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1 nned, and had at least three valid post-dose MRI scans).
2 oved by the Food and Drug Administration for MRI scanning).
3 ting physicians blinded to the result of the MRI scan.
4 ived one [123I]5-I-A-85380 SPECT scan and an MRI scan.
5 ructural MRI scan and co-registered to their MRI scan.
6 mination with (18)F-AZD4694 and a structural MRI scan.
7 -minute, eyes-open, resting-state functional MRI scan.
8 al) examinations as predictors of a positive MRI scan.
9 -5-IA-85380 ((123)I-5-IA) SPECT scan and one MRI scan.
10 PET scan of the chest, and a routine breast MRI scan.
11 were clinically assessed and 351 had a brain MRI scan.
12 hensive neuropsychological assessment and an MRI scan.
13 hyperenhancement in the early postoperative MRI scan.
14 (123)I-iodobenzovesamicol SPECT scan and an MRI scan.
15 ed medicine and varies considerably under an MRI scan.
16 o avoid shocks while undergoing a functional MRI scan.
17 nd healing were not evident clinically or on MRI scans.
18 se) availability while undergoing functional MRI scans.
19 Patients with MS also underwent annual brain MRI scans.
20 opsychiatric illnesses using only anatomical MRI scans.
21 We detected 34 enhancing lesions in 200 DCE-MRI scans.
22 es were collected, and 93% had corresponding MRI scans.
23 aging Initiative study with baseline CSF and MRI scans.
24 yses of 586 longitudinal and cross-sectional MRI scans.
25 he basis of blinded visual assessment of the MRI scans.
26 cohort of 613 children, 518 (85%) had usable MRI scans.
27 ortical and subcortical regions using serial MRI scans.
28 campal atrophy were assessed from anatomical MRI scans.
29 interpretation of standard contrast-enhanced MRI scans.
30 prone scans suitable for fusion with breast MRI scans.
31 ple-dose post-contrast T1-weighted spin echo MRI scans.
32 umber of lesions noted on pretreatment brain MRI scans.
33 were found to have an adrenal mass on CT or MRI scans.
34 rebellum and whole brain were collected from MRI scans.
35 ith a median of 40 (IQR, 15-65) days between MRI scans.
36 n measurements derived from structural brain MRI scans.
37 cient (ADC) values were acquired from the DW-MRI scans.
38 oth (68)Ga-PSMA-11 PET/CT and (68)Ga-RM2 PET/MRI scans.
39 ith 188 Gd-enhancing lesions on 48 pre-aHSCT MRI scans.
40 resolution coupled with conventional cardiac MRI scans.
41 rement of HF subfields in the human brain on MRI scans.
42 after a baseline magnetic resonance imaging (MRI) scan.
43 h are visible on magnetic resonance imaging (MRI) scans.
44 individual illusion magnitude and structural MRI scanning.
45 went structural and resting-state functional MRI scanning.
46 and 31 healthy comparison subjects underwent MRI scanning.
47 tched healthy controls (n=68) also underwent MRI scanning.
48 irements of immobilization or anesthesia for MRI scanning.
49 healthy subjects with one or more structural MRI scans (1,197 in total), machine learning algorithms
52 hange significantly from the first to second MRI scan (13.7 +/- 7.8 vs. 16.3 +/- 8.7 mm Hg, P = 0.239
53 82% of recruited subjects completed serial MRI scans (17 PSP, 9 MSA-P, 9 Parkinson's disease patien
55 ed tomography/magnetic resonance imaging (CT/MRI) scans, (2) subject the patient to a liver biopsy, o
56 els were extracted from 82 T1-weighted brain MRI scans (256 x 192 x 124 volumes) of 42 subjects with
57 d serial clinical assessments and volumetric MRI scans (41 scans: range 3-8 per patient) at different
59 ribution scans were compared with resting DE-MRI scans acquired within 24 h of SPECT acquisition.
60 graphy (PET) and magnetic resonance imaging (MRI) scans acquired in a total of 210 healthy individual
61 s calculated using single diffusion-weighted MRI scans (acute ischemic events that occurred within 10
63 iation across brain surfaces, extracted from MRI scans alone, can successfully diagnose the presence
65 ormal regional wall motion (WM) on a cardiac MRI scan also have abnormal BP regional ejection fractio
66 rior to development of lesions observable on MRI scans, an endeavor that may be facilitated by newbor
67 ent (i) clinical evaluations; and (ii) brain MRI scans analysed using whole-brain voxel-based morphom
71 ealthy controls (HCs) completed a structural MRI scan and provided blood sample for kynurenine metabo
73 , subjects received resting-state functional MRI scans and assessments of depressive symptoms using t
75 assessed by longitudinal gadolinium-enhanced MRI scans and clinical disease activity differ in their
77 averaged proton density-weighted structural MRI scans and drive its functional activity with a dual
79 based history of enuresis, volumetric brain MRI scans and neuropsychological testing were obtained i
80 o calculate hippocampal volume on all serial MRI scans and used linear mixed-effects regression model
81 ne changes in gray matter we used structural MRI scans and voxel-based morphometry (VBM) and to ident
82 high resolution magnetic resonance imaging (MRI) scans and a 3-day food diary were collected on 32 c
83 weighted cranial magnetic resonance imaging (MRI) scans and are associated with geriatric depression.
