ライフサイエンスコーパス: Mirabilis

1 condition in longjaw mudsuckers (Gillichthys mirabilis).

2 ted from the marine sponge Siliquariaspongia mirabilis.

3 the 3' end of the mrp fimbrial operon in P. mirabilis.

4 ntly reduced in an isogenic pta mutant of P. mirabilis.

5 ra from mice experimentally infected with P. mirabilis.

6 but were detected in every population of A. mirabilis.

7 an immunogenic outer membrane protein in P. mirabilis.

8 putrescine-deficient speA mutant of Proteus mirabilis.

9 ebsiella spp., Escherichia coli, and Proteus mirabilis.

10 sertion was mapped to a speA homologue in P. mirabilis.

11 nt virulence factor of uropathogenic Proteus mirabilis.

12 o prevent urinary tract infection by Proteus mirabilis.

13 erant burrow-dwelling goby fish, Gillichthys mirabilis.

14 theterized mice to infection by wild-type P. mirabilis.

15 ods for identifying individual strains of P. mirabilis.

16 so contributes to the uropathogenicity of P. mirabilis.

17 's-eye colonies typically associated with P. mirabilis.

18 ike (MR/P) fimbriae of uropathogenic Proteus mirabilis.

19 pe on an otherwise nonhemolytic mutant of P. mirabilis.

20 nfluenzae, Bacteroides fragilis, and Proteus mirabilis.

21 he isolates were E. coli and one was Proteus mirabilis.

22 colony often associated with cultures of P. mirabilis.

23 the formation of crystalline biofilms by P. mirabilis.

24 arboxylase that inhibits swarming in Proteus mirabilis.

25 putrescine importer, was characterized in P. mirabilis.

26 arming in the urinary tract pathogen Proteus mirabilis.

27 details of self-nonself discrimination in P. mirabilis.

28 nce of siderophore production in vivo for P. mirabilis.

29 iation of Klebsiella pneumoniae from Proteus mirabilis 16S rRNA target sequences differing by a singl

30 mmon species (68.6%), followed by Ochroconis mirabilis (21.5%).

31 Stenotrophomonas maltophilia (4.3%), Proteus mirabilis (4.0%), Klebsiella oxytoca (2.7%), and Citroba

32 Here we characterize PmDsbA from Proteus mirabilis, a bacterial pathogen increasingly associated

33 Proteus mirabilis, a cause of complicated urinary tract infectio

34 Proteus mirabilis, a cause of complicated urinary tract infectio

35 y, we identified a genomic island of Proteus mirabilis, a common agent of catheter-associated urinary

36 Proteus mirabilis, a common cause of nosocomial and catheter-ass

37 Proteus mirabilis, a common cause of urinary tract infections (U

38 Proteus mirabilis, a gram-negative bacterium associated with com

39 Proteus mirabilis, a gram-negative bacterium, is a frequent caus

40 Proteus mirabilis, a Gram-negative bacterium, represents a commo

41 Proteus mirabilis, a leading cause of catheter-associated urinar

42 Lautropia mirabilis, a pleomorphic, motile, gram-negative coccus,

43 solute tested, but the catalytic rate of G. mirabilis A4-LDH was decreased most by solutes and incre

44 To identify interactions among P. mirabilis accessory proteins, in vitro immunoprecipitati

45 Proteus mirabilis alternates between motile and adherent forms.

46 Proteus mirabilis, an etiologic agent of complicated urinary tra

47 wn previously using hyperflagellated Proteus mirabilis and a motile but non-swarming flgN transposon

48 dder colonization factor of uropathogenic P. mirabilis and also suggested that the ability to switch

49 ant, especially against the bacteria Proteus mirabilis and Antibiotic resistant Escherichia coli.

50 -negative bacterial cells, including Proteus mirabilis and Caulobacter crescentus, initiates asymmetr

51 metabolism as an adaptation strategy for P. mirabilis and contributes to better understand the ecolo

52 tedly and unlike other fliL mutants, both P. mirabilis and E. coli DeltafliL cells swarm (Swr(+)).

