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1 mesoderm (tongue) and cranial neural crest (Meckel's cartilage).
2 elements surrounding the rostral process of Meckel's cartilage.
3 hypoplastic mandibular process and truncated Meckel's cartilage.
4 ecreased differentiation of chondroblasts in Meckel's cartilage.
5 re connected to the mandible via an ossified Meckel's cartilage.
6 peptides partially rescued abnormalities in Meckel's cartilage.
7 ression in the epithelium and bifurcation of Meckel's cartilage.
8 sihyobranchial, palatoquadrate, and possibly Meckel's cartilages.
9 signaling is required for the elongation of Meckel's cartilage and FGFR2 and FGFR3 have roles during
10 e strongly expressed in the perichondrium of Meckel's cartilage and mesenchymal areas of embryonic da
13 cifically required for cell proliferation in Meckel's cartilage and the mandibular anlagen and for th
15 between the palatoquadrate and proximal part Meckel's cartilage (articular) has recently been shown t
16 er-dental mesenchyme and distal symphysis of Meckel's cartilage became hypoplastic, resulting in the
17 at HH26 did not affect the morphogenesis of Meckel's cartilage but resulted in truncations of the ma
21 ntum, periodontal ligaments, chondrocytes in Meckel's cartilage, mandible, the articulating disc of t
22 TGF rescues the cell proliferation defect in Meckel's cartilage of Tgfbr2(fl/fl);Wnt1-Cre mutants, de
23 t has molariform tooth replacement, ossified Meckel's cartilage of the middle ear, and specialized xe
24 dentary bone but still connect the ossified Meckel's cartilage (OMC); we hypothesize that the OMC se
25 as growth in the brain, limb buds, nose and Meckel's cartilage, remodeling of the intestine, and dea
27 abnormalities including the complete loss of Meckel's cartilage, the ceratohyal, and all of the cerat
28 , palatal clefting results from a failure of Meckel's cartilage to extend the mandible and thereby al
33 the maxillae into mandibles with duplicated Meckel's cartilage, which resulted in an absence of the
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