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1                                              MMSE (score and items) did not discriminate patients wit
2                                              MMSE has been used in hepatology but its usefulness in t
3                                              MMSE, RPM, AVLT, DS, and BD scores were higher in the AB
4  1.51 points [95% CI, 0.94-2.10]; P < .001), MMSE (mean difference, 0.56 points [95% CI, 0.32-0.80];
5 cline (ADAS_cog, P = 0.011; FAQ, P = 0.0016; MMSE, P = 0.004).
6  vs. post-procedure: 5 [3 to 7], p = 0.002), MMSE (pre-procedure: 27 [25 to 28] vs. post-procedure: 2
7 xicity were IRI arm (odds ratio [OR], 5.03), MMSE </= 27/30 (OR, 3.84), and impaired IADL (OR, 4.67);
8  interaction contributed an additional -0.16 MMSE points per year (95% CI, -0.29 to -0.03; P = .01).
9 e decline attributable to delirium was -0.37 MMSE points per year (95% CI, -0.60 to -0.13; P < .001).
10 e pathologic processes of dementia was -0.39 MMSE points per year (95% CI, -0.57 to -0.22; P < .001).
11 hologic processes of dementia declining 0.72 MMSE points per year faster than age-, sex-, and educati
12                       Cognition averaged 0.8 MMSE (mini-mental state examination) points better (95%
13  and 7 patients with Alzheimer disease (AD) (MMSE <19).
14 nd performance on cognitive tests (ADAS_cog, MMSE, and FAQ) was determined with 2 different correlati
15                               Combining age, MMSE score, Charlson's comorbidity index, and length of
16                      In conclusion, although MMSE score and single items are altered in patients with
17 howed individually divergent results with an MMSE rate of change of -3.2 points per year.
18 .003), PSP (p=0.022) and CBD (p=0.0002); and MMSE in CBD (p=0.004).
19 nt change on CDR-SB (r = 0.42, P < 0.01) and MMSE (r =-0.52, P < 0.01).
20      In the ABC21, folate, vitamin B-12, and MMSE score were positively correlated and homocysteine w
21  (MMSE) score and microglial activation, and MMSE score and rCMRGlc.
22                     The domain-composite and MMSE-type global cognitive function z scores both decrea
23 iation between use of psychotropic drugs and MMSE score (p = 0.004) is particularly potent in those c
24 otal score of SOFA (severity of illness) and MMSE (cognitive impairment).
25 correlation was found between VF indices and MMSE or H-Y scores.
26 he change of ipsilateral brain perfusion and MMSE (r = -0.33, p = 0.01) was also identified.
27                On the basis of age, sex, and MMSE score only, the 3-year progression risk to AD demen
28 henotypic sharing between RNFL thickness and MMSE (5%, 95% CI: 0-10%) or RT (7%, 95% CI: 1-12%).
29 he relation between level of alcohol use and MMSE score change between waves 2 and 3 of the study was
30 orrelation with any of MMSE items as well as MMSE summary score.
31 ly tested within 12 months of death (average MMSE 24.2).
32 ms experienced a significant gain in average MMSE score longitudinally over time, with no difference
33 erienced a statistically significant average MMSE score increase over time, with no difference betwee
34      Most patients with an abnormal baseline MMSE score (< 27) experienced significant increases.
35           Patients with an abnormal baseline MMSE were more likely to have an improvement in cognitiv
36 seline MMSE 20-26) and moderate AD (baseline MMSE 15-19) responded differently to tarenflurbil in the
37 evealed that patients with mild AD (baseline MMSE 20-26) and moderate AD (baseline MMSE 15-19) respon
38                       Age at onset, baseline MMSE, years of education, motor exam score, sex, depress
39 of aging 5 years in relation to the baseline MMSE score in study data.
40                                       The BC-MMSE association was greater only among individuals with
41                 However, CRP modified the BC-MMSE relationship, with stronger associations only at hi
42                                      Because MMSE could be impaired in several cognitive dysfunctions
43 n, smoking, alcohol intake, and time between MMSE tests.
44  well with cognitive status as determined by MMSE when the entire cohort of controls and AD patients
45 yme immunoassay, and the cognitive status by MMSE.
46 slightly lower in individuals with decreased MMSE scores.