84 h recent orbital magnetic resonance imaging (MRI) scans and normal eye examinations were consented fr
85 ions of at least 3 mm on a T2-weighted brain MRI scan, and an Expanded Disability Status Scale score
86 Score of 70 or higher, were able to have an MRI scan, and had a complete resection of one to three b
87 At 12 weeks, patients underwent a follow-up MRI scan, and were categorized as either treatment remit
88 re interrogated immediately before and after MRI scanning, and patients were continuously monitored.
89 patients with FCD IIa/b were submitted to 7T MRI scanning, and then analyzed histologically and ultra
90 valuation of response to therapy compared to MRI scans, and can predict outcomes, particularly for pa
92 went structural and resting state functional MRI scans, and spatial neglect was measured using the Po
93 clinical factors associated with 'positive' MRI scans; and (iii) the utilization of comprehensive ep
94 ed at the time of the first postradiotherapy MRI scan are prognostic for time to tumor recurrence and
95 ample, shape measurements derived from brain MRI scans are multidimensional geometric descriptions of
101 ecoming clinically apparent using structural MRI scans at multiple time points beginning at 1.5 years
102 er than minimal cerebral disease detected on MRI scans at the time of an HSCT are at risk for severe,
103 ast-enhanced magnetic resonance imaging (DCE-MRI) scanning at baseline and 15 +/- 2 days after initia
104 on T(2)-weighted magnetic resonance imaging (MRI) scans at 12 months and progression of disability th
105 oncurrently with magnetic resonance imaging (MRI) scans at multiple time points and were analyzed for
107 searchers are often skeptical of post mortem MRI scans because of uncertainty about whether the fixat
108 thological diagnosis who had antemortem head MRI scans between Jan 1, 1999, and Dec 31, 2012, and who
109 be, and whole brain were measured on coronal MRI scans by a single rater who was blind to the subject
110 with atrophy (as measured from T(1)-weighted MRI scans by region of interest analysis) in the amygdal
111 assessed by blinded central review of brain MRI scans by the study neuroradiologist in the modified
113 y control subjects were recruited for serial MRI scans, clinical assessments and formal neuropsycholo
115 diagnosed 1 to 4 years following the initial MRI scan, compared with those who would remain in the pr
117 enotype, more antipsychotic exposure between MRI scans correlated with greater volume reductions in f
119 as performed on 1.5-T structural T1-weighted MRI scans derived from the International OCD Brain Imagi
120 data support that the system is safe and the MRI scan does not adversely affect electrical performanc
125 ection with ferumoxytol-enhanced T1-weighted MRI scans for anatomical orientation, similar to the con
128 xytol-enhanced whole-body diffusion-weighted MRI scans for tumour detection with ferumoxytol-enhanced
129 icenter neuroimaging data, we analyzed brain MRI scans from 2028 schizophrenia patients and 2540 heal
130 fluid-attenuated inversion recovery (FLAIR) MRI scans from 262 participants in two phase 2 studies o
131 hmically calculated for 108 anatomical brain MRI scans from 50 patients (20 of whom were female) and
136 ver the past two decades, thousands of brain MRI scans from healthy youth and those with neuropsychia
140 1-weighted brain magnetic resonance imaging (MRI) scans from 2148 MDD patients and 7957 healthy contr
141 T), tau PET, and magnetic resonance imaging (MRI) scans from the population-based Mayo Clinic Study o
142 The presence of both DIS and DIT from two MRI scans has a higher specificity and risk for CDMS tha
144 he 32 participants, whereas the simultaneous MRI scan identified findings compatible with recurrent P
146 hypothesis that fusion of (18)F-FDG PET and MRI scans improves detection of breast cancer, 23 patien
147 and 83 of 140 in the control group underwent MRI scan in 2009 to identify progression of MRI-measured
153 comparative study of in vivo and post mortem MRI scans in healthy male Wistar rats at three age point
154 as well as knee magnetic resonance imaging (MRI) scans in each subject were obtained at baseline and
160 rebral microbleeds (CMBs) on prethrombolysis MRI scans is associated with an increased risk of ICH.
161 ombined analysis of computed tomographic and MRI scans may help indicate the diagnosis of adult-onset
162 ltrasound and/or magnetic resonance imaging (MRI) scan may be is necessary to confirm the diagnosis.
