ライフサイエンスコーパス: MSD

1 es flanked by basic amino acids in the HIV-1 MSD that function to anchor the glycoprotein in the lipi

2 polar head groups, is proposed for the HIV-1 MSD.

3 We aimed to enroll 220 MSD cases per year from selected health centers serving

4 atal in the community without treatment; (3) MSD seen at sentinel centers is a proxy for fatal diarrh

5 es were compared between 1,052 URD and 3,514 MSD BMT recipients with CML in CP1.

6 lts with advanced Hodgkin's lymphoma (UCB-9, MSD-12).

7 y ligase-PROTAC-target protein complex and a MSD assay to measure cellular degradation of the target

8 Furthermore, indications for a MSD were found by UPLC/MS/MS measurements.

9 In children lacking a MSD, increasingly MUD HSCT is being considered as first-

10 = 0.93), Abeta42-EI (r = 0.93), and Abeta42-MSD (r = 0.95) assays compared with the classic Abeta42-

11 ctors (including knowledge and beliefs about MSDs) between occupational groups should allow the study

12 In addition, MSD-IAA demonstrated better sensitivity than our mIAA fr

13 qPCR detected 155 additional MSD cases with high copy numbers of ipaH, a 90% increase

14 e functional significance of this additional MSD (MSD1) is currently unknown.

15 r rate of T-cell recovery was observed after MSD/MFD HCT.

16 re encouraging, with similar EFS rates after MSD, 6/6 matched UCB, 5/6 UCB, and 10/10 matched UD.

17 ds and were significantly protective against MSD in Mozambique and India.

18 In comparison, ion trap noise from Agilent MSD-Trap-SL is larger than the Q-TOF noise and is propor

19 ched at HLA-A, -B, and at -DRB1 alleles; all MSDs were genotypically identical at major histocompatib

20 An MSD case was defined as a child with a diarrheal illness

21 died 36 patients with obstructive HCM and an MSD and compared them with 15 patients with HCM and no o

22 These data favor an MSD over a MUD in patients age >/=50 years.

23 y exhibit subdiffusive transport and have an MSD scaling as t0.7.

24 ding to diabetes determine high affinity and MSD-IAA reactivity.

25 n back-propagation neural network (BPNN) and MSD analysis using a sliding window.

26 estimate the annual number of MSD cases and MSD incidence rate attributable to a pathogen or group o

27 tical copolymer including MMD, FTD, CCD, and MSD.

28 immunoassays (modified INNOTEST, FL, EI, and MSD) may correlate better with the antibody-independent

29 between sanitation and hygiene exposures and MSD.

30 significant difference between Haplo-HCT and MSD-HCT in terms of nonrelapse mortality (P = .06), prog

31 Therefore, LRRD and MSD are analog and digital force transducers, respective

32 re not significantly different after MUD and MSD transplants.

33 ll domain-swapping contacts between NBDs and MSD cytoplasmic loops in opposite halves of the protein

34 he established curative potential of URD and MSD marrow transplantation.

35 on at the last follow-up between the URD and MSD recipients; however, MSD recipients were more likely

36 Improvements in URD and MSD transplantation are still needed, and results of new

37 Fatigue and MSDs impact psychomotor performance; therefore, these re

38 integration of structural databases such as MSD and sequence databases like UniProt is the absence o

39 inished by mutating O-glycosylation sites at MSD.

40 f MSD and viscosity to the ensemble-averaged MSD and viscosity.

41 the longest molecules, the ensemble-averaged MSD increases linearly with time at all probed time scal

42 However, traditional time-averaged MSD analysis of individual trajectories remains linear a

43 of MSDs after 4 years among women (for back MSDs, risk ratio (RR) = 1.58, 95% confidence interval (C

44 1.59, 95% CI: 1.21, 2.07) and men (for back MSDs, RR = 1.50, 95% CI: 1.05, 2.15; for degenerative MS

45 both types of MSDs after 11 years (for back MSDs, RR = 1.72, 95% CI: 1.21, 2.43; for degenerative MS

46 hringer Ingelheim (Canada), Pfizer (Canada), MSD, Chest, Heart and Stroke Scotland, and The Stroke As

47 echnology from Meso Scale Discovery Company (MSD-IAA) detects as positive 61% (61 of 100) of new-onse

48 Among a subset (n = 2,874) comprising MSD cases and their age-, gender-, and location-matched

49 forms of NCAM, we found that NCAM containing MSD facilitates myoblast fusion, and this effect is dimi

50 yoblast system, we show that NCAM containing MSD is increasingly expressed on the cell surface as myo

51 ynamics analysis, which extends conventional MSD analysis to measure diffusive motion in confined tra

52 The Macromolecular Structure Database (MSD) group continues to enhance the quality and consiste

53 The Macromolecular Structure Database (MSD) group continues to enhance the quality and consiste

54 The Macromolecular Structure Database (MSD) group is one of the three partners in the worldwide

55 of information between the member databases (MSD, UniProt, Pfam, Interpro, SCOP and CATH).

