ライフサイエンスコーパス: MRS

1 MRS also showed an effect of fingolimod on glutamate lev

2 MRS and pathology associations were adjusted for time fr

3 MRS data were acquired from occipital cortex.

4 MRS may provide supplementary biochemical information an

5 MRS of whole-brain parenchyma showed decreases in N-acet

6 MRS plays a complementary role to MRI, by increasing its

7 MRS provides valuable information in the assessment of h

8 MRS revealed the number of neurons in the left hypothala

9 MRS was performed on these 25 subjects to examine cerebr

10 MRS was used to monitor intracellular and extracellular

11 MRS, plays significant complementary role and should be

12 to both real-time noninvasive imaging by 13C MRS as well as therapeutic response.

13 ate and glutamine (Glx), were measured by 1H MRS in the left dorsolateral prefrontal cortex (l-DLPFC)

14 g proton magnetic resonance spectroscopy (1H MRS), this study assessed whether dysregulation of the h

15 We measured MRI and 1H-MRS changes.

16 eased in relation to the pre-HSCT MRI and 1H-MRS.

17 d proton magnetic resonance spectroscopy (1H-MRS) as standard follow-up after HSCT with cord blood in

18 ) proton magnetic resonance spectroscopy (1H-MRS) to measure skeletal muscle acetylcarnitine concentr

19 h proton magnetic resonance spectroscopy (1H-MRS).

20 Cross-sectional design using 3-T 1H-MRS in participants recruited from university-based psyc

21 A long-TE 1H-MRS protocol was implemented for successful detection of

22 We applied long-TE 1H-MRS to measure acetylcarnitine in endurance-trained athl

23 ate ratio, it is not clear which of these 1H-MRS indices of glutamatergic neurotransmission is altere

24 Inclusion criteria were single voxel 1H-MRS studies reporting glutamate, glutamine or Glx values

25 uring acetylcarnitine concentrations with 1H-MRS is feasible on clinical MR scanners and support the

26 Criteria for 2HG MRS were established to make a presumptive molecular dia

27 e data provide a basis for incorporating 2HG MRS into clinical management of IDH-mutated gliomas.

28 Patients and Methods 2HG MRS was performed in 136 patients using point-resolved s

29 Results Quantitative 2HG MRS was technically and biologically reproducible.

30 1-specific agonist (N)-methanocarba-2MeSADP (MRS-2365), and inhibited by MRS-2500, a P2Y1-specific an

31 31P MRS showed trends for more rapid calculated adenosine di

32 scan for tissue segmentation followed by 31P MRS, chemical shift imaging scan with 84 voxels of data

33 orus-31 magnetic resonance spectroscopy (31P MRS) allows for the quantification of bioenergetic molec

34 function, as measured by phosphorus-MRS (31P-MRS), across groups.

35 ntagonists of protease-activated receptor 4, MRS 2179, and clopidogrel) were also protected from seve

36 -exposed normal controls (TENC) underwent 4T MRS of the left and right hippocampus.

37 Adding MRS and MRI to the protocol allows us to define the natu

38 left striatum during tDCS and an additional MRS measurement in the left DLPFC immediately after the

39 Responses to ADP, MRS-2365 and beta-NAD were absent in PDGFRalpha(+) cells

40 ssed only in mouse islets and the A3 agonist MRS 5698 inhibited glucose-induced insulin secretion fro

41 of P2Y6 by either UDP or a specific agonist (MRS 2693) induced a sustained increase in BSM tone (up t

42 Although MRS measures total concentration of GABA, magneto-enceph

43 stablished that the IDH1 mutation induces an MRS-detectable reprogramming of pyruvate metabolism, whi

44 ive fibers and mtDNA multiple deletions, and MRS displayed defective oxidative metabolism in muscle a

45 DTI and MRS seem to be more useful imaging methods to assess the

46 Our combined fMRI and MRS investigation showed that the stronger ATL BOLD resp

47 agnosis after deployment (combined MRS-I and MRS-II cohort meta-analysis odds ratio, 1.47; 95% CI, 1.

48 articles are highly irregular and jagged and MRS powder particles are rough, but primarily rounded.

