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1 MSC infusions were well tolerated.
2 MSC interrupted binding of the TF GATA-3 to the locus en
3 MSC morphology after IFN-gamma stimulation significantly
4 MSC therapy improves cardiac function in animal models o
5 MSC-Ex recovered the destruction of the epithelial barri
6 MSC-Ex shifted the macrophage functional phenotype from
7 MSC-induced changes in macrophage phenotype critically d
8 MSCs also expressed mannose receptor (MR) that was found
9 MSCs can be grown in large numbers in vitro, and autolog
10 MSCs can respond to tissue injury by anti- or proinflamm
11 MSCs significantly decreased natural killer cells in the
12 MSCs suppressed cytokine production, increased M2 macrop
13 MSCs were fluorescently labeled and injected into a rat
14 MSCs were transfected with recombinant minicircles encod
19 2 and CXCL5) expressed by TNFalpha-activated MSCs efficiently recruited CXCR2(+) neutrophils into tum
20 ast cancer, we found that TNFalpha-activated MSCs strikingly enhanced tumor metastasis compared with
24 timization of the dosing and frequency of AD-MSC therapy, either alone or used in, combination with o
25 the influence of timing and frequency of AD-MSC treatment on immunologic and graft survival as well
29 lar pathways through which IFN-gamma affects MSC plasticity and the consequence of their manipulation
30 hanced the immunosuppressive capacity of all MSC lines, and morphology predicted the magnitude of IFN
31 SCs/1 million CSCs, n = 7), 200 million allo-MSCs (n = 8), 1 million allo-CSCs (n = 4), or placebo (P
32 receive transendocardial injections of allo-MSCs + allo-CSCs (ACCT: 200 million MSCs/1 million CSCs,
34 andomized, controlled trial using allogeneic MSC, which are logistically more convenient than autolog
35 ardiac surgery, administration of allogeneic MSCs did not decrease the time to recovery of kidney fun
37 cells identifies in adult mouse synovium an MSC population largely negative for the skeletal stem ce
38 ffects of pFUS increased both MSC homing and MSC production of VEGF and IL-10, suggesting microenviro
44 Transcriptomes of many diverse tissues and MSCs further show that lamin-A,C's increase with tissue
48 ith nonatherosclerotic-MSCs, atherosclerotic-MSCs displayed higher levels of both intracellular (P=0.
49 ive oxygen species levels of atherosclerotic-MSCs promoted a phenotypic switch characterized by enhan
50 ed mitochondrial function of atherosclerotic-MSCs underlies their altered secretome and reduced immun
51 Furthermore, treatment of atherosclerotic-MSCs with the reactive oxygen species scavenger N-acetyl
52 These results indicate that FOXP1 attenuates MSC senescence by orchestrating their cell-fate switch w
53 wn in large numbers in vitro, and autologous MSCs transfused into tuberculosis patients have been fou
57 at bone marrow mesenchymal stromal cells (BM-MSC) protect AML blasts from spontaneous and chemotherap
63 human AML compared with AML cultured with BM-MSCs and found that macrophage migration inhibitory fact
65 The molecular effects of pFUS increased both MSC homing and MSC production of VEGF and IL-10, suggest
67 Intra-arterial route is very promising, but MSCs are missing machinery for diapedesis through blood-
74 nflammatory cytokines from activated cardiac MSCs of TLR4-deficient mice, compared with WT cardiac MS
75 improve LV remodeling and function, cardiac MSCs from LVD exacerbated anterior wall thinning 28 days
77 The inflammatory polarization of cardiac MSCs by LVD was mediated by TLR4, as we found less secre
78 ion of CD47 (don't eat me signal) on cardiac MSCs after both TLR4 stimulation in vitro and transplant
80 T cardiac MSCs and saline, TLR4(-/-) cardiac MSCs survived in the cardiac tissue and maintained their
84 hils and inflammation, mucus-secreting cell (MSC) numbers, type 2-associated cytokines (interleukin (
85 IONALE: Virtually all mesenchymal stem cell (MSC) studies assume that therapeutic effects accrue from
90 Bone marrow-derived mesenchymal stem cells (MSC) have been promoted for multiple therapeutic applica
91 ne marrow-derived mesenchymal stromal cells (MSC) and in vitro 3T3-L1 preadipocytes significantly inc
93 to originate from mesenchymal stromal cells (MSC), but their relationship with MSCs is not clear.