47 uce the Bayesian minimum mean squared error (MMSE) conditional error estimator and demonstrate its co
48 4) as well as on the mini-mental state exam (MMSE), digit symbol substitution (DSS) test, and a verba
49 airment (MCI) [minimental state examination (MMSE) >/=19], 2 patients with pre-MCI (normal MMSE), and
50 asured by the Mini-Mental State Examination (MMSE) and assessed by observers through the Informant Qu
51 determined by Mini-Mental State Examination (MMSE) and Cambridge Assessment of Mental Health for the
52 ing using the Mini-Mental State Examination (MMSE) and Clinical Dementia Rating scale.
53  standardized Mini-Mental State Examination (MMSE) and memory, processing speed, language, and execut
54 ng tests, the Mini-Mental State Examination (MMSE) and Mini-Cog, administered at hospital discharge,
55 s assessed by Mini-Mental State Examination (MMSE) at baseline and years 1, 2, 3, and 5.
56 alk tests and Mini-Mental State Examination (MMSE) at initial, two-month, and yearly visits.
57 completed the Mini-Mental State Examination (MMSE) at three time points in 1981, 1982, and 1993-1996.
58 completed the Mini-Mental State Examination (MMSE) during three study waves in 1981, 1982, and 1993-1
59 completed the Mini-Mental State Examination (MMSE) in 1991, and at that time, they or caregivers also
60               Mini-Mental State Examination (MMSE) is one of the most commonly used methods in the as
61 sessed by the Mini-Mental State Examination (MMSE) is reported here.
62 i type, and a Mini-Mental State Examination (MMSE) rate of change of +1.8 points per year, whereas pa
63 arameters and Mini-Mental State Examination (MMSE) results.
64 0.009], lower Mini-Mental State Examination (MMSE) score (MMSE, [27 (23-29) vs 28 (27-30) points; P =
65 gender, lower mini-mental state examination (MMSE) score and higher AD assessment scale cognitive sub
66 ation between mini-mental state examination (MMSE) score and microglial activation, and MMSE score an
67 ity who had a mini-mental state examination (MMSE) score of 15-26 were randomly assigned to receive t
68 </= 1 in 75%, Mini-Mental State Examination (MMSE) score was </= 27/30 in 31%, and Instrumental Activ
69 all score and Mini-Mental State Examination (MMSE) score was assessed at baseline and follow-up using
70  (FPI) score, Mini-Mental State Examination (MMSE) score, and handgrip and handheld dynamometer stren
71      Folstein Mini-Mental State Examination (MMSE) scores and neurologic function scores (NFS) at bas
72 subscores and Mini Mental State Examination (MMSE) scores at baseline (analyses of variance, receiver
73 es: Change in Mini-Mental State Examination (MMSE) scores during the 6 years before death.
74 and change in Mini-Mental State Examination (MMSE) scores in the Normative Aging Study, a cohort of e
75 s incapacity, Mini-Mental State Examination (MMSE) scores less than 20 increased the likelihood of in
76 ct changes in Mini-Mental State Examination (MMSE) scores over 24-months using standardized data.
77  and Folstein Mini-Mental State Examination (MMSE) scores recorded at baseline, 6, 12, 18, and 24 mon
78 esveratrol on Mini-Mental State Examination (MMSE) scores, macrophage M1M2 phenotype [the ratio of in
79 nts including Mini-Mental State Examination (MMSE), Alzheimer Disease Assessment Scale-Cognitive subs
80 atteries, the Mini-Mental State Examination (MMSE), and the Montreal Cognitive Assessment (MoCA).
81 ssment (MNA), Mini-Mental State Examination (MMSE), Geriatric Depression Scale (GDS15), and Timed Get
82 rmance by the Mini Mental State Examination (MMSE), National Adult Reading Test (NART), Raven's Progr
83 ormance using Mini Mental State Examination (MMSE), PD staging using modified Hoehn and Yahr (H-Y) sc
84 scores on the Mini-Mental State Examination (MMSE), the Brief Psychiatric Rating Scale, the Scale for
85 ubscales, the Mini-Mental State Examination (MMSE), the HD Activities of Daily Living (ADL) Scale, an
86 sted with the Mini-Mental State Examination (MMSE), the Mattis Dementia Rating Scale, and the Executi
87  (UPSIT), the Mini-Mental State Examination (MMSE), the Mattis Dementia Rating Scale-2 (DRS-2), and t
88  by using the Mini-Mental State Examination (MMSE), the Trail Making Test (TMT) A and B, and the Verb
89 /= 25) on the Mini-Mental State Examination (MMSE), which is a proxy of poor cognition.