49 The resulting LRS and MRS 3D-printed materials exhibit similar, but distinct i

50 inally, we discuss the potential for LRS and MRS ink components to be reclaimed and recycled, as well

51 Both LRS and MRS inks exhibit similar rheological and 3D-printing cha

52 The LRS and MRS powders are characterized by distinct, highly inhomo

53 Lunar and Martian regolith simulant (LRS and MRS, respectively) architectures using ambient condition

54 MCS and MRS only affected calcium solubility and dialysability w

55 MRI and MRS examinations were performed on variable dates (10-29

56 reased scanning time, combination of MRI and MRS is currently not recommended as a first line investi

57 In vivo MRI and MRS showed that at 17-23 months of age there was a signi

58 Combination of MRI and MRS showed the highest diagnostic accuracy among the ima

59 Cross-sectionally, MRI-PDFF and MRS-PDFF increased with increases in the histology-deter

60 rough a three-way comparison of MRI-PDFF and MRS-PDFF with the liver histology-determined steatosis g

61 evaluation with liver biopsy, MRI-PDFF, and MRS-PDFF at the baseline and 24 weeks.

62 ponding centers of mass in (18)F-FET PET and MRS imaging of Cho/NAA, determined by simultaneously acq

63 Thus, C-choline PET and MRS methods seemed to be complementary in strengths.

64 anomolar concentrations of the A3 antagonist MRS 1220, but not by the agonist NECA (up to 300 nM), co

65 n) that was blocked by the P2Y1R antagonist, MRS 2500, and by apamin, an inhibitor of Ca(2+)-activate

66 e have investigated the ability of bacterial MRSs from different clades to differentiate cognate tRNA

67 Both MRS and magneto-encephalography measures of the GABA sys

68 ne whether quantitative assessment of 2HG by MRS could serve as a noninvasive clinical imaging biomar

69 onses to nerve stimulation were abolished by MRS-2500 and not observed in muscles with genetic deacti

70 nts and IJPs elicited by EFS were blocked by MRS-2500, a P2Y1 antagonist, and absent in P2ry1((-/-))

71 purines and these responses were blocked by MRS-2500.

72 increase (>/= 1%) in MRI-PDFF (confirmed by MRS-PDFF) showed a parallel decrease or increase in thei

73 d both brain injury and glial dysfunction by MRS.

74 nocarba-2MeSADP (MRS-2365), and inhibited by MRS-2500, a P2Y1-specific antagonist.

75 Conclusion 2HG concentration as measured by MRS was reproducible and reliably reflected the disease

76 ry/inhibitory network activity as proxied by MRS measurements of GABA and glutamate.

77 of lysidine-dependent tRNA(Ile) rejection by MRS, we have investigated the ability of bacterial MRSs

78 Hyperpolarized (13)C MRS and the determination of metabolite exchange rates m

79 a novel agent for clinically relevant (13)C MRS studies of energy metabolism and further provides op

80 uating glycolysis using hyperpolarized (13)C MRS.

81 d time resolution using hyperpolarized (13)C MRS.

82 (13)C-MRS also revealed a reduction in glucose flux to glutama

83 (13)C-MRS dynamic acquisition demonstrated in an axial slab in

84 H probe, 3-aminopropylphosphonate, and (13)C-MRS measurements of the H(13)CO3(-)/(13)CO2 peak intensi

85 k(PL) (hyperpolarized (13)C-MRS) and lactate ((1)H-MRS) provide useful biomarkers fo

86 (13)C-magnetic resonance spectroscopy ((13)C-MRS) magnetization transfer measurements.

87 (13)C-magnetic resonance spectroscopy ((13)C-MRS), which revealed a reduction in metabolism of hyperp

88 Up4A caused MRS 2500-sensitive Ca(2+) transients in human 1321N1 ast

89 we also calculated ADC values and Cho Cr/Cit MRS ratios for all patients.

