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1 elopment of nosocomial infections (p < 0.05, Mann-Whitney U test).
2 .38, 4.7 letters) in the MTX arm (P = .0435, Mann-Whitney U test).
3 eplication remained asymptomatic (P < 0.001, Mann-Whitney U test).
4 nged in asthmatics than controls (p < 0.001, Mann-Whitney U test).
5 nd bilateral dural sinus stenosis [p=0.837], Mann-Whitney U test).
6 e multimodal group (5 vs. 7 days; P < 0.001, Mann-Whitney U test).
7 ian decrease 3%, range -13-16%; P = 0.029 by Mann-Whitney U test).
8 omplicated malaria (P, >0.1 for all enzymes; Mann-Whitney U test).
9 group compared with the CMV group (p <.005; Mann-Whitney U test).
10 and reduced or absent p27 levels (P = 0.02, Mann-Whitney U test).
11 o 91 copies for 31 subjects without (P=0.02, Mann-Whitney U test).
12 .1% CI difference medians 1.9-4.7, p<0.0001. Mann-Whitney U test).
13 n=4) vesus controls (8.2+/-1.3, n=5, P<0.02, Mann-Whitney U test).
14 ly longer than that in group 2 (P<0.05 using Mann-Whitney U test).
15 ptor motif, gp130 (0.833 mg ml-1) (P < 0.05, Mann-Whitney U test).
16 dopamine, respectively, p < .05 for both by Mann-Whitney U test).
17 ,256 vs. $11,234 + $12,146) costs (p < 0.01, Mann-Whitney U test).
18 nimals than in immunized animals (P = 0.014, Mann-Whitney U test).
19 istically significant difference (P < 0.001, Mann-Whitney U test).
20 between groups were significant (P = 0.001, Mann-Whitney U test).
21 minutes (SD, 18.5), respectively (P < 0.001; Mann-Whitney U test).
22 ces in activation volumes by tumor location (Mann-Whitney U test).
23 0.20 mg/L; 95% CI, 0.19-0.21 mg/L; P < .001; Mann-Whitney U test).
24 that of patients given the placebo (P = .02, Mann-Whitney U test).
25 re frequent in lesion tissue (all P < 0.005, Mann-Whitney U-test).
26 MSA subjects (0.9; 0.3-2.4 mU/l; P < 0.005, Mann-Whitney U-test).
27 ere 12.28 and 23.14, respectively (P < 0.01, Mann-Whitney U-test).
28 13] to 641 (IQR 507-694) (-31.5%, P = 0.001, Mann-Whitney U-test).
29 Statistical analysis was by ANOVA or Mann Whitney U test.
30 Differences in dose were assessed with the Mann-Whitney U test.
31 UCCA was compared between groups with the Mann-Whitney U test.
32 amyloidosis groups were compared by using a Mann-Whitney U test.
33 ing a Spearman correlation coefficient and a Mann-Whitney U test.
34 Results were compared with Student t test or Mann-Whitney U test.
35 cancer and positive control groups using the Mann-Whitney U test.
36 uated using a two-tailed Student's t-test or Mann-Whitney U test.
37 lesion sizes were compared with the Wilcoxon Mann-Whitney U test.
38 1, 2, 3, and 4 years were compared with the Mann-Whitney U test.
39 ent between cancer and healthy groups by the Mann-Whitney U test.
40 ficant differences were determined using the Mann-Whitney U test.
41 ificance was computed by using the t test or Mann-Whitney U test.
42 Data were tested statistically by Mann-Whitney U test.
43 ared using the independent samples t test or Mann-Whitney U test.
44 m factors among groups were sought using the Mann-Whitney U test.
45 ltiple logistic regression analyses, and the Mann-Whitney U test.
46 ed semiquantitatively and analyzed using the Mann-Whitney U test.
47 le nonparametric data were assessed with the Mann-Whitney U test.
48 tered treatment plans were assessed with the Mann-Whitney U test.
49 was evaluated with the independent t test or Mann-Whitney U test.
50 with that in control mice with a two-tailed Mann-Whitney U test.
51 d by chi2 test and continuous variables with Mann-Whitney U test.
