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1 acy of neutralizing antibodies in preventing mucosal acquisition of infection.
2  pathophysiological processes, including gut mucosal adaptation.
3 uman monoclonal antibody that binds to human mucosal addressin cell adhesion molecule-1 (MAdCAM-1) to
4     Fecal microbiota showed covariation with mucosal adherent microbiota.
5 nza HA and GPI-anchored CCL28 as antigen and mucosal adjuvant, respectively, when immunized intranasa
6                 This study demonstrates that mucosal administration of CTH522 adjuvanted with chitosa
7 on-obese diabetic (NOD) mouse model reported mucosal administration of T1D-related autoantigens [proi
8 healthy control samples (34 in plasma, 39 in mucosal ALS; 13 in both sample sets).
9 ich are found within the epithelial layer of mucosal and barrier tissues.
10 ved mast cells were used as "surrogates" for mucosal and connective tissue mast cells, respectively,
11                                          For mucosal and cutaneous melanoma, respectively, the incide
12 r of gut microbes may provide a link between mucosal and joint immunity in patients with RA.
13 ggests abnormal macrophage switching in both mucosal and luminal areas of COPD patients, that strongl
14  represent a broader category of NK cells in mucosal and non-mucosal pathological tissues.
15 ase in expression of CCL27, one of the major mucosal and skin cytokines, was found that correlated wi
16 complex interplay of the epithelial barrier, mucosal and systemic immune response, route of exposure,
17 n the development and function of the host's mucosal and systemic immune systems.
18 testinal apical membranes and segregation of mucosal and systemic immunity.
19 pitope specificities, locations (systemic or mucosal), and effector functions of antibodies elicited
20 th were assessed including periodontal, oral mucosal, and caries status in Eastern Finland from 2014
21                               Anti-TcdA26-39 mucosal antibodies obtained following immunization with
22 respective of not significantly boosting the mucosal antibody response, it augmented the frequency of
23 -anchored CCL28 induces significantly higher mucosal antibody responses, involved in providing long-t
24 Analysis revealed that all animals exhibited mucosal antigen-specific IgG and IgA with the IgA respon
25                                        After mucosal application, nanofibrous reservoir layers are in
26                                              Mucosal-associated invariant T (MAIT) cells are a recent
27                                              Mucosal-associated invariant T (MAIT) cells are innate T
28                                              Mucosal-associated invariant T (MAIT) cells might play a
29 tially affected NK, NKT (invariant NKT), and mucosal-associated invariant T cell populations in a dos
30 ng to significantly increased frequencies of mucosal-associated invariant T cells 6 mo postinfection.
31 iboflavin- and folate-related metabolites to mucosal-associated invariant T cells, but it is unknown
32 ntranasal vaccines, which deliver antigen to mucosal-associated lymphoid tissue and aim to elicit pro
33 nflammation is associated with low levels of mucosal ATP, highlighting the importance of mitochondria
34 oles in immunoregulation but their effect on mucosal autoimmunity remains largely unknown.
35 y CD8(+) T cells could impair the intestinal mucosal barrier already in the seronegative phase of acu
36 ighlight research trends at this distinctive mucosal barrier and identify crucial gaps in our current
37  signaling events activated by a compromised mucosal barrier and provide a novel in vivo screening pl
38 nce between these signaling pathways and key mucosal barrier components in regulating colon tumorigen
39  The cytokine IL-22 plays a critical role in mucosal barrier defense, but the mechanisms that promote
40                  Antagonizing IL-4 prevented mucosal barrier disruption and tight junction downregula
41 portant factors in the regulation of colonic mucosal barrier function and inflammation.
42 development, possibly indicating an impaired mucosal barrier function in allergic children.
43                                              Mucosal barrier function was measured by uptake of fluor
44 composition of the fecal microbiota, reduced mucosal barrier function, and altered epithelial prolife
45  stability is pivotal for the maintenance of mucosal barrier function.
46          Alterations in the integrity of the mucosal barrier have been indicated as a cause for chron
47  functions of IELs in the maintenance of the mucosal barrier integrity, as well as how dysregulated I
48 untered by Shigella upon its crossing of the mucosal barrier, are also mainly targeted by injection-o
49 re, we show that loss of PGANT4 disrupts the mucosal barrier, resulting in epithelial expression of t
50 helial cells (IECs) that form the intestinal mucosal barrier.
51 l transepithelial migration utilizing airway mucosal barriers differentiated from primary human airwa
52 nto the impact of DMPA and vaginal drying on mucosal barriers.