90 We collected MRS measurements in the left DLPFC and left striatum dur

91 of PTSD diagnosis after deployment (combined MRS-I and MRS-II cohort meta-analysis odds ratio, 1.47;

92 Methicillin resistant CoNS (MRS) and Methicillin resistance S. aureus (MRSA) were 31

93 oints using dynamic microPET imaging and CSI MRS.

94 h those of surrounding muscle, and (13)C-DHA MRS data were processed using custom software to compare

95 ABA was detected with a MEGA-PRESS J-editing MRS sequence.

96 es of interest (for example, metabolites for MRS or MRI) to fill the pore channels (incipient wetness

97 r, lysidine modification is not required for MRS rejection, indicating that this recognition strategy

98 oscopy-measured proton density fat fraction (MRS-PDFF) provides a biochemical measure of liver fat in

99 Distinct GABA/glutamate + glutamine MRS peaks were resolved from MEGA-PRESS difference spect

100 In addition, we used (1)H MRS and a fluorescent glucose analog to evaluate the eff

101 he interplay between fMRI connectivity, (1)H MRS and clinical data was explored by applying moderatio

102 The reviewed (1)H MRS and PET/SPECT studies support the theory of hypofunc

103 Using a multi-voxel (1)H MRS approach at 3 Tesla with high spatial resolution and

104 These results demonstrate potential of (1)H MRS at ultrahigh fields.

105 ched healthy control subjects underwent (1)H MRS scans at 3.0 T.

106 (1)H MRS was used to obtain estimates of combined glutamate a

107 proton magnetic resonance spectroscopy ((1)H MRS) and positron emission tomography (PET)/single-photo

108 Proton MR spectroscopy ((1)H MRS) was used to divide MDD subjects into two subgroups:

109 proton magnetic resonance spectroscopy ((1)H MRS) were performed immediately before and after the adm

110 proton magnetic resonance spectroscopy ((1)H MRS), especially in bipolar I disorder (BD-I).

111 HFC was assessed by (1)H MRS.

112 Metabolic flux analysis by (13)C and (2)H MRS showed that this was not due to dietary lipid absorp

113 0001) and liver triglyceride content by (1)H-MRS (P < 0.0001) were worse in NASH with elevated ALT.

114 roton magnetic resonance spectroscopy ((1) H-MRS) was established based on the 95th percentile in a g

115 roton magnetic resonance spectroscopy ((1) H-MRS), abdominal fat using magnetic resonance imaging (MR

116 ized with the following studies: liver (1) H-MRS; euglycemic insulin clamp with measurement of glucos

117 ipid content as assessed by noninvasive (1)H-MRS and lipid profiling independent of changes in hepati

118 egion above the lateral ventricles with (1)H-MRS and WMH volume in this region and throughout the bra

119 Conventional MR imaging and (1)H-MRS are important complementary tools in the diagnostics

120 concentrations were determined by using (1)H-MRS before and 3 and 5 h after a high-fat with added pro

121 (1)H-MRS data were processed using LCModel software to calcul

122 Consistent with the (1)H-MRS data, steatosis on liver biopsy was also significant

123 We prospectively collected high quality (1)H-MRS in 59 premature infants born </=32 weeks and 61 heal

124 inical trial, 18 successfully completed (1)H-MRS in dorsolateral prefrontal cortex (DLPFC) at baselin

125 in AD, pathologic underpinnings of the (1)H-MRS metabolite changes in AD are unknown.

126 (1)H-MRS metabolite levels reveal early neurodegenerative cha

127 Although (1)H-MRS metabolite ratios of N-acetyl aspartate (NAA)/creati

128 status each had a significant effect on (1)H-MRS metabolites (choline/creatine and myo-inositol/creat

129 Measurements of (1)H-MRS metabolites were obtained in the posterior cingulate

130 modulation of cognition was mediated by (1)H-MRS metabolites.

131 and 25 males) who underwent antemortem (1)H-MRS of the posterior cingulate gyrus at 3 tesla were inc

132 We correlated the (1)H-MRS PDFF findings with SPCs (r = 0.92; P < .001).

133 tive study to determine the accuracy of (1)H-MRS PDFF in the measurement of steatosis using histopath

134 (1)H-MRS PDFF results correlated with histopathology results

135 We collected clinical, serologic, (1)H-MRS PDFF, and liver biopsy data from 94 adult patients w

136 (1)H-MRS showed OAA increased brain lactate, GABA and glutath

137 rgeted metabolomics analysis of in vivo (1)H-MRS spectra discriminated between IDH-mutant tumors and

138 rations were measured continuously with (1)H-MRS using a specific lactate detection method.