52 mpared with those of normal subjects, by the Mann-Whitney U test.
53 (interquartile range) and compared using the Mann-Whitney U test.
54 neral phases of bone were compared using the Mann-Whitney U test.
55 frequency of motor seizures were done with a Mann-Whitney U test.
56 wise post hoc tests were conducted using the Mann-Whitney U test.
57 m admission to CT were compared by using the Mann-Whitney U test.
58 etween responders and nonresponders with the Mann-Whitney U test.
59 ion analysis (Pearson's coefficient) and the Mann-Whitney U test.
60 pletely responding lesions were evaluated by Mann-Whitney U test.
61 control subjects were assessed by using the Mann-Whitney U test.
62 met inhibitor after RF ablation by using the Mann-Whitney U test.
63 exact test, two-sample unpaired t test, and Mann-Whitney U test.
64 was performed with paired Student t test or Mann-Whitney U test.
65 tor expression included the median, IQR, and Mann-Whitney U test.
66 h cross tabulation, Pearson chi(2) test, and Mann-Whitney U test.
67 Relationships were assessed by using the Mann-Whitney U test.
68 tatistical difference was analyzed using the Mann-Whitney U test.
69 re assessed with the Wilcoxon signed rank or Mann-Whitney U test.
70 ps were tested using Kruskal-Wallis test and Mann-Whitney U test.
71 nerve (AC/C) strain ratio, analyzed with the Mann-Whitney U test.
72 ed using two-sample t test and nonparametric Mann-Whitney U test.
73 Data were analyzed with the Mann-Whitney U test.
74 s in the two groups were compared by using a Mann-Whitney U test.
75 ood-based markers was investigated using the Mann-Whitney U test.
76 to sets of unchanging control genes using a Mann-Whitney U-test.
77 chi(2) test and continuous variables by the Mann--Whitney U test.
78 nd carcinoma volumes were compared by use of Mann-Whitney U tests.
79 lysis was performed with the signed-rank and Mann-Whitney U tests.
80 scores in the two groups were compared with Mann-Whitney U tests.
81 analysis was performed by Kruskal-Wallis and Mann-Whitney U tests.
82 to individual questions were compared using Mann-Whitney U tests.
83 ses included: 1) Kolmogorov-Smirnov test; 2) Mann-Whitney U test; 3) Pearson chi(2) test; 4) Kruskal-
86 without an SSI, was tested using a nonpaired Mann-Whitney U test, an analysis of covariance, and a Pe
90 Between-group differences were tested by Mann-Whitney U test and correlations by Spearman's rank.
95 and nonresponders were compared by using the Mann-Whitney U test and receiver operating characteristi
102 for the 2 patient groups were compared with Mann-Whitney U tests and effect likelihood-ratio test.
103 heir predictive value was investigated using Mann-Whitney U tests and receiver-operating-characterist
104 Data were analyzed by Kruskal-Wallis and Mann-Whitney U tests and Spearman correlation analysis.
105 d MAIT cells between health and asthma using Mann-Whitney U tests and the Jonckheere-Terpstra test (l
106 p compared with increases of 66% (P = 0.004, Mann-Whitney U test) and 21% (P = 0.07) for patients who
108 ion exposure (0.06 versus 0.34 mSv; P=0.037, Mann-Whitney U test) and lower median costs ($934 versus
109 results were larger (68 vs. 34 mm2; P=0.08, Mann-Whitney U test) and were more likely to have papill
110 sub-100 bp nuclear genomic cfDNA (p 10(-5), Mann-Whitney U Test), and an increased relative abundanc
111 harm and procedural flow disruption scores (Mann-Whitney U test), and number of preventable failures
112 analyses were performed by Student's t-test, Mann Whitney U test, and Pearson product moment test.
113 ith intraclass correlation coefficients, the Mann Whitney U test, and the Wilcoxon signed rank test.
