mismatched MLRs either alone or in combination with MPA or sirolimu

114 ulation indices (SIs) than two DR-mismatched MLRs, 2 DR-matched MLRs generated more than twofold high

115 ion in MHC class I- and total MHC-mismatched MLRs, 6) neutralization of CXCR3 reduced MIG/CXCL9-induc

116 ct of high-frequency stimulation of the MLR (MLR-HFS) on gait impairment in a rodent stroke model.

117 mpacta (SNc) evokes increasing activation of MLR cells with a graded increase in the frequency of loc

118 However, the effect of MLR and MLR-associated proteins on neuronal aging is unk

119 Increasing the intensity of MLR stimulation produces a growing activation of reticul

120 Simultaneous to the onset of MLR-HFS, a significantly higher walking speed and improv

121 activation and suppression, respectively, of MLR glutamatergic neurons by direct and indirect pathway

122 Likewise, stimulation of MLR projections to the basal forebrain also enhanced cor

123 As the strength of MLR stimulation was increased, the responses increased i

124 ons from the MLR and that a subpopulation of MLR neurons plays a key role in the respiratory changes

125 ol from the plasma membrane leads to loss of MLRs, decreased presynaptic vesicle fusion, and changes

126 opaminergic pathway has a modulatory role on MLR cells that are known to receive glutamatergic projec

127 (+) T cells activated with CD3/CD28 beads or MLRs.

128 Our MLR projections are at least an order of magnitude below

129 Overall MLR suppression was increased when Treg cells were added

130 (MLR) is implemented in the software package MLR-tagging.

131 In addition, the PCA-MLR analysis confirmed that the industrial sections; cut

132 The peak MLR for the site nearest the leakage pathways ($7.5/tCO2

133 We performed MLRs with spleen T lymphocytes and APCs isolated from wi

134 The pooled MLR and BLM models predicted species-specific toxicity w

135 The pooled MLR model was then applied to the species sensitivity di

136 tion of allogeneic T cells both in a primary MLR and after restimulation.

137 ere added along with CSA and MP to a primary MLR culture, whereas MTX modestly reduced Treg suppressi

138 naive alloreactive CD4+ T cells in a primary MLR.

139 ion of regulatory T cells (Tregs) in primary MLR assays with SRL, demonstrating a uniquely supportive

140 responding cells in freshly prepared primary MLRs, to determine allospecific and nonspecific inhibito

141 was also backcrossed to the autoimmune prone MLR/lpr background.

142 onstrate that neuronal membrane/lipid rafts (MLRs) establish cell polarity by clustering progrowth re

143 Membrane lipid rafts (MLRs) within the plasma membrane of most cells serve as

144 tio (PLR), and monocyte-to-lymphocyte ratio (MLR) were optimized by receiver operating characteristic

145 We used the mixed leucocyte reaction (MLR), stimulating one partner's peripheral blood mononuc

146 alloreactivity in mixed leukocyte reaction (MLR) assays did not cause acute GVHD.

147 s to stimulate the mixed leukocyte reaction (MLR) in the presence of transforming growth factor beta

148 cific Tregs in the mixed leukocyte reaction (MLR) that develops from polyclonal populations of T cell

149 We established mixed leukocyte reaction (MLR) with dendritic cells (DCs) as stimulators and CD4+

150 g activity for the mixed leukocyte reaction (MLR), and the DCs producing HIV-1 gag p24 do not express

151 cific tolerance by mixed leukocyte reaction (MLR), cell-mediated lysis (CML), and antibody testing an

152 cells in a primary mixed leukocyte reaction (MLR).

153 were assessed by mixed lymphocyte reaction (MLR) and cell-mediated lympholysis coculture assays and

154 assays including mixed lymphocyte reaction (MLR) and flow cytometry to detect serum alloantibody.

155 IG]) inhibits the mixed lymphocyte reaction (MLR) and induces apoptosis primarily in B cells.

156 Mixed lymphocyte reaction (MLR) and skin grafting assessed donor-specific tolerance

157 okine analysis of mixed lymphocyte reaction (MLR) culture supernatants by ELISA was also carried out.

