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1                                              MSC infusions were well tolerated.
2                                              MSC interrupted binding of the TF GATA-3 to the locus en
3                                              MSC morphology after IFN-gamma stimulation significantly
4                                              MSC therapy improves cardiac function in animal models o
5                                              MSC-Ex recovered the destruction of the epithelial barri
6                                              MSC-Ex shifted the macrophage functional phenotype from
7                                              MSC-induced changes in macrophage phenotype critically d
8                                              MSCs also expressed mannose receptor (MR) that was found
9                                              MSCs can be grown in large numbers in vitro, and autolog
10                                              MSCs can respond to tissue injury by anti- or proinflamm
11                                              MSCs significantly decreased natural killer cells in the
12                                              MSCs suppressed cytokine production, increased M2 macrop
13                                              MSCs were fluorescently labeled and injected into a rat
14                                              MSCs were transfected with recombinant minicircles encod
15                                 In humans, a MSC subpopulation carrying markers for endothelial and p
16                                    Activated MSC were found to accumulate in the wound margins severa
17 eatment protocol was identified as activated MSC co-administered with antibiotic therapy.
18 r necrosis factor alpha (TNFalpha)-activated MSCs significantly promoted tumor growth.
19 2 and CXCL5) expressed by TNFalpha-activated MSCs efficiently recruited CXCR2(+) neutrophils into tum
20 ast cancer, we found that TNFalpha-activated MSCs strikingly enhanced tumor metastasis compared with
21 dispensable in supporting TNFalpha-activated MSCs to promote tumor metastasis.
22 on of the recipient immune response after AD-MSC treatment.
23                                  Although AD-MSC administration on POD 1 or POD 4, 8, and 15 resulted
24 timization of the dosing and frequency of AD-MSC therapy, either alone or used in, combination with o
25  the influence of timing and frequency of AD-MSC treatment on immunologic and graft survival as well
26                                 In addition, MSCs secreted nitric oxide after Mtb infection, and inhi
27       Because few intravenously administered MSCs engraft in the myocardium, studies have mainly util
28                   Intravenously administered MSCs for acute myocardial infarction attenuate the progr
29 lar pathways through which IFN-gamma affects MSC plasticity and the consequence of their manipulation
30 hanced the immunosuppressive capacity of all MSC lines, and morphology predicted the magnitude of IFN
31 SCs/1 million CSCs, n = 7), 200 million allo-MSCs (n = 8), 1 million allo-CSCs (n = 4), or placebo (P
32  receive transendocardial injections of allo-MSCs + allo-CSCs (ACCT: 200 million MSCs/1 million CSCs,
33                    Whereas allogeneic (allo-)MSCs are immunoevasive, the capacity of CSCs to similarl
34 andomized, controlled trial using allogeneic MSC, which are logistically more convenient than autolog
35 ardiac surgery, administration of allogeneic MSCs did not decrease the time to recovery of kidney fun
36                                     Although MSC-derived EVs (mEVs) are beneficial for acute lung inj
37  cells identifies in adult mouse synovium an MSC population largely negative for the skeletal stem ce
38 ffects of pFUS increased both MSC homing and MSC production of VEGF and IL-10, suggesting microenviro
39 tween left ventricular dysfunction (LVD) and MSC activation.
40 s model was established in C57BL/6 mice, and MSC-Ex was administered intraperitoneally.
41 timized seeding condition of NSCs, BMECs and MSCs.
42 ll cocultures of rat hippocampal neurons and MSCs.
43                Cotransplantation of NPIs and MSCs resulted in significantly earlier normoglycemia and
44   Transcriptomes of many diverse tissues and MSCs further show that lamin-A,C's increase with tissue
45 vidence of early AKI to receive intra-aortic MSCs (AC607; n=67) or placebo (n=68).
46                                     We armed MSCs with different oHSV variants (MSC-oHSV) and found t
47 )-purified Tbx18-expressing cells behaved as MSCs in vitro.
