ライフサイエンスコーパス: MMN

1 MMN area is useful to diagnose attention deficits and MH

2 MMN generation was well explained (>90% variance) by dip

3 MMN is a sensitive index of the neural systems engaged i

4 MMN occurrence reflects the individual JND for temporal

5 MMN results showed effects of language experience and at

6 MMN source analysis indicated dipoles in both auditory c

7 MMN supplementation was associated with a small increase

8 MMN to FM tones and functional connectivity together acc

9 MMN was also observable for both the 15 ms and 185 ms ri

10 MMN was assessed in N=28 SZ patients immediately before

11 MMN was found only for global deviant patterns, and only

12 MMN was present in all NH listeners and CI users when th

13 MMNs, literacy, and cognitive abilities were compared am

14 aims of this work were to assess whether (1) MMN is altered in cirrhotic patients with MHE, compared

15 Among primiparous women (IFA = 131; MMN = 110; LNS = 128), the LNS group had greater mean bi

16 e rate ratio [IRR] 1.03, 95% CI: 0.92, 1.16; MMN-0 versus MMN-12: 1.05, 95% CI: 0.93, 1.18).

17 with MHE, compared to those without MHE, (2) MMN changes in parallel with performance in attention te

18 and/or MHE in a longitudinal study, and (3) MMN predicts performance in attention tests and/or in th

19 his analysis included 1057 women (IFA = 349, MMN = 354, LNS = 354).

20 Additionally, MMN was examined in three related conditions: bipolar di

21 The relationship between AER, MMN to FM tones, and rsfMRI was assessed in the subset w

22 ive function, which are reflected in altered MMN.

23 alone at age 12 mo than in the IFA (49%) and MMN (49%) groups.

24 IFA (79.1 +/- 2.9 cm and -0.87 +/- 0.99) and MMN (79.1 +/- 2.9 cm and -0.91 +/- 1.01) groups (P = 0.0

25 ume overall and in the SCN, SON, VM, DM, and MMN.

26 in women who started consuming FA, IFA, and MMN before the 12th week of gestation (4.6%, 4.2%, and 3

27 nces of SPB for women consuming FA, IFA, and MMN were 5.7%, 5.6% and 5.1%, respectively.

28 rences were found when comparing the LNS and MMN groups among primiparous women, and no group differe

29 fically with psychoacoustic measurements and MMN recordings.

30 An oddball design was used and both N1 and MMN components were examined.

31 eating an illusory difference between N1 and MMN source loci when estimated by using equivalent curre

32 ed to test the effects of neurochemistry and MMN amplitude on DST performance.

33 rst time the effects of memantine on PPI and MMN in CPD subjects.

34 nderstanding the basis for deficient PPI and MMN in psychotic disorders, as reduced NMDA activity is

35 es a potential link between neuronal SSA and MMN.

36 l was used to assess the effects of iron and MMNs and the interaction between these factors.

37 tcentral gyrus in the absence of an auditory MMN.

38 This study examined auditory MMN in otherwise healthy chronic cannabis users compared

39 Behavioral and ERP data including auditory MMN and P300 were collected during each test day.

40 The amplitude of auditory MMN was unchanged by tDCS.

41 findings, showing generators of the auditory MMN along the superior temporal gyrus with no evidence o

42 C would reduce ketamine effects on behavior, MMN, and P300 in healthy humans.

43 th schizophrenia showed associations between MMN and Heschl gyrus (r=-0.52; P=.02) not present in the

44 Associations between MMN, cannabis use measures, and symptoms were examined.

45 Infants of women consuming MMN supplements had an 18% reduction in early infant mor

46 In contrast, MMN amplitude correlated significantly and independently

47 In contrast, MMN deficits were not associated with symptom severity o

48 (MMN-0 = 20 [1 death]; MMN-6 = 21 [1 death]; MMN-12 = 20 [0 death]).

49 imilar between groups (MMN-0 = 20 [1 death]; MMN-6 = 21 [1 death]; MMN-12 = 20 [0 death]).

50 d by the duration + frequency double deviant MMN, are compromised before psychosis onset and can enha

51 The duration + frequency double deviant MMN, but not the single deviant MMNs, significantly pred

52 In addition, NAC reduced frequency-deviant MMN amplitude and increased target and novelty P3 amplit

53 The decrements in frequency-deviant MMN amplitude produced by ketamine and NAC were not addi

54 The pitch-deviant MMN reductions present in patients with chronic schizoph

55 ons for the frequency- and intensity-deviant MMNs and P300.