139 proton magnetic resonance spectroscopy ((1)H-MRS) acquisition scheme that uses an ultrahigh magnetic

140 We determined liver fat ((1)H-MRS) and clinical characteristics including features of

141 proton magnetic resonance spectroscopy ((1)H-MRS) and fibrosis estimated as stiffness using transient

142 proton magnetic resonance spectroscopy ((1)H-MRS) and whether the addition of protein can modulate th

143 As proton MR spectroscopy ((1)H-MRS) can identify ischaemic tissue, we hypothesised that

144 proton magnetic resonance spectroscopy ((1)H-MRS) in the rACC to examine the function and neurochemis

145 Proton magnetic resonance spectroscopy ((1)H-MRS) is sensitive to early neurodegenerative processes a

146 (hyperpolarized (13)C-MRS) and lactate ((1)H-MRS) provide useful biomarkers for the autophagic proces

147 Proton magnetic resonance spectroscopy ((1)H-MRS) studies on healthy aging have reported inconsistent

148 proton magnetic resonance spectroscopy ((1)H-MRS) was conducted to explore the effects of davunetide

149 proton magnetic resonance spectroscopy ((1)H-MRS), instead of collecting and analyzing liver biopsy s

150 magnetic resonance spectroscopy ((31)P/(1)H-MRS).

151 proton magnetic resonance spectroscopy ((1)H-MRS).

152 proton magnetic resonance spectroscopy ((1)H-MRS); (2) severity of liver disease by biopsy (n = 293);

153 continuously with J-difference-editing (1)H-MRS, and time curves were analyzed using nonlinear mixed

154 rformance of a fractal n-back task, and (1)H-MRS, respectively.

155 er, which was detected by using in vivo (1)H-MRS.

156 nce spectroscopy (MRS) and steady-state (1)H-MRS.

157 olarized magnetic resonance spectroscopy (HP MRS) using dynamic nuclear polarization (DNP) is a techn

158 However, the HP MRS polarization decays in the liquid state according to

159 e dehydrogenase deficiency, in particular if MRS shows elevated succinate.

160 We also review several advances in MRS and PET technologies that have opened the door for n

161 Cases showed significant improvements in MRS scores (p<0.001), mobility (p<0.001) and ADL (p=0.00

162 We propose that the observed variation in MRS discrimination ability reflects differences in the e

163 patient the follow-up examinations included MRS with the assessment of metabolite ratios (NAA/Cr, Ch

164 We reviewed all clinically-indicated MRS studies conducted on preterm infants at a single ins

165 This is the largest MRS study of GABA in schizophrenia and the first to exam

166 ise protocol and muscle studied by localized MRS.

167 (LBG), modified corn and rice starches (MCS, MRS)) to an infant formula on both in vitro mineral avai

168 as better in group C (P = 0.02), and mesopic MRS was higher in groups B and C than in group A (P = 0.

169 MRI MRS has more diagnostic accuracy than MRI alone for dete

170 obese and 11 healthy subjects using a 3T MRI/MRS scanner, and IR in the obese subjects was documented

171 controls and a brain phantom using a 3 T MRI/MRS scanner.

172 However, publications of MRS, PET and SPECT are limited only providing anecdotal

173 The data also support use of MRS as a useful biomarker of baseline cognitive function

174 preclinical proof-of-concept for the use of MRS to measure lactate as a noninvasive metabolic biomar

175 gs provide definitive evidence of the use of MRS, CSI, and PET for imaging tumors in vivo.

176 evation of glutamate and glutamine levels on MRS (without associated typical tumor spectra) are commo

177 ols (13.2+/-2.2 years) provided at least one MRS scan.

178 eadily, but no published quantitative MRI or MRS studies were available for comparison.