163 of spatial distortions inherent in diffusion MRI scans, may enable more precise spatial targeting of
166 rther analysis of 102 hemispheres of in vivo MRI scans (N = 51 males, mean +/- SD 24.1 +/- 3.1 years
168 xamination, a baseline brain and spinal cord MRI scan obtained less than 3 months from clinical onset
170 f the menisci in the right knee on 1.5-tesla MRI scans obtained from 991 subjects (57% of whom were w
177 ospective study, we analysed early diffusion MRI scans of 14 patients with the E200K genetic form of
180 rior temporal gyrus (STG]) were drawn on the MRI scans of all subjects and used to measure volumes on
181 utational models reconstructed from clinical MRI scans of fibrotic patient atria to explore the feasi
183 bral injury, we assessed cerebral lesions on MRI scans of infants who participated in the Total Body
191 rmed dissections, histological sections, and MRI scans of the closest living relatives of tetrapods:
192 Sequential small-animal (18)F-FDG PET and MRI scans of the thighs were obtained and coregistered.
193 on oxide (IO) nanoparticle probe for PET and MRI scans of tumor integrin alphavbeta3 expression.
194 ckness, based on magnetic resonance imaging (MRI) scans of 164 brain hemispheres, identified a delimi
195 ied longitudinal magnetic resonance imaging (MRI) scans of 92 nondemented older adults (age 59-85 yea
196 ic subjects, and magnetic resonance imaging (MRI) scans of schizophrenic subjects have not consistent
197 occasionally on magnetic resonance imaging (MRI) scans of the elderly, and this type of striatum is
198 ed to structural Magnetic Resonance Imaging (MRI) scans of twenty social network site (SNS) users wit
200 Ninety-eight children received structural MRI scans on a Siemens head-only 3T scanner with magneti
201 ized them in detail clinically, and obtained MRI scans on admission and daily thereafter while coma p
208 participants had magnetic resonance imaging (MRI) scans, positron emission tomography (PET) scans wit
211 logical diagnostic methods (X-ray, CT scans, MRI scans) provide high precision monitoring of articula
212 ent nasogastric intubation before a baseline MRI scan, received 400 mL of Resource Energy (Nestle) as
216 oxel-based morphometry (VBM) analyses of the MRI scans revealed that absolute IQ scores were related
221 ting the inclusion criteria, with 816 usable MRI scans (spanning 1.0-11.2 years of the disease) avail
222 uroimaging reward paradigm during functional MRI scanning, structural scanning, and completed psychom
224 rogram, investigators interpreted a baseline MRI scan taken before treatment to establish whether the
226 ad injury model of TBI in mice, we showed by MRI scans that TBI caused substantial degeneration at th
227 l on T2-weighted magnetic resonance imaging (MRI) scans that most commonly reflect small vessel cereb
230 and a fluoro-deoxy-glucose-PET/MRI (FDG-PET/MRI) scan, the patient suffered from progressive dopamin
231 ography (CT) and magnetic resonance imaging (MRI) scans, the efficacy of routine radiologic staging i
232 nical criteria for aMCI and had three serial MRI scans: the first scan approximately 3 years before t
234 dissemination in space, change the timing of MRI scanning to show dissemination in time, and increase
235 tients (230 from each group) also had serial MRI scans to assess T2-weighted and gadolinium-enhancing
238 ]) in the forearm and foot; we also used 31P-MRI scans to study the cellular metabolism of the foot m
240 tal studies were coregistered with patients' MRI scans using automated software, and ictal minus inte
241 c measurements derived from brain structural MRI scans, using genome-wide SNP data from 1,320 unrelat
248 reconstructed from late gadolinium-enhanced MRI scans, we simulated channelrhodopsin-2 (ChR2) expres
251 e response and pharmacokinetic analysis, DCE-MRI scans were acquired at baseline and repeated at cycl
265 ces in infarct size were detected when the 2 MRI scans were compared, the 2 SPECT scans were compared
266 .8 ms, repetition time = 1 s, 8 interleaves) MRI scans were conducted at 3.0 T by using an extremity
271 een healthy children for whom anatomic brain MRI scans were obtained every 2 years, for 8-10 years, w
288 0 to 3 according to severity of the lesions, MRI scans were scored independently by 2 expert readers
292 hing T1-weighted magnetic resonance imaging (MRI) scans were obtained for 41 unmedicated patients wit
294 ent CD and 13 healthy participants underwent MRI scanning while performing a task that requires the u
295 d VAT, they had undergone a volumetric brain MRI scan with measurements of total brain volume (TCBV),
296 th 31 healthy controls using high-resolution MRI scans with an ROI approach focusing on the basal gan
298 d within 2 weeks of SRSE onset, (2) a second MRI scan within 6 months of SRSE resolution, and (3) a m
300 perfusion was assessed on an early follow-up MRI scan (within 12 h of the revascularisation procedure
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