56 uble deviant MMN, but not the single deviant MMNs, significantly predicted the time to psychosis onse

57 recordings pointed to statistically distinct MMN topographies across senses, implying differential un

58 In the meta-analysis, duration MMN achieved the highest effect size measures.

59 g-term users also exhibited reduced duration MMN relative to control subjects and short-term users an

60 PCP significantly reduced rodent duration MMN (p<0.001) and theta-band (p<0.01) response.

61 ction and support utility of rodent duration MMN as a translational biomarker for investigation of me

62 Overall, we demonstrate that rodent duration MMN shows neuro-oscillatory signature similar to human M

63 At baseline, rodent duration MMN was associated with increased theta (theta)-frequenc

64 y enhanced PPI in CPD subjects, and enhanced MMN across subject groups.

65 nd under some conditions, memantine enhances MMN; these findings present a challenge to understanding

66 4%, 35%), and 5% (2%, 9%) in the Fe, MMN, Fe+MMN, and placebo groups, respectively.Daily iron supplem

67 cronutrients; MMN group), iron plus MMNs (Fe+MMN group), or placebo capsules.

68 , 30% (24%, 35%), and 5% (2%, 9%) in the Fe, MMN, Fe+MMN, and placebo groups, respectively.Daily iron

69 y control subjects completed a multi-feature MMN paradigm, which included duration, frequency, and in

70 The magnitude of the 20 Hz FM MMN correlated with the degree of discrepancy between co

71 f language experience and attentional focus: MMN amplitudes were smaller for American compared to Jap

72 leted an electroencephalographic session for MMN, magnetic resonance spectroscopy for glutamate and G

73 res 24 wk after recruitment (effect size for MMN groups combined: 0.084 SD/24 wk, 95% CI: 0.005, 0.16

74 Frequency MMN was similarly attenuated in short- and long-term use

75 binding to MNs was detected using sera from MMN patients with and without detectable anti-GM1 IgM an

76 Sensory gating, MMN, and P300 have been demonstrated to be impaired in s

77 Children of mothers given MMN had a mean score of 0.11 SD (95% CI 0.01-0.20, p=0.0

78 , GABA, the ratio of glutamine to glutamate, MMN amplitude, and DST.

79 with clinical ratings, patients with greater MMN impairments were more likely to live in highly struc

80 to receive 12 wk of iron (60 mg; Fe group), MMNs (14 other micronutrients; MMN group), iron plus MMN

81 th 0.09 presentations per wk for all groups (MMN-0 versus MMN-6: adjusted incidence rate ratio [IRR]

82 supplementation were similar between groups (MMN-0 = 20 [1 death]; MMN-6 = 21 [1 death]; MMN-12 = 20

83 ans and rhesus macaques and found homologous MMN and P3a ERPs during an auditory oddball paradigm.

84 However, MMN and P3a reductions were unrelated in both UHR and FE

85 bin in nonpregnant Cambodian women; however, MMNs did not confer additional significant benefit.

86 However, unlike the human MMN, larger deviant responses were characterized by the

87 ble novelty correlates aligning to the human MMN.

88 e novelty in a manner analogous to the human MMN.

89 neuro-oscillatory signature similar to human MMN, along with sensitivity to the NMDAR antagonist and

90 and 49.9 +/- 2.1 cm (P = 0.104) in the IFA, MMN, and LNS groups, respectively.

91 ogic testing and magnetic resonance imaging: MMN and magnetic resonance imaging in 11 subjects with s

92 pathogenicity of anti-GM1 IgM antibodies in MMN patients and link their presence to the clinical cha

93 ge effect size (P=0.0004, d=1.0) deficits in MMN generation across deviant types.