56 generation in multiple sulfatase deficiency (MSD), a severe lysosomal storage disorder caused by muta

57 ting disorder multiple sulfatase deficiency (MSD).

58 interval (CI): 1.15, 2.18; for degenerative MSDs, RR = 1.59, 95% CI: 1.21, 2.07) and men (for back M

59 = 1.50, 95% CI: 1.05, 2.15; for degenerative MSDs, RR = 1.61, 95% CI: 1.16, 2.22) and both types of M

60 = 1.72, 95% CI: 1.21, 2.43; for degenerative MSDs, RR = 1.68, 95% CI: 1.25, 2.46) among men only, but

61 nfounders, job strain predicted degenerative MSDs among women after 4 and 11 years of follow-up.

62 Multi-specificity design (MSD), on the other hand, involves considering the stabil

63 ch we call Multivariate Shapelets Detection (MSD), that allows for early and patient-specific classif

64 hod called Multivariate Shapelets Detection (MSD), which extracts patterns from all dimensions of the

65 A more sustainable development (MSD) scenario introduces an impact fee that developers m

66 The mean standard deviation (MSD) was calculated to be 3.65 when alpha = 0.01.

67 m-maximum (MIN-MAX), map standard deviation (MSD), and pattern standard deviation (PSD), were calcula

68 uences of acute moderate-to-severe diarrhea (MSD) among a censused population of children aged 0-59 m

69 associated with moderate-to-severe diarrhea (MSD) among children <5 years of age, and thereby the att

70 d incidence) of moderate-to-severe diarrhea (MSD) in children <60 months of age at 7 sites in sub-Sah

71 isk factors for moderate-to-severe diarrhea (MSD) in children less than 5 y of age.

72 with or without moderate-to-severe diarrhea (MSD).

73 ut also in terms of mean squared-difference (MSD), Lloyd's K-means Clustering algorithm is more effic

74 ons (SAM) or the method of serial dilutions (MSD).

75 interpret using conventional two-dimensional MSD analysis.

76 ncentration of KS monosulfated disaccharide (MSD) Gal-beta-1,4-GlcNAc(6S) in E8 cornea equaled that a

77 ), EUROIMMUN (EI), and Meso Scale Discovery (MSD) assays.

78 Here we developed a Meso Scale Discovery (MSD)-based screening platform to quantify endogenous NMN

79 k and degenerative musculoskeletal diseases (MSDs).

80 tudy was to review musculoskeletal disorder (MSD) prevalence among surgeons performing minimally inva

81 hesis that common musculoskeletal disorders (MSDs) and associated disability are importantly influenc

82 ral scaling of the mean-square displacement (MSD proportional, variant tau (alpha), with alpha = 2/3)

83 Conventional mean-square displacement (MSD) analysis of single-particle trajectories often assu

84 A local mean-square displacement (MSD) analysis separates ballistic motion phases, which w

85 en evaluated using mean square displacement (MSD) analysis.

86 is examined by the mean-square displacement (MSD) and the fractional diffusion and fractional kinetic

87 ditionally use the mean square displacement (MSD) as an order parameter characterizing dynamics.

88 (t<7 s) with their mean-square displacement (MSD) Deltax(t)2 scaling as t1.25.

89 ensemble-averaged mean square displacement (MSD) exhibits superdiffusive behaviour.

90 the time-dependent mean-square displacement (MSD) of polystyrene microspheres imbedded in low- and hi

91 namics governs the mean square displacement (MSD) of water molecules, with a clear cage stage charact

92 tracked particle's mean-square displacement (MSD).

93 he combination of mean squared displacement (MSD) and cumulative distribution function (CDF) analysis

94 ution, from which mean-squared displacement (MSD) and viscosity distributions are generated.

95 Mean-squared displacements (MSD) and protein resilience on the picosecond-to-nanosec

96 on the measured mean squared displacements (MSD).

97 es as well as monomer sequence distribution (MSD), gradient separation only by CCD was not possible.

