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1 cal specimens from patients with intractable Meniere's disease.
2 an vestibular macula utricle from normal and Meniere's disease.
3 ffort to identify genetic causes of familial Meniere's disease.
4 ated vertigo), bilateral vestibulopathy, and Meniere's disease.
5 s hearing involvement may be misdiagnosed as Meniere's disease.
6 gy as well as the diagnosis and treatment of Meniere's disease.
7 he most debilitating symptom associated with Meniere's disease.
8 sac may be implicated in the pathogenesis of Meniere's disease.
9 will help define the role of vasopressin in Meniere's disease.
10 ablative, effective treatment for refractory Meniere's disease.
12 ntly no cure, more than 85% of patients with Meniere's disease are helped by either changes in lifest
20 cimens of three different types of patients (Meniere's disease, normal controls, and other otopatholo
21 micin, the standard treatment for refractory Meniere's disease, reduces vertigo, but damages vestibul
23 uch as sudden sensorineural hearing loss and Meniere's disease that are elicited by pathogens and pro
24 aged 18-70 years with refractory unilateral Meniere's disease were enrolled at Charing Cross Hospita
25 2009, and April 15, 2013, 256 patients with Meniere's disease were screened, 60 of whom were enrolle
26 th bilateral IMCVDs or symptoms of bilateral Meniere's disease were treated with etanercept (25 mg tw
27 e GLAST immunoreactive area of patients with Meniere's disease when compared to that of patients with
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