94 Initially, groups did not differ in MMN amplitude.

95 The role of glutamate and GABA in MMN and verbal working memory deficits in schizophrenia

96 Recently, our group showed a reduction in MMN to changes in the duration and intensity of backgrou

97 dicted, patients showed robust reductions in MMN across FM stimulus type (p = 0.005), particularly to

98 e groups no longer differed significantly in MMN amplitude during deviant pitch tones, and the degree

99 Larger MMN amplitudes recorded both before and after the traini

100 All 4 groups showed approximately 64% larger MMN to pitch-deviant tones over the right hemisphere com

101 arned English after age 10, exhibited larger MMN and P3a responses than early bilinguals, across all

102 had fewer years of L2 experience had larger MMN, P3a, and LN responses than those who learned it ear

103 on, hyperarticulated stimuli elicited larger MMNs for both language groups, suggesting vowel space ex

104 y deviants, along with At the network level, MMN deficits engaged canonical somatomotor, ventral atte

105 dings reinforce recent human studies linking MMN deficits to theta-band neuro-oscillatory dysfunction

106 At longitudinal MMN testing, schizophrenia showed MMN reduction (P=.004)

107 Maternal MMN supplementation might therefore be an important part

108 Maternal MMN supplementation, as compared with IFA, can reduce ea

109 INTERPRETATION: Maternal MMN had long-term benefits for child cognitive developme

110 Results showed that the mean MMN amplitude of the Chinese group was larger than that

111 The mean MMN amplitude was larger for the T1/T3 relative to the T

112 Multiple micronutrients (MMN) are commonly prescribed in pediatric primary health

113 upplementation with multiple micronutrients (MMN) has been hindered by little evidence of the effects

114 upplementation with multiple micronutrients (MMN) or iron + folic acid (IFA), versus folic acid (FA)

115 upplementation with multiple micronutrients (MMN) or iron and folic acid (IFA) in Indonesia.

116 ron with or without multiple micronutrients (MMNs) on hemoglobin concentration in nonpregnant Cambodi

117 capsule containing multiple micronutrients (MMNs), or one 20-g sachet of SQ-LNS (LNS, containing 118

118 g; Fe group), MMNs (14 other micronutrients; MMN group), iron plus MMNs (Fe+MMN group), or placebo ca

119 n analysis and structural equation modeling, MMN and diagnostic group were significant independent pr

120 otential studies as the mismatch negativity (MMN) and has been observed in several sensory modalities

121 Auditory mismatch negativity (MMN) and P300 event-related potentials (ERPs) are reduce

122 s is on sensory gating, mismatch negativity (MMN) and P300, thereby discussing which parameters allow

123 elated brain potentials mismatch negativity (MMN) and P300.

124 For example, the mismatch negativity (MMN) and P3a event-related potentials (ERPs), neurophysi

125 ated potential known as mismatch negativity (MMN) and provides a potential link between neuronal SSA

126 Mismatch negativity (MMN) and visual P1 are established event-related potenti

127 nses, as indexed by the mismatch negativity (MMN) component of the auditory event-related potential (

128 Here, we examined the mismatch negativity (MMN) component of the event-related potential elicited a

129 The mismatch negativity (MMN) component of the human event-related potential prov

130 response resembled the mismatch negativity (MMN) evoked by surprising or unlikely events in traditio

131 age study utilizing the mismatch negativity (MMN) evoked response was conducted to explore the influe

132 generation of auditory mismatch negativity (MMN) generation are among the most widely replicated neu

133 tone deviant elicited a Mismatch Negativity (MMN) in all participant groups before and after training

134 prepulse inhibition and mismatch negativity (MMN) in SZ patients and healthy subjects (HS).

135 Mismatch negativity (MMN) indexes pre-attentive information processing dysfun

136 Auditory mismatch negativity (MMN) is a biomarker for schizophrenia thought to reflect

137 The mismatch negativity (MMN) is a brain event-related potential marker of change

138 The mismatch negativity (MMN) is a preattentive component of the auditory event-r

139 Mismatch negativity (MMN) is an auditory event-related potential that reflect

140 The mismatch negativity (MMN) is an event related potential evoked by violations

141 Mismatch negativity (MMN) is an event-related potential measure that occurs i

142 The observation that mismatch negativity (MMN) is consistently impaired in schizophrenia has gener

143 Mismatch negativity (MMN) is reduced in chronic but not first-hospitalized sc

144 The mismatch negativity (MMN) is thought to index the activation of specialized n

145 auditory event-related mismatch negativity (MMN) potentials to frequency modulated (FM) tones at 5,

146 Chinese tones while the mismatch negativity (MMN) response was elicited using a passive oddball parad

147 electroencephalographic mismatch negativity (MMN) response.