48 ith nonatherosclerotic-MSCs, atherosclerotic-MSCs displayed higher levels of both intracellular (P=0.
49 ive oxygen species levels of atherosclerotic-MSCs promoted a phenotypic switch characterized by enhan
50 ed mitochondrial function of atherosclerotic-MSCs underlies their altered secretome and reduced immun
51    Furthermore, treatment of atherosclerotic-MSCs with the reactive oxygen species scavenger N-acetyl
52 These results indicate that FOXP1 attenuates MSC senescence by orchestrating their cell-fate switch w
53 wn in large numbers in vitro, and autologous MSCs transfused into tuberculosis patients have been fou
54 ety of intra-arterial infusion of autologous MSCs in patients with RVD.
55 ignant cells through which crosstalk between MSCs and TGFbeta regulates tumour metastasis.
56 ry AML, and that IL8 was increased in AML/BM-MSC cocultures.
57 at bone marrow mesenchymal stromal cells (BM-MSC) protect AML blasts from spontaneous and chemotherap
58     Finally, targeted IL8 shRNA inhibited BM-MSC-induced AML survival.
59 cteristics with bone marrow-derived MSCs (BM-MSC).
60  novel interaction between AML blasts and BM-MSCs, which benefits AML proliferation and survival.
61 combinant MIF increased IL8 expression in BM-MSCs via its receptor CD74.
62 ta (PKCbeta) regulated MIF-induced IL8 in BM-MSCs.
63 human AML compared with AML cultured with BM-MSCs and found that macrophage migration inhibitory fact
64 nic transcription factors in normal jaw bone MSCs.
65 The molecular effects of pFUS increased both MSC homing and MSC production of VEGF and IL-10, suggest
66 red retrotransposition is much lower in both MSCs and HSCs when compared to NPCs.
67  Intra-arterial route is very promising, but MSCs are missing machinery for diapedesis through blood-
68 CL21 on inflammatory cells were inhibited by MSC-Exo.
69                         Bacterial killing by MSC was found to be mediated in part by secretion of cat
70 s) in injured cortex were also attenuated by MSCs.
71                                Protection by MSCs entails three complementary mechanisms: 1) internal
72                  These cells (designated CAF-MSCs) enhanced in vitro neuroblastoma cell proliferation
73 dministration of activated allogeneic canine MSC.
74 nflammatory cytokines from activated cardiac MSCs of TLR4-deficient mice, compared with WT cardiac MS
75  improve LV remodeling and function, cardiac MSCs from LVD exacerbated anterior wall thinning 28 days
76          Although transplantation of cardiac MSCs and subcutaneous MSCs from LVD and sham hearts did
77     The inflammatory polarization of cardiac MSCs by LVD was mediated by TLR4, as we found less secre
78 ion of CD47 (don't eat me signal) on cardiac MSCs after both TLR4 stimulation in vitro and transplant
79                         LVD switched cardiac MSCs toward an inflammatory phenotype, with increased se
80 T cardiac MSCs and saline, TLR4(-/-) cardiac MSCs survived in the cardiac tissue and maintained their
81                Next, we transplanted cardiac MSCs from TLR4(-/-) and WT male mice into the infarcted
82                     Compared with WT cardiac MSCs and saline, TLR4(-/-) cardiac MSCs survived in the
83 LR4-deficient mice, compared with WT cardiac MSCs.
84 hils and inflammation, mucus-secreting cell (MSC) numbers, type 2-associated cytokines (interleukin (
85 IONALE: Virtually all mesenchymal stem cell (MSC) studies assume that therapeutic effects accrue from
86              Human mesenchymal stromal cell (MSC) lines can vary significantly in their functional ch
87                      Mesenchymal stem cells (MSC) are currently employed for the treatment of inflamm
88                      Mesenchymal stem cells (MSC) are promising therapeutics for critical limb ischem
89                      Mesenchymal stem cells (MSC) exert antibacterial activity in vitro and in acute
90  Bone marrow-derived mesenchymal stem cells (MSC) have been promoted for multiple therapeutic applica
91 ne marrow-derived mesenchymal stromal cells (MSC) and in vitro 3T3-L1 preadipocytes significantly inc
92        Autologous mesenchymal stromal cells (MSC) treatments have shown feasibility, safety and stron
93 to originate from mesenchymal stromal cells (MSC), but their relationship with MSCs is not clear.