53                                Ionization of mucosal biomass occurs directly from a standard rayon sw
54 nd postintervention stool samples and rectal mucosal biopsies were collected and used to quantify cal
55  healthy subjects); 232 fecal samples and 59 mucosal biopsy samples were collected and analyzed by 16
56 r an acute response (luminal obstruction and mucosal blanching); 2 other patients had a delayed wheal
57 ycle immunodeficiency virus, followed by two mucosal boosts with either recombinant adenovirus (rAd)
58                                    Thus, the mucosal bottleneck selects for viruses that are able to
59 d as yet unresolved, question is whether the mucosal bottleneck selects for viruses with increased tr
60 f subjects developing at least 1 small-bowel mucosal break at week 2.
61    To dissect the role of neutrophils during mucosal candidiasis, we took advantage of a new, transpa
62                All subjects with high airway mucosal CCL26 expression and moderate-to-severe asthma h
63 a minority (36%) of subjects with low airway mucosal CCL26 expression.
64 and 87% negative predictive value for airway mucosal CCL26-high status.
65 g HIV and had increased numbers of activated mucosal CD4(+) T cells compared to those with Lactobacil
66 gnal for MPECs and development of protective mucosal CD8 T cell memory.
67  In vivo, poly(I.C)-induced neutrophilia and mucosal chemokine production are blocked by a small-mole
68 ha-defective strains attained high levels of mucosal colonization but failed to induce robust vaginal
69 erienced severe disease, including increased mucosal colonization by C. rodentium, prolonged pathogen
70 anisms required for GBS to transition from a mucosal commensal lifestyle to bloodstream invasion, we
71             Candida albicans is a ubiquitous mucosal commensal that is normally prevented from causin
72 to be critical for their entry to these lung mucosal compartments.
73 ere constructed for the adjacent luminal and mucosal compartments.
74  the risk of Zika virus transmission through mucosal contact in rhesus macaques.
75 luding saliva, but transmission risk through mucosal contact is not well known.
76 roperties in parallel with analysis of local mucosal cytokine responses induced by nasal allergen exp
77      Our data indicate that the reduction in mucosal damage occurs through decline in stem/clonogenic
78 ing MCs were associated with the severity of mucosal damage, and their numbers were increased in pati
79 chemokine expression, neutrophil influx, and mucosal damage.
80 ids that are resistant to proteolytic/acidic mucosal delivery conditions.
81 duced increases in gut permeability, colonic mucosal destruction, and colonic IL-1beta expression.
82  paranasal sinuses was performed to look for mucosal disease of the paranasal sinuses, drainage pathw
83                             An ex vivo human mucosal disease tissue model was used for artificial inf
84 r location within the abomasum, being either mucosal-dwelling (MD) or lumen-dwelling (LD).
85 1 in the ileum, those lacking Mdr1 displayed mucosal dysfunction and induced Crohn's disease-like ile
86                                     Skin and mucosal epithelia deploy antimicrobial peptides (AMPs) t
87 anisms underlying disease progression on all mucosal epithelia, including those in the mouth, lungs,
88             Transplantation of cultured oral mucosal epithelial cells (OMECs) is a promising treatmen
89 lammatory signaling pathways associated with mucosal epithelial damage.
90  co-culture system consisting of a polarized mucosal epithelium and human neutrophils can provide a v
91 barrier, including increased permeability of mucosal epithelium and reduced production of important a
92 -ILK-dependent inflammatory signaling in the mucosal epithelium can be therapeutically targeted by th
93 amily, was used to infect equine respiratory mucosal explants and primary equine respiratory epitheli
94             EHV1 binding to and infection of mucosal explants was greatly enhanced upon destruction o
95 ural infection occurs through oral and nasal mucosal exposure to infectious prions.
96  those with colon-only CD based on increased mucosal expression of genes that mediate type 2 and type
97 th Crohn's disease and leprosy and implicate mucosal factors and the innate immune response to microb
98 at the modified preserved nasal and lacrimal mucosal flap technique in EES-DCR for treating PANDO is
99        Modified preserved nasal and lacrimal mucosal flap technique in EES-DCR was applied in all 27
100                  We conclude that esophageal mucosal food allergen injections induce acute and/or del
101 fficient technique facilitating ocular, oral mucosal, gastrointestinal, ungual and vaginal drug admin
102            The discovery that GLP-2 promotes mucosal growth in the intestine is described, and key fi
103 route with respect to efficacy were s.c. >/= mucosal > i.m.
104 re present in 16.3% of patients with IBD and mucosal healing (15.4% of patients with CD, 17.4% with U
105                     The primary endpoint was mucosal healing (CDEIS <4) with absence of deep ulcers 4
106 of IECs in mice with colitis and accelerates mucosal healing by activating STAT1.
107 n this cutoff, 36.2% of patients with IBD in mucosal healing have increased intestinal permeability.
108 ures of inflammatory bowel disease, complete mucosal healing occurs in fewer than 50% of patients.