158 cell-driven allo-mixed lymphocyte reaction (MLR) cultures by more than 90%.

159 lysis (m-CML) and mixed lymphocyte reaction (MLR) in recipients with quiescent grafts, but not in rec

160 ulator cells in a mixed lymphocyte reaction (MLR) in the presence of soluble Fas ligand (sFasL) to in

161 fting in vivo and mixed lymphocyte reaction (MLR) in vitro.

162 DC activation and mixed lymphocyte reaction (MLR) studies were performed to evaluate the immunogenici

163 roliferation in a mixed lymphocyte reaction (MLR) when compared with unpulsed Tregs, suggesting that

164 Using mixed lymphocyte reaction (MLR), the effect of atorvastatin on proliferation of hum

165 Proliferation and mixed lymphocyte reaction (MLR)-based assays were used to determine the immunosuppr

166 y inhibitory in a mixed lymphocyte reaction (MLR).

167 man was tested by mixed lymphocyte reaction (MLR).

168 the course of the mixed lymphocyte reaction (MLR).

169 d in an allogenic mixed lymphocyte reaction (MLR).

170 ression of canine mixed leukocyte reactions (MLR).

171 cells (Tregs) in mixed lymphocyte reactions (MLR) alone and in combination with maintenance agents us

172 Mixed lymphocyte reactions (MLR) and cell-mediated lympholysis (CML) assays were use

173 antibody blocked mixed lymphocyte reactions (MLR) in a dose-dependent manner similar to CTLA4-Ig, whe

174 FOXP3+ cells in mixed lymphocyte reactions (MLRs) ("the Treg MLR"), with varying human leukocyte ant

175 on of allogeneic mixed lymphocyte reactions (MLRs) (29 of 30 donors).

176 eptors over these neurons profoundly reduced MLR-induced excitation of reticulospinal neurons and mar

177 The mesencephalic locomotor region (MLR) has been shown to be capable of initiating running

178 ation of the mesencephalic locomotor region (MLR) in an in vitro isolated preparation, an effect that

179 ation of the mesencephalic locomotor region (MLR) is known to elicit gait movements, this area might

180 ation of the mesencephalic locomotor region (MLR) on one side evokes symmetrical locomotor movements

181 The mesencephalic locomotor region (MLR) plays a crucial role in locomotor control.

182 TATEMENT The mesencephalic locomotor region (MLR) plays a crucial role in the control of locomotion.

183 lly from the mesencephalic locomotor region (MLR) to reticulospinal neurons, which in turn project to

184 down to the mesencephalic locomotor region (MLR), a brainstem region controlling locomotion in verte

185 ntrol of the mesencephalic locomotor region (MLR), a brainstem target of BG that is critical for loco

186 ally defined mesencephalic locomotor region (MLR), is necessary for the production of L-DOPA-induced

187 The mesencephalic locomotor region (MLR), which includes the pedunculopontine nucleus (PPN)

188 ation of the mesencephalic locomotor region, MLR) with locomotion evoked by epidural stimulation of t

189 Multiple linear regression (MLR) and computational neural networks (CNNs) are employ

190 was built using multiple linear regression (MLR) and the OCT and VF data from the other eyes.

191 pe data based on multiple linear regression (MLR) is implemented in the software package MLR-tagging.

192 tribution to the multiple linear regression (MLR) model.

193 ing the best-fit multiple linear regression (MLR) models and Monte Carlo simulations as a function of

194 lysis (PCA), and multiple linear regression (MLR) models are compared.

195 veloped stepwise multiple linear regression (MLR) models for species that have been tested over wide

196 d then construct multiple linear regression (MLR) models with biological activity.

197 AO index using a multiple linear regression (MLR) technique with autumn conditions of sea-ice concent

198 each adulterant, Multiple Linear Regression (MLR) was applied, and a model was chosen and validated.

199 models based on multiple linear regression (MLR) were built using UV spectrophotometry (190-400 nm)

200 chniques include multiple linear regression (MLR), artificial neural networks (ANN), k-nearest neighb

201 h, combined with Multiple Linear Regression (MLR).