53 We focused on Lrp from Proteus mirabilis and E. coli, orthologs with 98% identity overa

54 The closely related Proteus mirabilis and Enterobacterlaceae plasmid-encoded urease

55 ed and characterized DeltafliL mutants of P. mirabilis and Escherichia coli.

56 flagellar operon, in vegetative cells of P. mirabilis and found that increased flagellum density pro

57 We sampled several populations of A. mirabilis and its photosynthetic sister groups to correl

58 and antigenicity of an autotransporter in P. mirabilis and its use in vaccine development.

59 ase-negative mutant of uropathogenic Proteus mirabilis and its wild-type parent strain was assessed b

60 UTI caused by the major uropathogens Proteus mirabilis and Klebsiella pneumoniae, in addition to UPEC

61 s a major assimilatory checkpoint in Proteus mirabilis and other Gram-negative bacteria and expand th

62 al catheter colonization, particularly by P. mirabilis and other urease-positive bacteria.

63 We conclude that P. mirabilis and P. stuartii coinfection promotes urolithia

64 iously demonstrated that coinfection with P. mirabilis and P. stuartii increased overall urease activ

65 The urease-positive species Proteus mirabilis and Providencia stuartii are two of the leadin

66 y urease-positive organisms, such as Proteus mirabilis and Providencia stuartii, commonly occurs with

67 moter sequences of Escherichia coli, Proteus mirabilis and Salmonella typhimurium allowed determinati

68 a coli, Pseudomonas aeruginosa PAO1, Proteus mirabilis and Serratia marcescens, possibly by interferi

69 e from other human pathogens such as Proteus mirabilis and Staphylococcus aureus that outcompete P. a

70 , the AT also promoted autoaggregation of P. mirabilis and this function was independent of its prote

71 etine and thioridazine) to act as EPIs in P. mirabilis, and control crystalline biofilm formation.

72 ies, lrp genes from Vibrio cholerae, Proteus mirabilis, and E. coli were introduced into the same E.

73 of wild-type Salmonella typhimurium, Proteus mirabilis, and Escherichia coli.

74 tine and thioridazine inhibited efflux in P. mirabilis, and molecular modelling predicted both drugs

75 nella muenchen, Serratia marcescens, Proteus mirabilis, and Proteus vulgaris).

76 siella pneumoniae, Escherichia coli, Proteus mirabilis, and Salmonella enterica serovar Typhimurium.

77 f Escherichia coli, Klebsiella spp., Proteus mirabilis, and Salmonella spp. and are associated with m

78 Thus, P. mirabilis appears to use a related mechanism to inhibit

79 ary tract infections (UTI) caused by Proteus mirabilis are associated with severe pathology in the bl

80 Fimbriae of the human uropathogen Proteus mirabilis are the only characterized surface proteins th

81 ften invasive, and, with the exception of P. mirabilis, are multiclonal.

82 Using Proteus mirabilis as a model, we investigate the role of choline

83 aracterizing trimeric autotransporters in P. mirabilis as afimbrial surface adhesins and autoagglutin

84 pplication of the mini-Tn7 system in Proteus mirabilis as an example of a bacterium with a secondary

85 These results support the hypothesis that P. mirabilis ascertains its location in the environment or

86 Proteus mirabilis, associated with complicated urinary tract inf

87 Ten strains of Lautropia mirabilis (ATCC 51599(T) and nine phenotypically similar

88 One of the six predicted Proteus mirabilis autotransporters (ATs), ORF c2341, is predicte

89 Fluorescent E. coli and P. mirabilis bacteria were observed by fluorescence microsc

90 P. mirabilis bacteriuria may lead to acute pyelonephritis,

91 ed from the 50 %-identical TEM-1 and Proteus mirabilis beta-lactamases.