96 sis and desensitizes mesenchymal stem cells (MSCs) against subsequent mechanical activation in vitro
97 tective potential of mesenchymal stem cells (MSCs) against the deleterious impact of AbetaOs on hippo
98 nation of autologous mesenchymal stem cells (MSCs) and cardiac stem cells (CSCs) synergistically redu
99 stem cells (iPSCs), mesenchymal stem cells (MSCs) and human umbilical vein endothelial cells (HUVECs
101 rogenitors and human mesenchymal stem cells (MSCs) appeared to contribute in replacement of 40% and 1
102 Bone marrow derived mesenchymal stem cells (MSCs) are regularly utilized for translational therapeut
103 tudies indicate that mesenchymal stem cells (MSCs) can stimulate angiogenesis and modify immune funct
104 he potential role of mesenchymal stem cells (MSCs) derived from human MT in the pathogenesis of bone
107 ment affects mesenchymal stromal/stem cells (MSCs) from umbilical cord's Wharton's Jelly (WJ) on a mo
109 of allogeneic human mesenchymal stem cells (MSCs) in reducing the time to recovery from AKI after ca
114 ieved to function as mesenchymal stem cells (MSCs), multipotent tissue-resident progenitors with grea
123 BMF that Gli1(+) mesenchymal stromal cells (MSCs) are recruited from the endosteal and perivascular
124 (P10) aging human mesenchymal stromal cells (MSCs) could be used for bone tissue regeneration as tiss
130 mesenchymal stem cell-conditioned medium (CM-MSC) as an alternative to cell therapy in an antigen-ind
134 T cells from arthritic mice treated with CM-MSC showed increases in FOXP3 and IL-4 expression and po
135 ght multi-aminoacyl-tRNA synthetase complex (MSC), restricting the pool of free LysRS-tRNA(Lys) Mount
137 atopoietic stem cells or bone marrow-derived MSC or dendritic cells) for optimization of appropriate
138 hly suppressive human adipose tissue-derived MSC (AdMSC) display and induce a differential immunologi
139 ollowing transplantation of human BM-derived MSCs, globule-containing hepatocytes declined and donor-
142 ssessed the effect human bone marrow-derived MSCs have on neonatal porcine islets (NPIs) in vitro and
145 ogenesis RNA), by RNA interference disrupted MSC chondrogenesis, concomitant with reduced cartilage-s
149 one outgrowth in MT, as mediated by enhanced MSC-driven osteogenic differentiation in the jaw bone.
153 reactive T cells and helps to understand how MSCs ameliorate symptoms in lupus-prone MRL.Fas(lpr) mic
154 n umbilical cord mesenchymal stem cells (hUC-MSCs), originating in Wharton's jelly, are multipotent s
155 d 2 are robust new players involved in human MSC immunoregulatory mechanisms, and the higher suppress
157 Ms) were cultured ex vivo with/without human MSC-derived EVs before adoptive transfer to LPS-injured
159 n this study, we show that transfer of human MSCs increased MRL.Fas(lpr) mouse survival, decreased T
165 n target of rapamycin (mTOR) manipulation in MSCs was studied in vivo in a mouse model of delayed-typ
166 Generating dysfunctional mitochondria in MSCs using rhodamine 6G pretreatment also abrogated thes
169 n synthesis in HEK293 cells only, whereas in MSCs, satisfactory results were obtained only after usin
173 tes that extensive long-term culture-induced MSC aging impaired their osteogenic ability and subseque
175 ALF and tissue eosinophils and inflammation, MSC numbers, reduced the production of type 2 cytokines
178 of lung resident mesenchymal stem cells (LR-MSCs) and in the lung tissues of a pulmonary fibrosis mo
179 We have devised an adult human bone marrow MSC (hMSC) delivery formula by investigating molecular e
180 e test group received allogeneic bone marrow MSCs by intradiscal injection of 25 x 10 cells per segme
181 onditional depletion of Foxp1 in bone marrow MSCs led to premature aging characteristics, including i
185 of allo-MSCs + allo-CSCs (ACCT: 200 million MSCs/1 million CSCs, n = 7), 200 million allo-MSCs (n =
186 ined whether administering extracts of MSCs (MSC-Ex) instead of MSCs could augment the beneficial eff
189 a (TGF-beta), we identified the TF musculin (MSC) as being critical for the development of induced Tr
190 hat upon HIV-1 infection, a free pool of non-MSC-associated LysRS is observed and partially relocaliz
194 nd found that intracarotid administration of MSC-oHSV, but not of purified oHSV alone, effectively tr
196 ll immunosuppressive capacity in a cohort of MSC lines derived from different donors and manufacturin
197 melanoma brain metastasis, a combination of MSC-oHSV and PD-L1 blockade increases IFNgamma-producing
199 al characteristics, and the effectiveness of MSC-based therapeutics may be realized by finding predic
200 MSCs could augment the beneficial effects of MSC therapy by overcoming the low homing efficiency of M
202 n group (n=14) received a single infusion of MSC (1.0 x 10(5) or 2.5 x 10(5) cells/kg; n=7 each) plus
204 Our findings reveal a novel mechanism of MSC effects on malignant cells through which crosstalk b
205 Here, we aimed to determine the mechanism of MSC recruitment by Dendritic Cells (DC), hypothesising t
209 esis is valid, intravenous administration of MSCs should improve outcomes in heart failure, an entity
210 ing the altered immunomodulatory capacity of MSCs in the context of atherosclerosis remain elusive.