94 ne cells and mesenchymal stem/stromal cells (MSC).
95        Here we use mesenchymal system cells (MSC) as a potential substitute for pericytes in a BBB mo
96 sis and desensitizes mesenchymal stem cells (MSCs) against subsequent mechanical activation in vitro
97 tective potential of mesenchymal stem cells (MSCs) against the deleterious impact of AbetaOs on hippo
98 nation of autologous mesenchymal stem cells (MSCs) and cardiac stem cells (CSCs) synergistically redu
99  stem cells (iPSCs), mesenchymal stem cells (MSCs) and human umbilical vein endothelial cells (HUVECs
100                Here, mesenchymal stem cells (MSCs) and their nuclei spread in response to thickness-c
101 rogenitors and human mesenchymal stem cells (MSCs) appeared to contribute in replacement of 40% and 1
102  Bone marrow derived mesenchymal stem cells (MSCs) are regularly utilized for translational therapeut
103 tudies indicate that mesenchymal stem cells (MSCs) can stimulate angiogenesis and modify immune funct
104 he potential role of mesenchymal stem cells (MSCs) derived from human MT in the pathogenesis of bone
105 Adipose-derived (AD) mesenchymal stem cells (MSCs) especially have shown encouraging potential.
106                Human mesenchymal stem cells (MSCs) express scavenger receptors that internalize lipid
107 ment affects mesenchymal stromal/stem cells (MSCs) from umbilical cord's Wharton's Jelly (WJ) on a mo
108                      Mesenchymal stem cells (MSCs) have multiple properties including anti-inflammato
109  of allogeneic human mesenchymal stem cells (MSCs) in reducing the time to recovery from AKI after ca
110              Mesenchymal stromal stem cells (MSCs) isolated from adult tissues offer tangible potenti
111  bone marrow-derived mesenchymal stem cells (MSCs) on SE-induced adverse changes.
112                      Mesenchymal stem cells (MSCs) possess immunoregulatory, anti-inflammatory, and p
113                      Mesenchymal stem cells (MSCs) represent promising resource of cells for regenera
114 ieved to function as mesenchymal stem cells (MSCs), multipotent tissue-resident progenitors with grea
115 ls (BMECs) and human mesenchymal stem cells (MSCs).
116 h to magnetize human mesenchymal stem cells (MSCs).
117 esis of human marrow mesenchymal stem cells (MSCs).
118 c differentiation of mesenchymal stem cells (MSCs).
119                   Mesenchymal stromal cells (MSCs) are a promising candidate for a cell-based therapy
120                As mesenchymal stromal cells (MSCs) are continuously exposed in vivo to a dynamically
121                   Mesenchymal stromal cells (MSCs) are one of major components of the tumour microenv
122        RATIONALE: Mesenchymal stromal cells (MSCs) are promising therapeutic strategies for coronary
123  BMF that Gli1(+) mesenchymal stromal cells (MSCs) are recruited from the endosteal and perivascular
124 (P10) aging human mesenchymal stromal cells (MSCs) could be used for bone tissue regeneration as tiss
125  from bone marrow mesenchymal stromal cells (MSCs) in vitro.