148 , with the multisensory mismatch negativity (MMN) significantly different from the sum of the unisens

149 We used mismatch negativity (MMN) to examine structural encoding of local and global

150 Mismatch negativity (MMN) to FM tones was assessed in 43 patients/36 controls

151 Mismatch negativity (MMN) was used to index discrimination of the temporally-

152 e two systems using the mismatch negativity (MMN), a well studied EEG effect evoked by acoustic devia

153 ed to determine whether mismatch negativity (MMN), an event-related potential index of auditory senso

154 The significance of the mismatch negativity (MMN), an event-related potential measured in humans whic

155 Mismatch negativity (MMN), an evoked potential calculated by subtracting the

156 between groups for the mismatch negativity (MMN), but the late discriminative negativity (LDN) was r

157 ed PTSD showed enhanced mismatch negativity (MMN), increased theta power (5-7 Hz), and stronger suppr

158 itory change detection, mismatch negativity (MMN), is one of the most consistent findings in schizoph

159 f neurodynamics, namely mismatch negativity (MMN), P300, and contingent negative variation (CNV), as

160 EG/ERP study, using the mismatch negativity (MMN), P3a, and late negativity (LN), we examined differe

161 ic brain potential, the mismatch negativity (MMN), when listeners did not attend to the auditory stim

162 ere presented to derive mismatch negativity (MMN), which reflects the ability to automatically detect

163 esponses--including the mismatch negativity (MMN).

164 rical recordings of the mismatch negativity (MMN).

165 ogram (EEG) paradigm of mismatch negativity (MMN).

166 n (PPI) of startle) and mismatch negativity (MMN).

167 ity to acoustic change [mismatch negativity (MMN)] were measured in a group of children with learning

168 itory change detection (mismatch negativity [MMN]) was used to assess whether sensory memory represen

169 m patients with multifocal motor neuropathy (MMN) using human induced pluripotent stem cell (iPSC)-de

170 ial nucleus (VM), medial mammillary nucleus (MMN), and lateral hypothalamic area (LHA).

171 hical Cox regression examined the ability of MMN to predict time to psychosis onset in CHR patients.

172 udy expands upon an earlier meta-analysis of MMN impairment in schizophrenia by examining impairment

173 A regional analysis of MMN revealed that the largest correlations of MMN to eve

174 of 21 tests showed a positive coefficient of MMN versus IFA (p=0.0431) with effect sizes from 0.00-0.

175 MN revealed that the largest correlations of MMN to everyday functioning were present at frontocentra

176 for the MMN, which models the disruption of MMN observed in schizophrenia, may occur at a mechanisti

177 the mechanisms behind the lack of effect of MMN supplementation on morbidity measures and limited ef

178 The effectiveness of MMN in improving morbidity or growth in sick children pr

179 We assessed the effects of MMN and associations of biomedical (ie, maternal and chi

180 indered by little evidence of the effects of MMN on fetal loss and infant death.

181 , that accounts for the critical features of MMN.

182 ficits of large effect size in generation of MMN (d>1.26) and P300 (d=1.08) relative to comparison su

183 Greater levels of MMN impairment were associated with lower Global Assessm

184 MMN elicitation, and that the source loci of MMN and N1 are different.

185 for a better understanding of the nature of MMN impairment in schizophrenia, as well as its potentia

186 es remain unresolved regarding the nature of MMN impairment.

187 The pattern of MMN alterations in cannabis users differed from that typ

188 s and caution with systematic prescribing of MMN.

189 rt glutamatergic and GABAergic regulation of MMN and verbal working memory function in schizophrenia.

190 s known about the functional significance of MMN deficits in schizophrenia patients.

191 These findings encourage future trials of MMN as a biomarker for individual assignment, prediction

192 dent in older CPD patients, whereas those on MMN were most evident in younger subjects.

193 262 midwives to distribute IFA (n=15 ,86) or MMN (n=15,804) supplements to 31 290 pregnant women thro

194 Demyelinating neuropathy (CIDP or MMN) occurred in close to 30% of the patients, in a high

195 ndomly assigned to receive daily FA, IFA, or MMN from the period before 20 weeks' gestation to delive

196 Starting use of FA, IFA, or MMN supplements before the 12th week of gestation produc

197 We performed MMN analysis as well as attention and coordination tests

198 other micronutrients; MMN group), iron plus MMNs (Fe+MMN group), or placebo capsules.