109 eability in patients with symptomatic IBD in mucosal healing vs patients with asymptomatic IBD in muc
110                                              Mucosal healing was defined as CD Endoscopic Index of Se
111 oing bowel symptoms in patients with IBD and mucosal healing with 95.2% sensitivity and 97.6% specifi
112 stinal permeability in patients with IBD and mucosal healing, we associated impaired intestinal perme
113 healing vs patients with asymptomatic IBD in mucosal healing.
114 t; at day 0, after reinstitution of gingival/mucosal health; at days 7, 14, and 21, during stent-medi
115 nital bacteria increase HIV risk by inducing mucosal HIV target cells.
116 ese gut bacteria, fungi, and viruses mediate mucosal homeostasis via their composite genes (metagenom
117 t maintain and restore gastrointestinal (GI) mucosal homeostasis.
118 TK (TAM) receptor family, in regulating oral mucosal homeostasis.
119  populations critical for the maintenance of mucosal homeostasis.
120 esolution of inflammation and contributes to mucosal host defense of the nasopharyngeal niche, a rese
121 rafish model a unique tool for understanding mucosal host-pathogen interactions.
122 romised ME bacterial clearance and prolonged mucosal hyperplasia.
123                                              Mucosal IgA or secretory IgA (SIgA) are structurally equ
124 o define type 2 inflammation based on airway mucosal IL-13-driven gene expression and how this relate
125  in regulating functional responses of human mucosal ILC3s to IL-23 plus IL-1beta stimulation.
126               The ocular surface is a unique mucosal immune compartment in which anatomical, physiolo
127 ry 'dipstick' tests to assess non-accessible mucosal immune development and identify early precursors
128 , possibly due to antigen digestion prior to mucosal immune exposure.
129 es, avidities, and functions of systemic and mucosal immune responses elicited by a vaccine regimen c
130                       Importantly, prominent mucosal immune responses in CCR7-deficient mice increase
131                   Increased antigen-specific mucosal immune responses were induced in the lungs and t
132 h the mucoadhesive polymer chitosan enhances mucosal immune responses.
133 ells; thus, they are crucial participants in mucosal immune surveillance.
134 malian gastrointestinal tract and associated mucosal immune system harbor a large repertoire of metab
135 ternal environment; however, the role of the mucosal immune system in chronic lung diseases is incomp
136                                   The host's mucosal immune system must tolerate commensals while fig
137            One of the mechanisms used by the mucosal immune system to maintain homeostasis is the sec
138  the GI tract by focusing on the ENS and the mucosal immune system.
139 testinal epithelial cells and the underlying mucosal immune system.
140 ation promotes competent innate and acquired mucosal immune systems, epithelial renewal, barrier inte
141 than inactivated vaccines by eliciting local mucosal immunity and systemic B cell- and T cell-mediate
142                                              Mucosal immunity is considered important for protection
143                                              Mucosal immunity remodeled with increased in anti-inflam
144 ent for IgA nephropathy targeting intestinal mucosal immunity upstream of disease manifestation.
145 nature of antipneumococcal antibody-mediated mucosal immunity while informing vaccine development.
146                                 Induction of mucosal immunity with vaccines is attractive for the imm
147 lls, rendering the metastases susceptible to mucosal immunity.
148                                        Thus, mucosal immunization with FWPV-based vaccines should be
149  malonate, phenylacetylglycine, alanine) and mucosal immunoglobulin (IgM) and cytokine (IL-10, IL-4)
150 ssed the impact of the genital microbiota on mucosal immunology in ACB women and microbiome-HSV-2 int
151            In the current study, we examined mucosal infection in several immunocompetent and immunoc
152 ty may display differential activity against mucosal infection.
153 ee strains of immunocompetent mice supported mucosal infections.
154  (grade 3 acne [n=1] and intolerable grade 2 mucosal inflammation [n=1]); hence, doses of 200 mg and
155   During Salmonella-induced gastroenteritis, mucosal inflammation creates a niche that favors the exp
156 r study of the role of RNASET2 in regulating mucosal inflammation may lead to development of novel th
157  intestinal microbiota affect development of mucosal inflammation or inflammatory bowel disease.
158   Allergic rhinitis (AR) is characterized by mucosal inflammation, driven by activated immune cells.
159 r a specific parasite protein that increases mucosal inflammation, expands our knowledge of host-para
160  environment in pathological processes, like mucosal inflammation, preneoplasia, and neoplasia, altho
161 th IBD vs controls; some were independent of mucosal inflammation.
162  stable before and during the development of mucosal inflammation.
163 n contribute to innate IEL activation during mucosal inflammation.
164 ropathy (and possibly other gastrointestinal mucosal inflammatory diseases).
165 tion allowed measurement of RSV load and the mucosal inflammatory response in infants.