126              Mesenchymal stem/stromal cells (MSCs) play crucial roles in maintaining tissue homeostas
127                   Mesenchymal stromal cells (MSCs) tend to infiltrate into tumors and form a major co
128                                           CM-MSC treatment reduces cartilage damage and suppresses im
129                                           CM-MSC treatment significantly reduced knee-joint swelling,
130 mesenchymal stem cell-conditioned medium (CM-MSC) as an alternative to cell therapy in an antigen-ind
131                        In the presence of CM-MSC or MSCs, increases in IL-10 concentration were obser
132  from healthy mice following culture with CM-MSC or co-culture with MSCs.
133 ymph nodes of arthritic mice treated with CM-MSC or MSCs.
134  T cells from arthritic mice treated with CM-MSC showed increases in FOXP3 and IL-4 expression and po
135 ght multi-aminoacyl-tRNA synthetase complex (MSC), restricting the pool of free LysRS-tRNA(Lys) Mount
136                  In contrast, CCL2 deficient MSCs did not induce T cell migration and VCAM-1 expressi
137 atopoietic stem cells or bone marrow-derived MSC or dendritic cells) for optimization of appropriate
138 hly suppressive human adipose tissue-derived MSC (AdMSC) display and induce a differential immunologi
139 ollowing transplantation of human BM-derived MSCs, globule-containing hepatocytes declined and donor-
140 regulated in the same manner in iPSC-derived MSCs.
141 nal characteristics with bone marrow-derived MSCs (BM-MSC).
142 ssessed the effect human bone marrow-derived MSCs have on neonatal porcine islets (NPIs) in vitro and
143           METHODS AND Adipose tissue-derived MSCs were isolated from atherosclerotic (n=38) and nonat
144 neering a variety of significantly different MSC shapes.
145 ogenesis RNA), by RNA interference disrupted MSC chondrogenesis, concomitant with reduced cartilage-s
146 ated the anti-arthritic effect of engineered MSCs in a collagen-induced arthritis (CIA) model.
147 or the delivery of biologics and engineering MSCs.
148 e from local myocardial effects of engrafted MSCs.
149 one outgrowth in MT, as mediated by enhanced MSC-driven osteogenic differentiation in the jaw bone.
150                           In this study, GFP-MSCs were topically applied to the surface of cerebral c
151          Combined injection of sRAGE and hBD-MSCs resulted in enhanced survival of hBD-MSCs and angio
152 BD-MSCs resulted in enhanced survival of hBD-MSCs and angiogenesis in PIRI-CLI mice.
153 reactive T cells and helps to understand how MSCs ameliorate symptoms in lupus-prone MRL.Fas(lpr) mic
154 n umbilical cord mesenchymal stem cells (hUC-MSCs), originating in Wharton's jelly, are multipotent s
155 d 2 are robust new players involved in human MSC immunoregulatory mechanisms, and the higher suppress
156  the effect of local administration of human MSC-Exo on established EAU in the same species.
157 Ms) were cultured ex vivo with/without human MSC-derived EVs before adoptive transfer to LPS-injured
158                                        Human MSCs, grown chronically at 5% O2, were administered intr
159 n this study, we show that transfer of human MSCs increased MRL.Fas(lpr) mouse survival, decreased T
160                Together, these data identify MSC morphology as a predictive feature of MSC immunosupp
161 adiposity, decreased bone mass, and impaired MSC self-renewal capacity in mice.
162 er application, scientists sought to improve MSC functions by engineering.
163 e ligand 2 in the MSC secretome and improved MSCs immunosuppressive capacity (P=0.03).
164 ing drug effects will enable improvements in MSC therapies for renal disease.
165 n target of rapamycin (mTOR) manipulation in MSCs was studied in vivo in a mouse model of delayed-typ
166     Generating dysfunctional mitochondria in MSCs using rhodamine 6G pretreatment also abrogated thes
167 m memory keeper of the fibrogenic program in MSCs.
168 eases LBR:lamin-A,C protein stoichiometry in MSCs versus osteogenesis (stiff).
169 n synthesis in HEK293 cells only, whereas in MSCs, satisfactory results were obtained only after usin
170  theoretically produced from cells including MSCs.