199 Significant pretraining vs posttraining MMN amplitude reduction was also observed (p<0.02).

200 stic startle magnitude and habituation, PPI, MMN, autonomic indices, and subjective self-rating scale

201 Furthermore, it extends previous MMN findings that have revealed indexing of complex abst

202 Unlike previous MMN studies of local-global auditory perceptual organiza

203 The resulting scheme generates realistic MMN waveforms, explains the qualitative effects of devia

204 Reduced MMN amplitude was linked to poor verbal working memory i

205 Patients also exhibited reduced MMN to all deviant types.

206 zophrenia patients had significantly reduced MMN (P<.001).

207 In this report, we examine whether reduced MMN, as well as P3a, another index of auditory deviance

208 in 5 years of illness onset, whereas reduced MMNs to changes in sound frequency were only seen in pat

209 While many studies have reported MMN deficits in schizophrenia patients, little is known

210 icitation of a mismatch negativity response (MMN) by changes in repetitive aspects of auditory stimul

211 ur electroencephalography data show a robust MMN after a single standard event when the interval betw

212 evaluate physiological properties of rodent MMN, along with sensitivity to NMDAR agonist and antagon

213 At first hospitalization for schizophrenia, MMN indexed left hemisphere Heschl gyrus gray matter vol

214 rly to those with schizophrenia, should show MMN alterations related with attention deficits.

215 ngitudinal MMN testing, schizophrenia showed MMN reduction (P=.004).

216 stage of processing, we observed significant MMN effects for both head-independent and head-centered

217 Simultaneously, MMN was obtained to unattended pitch, duration, and inte

218 Smaller MMN amplitude was significantly associated with lower GA

219 d that Greek speakers in general had smaller MMNs compared to English speakers, confirming previous s

220 dyslexic group showed significantly smaller MMNs in the 20 Hz FM condition in both the early (150-30

221 gh rising deviant elicited no, or a smaller, MMN, which became larger after training only in the Engl

222 al gyrus with no evidence of a somatosensory MMN in this region, whereas a robust somatosensory MMN w

223 increase in peak amplitude of somatosensory MMN after anodal tDCS (F(1,9) = 8.98, P < 0.02, mean dif

224 reduction in peak amplitude of somatosensory MMN after cathodal tDCS (F(1,9) = 7.15, P < 0.03, mean d

225 se bidirectional modulation of somatosensory MMN and the dependence of somatosensory MMN on the cereb

226 sory MMN and the dependence of somatosensory MMN on the cerebellum.

227 se to explore the mechanism of somatosensory MMN, and specifically its dependence on the cerebellum.

228 this region, whereas a robust somatosensory MMN was recorded from postcentral gyrus in the absence o

229 In the longitudinal study, MMN area improved in parallel with performance in attent

230 For this study, MMN data to frequency, intensity and duration-deviants w

231 In summary, MMN studies have produced the most convincing results un

232 Longitudinal electrophysiologic testing: MMN in 16 subjects with schizophrenia, 17 subjects with

233 y chi2 test), supporting the contention that MMN serves as an intermediate biomarker linking glutamat

234 We found that MMN impairment 1) likely reflects a vulnerability to dis

235 ng characteristic curve analyses showed that MMN area predicts attention deficits in the number conne

236 Logistic regression analyses showed that MMN area predicts performance in attention tests and in

237 These results suggest that MMN, as well as P3a, to duration deviants are reduced in

238 This pattern of results suggests that MMN deficits represent a core neurophysiological dysfunc

239 The MMN amplitude was significantly reduced to frequency but

240 The MMN deficits are independently related to premorbid func

241 The MMN is reported to be reduced in patients with chronic s

242 The MMN is then modelled as the superposition of the electri

243 The MMN is thought primarily to reflect the activity of sens

244 The MMN to duration versus other deviants was differentially

245 The MMN was compared between users and control subjects as w

246 The MMN was visualized by subtracting the averaged event-rel

247 The MMN wave area was reduced in patients with MHE, but not

248 issociating SSA from sensory novelty and the MMN.

