ライフサイエンスコーパス: Mendelian ratio

1 bearing progeny rather than the expected 1:1 Mendelian ratio.

2 s, and male Pgam5(-/-) mice were born at sub-Mendelian ratio.

3 ding of the mutant mice yielded the expected mendelian ratio.

4 type, heterozygous, and homozygous mice in a Mendelian ratio.

5 ly normal homozygous mutant mice at a normal Mendelian ratio.

6 ll mice were viable and born at the expected Mendelian ratio.

7 fl);Nr5a1-Cre(/+) mice were born at a normal Mendelian ratio.

8 c loci, with inheritance conforming to a 3:1 Mendelian ratio.

9 disease phenotype deviated from the expected Mendelian ratio.

10 kout (mVps34(pdKO)) mice, which were born at Mendelian ratios.

11 expressing Y265C pol beta are born at normal Mendelian ratios.

12 ull for Eset were recovered but in less than Mendelian ratios.

13 mice are viable but not born in the expected Mendelian ratios.

14 s a semidominant transgene and segregated in Mendelian ratios.

15 (-) mouse crosses, were born at the expected Mendelian ratio (26.5%; n = 1,080 pups), indicating no e

16 ing of heterozygous mutants yielded a normal Mendelian ratio among embryos on gestation day 9.5; howe

17 IKfyve(WT/KO) mice were born at the expected Mendelian ratio and developed into adulthood.

18 onal knockout mice were born at the expected Mendelian ratio and developed normally to adulthood, ind

19 ozygous knock-in animals were born in normal Mendelian ratio and developed normally to adulthood.

20 mafF null mutant mice were born in a normal Mendelian ratio and displayed no obvious functional defi

21 CCR2-/- mice were born at the expected Mendelian ratios and developed normally.

22 Parc knockout mice were born at the expected Mendelian ratios and exhibited no apparent phenotype.

23 sphatase-negative mice were born in expected Mendelian ratios and exhibited normal growth and develop

24 Global tbeta4-knockout mice were born at mendelian ratios and exhibited normal heart and blood ve

25 Nlrp2-deficient mice were born with expected Mendelian ratios and that Nlrp2 was dispensable for inna

26 e is well under that predicted by the normal Mendelian ratio, and TR4(-/-) mice demonstrate high rate

27 The Mcoln1(-/-) mice are born in Mendelian ratios, and both male and female Mcoln1(-/-) m

28 ADAM33-null mice were born at expected Mendelian ratios, and both male and females developed no

29 nd colonic enterocytes were born in expected Mendelian ratios, and RelA-null epithelia differentiated

30 The lal-/lal- mice are born in Mendelian ratios, are normal appearing at birth, and fol

31 s Nrp1(Y297A/Y297A) mice were born at normal Mendelian ratios, arguing against NRP1 functioning exclu

32 pups were represented at the expected normal Mendelian ratios at 1 to 3 days of age but at only 10% o

33 Klf5 mutant mice were born at a normal Mendelian ratio but had high mortality compared with con

34 rdial Dicer mutant mice are born in expected Mendelian ratios but die immediately after birth with pr

35 These mice are born at the expected Mendelian ratio, but die prematurely from spontaneous ca

36 zygous mutant mice were born at the expected Mendelian ratio, but their growth was impaired.

37 f4(loxP/loxP) mice were born at the expected Mendelian ratio, but they gradually died after birth.

38 These mice are born at normal Mendelian ratios, but display a progressive, systemic hy

39 ed knockouts develop normally, being born in Mendelian ratios, but fail to thrive within 2 weeks, dis

40 xpress KrasG12D in utero, are born at normal Mendelian ratios, develop hepatosplenomegaly, anemia, an

41 f the mutated gene were born in the expected Mendelian ratio, developed normally, and showed no appar

42 FLKO mice, while born at a normal Mendelian ratio, developed severe anemia and exhibited p

43 unable to interpret his own data that showed Mendelian ratios, even though he shared with Mendel a mo

44 VENIRKO mice were born at the expected Mendelian ratio, grew normally, were fertile, and exhibi

45 omozygous cardiac betaStop mice were born at Mendelian ratio, had a normal life expectancy, and norma

46 Btbd12-deficient animals are born at sub-Mendelian ratios, have greatly reduced fertility, are de

47 eficient mice are grossly normal and born in Mendelian ratios; however, deficiency of Mmp1a results i

48 onditional mutants were born at the expected Mendelian ratios; however, these died shortly after birt

49 gene was found to be transmitted at a normal Mendelian ratio in mice, in striking contrast to the pro

50 PAK4 knock-out (KO) mice were born at Mendelian ratios in both genders.

51 TAFI mice produced offspring in the expected Mendelian ratio, indicating that transmission of the mut

52 c25B and Cdc25C are obtained at the expected Mendelian ratios, indicating that Cdc25B and Cdc25C are

53 minant involved in the generation of the non-Mendelian ratio is epigenetic.

54 In mice lacking Msy2, a normal Mendelian ratio is observed after matings between hetero

55 Although born in a normal mendelian ratio, no PGT(-/-) mice survived past postnata

56 (a/a) to A(vy)/a offspring from the expected Mendelian ratio of 1:1.

57 duced normal-sized and shrunken seeds in the Mendelian ratio of 3:1, and the shrunken seeds could not

58 The unexpected Mendelian ratio of 4.4% cKO mice was confirmed by genoty

59 his background, we also obtained less than a Mendelian ratio of null offspring suggesting development

60 ned as a significant departure from expected Mendelian ratios of inheritance of an allele or chromoso

61 lta4A/tmDelta4A) offspring with the expected Mendelian ratios of inheritance, and these mice expresse

62 In contrast, we observed appropriate Mendelian ratios of RA+ mAgt+/+, RA+ mAgt+/-, and RA+ mA

63 Despite appropriate Mendelian ratios of RA- mice that were wildtype (+/+), h

64 heterozygous matings produced mutant mice at Mendelian ratios that had normal viability and fertility

65 Arl3 (-/-) mice were born at a sub-Mendelian ratio, were small and sickly, and had markedly

66 breviated Sca(+/AE)) mutant mice are born in Mendelian ratios with no apparent abnormalities in growt

67 homozygous mutants were born at the expected Mendelian ratio without apparent developmental abnormali

68 us mutant mice (Brca1(FL/FL)) were born at a Mendelian ratio without obvious developmental defects.

69 Appl1-null mice were born at expected Mendelian ratios, without obvious phenotypic abnormaliti