98 ) and juxtamembrane mechanosensitive domain (MSD).

99 ces unfolding of this mechanosensory domain (MSD) on the platelet surface.

100 ately 1-395) and a membrane-spanning domain (MSD) (residues approximately 396-655); however, its exac

101 eucine) within the membrane-spanning domain (MSD) abolished protein functionality in infectivity assa

102 he residues in the membrane-spanning domain (MSD) and also immediately flanking it (T140 to L163).

103 yl terminus of the membrane-spanning domain (MSD) have been characterized.

104 ing domain and one membrane-spanning domain (MSD) likely work as dimers, whereas full-length transpor

105 The membrane-spanning domain (MSD) of a number of retroviral transmembrane (TM) glycop

106 The membrane-spanning domain (MSD) of the envelope (Env) glycoprotein from human (HIV)

107 The 27-residue membrane-spanning domain (MSD) of the HIV-1 glycoprotein gp41 bears conserved sequ

108 The membrane-spanning domain (MSD) of the human immunodeficiency virus type 1 (HIV-1)

109 y of the F-MLV Env membrane-spanning domain (MSD) to its incorporation into HIV-1 particles.

110 have an additional membrane-spanning domain (MSD) with a putative extracellular amino terminus.

111 D) followed by one membrane-spanning domain (MSD).

112 le tissue contains a muscle-specific domain (MSD) to which mucin type O-glycans are attached.

113 loops (CL) of the membrane-spanning domains (MSD).

114 ssembled from two membrane-spanning domains (MSDs) and two nucleotide-binding domains (NBDs).

115 oops of the three membrane-spanning domains (MSDs) of the transporter.

116 region (MPER) and membrane-spanning domains (MSDs) of viral glycoproteins have been shown to be criti

117 d consists of two membrane spanning domains (MSDs), two cytosolic nucleotide binding domains (NBDs),

118 structed from two membrane-spanning domains (MSDs), two nucleotide-binding domains (NBD) and a regula

119 CFTR contains two membrane-spanning domains (MSDs), two nucleotide-binding domains (NBDs), and a regu

120 ts interface with membrane-spanning domains (MSDs).

121 EFS after HLA-matched sibling donor (MSD) and 6/6 matched unrelated cord blood (CB) donor wer

122 ukocyte antigen (HLA)-matched sibling donor (MSD) HSCT remains the gold standard for SAA patients you

123 th 450 HLA-identical, matched sibling donor (MSD) transplantations performed at collaborating Nationa

124 do not have an HLA-identical sibling donor (MSD).

125 rom a matched histocompatible sibling donor (MSD).

126 he sequences between the major splice donor (MSD) and the start codon of gag contribute negligibly to

127 -cord blood (UCB) or matched-sibling donors (MSD) in 21 high-risk adults with advanced Hodgkin's lymp

128 patients lacking HLA-matched sibling donors (MSD).

129 ved transplants from matched sibling donors (MSD; n = 21) and unrelated donors (UD; n = 29), using fl

130 HCT from matched sibling and family donors (MSDs, MFDs) had significantly better overall survival (8

131 ncy disease who have matched sibling donors (MSDs) can proceed to hematopoietic cell transplantation

132 oviruses with defective major splice donors (MSDs) can activate novel splice sites to produce HIV-1 t

133 HLA-matched sibling (matched sibling donors [MSDs]) BMT in 66 patients.

134 estigated the cause of the midsystolic drop (MSD) in left ventricular (LV) ejection velocities that a

135 KS disulfated/monosulfated disaccharide (DSD/MSD) ratios.

136 The E-MSD macromolecular structure relational database is desi

137 iffusive" regime is only a transient effect, MSD proportional, varianttau(alpha),alpha<1.

138 rved after receipt of transplant from either MSD or URD.

139 ighly conserved core residues of the HIV Env MSD, in addition to serving as a membrane anchor, play a

140 Using HPLC/ESI-MSD, the proanthocyanidin monomers, (+)-catechin (C), (-

141 ization-mass spectrometric detection (LC/ESI-MSD) for raw grape products.

142 592 case children aged <5 y old experiencing MSD and for 12,390 asymptomatic age, gender, and neighbo

143 , 0-55%) for UCB vs. 20% (95% CI, 0-44%) for MSD alloSCT (p=0.67).

144 tive incidence of graft failure was 9.5% for MSD and 14.5% for UD HSCT.

145 erall survival at 2 years was 95% +/- 5% for MSD and 83% for UD HSCT (p 0.34).

