1 and phased haplotypes in people of Amish and Mennonite ancestry.

2 P, and I268T, the last found in a patient of Mennonite ancestry.

3                                     Affected Mennonite and Dutch patients were homozygous and doubly

4 ypical early-onset torsion dystonia of Swiss-Mennonite and non-Jewish Russian origin, respectively, t

5 a group of seven distantly related Old Order Mennonite children.

6 mutation is estimated to be present on 3% of Mennonite chromosomes and the disease affects 0.1% of th

7 of mental illnesses in traditional Amish and Mennonite communities--known collectively as the Plain p

8  prominent cranial-cervical ITD in two large Mennonite families to chromosome 8.

9  a mutation in the THAP1 gene in three Amish-Mennonite families with mixed-onset primary torsion dyst

10 ia; a founder mutation was detected in Amish-Mennonite families, and a different mutation was identif

11       We identified 11 members of a Canadian Mennonite family suffering from adult onset, slowly prog

12 ducted a genome-wide association study in 43 Mennonite family trios using 2,083 microsatellites and s

13 molecular studies have been conducted on the Mennonite family with X-linked mental retardation (XLMR)

14              The patient came from an inbred Mennonite family, and though the mutation identified her

15                     One family had the Amish-Mennonite founder mutation, whereas the other eight fami

16 unds (Amish-Swiss from United States, n = 8; Mennonite-German from Canada, n = 1; mixed European from

17                                              Mennonite GSD6 was linked to the PYGL locus with a multi

18                                We analyzed a Mennonite kindred with an autosomal recessive form of GS

19                   We studied a 13-generation Mennonite pedigree with autosomal recessive myopathy and