92 encrusted and blocked by crystalline Proteus mirabilis biofilms.

93 sed internalization of S. typhimurium and P. mirabilis by both HT-29 and Caco-2 enterocytes and with

94 site-specific transposition pathway into P. mirabilis by transformation, followed by selection of in

95 The crystalline biofilms of P. mirabilis can cause serious complications for patients u

96 tients revealed that a single genotype of P. mirabilis can persist in the urinary tract despite many

97 Proteus mirabilis can rapidly utilize choline to enhance growth

98 first report of carbapenem resistance in P. mirabilis caused by the acquisition of bla(KPC).

99 Recent work has elucidated how P. mirabilis causes all of these disease states.

100 Proteus mirabilis causes complicated urinary tract infections (U

101 In this study, we show that P. mirabilis CaUTI isolates initiate swarming in response t

102 clear how energetically costly changes in P. mirabilis cell morphology translate into an advantage fo

103 ntal behaviors of motile, rod-shaped Proteus mirabilis cells (3 mum in length) adsorbed to the LC int

104 The number of P. mirabilis cells adhering to bladder tissue did not appea

105 In liquid, P. mirabilis cells are 1.5- to 2.0-mum swimmer cells with 4

106 In the lab, P. mirabilis cells become long and multinucleate and increa

107 The majority of wild-type P. mirabilis cells in transurethrally infected mice produce

108 that MR/P fimbriae are expressed by most P. mirabilis cells infecting the urinary tract, dictate the

109 ility to sense a surface: e.g., DeltafliL P. mirabilis cells swarm precociously over surfaces with lo

110 found in cell-free supernatants from Proteus mirabilis, Citrobacter freundii and Enterobacter agglome

111 A total of 63 clinical isolates of Proteus mirabilis collected over a 19-month period were typed by

112 creening for the presence of ICEPm1 among P. mirabilis colonizing isolates showed that ICEPm1 is more

113 On agar, a P. mirabilis colony grows outward in a bull's-eye pattern f

114 Proteus mirabilis commonly infects the complicated urinary tract

115 Proteus mirabilis compromises the care of many patients undergoi

116 found to significantly reduce the rate of P. mirabilis crystalline biofilm formation on catheters, an

117 dentified as important for development of P. mirabilis crystalline biofilms, highlighting the potenti

118 ibitors in the treatment or prevention of P. mirabilis crystalline biofilms.

119 Further analysis revealed that P. mirabilis DeltafliL cells also exhibit an alteration in

120 The HIV-infected children with L. mirabilis did not differ from the HIV-infected children

121 ia availability due to urease activity in P. mirabilis did not drive this gene expression.

122 We conclude that prior infection with P. mirabilis does not protect significantly against homolog

123 fimbriae, a surface antigen expressed by P. mirabilis during experimental urinary tract infection, v

124 Examination of a set of 55 isolates of P. mirabilis, each from a different clinical or environment

125 , is a functional homolog of MrpJ of Proteus mirabilis; ectopic expression of papX in P. mirabilis re

126 When P. mirabilis encounters a highly viscous environment, e.g.,

127 When P. mirabilis encounters a solid surface, where flagellar ro

128 Overall, P. mirabilis exhibits an extraordinary lifestyle, and furth

129 ns (RIPs) were found in the storage roots of Mirabilis expansa, an underutilized Andean root crop.

130 mmunoreactivity with antibodies of RIPs from Mirabilis expansa.

131 results establish a relationship between P. mirabilis flagellum density and cell motility in viscous

132 d into the culture medium by the original P. mirabilis flgN mutant demonstrated that export of FlgK a

133 and motility, a result also observed when P. mirabilis fliL+ was expressed in Escherichia coli.