171         Further, functionalization increased MSC adhesion to the surfaces and the alphavbeta3-selecti
172                                      Indeed, MSCs exhibited intrinsic autophagy, which was up-regulat
173 tes that extensive long-term culture-induced MSC aging impaired their osteogenic ability and subseque
174  of Notch signaling fully blocked OA induced MSC osteogenic differentiation.
175 ALF and tissue eosinophils and inflammation, MSC numbers, reduced the production of type 2 cytokines
176                    Subsequently, we injected MSCs or saline into the infarcted myocardium of mice and
177                                  Intravenous MSCs eliminated the progressive deterioration in LV end-
178  of lung resident mesenchymal stem cells (LR-MSCs) and in the lung tissues of a pulmonary fibrosis mo
179   We have devised an adult human bone marrow MSC (hMSC) delivery formula by investigating molecular e
180 e test group received allogeneic bone marrow MSCs by intradiscal injection of 25 x 10 cells per segme
181 onditional depletion of Foxp1 in bone marrow MSCs led to premature aging characteristics, including i
182                               In bone marrow MSCs, FOXP1 expression levels declined with age in an in
183 nd PTC MSCs were comparable with bone marrow MSCs.
184                           On a stiff matrix, MSC cultured with conditioned media from mammary cancer
185  of allo-MSCs + allo-CSCs (ACCT: 200 million MSCs/1 million CSCs, n = 7), 200 million allo-MSCs (n =
186 ined whether administering extracts of MSCs (MSC-Ex) instead of MSCs could augment the beneficial eff
187          The effect of HA-coatings on murine MSC was functionally determined both, in vitro and in vi
188            Current methods to culture murine MSCs (mMSCs) select for rapidly dividing colonies and re
189 a (TGF-beta), we identified the TF musculin (MSC) as being critical for the development of induced Tr
190 hat upon HIV-1 infection, a free pool of non-MSC-associated LysRS is observed and partially relocaliz
191             Compared with nonatherosclerotic-MSCs, atherosclerotic-MSCs displayed higher levels of bo
192 hanced tumor metastasis compared with normal MSCs.
193                           Only ACCT, but not MSCs or CSCs, prevented ongoing negative remodeling by o
194 nd found that intracarotid administration of MSC-oHSV, but not of purified oHSV alone, effectively tr
195  predicted the immunosuppressive capacity of MSC lines in a validation cohort.
196 ll immunosuppressive capacity in a cohort of MSC lines derived from different donors and manufacturin
197  melanoma brain metastasis, a combination of MSC-oHSV and PD-L1 blockade increases IFNgamma-producing
198 box P1 (FOXP1) in transcriptional control of MSC senescence.
199 al characteristics, and the effectiveness of MSC-based therapeutics may be realized by finding predic
200 MSCs could augment the beneficial effects of MSC therapy by overcoming the low homing efficiency of M
201 fy MSC morphology as a predictive feature of MSC immunosuppressive function.
202 n group (n=14) received a single infusion of MSC (1.0 x 10(5) or 2.5 x 10(5) cells/kg; n=7 each) plus
203                                      Loss of MSC reduced expression of the Treg cell master TF Foxp3
204     Our findings reveal a novel mechanism of MSC effects on malignant cells through which crosstalk b
205 Here, we aimed to determine the mechanism of MSC recruitment by Dendritic Cells (DC), hypothesising t
206 ent and increases the chances for success of MSC therapy in tissue-repair applications.
207          At 3 weeks after transplantation of MSC sheets, results showed more bony callus formed betwe
208               The protumorigenic activity of MSCs in vitro and in xenografted mice was dependent on t
209 esis is valid, intravenous administration of MSCs should improve outcomes in heart failure, an entity
210 ing the altered immunomodulatory capacity of MSCs in the context of atherosclerosis remain elusive.
211 r level, FOXP1 regulated cell-fate choice of MSCs through interactions with the CEBPbeta/delta comple
212 on offers a direct and efficient delivery of MSCs to the brain.