90 y of 7 tests: Mini-Mental State Examination (MMSE), word list learning (verbal memory), digit span (a
91 individuals); Mini-Mental State Examination (MMSE)-type tests were available at the end of treatment
92 ally with the mini-mental state examination (MMSE).
93 ing Scale and Mini-Mental State Examination (MMSE).
94 rmance on the Mini-Mental State Examination (MMSE).
95 n measured by Mini-Mental State Examination (MMSE).
96 e severity on Mini Mental State Examination (MMSE).
97  'passed' the Mini-Mental State Examination (MMSE).
98 erformance on mini mental state examination (MMSE, F(5,883) = 5.8, p < 0.001), and with faster reacti
99 scores on the Mini-Mental State Examination (MMSE; -2.4 points over 36 weeks) and the cognitive subsc
100 performance), Mini-Mental State Examination (MMSE; 0 [worst] to 30 [best] points), Clinical Dementia
101  p=0.011) and Mini-Mental State Examination (MMSE; p=0.004) at 1 year; these differences were not pre
102 he use of the Mini-Mental State Examination (MMSE; score range, 0 to 30, with lower scores indicating
103 pression with MiniMental Status Examination (MMSE) and neurofibrillary tangle (NFT) scores across all
104  underwent a Mini-Mental Status Examination (MMSE) for correlation with the whole-brain NAA.
105 er, race and Mini-Mental Status Examination (MMSE) result were not predictive.
106 ted with the mini-mental status examination (MMSE) score.
107 ividuals with MiniMental Status Examination (MMSE) scores lower than 10 were testable by recognition
108       Hourly Mini-Mental Status Examination (MMSE) scores showed an increase during stimulation when
109 AS_cog), the Mini-Mental Status Examination (MMSE), and the Functional Activities Questionnaire (FAQ)
110 t (defined as Mini Mental State Examination [MMSE] </=25) using data from nine cohorts of patients wi
111  10.6 y; mean Mini-Mental State Examination [MMSE] score +/- SD, 22.2 +/- 6.0) or frontotemporal loba
112 th severe AD (mini-mental state examination [MMSE] score 5-12 points), in a nursing home setting were
113 age, sex, and Mini-Mental State Examination [MMSE] score), magnetic resonance imaging (MRI) biomarker
114 and mean [SE] Mini-Mental State Examination [MMSE] score, 28 [0.3]), 61 patients with mild cognitive
115 er's disease (mini-mental state examination [MMSE] scores 10-24) at 11 sites in Russia.
116  the 30-point Mini Mental State Examination [MMSE]), and adverse events.
117 irrhosis underwent neurological examination, MMSE and electroencephalography (EEG).
118   Sequential Mini-Mental State Examinations (MMSE) demonstrated an 80% reduction in new cognitive def
119 s in the proportion of patients experiencing MMSE score decline between the randomized study arms at
120                           Geriatric factors (MMSE and IADL) are predictive of severe toxicity or unex
121 re for PACC at baseline was 0.00 (2.60); for MMSE, 29.0 (1.2); for CDR-Sum of Boxes, 0.04 (0.14); and
122 ifferences were found between PD and PSP for MMSE and ACE-R (total score and subscores for attention
123 readmission performance status, IADL, GDS15, MMSE, GUG, and MNA were associated with increased likeli
124 items are altered in patients with overt HE, MMSE has no value in the assessment of minimal HE.
125 iving independently had significantly higher MMSE scores, lower SANS scores, more years of education,
126 vere toxicity or unexpected hospitalization (MMSE) in a randomized prospective phase III study in mCR
127 f Abeta, and are associated with an improved MMSE rate of change in ApoEe3/e3 vs. ApoEe3/e4 patients.
128  -2.7 p = 0.004, MWU Z = -3.0 p = 0.005), in MMSE at one and 52 weeks (MWU Z = -2.9 p = 0.003, MWU Z
129 oncentration was associated with a change in MMSE score of -0.24 (95% confidence interval: -0.44, -0.
130 ge was positively correlated with changes in MMSE and memory scores after 24 months in the drug group
131                               The changes in MMSE score in response to the 3 different treatments wer
132  patients experienced significant decline in MMSE score.