134 Proteus mirabilis forms extensive crystalline biofilms on indwel

135 Proteus mirabilis forms extensive crystalline biofilms on urethr

136 en of the bizarrely specialised Malleodectes mirabilis from middle Miocene deposits in the Riversleig

137 s study, microarrays were used to analyze P. mirabilis gene expression in vivo from experimentally in

138 In this study, we describe wosA, a Proteus mirabilis gene identified by its ability to increase swa

139 In a search for Proteus mirabilis genes that were regulated by cell-to-cell sign

140 to be localized to a 5.4-kb locus on the P. mirabilis genome encoding RsbA (regulator of swarming be

141 Sequencing of the P. mirabilis genome revealed 14 additional paralogues of mr

142 idence for ancient polyploidy in Welwitschia mirabilis (Gnetales) and no evidence for polyploidy in p

143 institution, E. coli, K. pneumoniae, and P. mirabilis harbor plasmid-mediated AmpC enzymes.

144 tility by the urinary tract pathogen Proteus mirabilis has been a long-studied but little understood

145 ed significantly more biofilm than either P. mirabilis HI4320 (P = 0.03) or MR/P OFF (P = 0.05).

146 ract, a ureR mutation was introduced into P. mirabilis HI4320 by homologous recombination.

147 P. mirabilis HI4320 carrying the UreD-GFP fusion plasmid wa

148 Microarray analysis of P. mirabilis HI4320 cultured under iron limitation identifi

149 After 7 days, however, P. mirabilis HI4320 formed a 65-mum-thick biofilm, while th

150 In addition, the P. mirabilis HI4320 genome possesses four tandem copies of

151 Immunogold electron microscopy of P. mirabilis HI4320 revealed that MrpH was located at the t

152 o evaluate the colonization of mutants of P. mirabilis HI4320 that were generated by signature-tagged

153 we report the complete genome sequence of P. mirabilis HI4320, a representative strain cultured in ou

154 Although in P. mirabilis HI4320, ICEPm1 is annotated as integrated into

155 th the newly completed genome sequence of P. mirabilis HI4320, was used to identify surface-exposed a

156 rea in both Escherichia coli DH5alpha and P. mirabilis HI4320.

157 I null mutants from a clinical isolate of P. mirabilis, HI4320.

158 A recombinant plasmid containing cloned P. mirabilis hns was able to complement and restore repress

159 (regulator of swarming behavior) and the P. mirabilis homologs to RcsB and RcsC.

160 ese data underscore the importance of the P. mirabilis IgA-degrading metalloprotease in UTI.

161 d TaaP individually offered advantages to P. mirabilis in a murine model.

162 stal structure of a Crl homolog from Proteus mirabilis in conjunction with in vivo and in vitro appro

163 er from the HIV-infected children without L. mirabilis in immunological status, clinical status, or s

164 ated and important for the persistence of P. mirabilis in the host, it was selected as a vaccine cand

165 ncomitant factors in the establishment of L. mirabilis in the oral cavity remains to be elucidated.

166 Modeling CaiT from P. mirabilis in the outward-open and closed states on the c

167 n of urease by urea, and for virulence of P. mirabilis in the urinary tract.

168 ontributes to the pathogenic potential of P. mirabilis in the urinary tract.

169 y be a significant virulence component of P. mirabilis in urinary tract infections.

170 ght into crystalline biofilm formation by P. mirabilis, including the link between biofilm formation

171 In wild-type P. mirabilis, increased expression of the flhDC operon occu

172 tant Proteus-like (MR/P) fimbriae of Proteus mirabilis, indicate that MrpB functions as the terminato

173 eae and in particular the pathobiont Proteus mirabilis, induced robust IL-1beta release that was comp

174 al. (2015) show that the pathobiont Proteus mirabilis induces NLRP3 inflammasome-dependent interleuk

175 deposits in bladder and kidney tissues of P. mirabilis-infected mice.

176 ides a clear visual early warning of Proteus mirabilis infection and subsequent blockage.

177 ey metabolic pathways as requirements for P. mirabilis infection of the urinary tract.