179 nfluences of local gray matter variations or MRS voxel-placement deviations.

180 horus Magnetic Resonance Spectroscopy ((31)P MRS) was used to determine pH, phopshocreatine (PCr) and

181 Hepatic energy status, assessed by (31)P MRS, was similar for fructose- and glucose-fed mice.

182 ial capacity, and, in addition, static (31)P-MRS also revealed differences in the Pi-to-ATP exchange

183 iduals through dynamic, static, and ST (31)P-MRS at 7T.

184 in spectral and temporal resolution of (31)P-MRS at ultra high fields (i.e., 7T) uncovers new potenti

185 hyperinsulinemic-euglycemic clamp and (31)P-MRS before, during, and after near-maximal isometric cal

186 We acquired (31)P-MRS data from skeletal muscle of subjects of mixed gende

187 lity of using high spectral resolution (31)P-MRS data, acquired at rest, as a marker of oxidative met

188 estigate this hypothesis, we performed (31)P-MRS imaging in mouse xenograft models of four human glio

189 (31)P-MRS measurements of the chemical shift of the pH probe,

190 In addition, as the dynamic (31)P-MRS requires a complex setup and patient exercise, our a

191 horus magnetic resonance spectroscopy ((31)P-MRS), in particularly dynamic (31)P-MRS, provides a powe

192 y ((31)P-MRS), in particularly dynamic (31)P-MRS, provides a powerful tool for the non-invasive inves

193 F with magnetic resonance spectroscopy-PDFF (MRS-PDFF), biochemical triglyceride extraction, and hist

194 on echo-planar spectroscopic imaging (PEPSI) MRS scans of pregenual anterior cingulate cortex (pACC)

195 hondrial function, as measured by phosphorus-MRS (31P-MRS), across groups.

196 chosis by means of echo time-averaged proton MRS at 4T.

197 jects by using the echo time-averaged proton MRS technique at 4T (i.e., modified point resolved spect

198 arametric liver MR, including hepatic proton MRS, T1- and T2*-mapping yielding "iron-corrected T1" [c

199 )F-FDG PET and hyperpolarized (13)C-pyruvate MRS imaging simultaneously for tumor tissue characteriza

200 at combined (18)F-FDG PET and (13)C-pyruvate MRS imaging was possible in a single session of approxim

201 With this spectral quality, MRS was used to interrogate a number of metabolic proper

202 Here, an enabling relaxation-enhanced (RE) MRS approach is demonstrated that by combining highly se

203 y acquired, 3-dimensional spatially resolved MRS imaging data, were compared.

204 ily living (ADLs) and Modified Rankin Scale (MRS) score at admission and discharge were extracted.

205 rapeutic candidate from our in vitro screen (MRS 2179, a P2Y1 receptor antagonist) also improves hist

206 etic divergence from multiregion sequencing (MRS) data, we demonstrate that it is feasible to disting

207 troduction of combined PET/MR spectroscopic (MRS) imaging, it is now possible to directly and indirec

208 s, hyperpolarized (13)C-DHA MR spectroscopy (MRS) and (18)F-FDG PET were applied as complementary tec

209 MR spectroscopy (MRS) changes of increased Glutamate/glutamine, reduced m

210 ng the results of series of MR spectroscopy (MRS) examinations in the course of BBE.

211 sine ((18)F-FET) and proton MR spectroscopy (MRS) imaging of cell turnover measured by the ratio of c

212 ic resonance imaging (MRI), MR spectroscopy (MRS), and diffusion weighted imaging (DWI), was used in

213 bution of cerebral CT, MRI, MR spectroscopy (MRS), positron emission tomography (PET) and single-phot

214 s in perfusion MR (PWI) and MR spectroscopy (MRS).

215 lism, (13)C magnetic resonance spectroscopy (MRS) allows following the fate of (13)C-enriched substra

216 Magnetic resonance spectroscopy (MRS) allows for noninvasive measurement of metabolites i

217 strates for magnetic resonance spectroscopy (MRS) and imaging (MRI) by dissolution dynamic nuclear po

218 we combined magnetic resonance spectroscopy (MRS) and resting state fMRI and demonstrated an inverse

219 rized (13)C-magnetic resonance spectroscopy (MRS) and steady-state (1)H-MRS.