133 ifying subjects with significant declines in MMSE scores, and (3) incorporating SNPs of top 10 genes
134 sion, there was significant deterioration in MMSE scores for patients who were to experience progress
135 ion, clinically significant deterioration in MMSE scores was a strong predictor of a more rapid time
136 lly significant difference between groups in MMSE score.
137         No corresponding changes occurred in MMSE scores.
138                                   Individual MMSE score differences between waves 2 and 3 were calcul
139                                Their initial MMSE score of 23.3 (SD=4.2) improved to 26.6 (SD=3.5) at
140  in cognitive function over 2 years (initial MMSE score: mean=26.3, SD=3.1; score at 2-year follow-up
141 95% CI: 1.20, 2.05) times higher odds of low MMSE scores.
142 ypertension, older age, female gender, lower MMSE score and higher ADAS-cog score, had a high risk fo
143 hose who ate more calories in 1976 had lower MMSE scores in 1991 (p = 0.03), an association strengthe
144             Less-educated subjects had lower MMSE scores, especially among the very elderly.
145 ls (CIs) = 20.95 to 23.13; 124 control, mean MMSE = 22.59, 95% CI = 21.58 to 23.6; p = 0.48).
146 rences, the between-group difference in mean MMSE scores was significant 30 days after surgery (P<0.0
147 severity at diagnosis (99 intervention, mean MMSE = 22.04, 95% confidence intervals (CIs) = 20.95 to
148 hose without delirium, both at 1 month (mean MMSE score, 24.1 vs. 27.4; P<0.001) and at 1 year (25.2
149  postoperatively had lower preoperative mean MMSE scores than those in whom delirium did not develop
150                                     The mean MMSE score 6 years before death was 24.7 points.
151                                     The mean MMSE score of patients with MCI and pre-MCI was 25.9 at
152 D) (n = 31; age +/- SD, 63.9 +/- 7.1 y, mean MMSE score +/- SD, 23.8 +/- 6.7).
153                                   The median MMSE score for survivors was within the reference range
154                      At baseline, the median MMSE score in patients in both the apolipoprotein E (Apo
155 pulation, including nonsurvivors, the median MMSE score was 14 in the 33 degrees C group (interquarti
156 MSE) >/=19], 2 patients with pre-MCI (normal MMSE), and 7 patients with Alzheimer disease (AD) (MMSE
157 items, MDF showed no correlation with any of MMSE items as well as MMSE summary score.
158  not result in significantly higher rates of MMSE score decline than RT alone through 5 years of foll
159 HE and with overt HE were seen in respect of MMSE score (p<0.02), orientation to place (p<0.003), rep
160 of the study was to investigate the value of MMSE in detection of HE in patients with cirrhosis.
161  95% confidence interval (CI) -0.06-0.02) or MMSE score (0.06, 95% CI -0.002-0.12).
162 nce, 2.4 [95% CI, -1.2 to 6.0], P = .19), or MMSE score (difference, 0.6 [95% CI, -0.2 to 1.4], P = .
163 diovascular Society Angina Classification or MMSE scores.
164 ive function especially the primary outcomes MMSE and episodic memory with Bushen capsule treatment.
165 o cerebellum FDG metabolism ratios predicted MMSE (beta=0.38, p=0.001) and MoCA (beta=0.3, p=0.002) a
166  stimulation when compared to prestimulation MMSE, largely due to improvement in recall, possibly rep
167 ementia (mini-mental state examination score MMSE=23), mean yearly loss of brain volume was 2.8% (95%
168  Mini-Mental State Examination (MMSE) score (MMSE, [27 (23-29) vs 28 (27-30) points; P = 0.021], leng
169 age, 65 [1] years; 45% female; and mean [SE] MMSE score, 22 [0.7]).
170 age, 68 [1] years; 38% female; and mean [SE] MMSE score, 27 [0.3]), and 65 patients with AD (mean [SE
171 under ROC of 0.814 in predicting significant MMSE decline: our model has 100% precision at 5% recall,
172 ross only control and incipient AD subjects (MMSE > 20).
173     Test performance analysis has shown that MMSE has no value as a prediction method in determining
174                                          The MMSE is a relatively insensitive tool, and subtle change
175                                          The MMSE is useful only at extreme scores.
176                                          The MMSE rate of change increased in the ApoE epsilon3/epsil
177 d greater declines in verbal fluency and the MMSE (P < 0.05).
