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1 nonpathogenic bacteria (Escherichia coli and Micrococcus luteus).
2 d yeast and interacts with peptidoglycan and Micrococcus luteus.
3 , H2B, and H4, for growth inhibition against Micrococcus luteus.
4 a means to distinguish Escherichia coli from Micrococcus luteus.
5 ium tuberculosis, Neisseria gonorrhoeae, and Micrococcus luteus.
6 d prophenoloxidase activation in response to Micrococcus luteus.
7 y from hemolymph activated by treatment with Micrococcus luteus.
8  the resuscitation-promoting factor (Rpf) of Micrococcus luteus.
9 enic and highly lysozyme-sensitive bacterium Micrococcus luteus.
10 s whose predicted products resemble Rpf from Micrococcus luteus.
11 the most common erroneous identification was Micrococcus luteus.
12  studies using gram-positive model bacterium Micrococcus luteus.
13   Four genomic DNAs of differing GC content (Micrococcus luteus, 72% GC; Escherichia coli, 50% GC; ca
14                                              Micrococcus luteus , a Gram-positive bacterium, is incap
15    Transcription termination factor Rho from Micrococcus luteus, a high G + C Gram-positive bacterium
16                                The growth of Micrococcus luteus, a soil microorganism that belongs to
17 al activity of dendrigraft poly-L-lysines on Micrococcus luteus and Erwinia carotovora.
18       After the intraperitoneal injection of Micrococcus luteus and Escherichia coli, the peptide was
19 opin A gene was induced by the G(+) bacteria Micrococcus luteus and Staphylococcus aureus, but not by
20 Escherichia coli, Staphylococcus aureus, and Micrococcus luteus and the yeast, Candida albicans.
21 , Escherichia coli, Lactobacillus plantarum, Micrococcus luteus, and Staphylococcus aureus support th
22 as purified from the Gram-positive bacterium Micrococcus luteus, and the complete gene sequence was d
23 ssessed by qualitative agar plate test using Micrococcus luteus as substrate showing that both the un
24 y against Bacillus subtilis (but not against Micrococcus luteus), as well as against the parental and
25 te inhibited the growth of Escherichia coli, Micrococcus luteus, Bacillus subtilis, and Klebsiella pn
26                  Staphylococcus epidermidis, Micrococcus luteus, Brevibacterium linens, Pseudomonas f
27 exhibit biphasic kinetics in the clearing of Micrococcus luteus cell suspensions, suggesting preferen
28  the biosynthesis of the teichuronic acid of Micrococcus luteus cell walls.
29  the D52A and D52A/N46A ChEWL complexes with Micrococcus luteus cells are 3- and 4-fold higher, respe
30 nging from 45% identity for the homolog from Micrococcus luteus (FtsZ[Ml]) to 91% identity for the ho
31                          The best ligand was Micrococcus luteus lipomannan, followed by Enterococcus
32 lf thymus, salmon testes, and the bacterium, micrococcus luteus (lysodeikticus) containing different
33                                              Micrococcus luteus (NCTC2665, "Fleming strain") has one
34                                              Micrococcus luteus secretes a small protein called Rpf,
35                         In contrast, against Micrococcus luteus the TA(-) mutant exhibited no defect
36                                     Rpf from Micrococcus luteus, the founder member of this protein f
37                 In contrast to Uvr(A)BC, the Micrococcus luteus UV endonuclease efficiently incises u
38 uence similarity to the Escherichia coli and Micrococcus luteus UvrA proteins involved in excision re
39 of resuscitation-promoting factor (Rpf) from Micrococcus luteus, which is an extremely potent anti-do

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