146 ticing open defecation was a risk factor for MSD in children <5 y old in Kenya.

147 a statistically significant risk factor for MSD in Kenya, Mali, Mozambique, and Pakistan.

148 ted donor (MUD) despite trial guidelines for MSD transplantations only.

149 pathogen was isolated and the odds ratio for MSD and the pathogen from conditional logistic regressio

150 worse than those after receipt of BMTs from MSDs (5-year survival, 55% v 63%; RR, 1.35; 95% CI, 1.17

151 ansplants from URDs compared with those from MSDs remains undefined.

152 l cord blood, are comparable with those from MSDs.

153 than those after receipt of transplant from MSDs.

154 les gas chromatography/mass spectrometry (GC/MSD) with a novel scheme for cryogen-free low-temperatur

155 conformational variability of the HIV-1 gp41 MSD.

156 ), more negative MIN-MAX (P<0.0001), greater MSD (P<0.0001), and larger PSD (P<0.0001).

157 A mean deviation (MD) > MSD indicates a significant difference.

158 It is of interest that in holo-Mb, higher MSD values are observed for the residues outside the hem

159 However, MSD measurements are highly susceptible to static error

160 between the URD and MSD recipients; however, MSD recipients were more likely to achieve B-cell recons

161 95% confidence interval [95% CI], 6%-21%) in MSD groups (P = .15), with respective thalassemia-free s

162 nd control households, are evaluated both in MSD cases and in the population.

163 of the time after 30 years of development in MSD or business as usual (BAU) scenarios, respectively.

164 Neutrophil recovery occurred earlier in MSD group (median 7 vs. 10 days, p=0.02).

165 integral components of the neuropathology in MSD and that modulation of astrocyte function may impact

166 r T-cell engraftment similar to that seen in MSD recipients, and reconstitution rates are adequate.

167 materials, coincident with the cage stage in MSD, the non-Gaussian parameter indicates a significant

168 essential statistical features of the local MSD algorithm.

169 Specifically, the values of the mean MSD exponent and effective diffusion coefficients can be

170 etiologic agents may be responsible for most MSD; (2) a definition of MSD can be crafted that encompa

171 nalization signal in the context of a mutant MSD.

172 binding small molecules with or without NBD1/MSD interface mutation.

173 ether, our findings indicate that the NMNAT2-MSD platform provides a sensitive phenotypic screen to d

174 ivity and large dynamic range of this NMNAT2-MSD platform allowed us to screen the Sigma LOPAC librar

175 The IAA detectable by radioassay, but not MSD-IAA, were usually of lower affinity compared with th

176 a subset of IAA with current radioassay (not MSD-IAA) represents biologic false positives in terms of

177 We have developed a novel MSD algorithm, which we refer to as REstrained CONvergen

178 The entire RD group and 88% of MSD group had sustained engraftment.

179 roach that greatly increases the accuracy of MSD measurements is presented herein by combining experi

180 nitation facilities may reduce the burden of MSD in children.

181 esponsible for most MSD; (2) a definition of MSD can be crafted that encompasses cases that might oth

182 ic strategies will calculate the fraction of MSD attributable to each pathogen and the incidence, fin

183 would substantially increase the fraction of MSD cases that are attributable to Shigella.

184 d location-matched controls, the fraction of MSD cases that were attributable to Shigella infection i

185 hogen-specific population-based incidence of MSD, to guide investments in research and public health

186 k a subset of neurological manifestations of MSD to astrocyte dysfunction.

187 on are used to estimate the annual number of MSD cases and MSD incidence rate attributable to a patho

188 of the incidence, etiology, and outcomes of MSD among infants and young children in sub-Saharan Afri

189 hogens, is estimated using the proportion of MSD cases from whom the pathogen was isolated and the od

190 djusted attributable fraction, proportion of MSD cases taken to a sentinel health center (SHC), numbe

191 nalyzed comparing outcomes for recipients of MSD >/=50 (n = 1415) versus MUD <50 years (n = 757).

192 The repeatability of MSD was better than 0.4 mum.

193 wo other households can increase the risk of MSD in young children, compared to using a private facil

194 molar percent of DSD was higher than that of MSD from E8 to E18, equivalent at E20, and less than tha

195 18, equivalent at E20, and less than that of MSD in adult corneas.

196 yields survival and DFS approaching that of MSD transplantation.

197 s of the 10%, 25%, and 50% highest values of MSD and viscosity to the ensemble-averaged MSD and visco

198 but there may be other pathways of onset of MSDs in old age.