220 ies, proton magnetic resonance spectroscopy (MRS) and tissue biochemistry, sampling individuals with

221 ging (MRI), magnetic resonance spectroscopy (MRS) and transrectal ultrasound (TRUS) in detecting pros

222 with (31)P magnetic resonance spectroscopy (MRS) and whole-body VO2 during moderate (MOD) and heavy

223 ) and novel magnetic resonance spectroscopy (MRS) approaches.

224 assessed by magnetic resonance spectroscopy (MRS) are used as markers of neuroinflammation in neurode

225 )C-pyruvate magnetic resonance spectroscopy (MRS) combined with a bioreactor platform and interrogate

226 I) and (1)H magnetic resonance spectroscopy (MRS) data were obtained from participants with OCD (n=49

227 muscle 31P magnetic resonance spectroscopy (MRS) for estimation of mitochondrial oxidative capacity.

228 ed by (13)C-magnetic resonance spectroscopy (MRS) has been suggested as one marker for cell death and

229 (HP) (13)C magnetic resonance spectroscopy (MRS) has the unique ability to detect real-time metaboli

230 e GABA with magnetic resonance spectroscopy (MRS) have been inconsistent.

231 ng tDCS and magnetic resonance spectroscopy (MRS) in 17 healthy participants.

232 ole of (1)H magnetic resonance spectroscopy (MRS) in demonstrating these metabolic changes and to rev

233 Magnetic resonance spectroscopy (MRS) is well suited to the task of noninvasive D-2HG det

234 (31)P magnetic resonance spectroscopy (MRS) is widely used for non-invasive investigation of mu

235 ical exams, magnetic resonance spectroscopy (MRS) measurements, and Purdue pegboard motor testing.

236 Magnetic resonance spectroscopy (MRS) measures of glutamate in the striatum and hippocamp

237 Magnetic resonance spectroscopy (MRS) of glutamatergic or GABAergic measures in anterior

238 ined by 13C magnetic resonance spectroscopy (MRS) of hyperpolarized pyruvate, we confirmed that FX11

239 pose Proton magnetic resonance spectroscopy (MRS) of the brain can detect 2-hydroxyglutarate (2HG), t

240 rized (13)C magnetic resonance spectroscopy (MRS) provides unprecedented opportunities to obtain clin

241 Magnetic resonance spectroscopy (MRS) showed alterations characteristic of AD (i.e. incre

242 Magnetic resonance spectroscopy (MRS) studies have consistently demonstrated reduced cort

243 novel (17)O magnetic resonance spectroscopy (MRS) technique on R6/2 mice to directly determine rates

244 ed pyruvate magnetic resonance spectroscopy (MRS) to determine changes in hepatic gluconeogenesis in

245 ield proton magnetic resonance spectroscopy (MRS) to determine whether PTSD is associated with lower

246 s underwent magnetic resonance spectroscopy (MRS) to measure markers of brain injury.

247 sting-state magnetic resonance spectroscopy (MRS) to measure task-related blood-oxygen level-dependen

248 asured with magnetic resonance spectroscopy (MRS) was associated with OCD and/or CBT response.

249 ingle-voxel magnetic resonance spectroscopy (MRS) was employed to measure glutamate concentrations no

250 Proton magnetic resonance spectroscopy (MRS) was used to test the hypothesis that older particip

251 Using magnetic resonance spectroscopy (MRS) we found that R-flurbiprofen treatment decreased th

252 (1)H magnetic resonance spectroscopy (MRS) yields site-specific signatures that directly repor

253 pairment by Magnetic resonance spectroscopy (MRS), an advanced MR imaging technique, which could not

254 ain MRI and magnetic resonance spectroscopy (MRS), and electron microscopic analyses of leukocytes fo

255 using (31)P magnetic resonance spectroscopy (MRS), and glycolysis and fatty acid oxidation were measu

256 e and brain magnetic resonance spectroscopy (MRS), and muscle biopsy and fibroblasts were analyzed.