213 uired for the odontogenic differentiation of MSCs.
214 ion, promotes odontogenic differentiation of MSCs.
215 study, we focused on the paracrine effect of MSCs on macrophage polarization and the role of extracel
216 responsible for the pro-metastatic effect of MSCs since inhibition of this chemotaxis abolished incre
217 y by overcoming the low homing efficiency of MSCs systemically administered in inflammatory bowel dis
218 e examined whether administering extracts of MSCs (MSC-Ex) instead of MSCs could augment the benefici
219 stering extracts of MSCs (MSC-Ex) instead of MSCs could augment the beneficial effects of MSC therapy
220  efficient and minimally invasive methods of MSCs delivery to the brain still have to be developed.
221  changes from pFUS also increased potency of MSCs in situ to further enhance their efficacy.
222  the impaired immunomodulatory properties of MSCs from patients with atherosclerosis.
223 ly important in promoting the recruitment of MSCs to the site of skin injury, which in turn modulates
224                    Following transfection of MSCs with the mms6 gene there is bio-assimilated synthes
225  This study thus demonstrates the utility of MSCs as OV carriers to disseminated brain lesions, and p
226  the hyaluronic acid (HA) receptor, CD44, on MSC membranes, to improve their homing potential towards
227 and the consequence of their manipulation on MSC functions.
228 ng is required for its osteogenic effects on MSCs.
229                 In the presence of CM-MSC or MSCs, increases in IL-10 concentration were observed in
230 des of arthritic mice treated with CM-MSC or MSCs.
231 area of the chondriod callus in the aged P10 MSC sheet groups was significantly larger than in P3 MSC
232  bone ends in both young P3 MSC and aged P10 MSC sheet-wrapped groups when compared to allograft alon
233 t groups was significantly larger than in P3 MSC sheet groups.
234 llograft and host bone ends in both young P3 MSC and aged P10 MSC sheet-wrapped groups when compared
235                                       pFUS + MSC improved perfusion and vascular density in this clin
236            By 7 weeks post-treatment, pFUS + MSC significantly increased perfusion and CD31 expressio
237 %, demonstrating a prototype photoswitchable MSC.
238 slational progress has been hampered by poor MSC graft survival, jeopardizing cellular and molecular
239 EBVs fabricated from normal iSMCs or primary MSCs.
240 demonstrates that CCL2 enables the prolonged MSC-T cell interactions needed for sufficient suppressio
241 ry following cell culture expansion protects MSCs from fibrogenesis in the host wound environment and
242 tion characteristics of Thyroid MSCs and PTC MSCs were comparable with bone marrow MSCs.
243                 Fluorescent and radiolabeled MSCs (1x10(6)) were injected 24 hours post-myocardial in
244  significance of BMP signaling in regulating MSC fate during root development and shed light on how B
245 mmary, our data show that CD34(+) derived SG-MSCs could be a promising cell source for adoptive cell-
246 f female WT mice and evaluated infarct size, MSC retention, inflammation, remodeling, and function af
247 splantation of cardiac MSCs and subcutaneous MSCs from LVD and sham hearts did not improve LV remodel
248 ine-filtering on-chip micro-supercapacitors (MSCs) based on coordination polymer frameworks were fabr
249                      Finally, human synovial MSCs transduced with Bmp7 display morphogenetic properti
250  ARDS is a heterogeneous syndrome, targeting MSCs to patients with ARDS with a more hyperinflammatory
251                                We found that MSC-Exo greatly reduced the intensity of ongoing EAU as
252                 Interestingly, we found that MSC-produced CXCL12, an important chemokine in tumour me
253           Further experiments indicated that MSC contributed in a similar manner to pericytes in a co
254               Recent studies have shown that MSC tumour residence and their close interactions with i
255 lagen nanofilms serve as a model matrix that MSCs can easily deform unless the film is enzymatically
256              Results support the notion that MSCs may represent a promising alternative for cell-base
257                               We report that MSCs phagocytose Mycobacterium tuberculosis (Mtb) throug
258             Time-lapse imaging revealed that MSCs recruited MRL.Fas(lpr) T cells establishing long-la
259 timuli, but how LysRS is redirected from the MSC to viral particles for packaging is unknown.