199 To summarize the prevalence of MSDs among surgeons performing laparoscopic surgery, we

200 chanical exposure for possible prevention of MSDs in working life and around the time of retirement,

201 1.61, 95% CI: 1.16, 2.22) and both types of MSDs after 11 years (for back MSDs, RR = 1.72, 95% CI: 1

202 Joint exposure predicted both types of MSDs after 4 years among women (for back MSDs, risk rati

203 ears, both exposures predicted both types of MSDs among men.

204 nts undergoing either Haplo-HCT (n = 180) or MSD-HCT (n = 807) following reduced-intensity conditioni

205 deletions and insertions within the MPER or MSD result in functionally inactive proteins.

206 concentration, quantified via either SAM or MSD or simply a calibration curve, is of great importanc

207 s for reduced-intensity alloSCT using UCB or MSD source in high-risk adults with advanced Hodgkin's l

208 relapse was low after either matched URD or MSD transplantations.

209 conditioned HCT from either URDs (n = 37) or MSDs (n = 66).

210 xplanations for the roles of these and other MSD features remain unclear.

211 Differences in thermodynamic parameters, MSD, and resilience were observed for both proteins.

212 separate and joint exposures did not predict MSDs.

213 ability of truncations within the predicted MSD to remain membrane anchored and maintain biological

214 l ergonomics as a determinant, and reporting MSD prevalence.

215 interface that begins to integrate separate MSD search services in a single graphical tool.

216 D) rather than an older HLA-matched sibling (MSD).

217 lecular dynamics simulations yielded similar MSD and resilience values for the two proteins.

218 el method for quantifying FAE using a single MSD immunoassay is also reported and confirms previous f

219 d the heme group in holo-Mb showed a smaller MSD and higher resilience compared to the same residue g

220 (SWISS-PROT and TrEMBL), protein structure (MSD), whole genomes (Ensembl) and gene expression (Array

221 lly poorer 5-year DFS after matched URD than MSD BMT (CP 1-2 years: URD 39% +/- 6% vs MSD 63% +/- 12%

222 were younger (median 36 vs 39, P =.001) than MSDs and underwent BMT later after diagnosis (median 17

223 The MSD 'atlas pages' show all of the information in the MSD

224 The MSD distribution of a 2.6-microM F-actin solution is sym

225 The MSD group received calcineurin inhibitor-based GVHD prop

226 The MSD in velocity and flow occurs in patients with gradien

227 The MSD scenario generates more tax revenue and water saving

228 reduced fusogenicity and infectivity as the MSD length was shortened.

229 ortant role in the communication between the MSD and NBD as it is predicted to be located at the inte

230 mation about stochastic processes beyond the MSD.

231 delays the expression of NCAM containing the MSD domain.

232 In our experiments, the MSD method outperformed the baseline methods, achieving

233 these mutants, an alternative model for the MSD of SIV is proposed.

234 sequence cross-reference information in the MSD and UniProt databases.

235 In the MSD approach instead of the sequence of standard additio

236 s enriched the structural information in the MSD database with annotation from wider sequence-oriente

237 as pages' show all of the information in the MSD for a particular PDB entry.

238 the RD group and 8% (95% CI, 3%-16%) in the MSD group (P = .83).

239 omy and Sequences (SIFTS)' initiative in the MSD group.

240 lenced by a counteracting determinant in the MSD that acts to prevent clustering of Env into endocyti

241 r deleted one, two, or three leucines in the MSD were constructed.

242 n HIV-1 Env upon addition of leucines in the MSD, with +1 and +2 leucine mutations greatly reducing E

243 Since its inception, the MSD group has worked with partners around the world to i

244 ide chain snorkels to the inner leaflet, the MSD peptide assumes a metastable conformation consistent

245 the outer leaflet of the viral membrane, the MSD assumes a metastable conformation where the highly-c

246 from the cell body contain MSP, but not the MSD proteins, which counteract MSP filament assembly.

247 With medical abolition of obstruction the MSD disappears and the duration and length of contractio

248 144) and a proline near the beginning of the MSD (P148).

249 (T139 and T140) and at the N terminus of the MSD (S143 and T144) and a proline near the beginning of

250 he C terminus (positions 159 and 160) of the MSD as being the boundaries of the two interfaces.

