1 hat include chorea-acanthocytosis (ChAc) and McLeod syndrome (MLS).

2 nical features with Huntington's disease and McLeod syndrome.

3 ive membrane transport protein implicated in McLeod Syndrome, a form of hereditary neuroacanthocytosi

4 eably reduced in ChAc patient cells, but not McLeod syndrome and Huntington's disease cells.

5 MA1 failed to bind trypsin-treated Kx(null) (McLeod) erythrocytes, which lack the Kx protein.

6 e deletion breakpoints of two novel cases of McLeod phenotype with extensive deletions are reported.

7                                          The McLeod phenotype is derived from various forms of XK gen

8 ophy (DMD), mild mental retardation, and the McLeod phenotype.

9 set neuromuscular abnormalities known as the McLeod syndrome (MLS).

10 and neuromuscular abnormalities known as the McLeod syndrome.

11 10 times and whose absence, as occurs in the McLeod phenotype, is associated with a set of clinical s

12 but absence of the protein, as occurs in the McLeod phenotype, is associated with red cell acanthocyt

13                         Individuals with the McLeod phenotype usually develop late-onset neuromuscula

14 mes, is lacking in rare individuals with the McLeod syndrome.