114 Data were analysed using Pearson chi(2), the Mann-Whitney U test, and binary logistic regression.
117 For statistical analysis, Student t test, Mann-Whitney U test, and Spearman's correlation coeffici
118 rank composite is typically analyzed using a Mann-Whitney U test, and the results are summarized by t
122 stical analysis comprised paired t tests and Mann-Whitney U tests, as well as Pearson r and Spearman
124 ndicated no significant difference (P >0.05, Mann-Whitney U test) between the S or F inserts in the a
125 ients (</=55 years) were compared by t-test, Mann-Whitney U test, chi-square, or Fisher's exact test.
126 cal analysis was performed with the Wilcoxon Mann-Whitney U test, chi2 test, Wilcoxon matched-pairs s
128 ts), (c) pairwise tests between tumor types (Mann-Whitney U test), (d) relationships between fast flu
129 compared by using unpaired Student t test or Mann-Whitney U test, depending on data distribution.
130 f clinical EAE (p = 0.0002 vs control by the Mann-Whitney U test) enough to completely prevent fatal
131 used to compare paired samples, such as the Mann-Whitney U test (equivalent to the Wilcoxon rank sum
136 11-514 min] vs 30 min [5-90 min]; p<0.0001, Mann-Whitney U test); for each minute delay from onset o
138 there was a significant reduction (P <0.05, Mann-Whitney U test) in the amount of contamination for
140 y markers with QODD scores were tested using Mann-Whitney U tests, Kruskal-Wallis tests, or Spearman'
141 atalase quantification.Data were analyzed by Mann-Whitney U-test, Kruskal-Wallis test and Cuzick's te
142 between groups was significant (P = 0.005 on Mann-Whitney U test; mean ranks 13.9 and 6.3 [of 21], fo
143 g for interobserver agreement, McNemar test, Mann-Whitney U test, multiple regression analysis, Spear
144 n adenocarcinoma (AC), whereas VB was lower (Mann-Whitney U test or t test, P = .003, P = .036, and P
146 ysis was carried out using Student's t-test, Mann-Whitney U test, or chi-square test (significance, p
147 were compared between both groups by t test, Mann-Whitney U test, or likelihood ratio chi-square test
150 2 +/- 35 versus 73 +/- 24 nmol L(-1) d(-1) , Mann-Whitney U-test p < 0.0001), and the South Atlantic
153 basic tasks demonstrated construct validity (Mann-Whitney U test, P < 0.05), and learning curves for
157 g infarct growth in the upper tertile range (Mann-Whitney U test, p = 0.04) but not in the middle ter
162 were analyzed using chi-square analysis and Mann-Whitney U-tests; P < 0.05 was used to define signif
169 Kruskal-Wallis ANOVA-on-ranks with post hoc Mann-Whitney U tests showed significant pairwise between
170 s of variance (ANOVA)-on-Ranks with post-hoc Mann-Whitney U-tests showed significant pairwise between
171 5, multiple regression analysis; P =.25-.75, Mann-Whitney U test; Spearman correlation coefficients b
172 categorical variables and the t test or the Mann-Whitney U test to compare continuous variables.
174 d Student's t test for continuous variables (Mann-Whitney U test used for nonnormally distributed var
183 llis test was used for significance, and the Mann-Whitney U test was used for pairwise comparison of
190 analysis of functional development, Wilcoxon-Mann-Whitney U test was used to compare the medians of 2
192 valuate the masticatory performance, and the Mann-Whitney U test was used to determine quality of lif
195 the data were not normally distributed, the Mann-Whitney U-test was employed to assess the statistic
200 npaired Student t, chi(2), Fisher exact, and Mann-Whitney U tests were applied to analyze the differe
201 is followed by Bonferroni-corrected post hoc Mann-Whitney U tests were used to analyze the data.
204 The chi-square test, Student's t-test, and Mann-Whitney U-test were used for statistical analysis.
205 he gene sets is performed by an extension of Mann-Whitney U test which is based on weighted rank sums
206 test, Wilcoxon's matched pairs test, and the Mann Whitney U Test with P < 0.05 considered significant
208 versus 23 +/- 1.4 in controls; P < 0.0001 by Mann-Whitney U test), with virtually no overlap between
210 iability and differentiated VCD vs. healthy (Mann-Whitney U-test: z = -5.390, P < 0.001) and asthma (
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