211 r level, FOXP1 regulated cell-fate choice of MSCs through interactions with the CEBPbeta/delta comple
215 study, we focused on the paracrine effect of MSCs on macrophage polarization and the role of extracel
216 responsible for the pro-metastatic effect of MSCs since inhibition of this chemotaxis abolished incre
217 y by overcoming the low homing efficiency of MSCs systemically administered in inflammatory bowel dis
218 e examined whether administering extracts of MSCs (MSC-Ex) instead of MSCs could augment the benefici
219 stering extracts of MSCs (MSC-Ex) instead of MSCs could augment the beneficial effects of MSC therapy
220 efficient and minimally invasive methods of MSCs delivery to the brain still have to be developed.
223 ly important in promoting the recruitment of MSCs to the site of skin injury, which in turn modulates
225 This study thus demonstrates the utility of MSCs as OV carriers to disseminated brain lesions, and p
226 the hyaluronic acid (HA) receptor, CD44, on MSC membranes, to improve their homing potential towards
231 area of the chondriod callus in the aged P10 MSC sheet groups was significantly larger than in P3 MSC
232 bone ends in both young P3 MSC and aged P10 MSC sheet-wrapped groups when compared to allograft alon
234 llograft and host bone ends in both young P3 MSC and aged P10 MSC sheet-wrapped groups when compared
238 slational progress has been hampered by poor MSC graft survival, jeopardizing cellular and molecular
240 demonstrates that CCL2 enables the prolonged MSC-T cell interactions needed for sufficient suppressio
241 ry following cell culture expansion protects MSCs from fibrogenesis in the host wound environment and
244 significance of BMP signaling in regulating MSC fate during root development and shed light on how B
245 mmary, our data show that CD34(+) derived SG-MSCs could be a promising cell source for adoptive cell-
246 f female WT mice and evaluated infarct size, MSC retention, inflammation, remodeling, and function af
247 splantation of cardiac MSCs and subcutaneous MSCs from LVD and sham hearts did not improve LV remodel
248 ine-filtering on-chip micro-supercapacitors (MSCs) based on coordination polymer frameworks were fabr
250 ARDS is a heterogeneous syndrome, targeting MSCs to patients with ARDS with a more hyperinflammatory
255 lagen nanofilms serve as a model matrix that MSCs can easily deform unless the film is enzymatically
261 tractant protein-1/chemokine ligand 2 in the MSC secretome and improved MSCs immunosuppressive capaci
263 studies demonstrated that components of the MSC can be mobilized in response to certain cellular sti
264 Three days after topical application, the MSCs homed to the injured parenchyma and improved the ne
265 ytokeratin(+) cells in the bone marrow, this MSC subpopulation could prove useful in determining the
266 d differentiation characteristics of Thyroid MSCs and PTC MSCs were comparable with bone marrow MSCs.
268 intravenously administered ischemia-tolerant MSCs (itMSCs) in patients with nonischemic cardiomyopath
270 teogenic differentiation of mandibular torus MSCs was associated with the suppression of Notch3 signa
274 myocardium drives resident and transplanted MSCs toward a proinflammatory phenotype and restricts th
276 This increased accumulation for HA-treated MSC yielded a substantial reduction in inflammation as d
278 d efficacy of the intravenous infusion of UC-MSC in patients with chronic stable heart failure and re
281 ized to intravenous infusion of allogenic UC-MSCs (Cellistem, Cells for Cells S.A., Santiago, Chile;
283 We armed MSCs with different oHSV variants (MSC-oHSV) and found that intracarotid administration of
285 sults on histology demonstrate promises when MSCs are injected endotracheally (but not intravenously)
289 Adoptive transfer of AMs pretreated with MSC-derived EVs reduced inflammation and lung injury in
290 t with MSC-Ex was more potent than that with MSC in reducing DAI, the histological score, and nitrite
299 triple positive (CD73(+)CD90(+)CD105(+)) WJ MSCs found 67 genes with at least one CpG site where the
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