249 auditory novelty detection as indexed by the MMN is dissociable from SSA at the level of activity enc

250 Early first-level processing indexed by the MMN was sensitive to stimulus predictability: here, atte

251 duration discriminations, represented by the MMN, were generated in the same cortical regions regardl

252 antagonists, which are known to disrupt the MMN, suppressed the magnitude of multiunit responses in

253 rther, the NMDA sensitivity reported for the MMN, which models the disruption of MMN observed in schi

254 However, the MMN may also reflect the violation of a prediction based

255 ented that validates our key hypothesis: the MMN results from active cortical prediction rather than

256 normal subjects, including reductions in the MMN and the P3a.

257 with 13.7% in the IFA group and 12.9% in the MMN group (P = 0.12).

258 Children of anaemic mothers in the MMN group scored 0.18 SD (0.06-0.31, p=0.0047) higher in

259 ighest in the LNS group, intermediate in the MMN group, and lowest in the IFA group, but except for m

260 sed risk of low birthweight for those in the MMN group, with a 33% (RR 0.67, 0.51-0.89, p=0.0062) dec

261 Moreover, the MMN amplitudes evoked from the left forebrain and midbra

262 work advances a formal understanding of the MMN and--more generally--illustrates the potential for d

263 We investigate possible homologs of the MMN in macaque primary auditory cortex (A1) using a freq

264 ous studies demonstrating sensitivity of the MMN to language background.

265 to sound omission, and a sensitivity of the MMN to NMDA receptor antagonists.

266 sensory novelty, a defining property of the MMN, however, has not been resolved.

267 nt for the major empirical properties of the MMN.

268 of deviant probability and magnitude on the MMN - in terms of latency and amplitude--and makes quant

269 Source solution further revealed the MMN involved the neural activations from the visual cort

270 rmance on auditory psychophysical tasks, the MMN responses of some dyslexic adults were found to be a

271 nd standing with assistance earlier than the MMN group (B = 0.51; 95% CI: 0.12, 0.89; P = 0.029).

272 The results showed that the MMN amplitude for the first call note was significantly

273 This has been taken to suggest that the MMN is a correlate of true change or "deviance" detectio

274 c, and psychophysical data indicate that the MMN is generated as a result of differential adaptation

275 Results showed that the MMN reflects sensitivity only to the formant structure o

276 he neurobiological mechanisms underlying the MMN is still lacking, and its computational foundations

277 requency analysis revealed that, whereas the MMN reflected enhanced intertrial coherence in the theta

278 for explicit deviant detection (as with the MMN).

279 hich shares several characteristics with the MMN.

280 from the ratio of glutamine to glutamate to MMN to verbal working memory (P = .38 [root-mean-square

281 novel" stimulus constitute a prerequisite to MMN elicitation, and that the source loci of MMN and N1

282 ldhood development, and policy change toward MMN.

283 Irrespective of deviant type, MMN was significantly reduced in ESZ and CHR patients re

284 his study investigates mechanisms underlying MMN impairments in schizophrenia using event-related pot

285 tly different from the sum of the unisensory MMNs.

286 A, the relative risks of SPB for those using MMN and IFA were 0.99 (95% confidence interval: 0.85, 1.

287 [IRR] 1.03, 95% CI: 0.92, 1.16; MMN-0 versus MMN-12: 1.05, 95% CI: 0.93, 1.18).

288 ntations per wk for all groups (MMN-0 versus MMN-6: adjusted incidence rate ratio [IRR] 1.03, 95% CI:

289 In agreement with visual MMN studies, single-unit IT responses were greater for t

290 To determine whether MMN shows associations with underlying auditory cortex g

291 We also examined whether MMN predicted subsequent conversion to psychosis in CHR

292 While MMN has been extensively used in human electrophysiologi

293 Community-wide MMN supplementation has shown limited and heterogeneous

294 s reduction (P=.003), highly correlated with MMN reduction (r=0.6; P=.04).

295 ients, similar to our reported findings with MMN.

296 ver, the association of glutamate level with MMN has not been directly examined in patients with schi

297 the effect of maternal supplementation with MMN, compared with IFA, on fetal loss and infant death i

298 mentation with unfortified SQ-LNS for 12 wk (MMN-0) to be consumed in daily portions.

299 th micronutrient-fortified SQ-LNS for 12 wk (MMN-12), (2) supplementation with micronutrient-fortifie

300 wk followed by unfortified SQ-LNS for 6 wk (MMN-6), or (3) supplementation with unfortified SQ-LNS f