257 detected by magnetic resonance spectroscopy (MRS), as a metabolic biomarker for the pharmacodynamic r

258 asured with magnetic resonance spectroscopy (MRS), in 12 healthy volunteers.

259 using (31)P magnetic resonance spectroscopy (MRS), we demonstrated that pHLIP Var3 was retained in tu

260 Using magnetic resonance spectroscopy (MRS), we tested the hypothesis that glutamate and glutam

261 and proton-magnetic resonance spectroscopy (MRS)-based metabolites.

262 measured by magnetic resonance spectroscopy (MRS).

263 uccinate on magnetic resonance spectroscopy (MRS).

264 I) and (1)H magnetic resonance spectroscopy (MRS).

265 ith [(19)F] magnetic resonance spectroscopy (MRS).

266 bjects with magnetic resonance spectroscopy (MRS).

267 lution (1)H magnetic resonance spectroscopy (MRS).

268 sting-state magnetic resonance spectroscopy (MRS).

269 le spinning magnetic resonance spectroscopy (MRS).

270 chniques, such as single voxel spectroscopy (MRS) and diffusion tensor imaging (DTI), in children wit

271 Both magnetic resonance spectroscopy [MRS using chemical shift imaging (CSI) total choline (tC

272 first phase of the Marine Resiliency Study (MRS-I) included 1415 male Marines, 59 of whom developed

273 second phase of the Marine Resiliency Study (MRS-II) included 745 male Marines, 25 of whom developed

274 nt recognition by methionyl-tRNA synthetase (MRS) and production of a chimeric Met-tRNA(Ile) that wou

275 Using single-voxel 3-T MRS, healthy term neonates (n = 31, mean postconception

276 ns with re-experiencing symptoms affirm that MRS indices of hippocampus neuron integrity and glutamat

277 ntify ischaemic tissue, we hypothesised that MRS detectable brain metabolites would be superior to WM

278 t not necessarily in DEHSI, and suggest that MRS provides a useful biomarker for determining the path

279 Neither the postmortem nor the MRS studies directly address the physiological propertie

280 nd interrogated the biochemical basis of the MRS data with respect to cancer aggressiveness.

281 imal activation and from the location of the MRS voxel to assess the relationship of GABA with gamma.

282 Short-echo-time MRS was used to measure N-Acetyl-aspartate (NAA) and myo

283 No relationships to MRS glutamate and amphetamine-induced subclinical positi

284 Using MRS quantification, a good agreement was found between C

285 Using MRS, we monitored the brain metabolic response to lipopo

286 and tumor glutamine pool size measured using MRS (r(2) = 0.71).

287 Taurine levels, measured using MRS, showed a very strong inverse correlation with GFAP

288 ecent efforts to translate methodology using MRS to reliably measure in vivo D-2HG into clinical rese

289 ee, depressed outpatients using single-voxel MRS, to measure absolute glutamate concentrations in bas

290 ll nine Couinaud segments using single-voxel MRS-PDFF (n=117) and tissue wedges for biochemical trigl

291 lasma and CSF neopterin (p = 0.023) and with MRS markers of neuronal injury (lower N-acetyl aspartate

292 he first report that PTSD is associated with MRS markers of hippocampus Glu excess, together with ind

293 n dACC and basal ganglia are consistent with MRS findings in bipolar depression and suggest that infl

294 ies have shown that MRI-PDFF correlates with MRS-PDFF.

295 ilament light chain and its correlation with MRS-based metabolites suggest early neuronal injury in a

296 RI-PDFF showed an excellent correlation with MRS-PDFF (r=0.984, confidence interval 0.978-0.989) and

297 MRI-PDFF showed a robust correlation with MRS-PDFF both at week 0 and at week 24 (r = 0.98, P < 0.

298 ompared between groups and correlations with MRS glutamate, subclinical psychopathological and neuroc

299 was assessed through linear regression with MRS-PDFF, triglyceride extraction, and histology.

300 tudinal study, MRI-PDFF correlated well with MRS-PDFF and was more sensitive than the histology-deter