202 ed less allogeneic mixed leukocyte response (MLR).

203 rimary allogeneic mixed lymphocyte response (MLR) response.

204 pair, and genetic interventions that restore MLRs to normal cellular levels may be exploited as poten

205 Trauma patient Mphi that retained MLR-inducing capacity had a nonsignificant reduction in

206 on of a stimulating electrode into the right MLR.

207 s with estimation of monetized leakage risk (MLR).

208 had negligible alloreactivity in a secondary MLR.

209 cells suppressed both primary and secondary MLR in vitro in an antigen-specific manner.

210 ed, resulting in abrogation of the secondary MLR.

211 specifically inhibited the same freshly set MLR SI and caused recruitment of new CD4+CD25 high FOXP3

212 In addition, sFasL/MLR-treated cell populations contained influenza-specifi

213 These data indicate that this ex vivo sFasL/MLR depletion of alloreacting human donor anti-host T ce

214 re, mice transplanted with the ex vivo sFasL/MLR-treated cells had prolonged time to fatal GVHD in an

215 on of our model with traditional and simpler MLR and PLS regression models shows that KPLS better per

216 f reticulospinal neurons and markedly slowed MLR-evoked locomotion.

217 SRL-MLR-generated Tregs added as third components allospecif

218 on, as evidenced by a comparatively superior MLR stimulatory capacity.

219 alloTregs suppressed MLR at a 16:1 responder to suppressor ratio, whereas nTr

220 DCs reduced HLA-DR expression and suppressed MLR, whereas silencing of SUFU enhanced HLA-DR expressio

221 ld less potent than were MSCs in suppressing MLR.

222 contrast with previous studies using AM14 Tg MLR.Fas(lpr) mice, we found that a significant number of

223 changed cortical processing, revealing that MLR's effects on cortex are dissociable from locomotion.

224 The MLR can thus be considered as a command center for forwa

225 The MLR SNP prediction algorithm predicts a complete genotyp

226 sumed that higher brain regions activate the MLR in a graded fashion, but this has not been confirmed

227 f a new glutamatergic pathway activating the MLR in a graded fashion, and consequently evoking a prog

228 sult from higher brain inputs activating the MLR like a dimmer switch.

229 that the brainstem circuits activated by the MLR in the salamander are organized similarly to those p

230 with subsurface resources, and estimates the MLR reduction achievable by remediating leaks.

231 inus) that received parallel inputs from the MLR and projected back to reticulospinal cells to amplif

232 ceived direct muscarinic excitation from the MLR and projected glutamatergic excitation to reticulosp

233 ve direct glutamatergic connections from the MLR and that a subpopulation of MLR neurons plays a key

234 etrapods the downstream projections from the MLR to brainstem neurons are not fully understood.

235 we examined the brainstem circuits from the MLR to identified reticulospinal neurons in the salamand

236 esence of glutamatergic projections from the MLR to reticulospinal neurons.

237 When purified from the MLR, iT reg cells suppressed both primary and secondary

238 nally, at low and intermediate hardness, the MLR model is less responsive than the BLM to pH, but the

239 Local blockade of D1 receptors in the MLR decreased locomotor frequency, but did not disrupt t

240 Microinjections of a D1 antagonist in the MLR decreased the locomotor output elicited by PT stimul

241 In the MLR, B box-activated DCs acted as potent stimulators of

242 that PT stimulation evoked DA release in the MLR, together with the activation of reticulospinal cell

243 57BL/6 (B6) CD11c+ DCs+IL-2+TGF-beta1 in the MLR.

244 sfected DCs were poor allostimulators in the MLR.

245 reas enhancing the production of IL-4 in the MLR.

246 DCs to stimulate T cell proliferation in the MLR.

247 or augments apoptosis induced by IVIG in the MLR.

248 proliferation and apoptosis induction in the MLR.

249 ntrations inhibits cell proliferation in the MLR.

250 responses, suggesting a pathway linking the MLR to cortex.

