ライフサイエンスコーパス: MVV

コーパス検索結果 (left1)

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1 MVV infection induces axonal damage with some areas of n

2 +/- 0.24 versus 0.95 +/- 0.31 L, p < 0.001), MVV (28 +/- 11 versus 41 +/- 13 L, p < 0.001), resting P

3 ctional receptors for the two North American MVV strains tested, unlike the Icelandic MVV and similar

4 EV CO, and lytic and nonlytic North American MVV strains to cells of different species.

5 tepwise multiple regression model, %DCO and %MVV combined were the best predictors of dyspnea severit

6 icate a differential receptor recognition by MVV strains which is unrelated to cytopathic phenotype.

7 ral constructs and plasmids expressing CAEV, MVV, or vesicular stomatitis virus envelope glycoprotein

8 in), Watts (45 +/- 48 versus 71 +/- 59), V E/MVV (88 +/- 33 versus 79 +/- 14), and HRmax (117 +/- 17

9 h mechanical ventilatory limitation (V Emax/ MVV 0.81 versus 0.58; p = 0.02).

10 Icelandic MVV strains, which are lytic in tissue culture, have a w

11 d the wide species distribution of Icelandic MVV receptors and the narrow host range of CAEV.

12 can MVV strains tested, unlike the Icelandic MVV and similar to CAEV, were limited to cells of rumina

13 ve improvement in dyspnea were the change in MVV, change in resting arterial PaO2, and change in FEV1

14 en demonstrated not only to carry infectious MVV but also to be hosts of the virus themselves.

15 eudotyped with the envelope glycoproteins of MVV K1514, CAEV CO, and lytic and nonlytic North America

16 functional receptors is a common property of MVV strains or related to cytopathic phenotype, we teste

17 tudies suggest that the early effects of the MVV tat peptide may involve glutamate neurotoxicity via

18 peptide derived from the basic region of the MVV transactivating protein Tat causes considerable neur

19 4 L/min), which represented 60 +/- 2% of the MVV.

20 ception of the change in Dyspneic Index [(VE/MVV)100] at maximum exercise.

21 s 12.1 [10.4-14.8] vs 9.4 [9.1-12.4]), or VE/MVV (80.4% [72.6-88.3] vs 57.8 [52.1-92.6] vs 63.9 [34.5

22 ilation/maximum ventilatory volume ratio [VE/MVV]) were measured continuously in patients with COPD d

23 VEmax/MVV was 67.2 +/- 4.0% in SLT recipients and 48.5 +/- 3.6

24 of their 12-s maximum voluntary ventilation (MVV) until task failure.

25 and increased maximum voluntary ventilation [MVV]) following the surgically induced reduction in resi

26 minant lentiviruses ovine maedi-visna virus (MVV) and caprine arthritis-encephalitis virus (CAEV) cau

27 nodeficiency virus (BIV), maedi-visna virus (MVV) and equine infectious anemia virus (EIAV) readily i

28 Lentiviruses such as Maedi Visna virus (MVV) in sheep, and human immunodeficiency virus (HIV) in

29 Maedi-visna virus (MVV) is a natural pathogen of sheep with a tropism for m

30 Maedi-visna virus (MVV) Vif is similarly promiscuous, degrading not only sh

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