251 However, the amino acid composition of the MSD core does influence the ability of Env to mediate ce

252 We show here that conservation of the MSD core sequence is not required for normal expression,

253 The implementation of the MSD database, together with the parallel development of

254 Quantitative changes in the shape of the MSD distribution correlate qualitatively with the presen

255 P with the amino and carboxyl termini of the MSD located intracellularly.

256 Env constructs were deleted, all six of the MSD mutants were able to produce infectious particles.

257 e nature of the fusion defect in five of the MSD mutants: TM185, TM186, TM187, TM188, and TM189.

258 In contrast, sequences upstream of the MSD were important for encapsidation, and deletion of th

259 in its predicted hydrophilic regions of the MSD.

260 fects from the local microenvironment on the MSD and potentially generate misleading conclusions abou

261 SRSF2, we analyzed splicing responses on the MSD-L cell line and found that the missense mutation of

262 sed interface that allows users to query the MSD directly.

263 elated projects in the NMR and EM realms the MSD continues to improve the quality of publicly availab

264 , increasing actin concentration renders the MSD distribution wide and asymmetric, an effect enhanced

265 These data indicate that the MSD is caused by premature termination of LV segmental s

266 We showed that the MSD method can classify the time series early by using a

267 We find that the MSD peptide assumes a stable tilted alpha-helical confor

268 this data viewer is now used throughout the MSD services for the visualization and presentation of s

269 etime is prolonged by LRRD unfolding) to the MSD to enhance its unfolding, resulting in unfolding coo

270 n the haploidentical group compared with the MSD group (63% v 91%; P = .001).

271 l is shorter in obstructed patients with the MSD than in nonobstructed HCM patients: septal contracti

272 the role of specific amino acids within the MSD core, we initially replaced the core region with 12

273 diverse transmembrane pathways formed by the MSDs.

274 By contrast, in CFTR the MSDs form a pathway for passive anion flow that is gated

275 nucleotide-binding domains and two or three MSDs function as monomers.

276 ad similar 5-year overall survival (100%) to MSD recipients.

277 -Cy provides survival outcomes comparable to MSD-HCT, with a significantly lower risk of chronic GVHD

278 Compared to MSD algorithms that enforce (constrain) an identical seq

279 received a first-remission transplantation (MSD and MUD) than for the 351 patients treated with chem

280 nus in addition to the core structure of two MSDs and two NBDs (nucleotide-binding domains).

281 The unfolded MSD, particularly the juxtamembrane 'Trigger' sequence t

282 ing members of the wwPDB are RCSB PDB (USA), MSD-EBI (Europe) and PDBj (Japan).

283 The members of the wwPDB-RCSB PDB (USA), MSD-EBI (Europe), PDBj (Japan) and BMRB (USA)-have remed

284 Using MSD as the reference standard, we determined the optimal

285 For Haplo-HCT v MSD-HCT, 3-year rates of nonrelapse mortality (15% v 13%

286 y slightly inferior after matched URD versus MSD transplantation (age < 30: URD 61% +/- 8% vs MSD 68%

287 han MSD BMT (CP 1-2 years: URD 39% +/- 6% vs MSD 63% +/- 12%; beyond 2 years: URD 33% +/- 7% vs MSD 5

288 % +/- 12%; beyond 2 years: URD 33% +/- 7% vs MSD 50% +/- 20%).

289 transplantation (age < 30: URD 61% +/- 8% vs MSD 68% +/- 15%, P =.18; 30-40: URD 57% +/- 9% vs MSD 67

290 67% +/- 10%, P =.05; > 40: URD 46% +/- 9% vs MSD 57% +/- 9%, P =.02).

291 8% +/- 15%, P =.18; 30-40: URD 57% +/- 9% vs MSD 67% +/- 10%, P =.05; > 40: URD 46% +/- 9% vs MSD 57%

292 unfolding intensifies Ca(2+) signal whereas MSD unfolding affects the type of Ca(2+) signal.

293 RD (50% and 39%, respectively) compared with MSD (22% and 5%, respectively) recipients (P < .01 for b

294 ts (71% and 60%, respectively) compared with MSD recipients (92% and 89%, respectively; P < .01 for b

295 < .0001) were higher after MUD compared with MSD transplants.

296 ed outcomes after Haplo-HCT using PT-Cy with MSD-HCT in patients with lymphoma, using the Center for

297 transplant outcomes similar to patients with MSD grafts.

298 r progression to diabetes were positive with MSD-IAA assay.

299 her islet autoantibodies) were positive with MSD-IAA.

300 engraftment compared with 97% for those with MSDs; however, estimated 5-year overall and event-free s