251 effect of high-frequency stimulation of the MLR (MLR-HFS) on gait impairment in a rodent stroke mode

252 In vertebrates, stimulation of the MLR at increasing intensities elicits locomotion of grow

253 we find that optogenetic stimulation of the MLR in awake, head-fixed mice can induce both locomotion

254 With the exception of the MLR model, the ANN, kNN, and SVM QSARs were ensemble mod

255 the function and the precise anatomy of the MLR remain unclear in humans.

256 Since the discovery of the MLR some 50 years ago, it has been presumed that higher

257 rent but anatomically connected parts of the MLR were activated during the fast condition of each tas

258 -4, and IL-5 by T cells in the course of the MLR, was further enhanced when DC were treated with TSLP

259 nculopontine nucleus, considered part of the MLR, was recently identified in the monkey.

260 Overall, the MLR is less responsive to DOC than the BLM across a rang

261 xpansion phase of culture did not reduce the MLR suppressive capacity of Treg cells.

262 could suppress another immune response, the MLR.

263 ropriate under these circumstances since the MLR models suffer from "selection bias".

264 + T cells as responders and supplemented the MLR with IL-2 and TGF-beta1 and investigated whether DCs

265 luding 10 regions from HapMap shows that the MLR prediction combined with stepwise tag selection uses

266 These studies demonstrate that the MLR regulates cortical state in parallel with locomotion

267 Using neural tracing, we show that the MLR sends bilateral projections to the middle reticular

268 spinal neuron responses, indicating that the MLR sends glutamatergic inputs to reticulospinal neurons

269 the presence of a glutamatergic input to the MLR originating from the primal SNc that evokes graded l

270 Added to the MLR, atorvastatin reduced (i) the human PBMC response to

271 5) and severe lens phenotype compared to the MLR-10/Rac1 cKO (E15.5) mice.

272 found to project from the primal SNc to the MLR.

273 tum, but send a descending projection to the MLR.

274 or ventral tegmental area of mammals) to the MLR.

275 urochemically distinct cell types within the MLR: glutamatergic, GABAergic, and cholinergic neurons.

276 the closely linked GzmC and F genes in their MLR-derived CTLs and lymphokine-activated killer cells;

277 This MLR model also provides skilful seasonal outlooks of win

278 We suggest that this MLR-based approach, which includes the mechanistic found

279 Thus, MLRs provide an active membrane domain that tethers and

280 for intervention 96% vs 46%) and similar to MLR.

281 r intervention (94%), and it was superior to MLR (70%, 73%, and 70%, respectively).

282 edia without agents were added separately to MLRs using human leukocyte antigen two DR-matched and -m

283 eg) cells in mixed lymphocyte reaction (Treg MLR) and now report additional findings on the effects o

284 lerance protocol) caused donor-specific Treg MLR inhibition or recruitment.

285 As an example of the "Treg MLR" immune monitoring potential, addition of third comp

286 mixed lymphocyte reactions (MLRs) ("the Treg MLR"), with varying human leukocyte antigen (HLA) dispar

287 In the Treg MLR, the generation of CD4+CD25 high FOXP3+ cells is mor

288 allospecific Treg generation using the Treg MLR.

289 nation, were added to healthy volunteer Treg-MLR, testing (a) H-TdR incorporation for inhibition of l

290 spinal neurons on both sides to a unilateral MLR stimulation.

291 In vitro MLR and in vivo adoptive transfer experiments were condu

292 In a 1-way MLR, KA1010 (5 muMu) reduced parent cell proliferation f

293 A 2-way MLR, adopting IFN-gamma production as a marker of allore

294 ed recipient lymphocytes in a direct one-way MLR, and also analyzed the kinetics of expression of IFN

295 Two-way MLR-stimulated T cell proliferation was reduced by ATL31

296 By one-way MLRs, we observed that ATL313's inhibitory effect was du

297 diated lympholysis and interferon-gamma with MLR tests.

298 ly validated and direct comparison made with MLR in patients admitted to an independent institution i

299 Within MLRs are the scaffolding and cholesterol binding protein

300 4 after experimental stroke with and without MLR-HFS.