178 of urease-mediated urea hydrolysis during P. mirabilis infection.

179 d the epidemiology of catheter-associated P. mirabilis infections by use of pulsed-field gel electrop

180 The enteric bacterium Proteus mirabilis is a common cause of complicated urinary tract

181 Proteus mirabilis is a common cause of urinary tract infection (

182 Proteus mirabilis is a common uropathogen in patients with long-

183 Proteus mirabilis is a dimorphic motile bacterium well known for

184 Proteus mirabilis is a dimorphic, motile bacterium often associa

185 The gram-negative enteric bacterium Proteus mirabilis is a frequent cause of urinary tract infection

186 Proteus mirabilis is a Gram-negative bacterium that exists as a

187 Proteus mirabilis is a Gram-negative bacterium that undergoes a

188 Proteus mirabilis is a model organism for urease-producing uropa

189 Proteus mirabilis is a urinary tract pathogen and well known for

190 Proteus mirabilis is a urinary tract pathogen that differentiate

191 Proteus mirabilis is an opportunistic pathogen that is frequentl

192 The enteric bacterium Proteus mirabilis is associated with a significant number of cat

193 The bacterium Proteus mirabilis is capable of movement on solid surfaces by a

194 P. mirabilis is capable of swarming, a form of multicellula

195 Swarming by Proteus mirabilis is characterized by cycles of rapid and coordi

196 ZapA, of the urinary tract pathogen Proteus mirabilis is co-ordinately expressed along with other pr

197 eding and biochemical analysis shows that P. mirabilis is unable to utilize or produce yersiniabactin

198 solates, 2 S. marcescens isolates, 1 Proteus mirabilis isolate, and 2 A. baumannii isolates) producin

199 Swarming colonies of independent Proteus mirabilis isolates recognize each other as foreign and d

200 lebsiella sp. isolates, and 7 (100%) Proteus mirabilis isolates tested were CTX-M positive, with 15%

201 f 683 E. coli, 371 K. pneumoniae, and 232 P. mirabilis isolates tested, 13 (1.9%), 28 (7.6%), and 4 (

202 One of seven positive P. mirabilis isolates was in group II, with the remainder b

203 e CTX-M-positive isolates showed that six P. mirabilis isolates were clonal and that there were seven

204 Five of seven P. mirabilis isolates were from blood cultures.

205 pneumoniae, Klebsiella oxytoca, and Proteus mirabilis isolates, including phenotypically ESBL-positi

206 tion, is useful for the identification of L. mirabilis isolates.

207 initial epidemiologic characterization of P. mirabilis isolates.

208 V-uninfected controls; the association of L. mirabilis isolation with HIV infection is significant (P

209 ME2 showed high similarity to the Mirabilis jalapa antiviral protein, a type I RIP.

210 red beet (Beta vulgaris) and four o'clocks (Mirabilis jalapa) to identify a novel, betalain-related

211 net exchange rate of a plant-soil ecosystem (Mirabilis jalapa).

212 bilis, which is responsible for infection of Mirabilis jalapa, are adapted to protease targets unique

213 of similarity to sequences encoding Proteus mirabilis mannose-resistant fimbriae.

214 ragment was isolated from a genomic clone of mirabilis mosaic virus (MMV), a double-stranded DNA plan

215 While there are similarities between P. mirabilis MR/P and E. coli P fimbriae, there are more no

216 ted zinc present in the urinary tract and P. mirabilis must scavenge this ion to colonize and persist

217 gration in a non-swarming but motile Proteus mirabilis mutant lacking the FIgN facilitator of flagell

218 4), Pseudomonas aeruginosa (n = 14), Proteus mirabilis (n = 3), Serratia spp. (n = 10), Stenotrophomo

219 infection (mean log(10) number of CFU of P. mirabilis Nal(r) HI4320 per milliliter or gram in vaccin

220 s parainfluenzae, Kingella oralis, Lautropia mirabilis, Neisseria elongata, Rothia dentocariosa, Stre

221 Mice were infected with P. mirabilis or a urease mutant, P. stuartii, or a combinat

222 In particular, for both the E. coli and P. mirabilis orthologs, Lrp responsiveness to methionine wa

223 ection against urinary tract infection by P. mirabilis (P < 0.002).