178 ven patients (39%) were impaired on both the MMSE and Mini-Cog, whereas only 20 patients (28%) had sc
179 ad cognitive impairments, as assessed by the MMSE and Mini-Cog, at hospital discharge.
180 op in cognitive function (as measured by the MMSE score) 2 days after surgery than did those without
181 al cognitive performance, as assessed by the MMSE score.
182  after focal radiotherapy as measured by the MMSE.
183 eventy critically ill patients completed the MMSE and Mini-Cog just before hospital discharge.
184 th a rate of decline slightly slower for the MMSE (0.004 SD/decade less, 95% CI [0.002, 0.006], p = 0
185                                 However, the MMSE and Mini-Cog scores did not predict long-term cogni
186 le cases of incident dementia (a fall in the MMSE score to <24 points or a drop of three points in 1
187               The adjusted difference in the MMSE score was -0.90 (95% CI: -1.6, -0.19; P for trend =
188 n multiple cognitive measures, including the MMSE, the cognitive subscale of the Brief Psychiatric Ra
189  regression analyses showed that neither the MMSE nor the Mini-Cog predicted cognitive sequelae at 6
190 ive decline not apparent with the use of the MMSE.
191 w the mean of the comparison subjects on the MMSE (N=8, 44%) and the Mattis Dementia Rating Scale tot
192 tients (64%) had impaired performance on the MMSE (score < 27, mean = 24.4) and 32 (45%) on the Mini-
193 scores declined a mean of 1.20 points on the MMSE (standard deviation 1.90), with 66% having scores t
194                        Males declined on the MMSE at a slightly slower rate than females (difference
195 a slightly faster decline (p = 0.021) on the MMSE for Asians (-0.20 SD/decade, 95% CI [-0.28, -0.12],
196               We examined performance on the MMSE in the first year after surgery, controlling for de
197 eper decline over time in performance on the MMSE test among nonoccupationally exposed elderly men.
198 7.4 (standard deviation, 6.6) years took the MMSE on two occasions that were an average of 3.5 (stand
199  subsequent cognitive decline rated with the MMSE and MoCA.
200 ed cognitive impairment as measured with the MMSE and the Clinical Dementia Rating scale sum-of-boxes
201                  A change of more than three MMSE points was considered clinically significant.
202 o had no significant effect on end-treatment MMSE-type global cognitive function (z score difference:
203 he TYM-MCI, the Test Your Memory test (TYM), MMSE and revised Addenbrooke's Cognitive Examination (AC
204 la were significantly correlated with UPSIT, MMSE, DRS-2, and CDR scores.
205 , overt HE (West-Haven criteria) and various MMSE items, MDF showed no correlation with any of MMSE i
206 ization, significant predictive factors were MMSE </= 27/30 (OR, 4.56) and Geriatric Depression Scale
207  whole-brain NAA loss was detected even when MMSE scores were unchanged, the former seems to be a mor
208 r combinations of parameters associated with MMSE could help provide better group discrimination.
209                       When a comparison with MMSE scores was made, (18)F-FDG significantly correlated
210  volume and hippocampal were correlated with MMSE results.
211 betapir scores significantly correlated with MMSE scores only when both controls and AD patients were
212 ade, (18)F-FDG significantly correlated with MMSE when both controls and AD patients were included (r
213 munoreactive neurons was not correlated with MMSE, age at death, education, apolipoprotein E allele s
214 but some evidence of a weak correlation with MMSE (r =-0.22, P = 0.09).
215  gender, but had an inverse correlation with MMSE score.
216 om 26% (95% CI, 19%-34%) in younger men with MMSE scores of 29 to 76% (95% CI, 65%-84%) in older wome
217  associated neither with each other nor with MMSE.
218                            235 patients with MMSE </=25 at baseline and 135 whose first study visit o
219                Neuropsychological tests with MMSE and episodic memory as the primary outcomes and res
220 to 76% (95% CI, 65%-84%) in older women with MMSE scores of 24 (1-year risk: 6% [95% CI, 4%-9%] to 24
221 d 66 control subjects (age = 73.5+/-7.3 yrs; MMSE = 29+/-1.3) from the Australian Imaging Biomarkers

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