260                      We first identified the MSC population supporting mouse molar root growth as Gli
261 tractant protein-1/chemokine ligand 2 in the MSC secretome and improved MSCs immunosuppressive capaci
262 tified by Pfirrmann grading, improved in the MSC-treated patients and worsened in the controls.
263  studies demonstrated that components of the MSC can be mobilized in response to certain cellular sti
264    Three days after topical application, the MSCs homed to the injured parenchyma and improved the ne
265 ytokeratin(+) cells in the bone marrow, this MSC subpopulation could prove useful in determining the
266 d differentiation characteristics of Thyroid MSCs and PTC MSCs were comparable with bone marrow MSCs.
267 the extracellular matrix (ECM) contribute to MSC phenotype in cancer remains poorly understood.
268 intravenously administered ischemia-tolerant MSCs (itMSCs) in patients with nonischemic cardiomyopath
269                                      Topical MSCs triggered earlier astrocytosis and reactive microgl
270 teogenic differentiation of mandibular torus MSCs was associated with the suppression of Notch3 signa
271 m contained only a small proportion of total MSCs.
272 omicroscopy as a novel imaging tool to track MSCs in vivo.
273 enous ITGA4-mRNA was detected in transfected MSCs.
274  myocardium drives resident and transplanted MSCs toward a proinflammatory phenotype and restricts th
275 ase in the migratory potential of HA-treated MSC compared to untreated cells.
276   This increased accumulation for HA-treated MSC yielded a substantial reduction in inflammation as d
277        However, the role of TNFalpha-treated MSCs in tumor metastasis remains elusive.
278 d efficacy of the intravenous infusion of UC-MSC in patients with chronic stable heart failure and re
279                                  Only the UC-MSC-treated group exhibited significant improvements in
280                                           UC-MSCs in vitro, compared with bone marrow-derived mesench
281 ized to intravenous infusion of allogenic UC-MSCs (Cellistem, Cells for Cells S.A., Santiago, Chile;
282 fe were observed in patients treated with UC-MSCs.
283  We armed MSCs with different oHSV variants (MSC-oHSV) and found that intracarotid administration of
284                                    In vitro, MSCs from individuals with coronary artery disease have
285 sults on histology demonstrate promises when MSCs are injected endotracheally (but not intravenously)
286  finding predictive features associated with MSC function.
287 US in mouse kidneys and its association with MSC tropism.
288 muscle actin, a marker of CAF, compared with MSC cultured on a soft matrix.
289     Adoptive transfer of AMs pretreated with MSC-derived EVs reduced inflammation and lung injury in
290 t with MSC-Ex was more potent than that with MSC in reducing DAI, the histological score, and nitrite
291                             AMs treated with MSC-derived EVs ameliorate lung injury in vivo.
292                               Treatment with MSC-Ex completely blocked the induction of inflammatory
293                               Treatment with MSC-Ex was more potent than that with MSC in reducing DA
294                         NPIs cocultured with MSCs had greater cellular insulin content and increased
295 owing culture with CM-MSC or co-culture with MSCs.
296 ch HE-iPSCs were separately co-cultured with MSCs and/or HUVECs.
297 mal cells (MSC), but their relationship with MSCs is not clear.
298     NPIs were cotransplanted with or without MSCs in diabetic B6.129S7-Rag1(tm1Mom)/J mice.
299  triple positive (CD73(+)CD90(+)CD105(+)) WJ MSCs found 67 genes with at least one CpG site where the
300 ody weight might bear consequences on the WJ MSCs.
301 e model similar to fresh passaged (P3) young MSCs.

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