224 from ascending urinary tract infection by P. mirabilis (P < 0.05).

225 al related Phytophthora species including P. mirabilis, P. ipomoeae, and possibly P. phaseoli.

226 Transurethral inoculation of P. mirabilis(pBAC001) resulted in ascending urinary tract i

227 P. mirabilis(pBAC001), which expresses green fluorescent pr

228 olecular analyses have revealed that Proteus mirabilis possesses two genes, flaA and flaB, that are h

229 e consolidation phase is a state in which P. mirabilis prepares for the next wave of swarming.

230 Furthermore, colonization with P. mirabilis promoted intestinal inflammation upon intestin

231 designated ICEPm1, that is common to Proteus mirabilis, Providencia stuartii, and Morganella morganii

232 This chromosome integration system in P. mirabilis provides an important tool for animal and biof

233 ncluded probes for Escherichia coli, Proteus mirabilis, Pseudomonas aeruginosa, Enterocococcus spp.,

234 ative bacteria are Escherichia coli, Proteus mirabilis, Pseudomonas aeruginosa, Klebsiella pneumonia.

235 An isolate of Proteus mirabilis recovered from blood cultures of a diabetic pa

236 mirabilis; ectopic expression of papX in P. mirabilis reduces motility.

237 E. coli and P. mirabilis remained susceptible to most of the drugs test

238 hia coli, Klebsiella pneumoniae, and Proteus mirabilis remains unknown.

239 One population of A. mirabilis revealed a unique deletion of 541 bp in the ps

240 es of CaiT from Escherichia coli and Proteus mirabilis revealed an inverted five-transmembrane-helix

241 e sequence of the parasitic liverwort Aneura mirabilis revealed the loss of five chlororespiration (n

242 How P. mirabilis senses a surface is not fully understood; howe

243 d autoagglutination, and a taaP mutant of P. mirabilis showed significantly (P < 0.05) more reduced a

244 a Foley catheterised bladder infected with P mirabilis showed that when retention balloons were infla

245 In a P. mirabilis speA mutant with greatly reduced levels of int

246 colonization by common uropathogens Proteus mirabilis, Staphylococcus aureus and Escherichia coli fo

247 The isogenic mrpJ null mutant of wild-type P.mirabilis strain HI4320 is attenuated in the murine mode

248 rm between swarming colonies of different P. mirabilis strains but not between colonies of a single s

249 e prevalent in urine isolates compared to P. mirabilis strains isolated from other body sites (P<0.00

250 Inactivation of aipA in P. mirabilis strains significantly (P < 0.01) reduced the m

251 o assess the relatedness of swarming Proteus mirabilis strains, was used to study 15 P. aeruginosa is

252 erichia coli, Klebsiella pneumoniae, Proteus mirabilis, Streptococcus pyogenes, Bacillus subtilis, St

253 ar basal body protein FliL is involved in P. mirabilis surface sensing.

254 Proteus mirabilis swarming behavior is characterized by the deve

255 dentify rhomboid-encoding genes from Proteus mirabilis, tatA was identified as a multicopy suppressor

256 d flagellum are among virulence factors of P.mirabilis that contribute to its colonization in a murin

257 Here, we identified a gene from Proteus mirabilis that encodes a 135-amino acid residue protein,

258 laboratory models of colonization by Proteus mirabilis, the sensor signaled encrustation at an averag

259 ids genes are involved in the ability of P. mirabilis to distinguish self from nonself.

260 In order for P. mirabilis to swarm, it first needs to detect a surface.

261 in behaviour characterization in Welwitschia mirabilis to test whether the known regulatory links bet

262 s a 135-amino acid residue protein, PMTR (P. mirabilis transcription regulator), a new member of the

263 ification of 37 consistently out-competed P. mirabilis transposon mutants, 25 of which were out-compe

264 By performing primary screening of 2088 P. mirabilis transposon mutants, we identified 502 mutants

265 s swarming-associated colony expansion of P. mirabilis under anaerobic conditions on a solid surface.

266 MR/P fimbriae of uropathogenic Proteus mirabilis undergo invertible element-mediated phase vari

267 this organism, was sufficient to enhance P. mirabilis urease activity and increase disease severity,

268 Other uropathogens also enhanced P. mirabilis urease activity in vitro, including recent cli

269 Proteus mirabilis urease catalyzes the hydrolysis of urea to CO(

270 Expression of Proteus mirabilis urease is governed by UreR, an AraC-like posit

271 re not observed during coinfection with a P. mirabilis urease mutant.

272 To investigate in vivo expression of P. mirabilis urease, the gene encoding green fluorescent pr

273 This report suggests that in vivo, P. mirabilis UreD may be important for recruitment of UreF

274 ing sites in both the plasmid-encoded and P. mirabilis ureRp-ureDp intergenic regions.

275 ted the hypothesis that experimental Proteus mirabilis urinary tract infection in mice would protect

276 ot utilize citrate, the data suggest that P. mirabilis uses glutamate dehydrogenase to monitor carbon

277 d form of hemolysin A (HpmA265) from Proteus mirabilis using a series of functional and structural st

278 transconjugants/donor to ICEPm1-deficient P. mirabilis using plate mating assays with clinical isolat

279 Patients with Proteus mirabilis UTIs were more likely to have a foreign body i

280 than the averages were observed with Proteus mirabilis versus imipenem and with Klebsiella pneumoniae

281 airpins from Haemophilus influenzae, Proteus mirabilis, Vibrio fischeri, and Pasteurella multocida ar

282 We show that the P. mirabilis, Vibrio harveyi, and E. coli Crl homologs func

283 oach to investigate in vivo expression of P. mirabilis virulence genes in experimental urinary tract

284 t a role of MrpJ as a global regulator of P. mirabilis virulence.

285 ence in other pathogens, its role in Proteus mirabilis was investigated by constructing a strain with

286 A TnphoA mutant of Proteus mirabilis was isolated, which had lost the ability to sw

287 Wild-type P. mirabilis was usually found colonizing intact uroepithel

288 ncoding the entire urease gene cluster of P. mirabilis) was equivalent in both the H-NS(-) background

289 ng strains of Myxococcus xanthus and Proteus mirabilis, we found the rates of killing between the str

290 Swimming and swarming motilies in P. mirabilis were also significantly reduced by both EPIs.

291 a, Haemophilus parainfluenzae, and Lautropia mirabilis were most abundant in healthy children, while

292 lpha and an isogenic mrpH::aphA mutant of P. mirabilis were unable to produce normal MR/P fimbriae ef

293 s from mice transurethrally infected with P. mirabilis were used to prepare template DNA for PCR ampl

294 athogens, Pseudomonas aeruginosa and Proteus mirabilis, were made bioluminescent by stable insertion

295 -LDH from a warm-temperate goby (Gillichthys mirabilis), which has lower values for kcat and the Mich

296 estans, and its sister species, Phytophthora mirabilis, which is responsible for infection of Mirabil

297 pneumoniae, Klebsiella oxytoca, and Proteus mirabilis with an ertapenem-susceptible extended-spectru

298 Strains of P. mirabilis with mutations in three of the corresponding g

299 croscopy, we demonstrated the presence of P. mirabilis within the urease-induced stone matrix.

300 tion studies with hemolysin-negative Proteus mirabilis WPM111 (a HpmA(-) mutant of BA6163) transforme