166                                          The mucosal inflammatory response induced by Salmonella sero
167 -10 was rapidly induced following intestinal mucosal injury and was required for optimal intestinal m
168  histologic evidence of significant duodenal mucosal injury, latiglutenase did not improve histologic
169                           Further, following mucosal injury, macrophage-derived IL-10 resulted in epi
170                      In response to acute GI mucosal injury, TFF peptides accelerate cell migration t
171 e to have symptoms and have small intestinal mucosal injury.
172 superficial wounds in the colon and impaired mucosal innate immune responses against C. rodentium inf
173 activity arising from acid-sensitive general mucosal innervation by comparing acid-evoked c-Fos in wi
174 iceptors, suggesting that trigeminal general mucosal innervation carries information about acids in t
175 l and glossopharyngeal nerves, while general mucosal innervation is carried by the trigeminal and glo
176      We observe that EMT silences protective mucosal interferon (IFN)-I and III production associated
177 reproductive tract (FRT) is one of the major mucosal invasion sites for HIV-1.
178 erently in SI; Mf3 formed a dense network in mucosal lamina propria, whereas Mf4 was enriched in subm
179                                          The mucosal layer of conducting airways is the primary tissu
180 part of the host diet, lining the intestinal mucosal layer, and decorating the surface of other micro
181 aniasis (LCL), which heals spontaneously, or mucosal leishmaniasis (ML), characterized by chronic and
182  of teeth, dental plaque, or detectable oral mucosal lesions and PD risk, although there was a sugges
183 points were the change in the mean number of mucosal lesions and the number of subjects with large er
184 t role of C. difficile flagella in eliciting mucosal lesions as long as the toxins exert their action
185 estive association with Candida-related oral mucosal lesions in males (hazard ratio = 1.56, 95% CI: 0
186    Active EPIT significantly reduced gastric mucosal lesions induced by PPE oral intake (macroscopic
187 ramural nodules, continuous or discontinuous mucosal lining and cholelithiasis may indicate XGC rathe
188                The upper aerodigestive tract mucosal lining is often disrupted during surgery, which
189 inated field and the need to reconstruct the mucosal lining.
190 jority of cases (22/30) showed discontinuous mucosal lining.
191 ng a transparent juvenile zebrafish to model mucosal lung infection and show that C. albicans and P.
192                                      Purpose Mucosal melanoma is an aggressive malignancy with a poor
193  data for anti-programmed death-1 therapy in mucosal melanoma to date.
194 lone, and although the activity was lower in mucosal melanoma, the safety profile was similar between
195  NRAS and NF1 in acral melanoma and SF3B1 in mucosal melanoma.
196 ofiling, the mutational landscape of primary mucosal melanomas (PMM) remains unclear.
197                           However, acral and mucosal melanomas were dominated by structural changes a
198 ucoadhesion between polymeric substances and mucosal membranes, widely exploited in the pharmaceutics
199 sponsible for the modification of the cancer mucosal metabonome.
200                       In analyzing fecal and mucosal microbiota from patients with IBS and healthy in
201   The 10% CP dietary treatment damaged ileal mucosal morphology, and decreased the expression of biom
202 binant gluten-targeting proteases, to reduce mucosal morphometric measures in biopsy specimens from p
203 ic formation of lymphocyte aggregates within mucosal nonlymphoid tissues, including the FRT.
204                                              Mucosal or epicutaneous delivery of vaccines helps targe
205        Animals challenged with SHIVAD8-EO by mucosal or intravenous routes received a single 2-week c
206 both innate and adaptive immune responses in mucosal organs.
207 cted to be a valuable model for the study of mucosal papillomavirus disease, progression, and host im
208            Images from the vermillion and/or mucosal part of the lip were recorded from 10 patients w
209 r the diagnosis of MEL of the vermillion and mucosal part of the lip.
210 tory syncytial virus (RSV) is an exceptional mucosal pathogen.
211 ader category of NK cells in mucosal and non-mucosal pathological tissues.
212 n one major etiologic factor involved in the mucosal pathology.
213      IL-4, IFN-gamma, and TNF-alpha enhanced mucosal permeability in mice.
214  of IL-4, IL-13, IFN-gamma, and TNF-alpha on mucosal permeability was tested in vivo.
215 termine if the same macaques maintained high mucosal plasma cell frequencies postinfection and if thi
216 l and contrast the potential contribution of mucosal plasma cells to mediate protection at sites of i
217 bodies for preventing HIV-1 infection at the mucosal portals of infection.
218                                      Jejunum mucosal pressure increased 5-HT release and short-circui
219  hypothesis that smoking increases bronchial mucosal production of IL-17A which acts on bronchial epi
220 sis, but its exact role in the regulation of mucosal repair after injury remains unknown.
221  Bone marrow (BM) chimera mice revealed that mucosal repair depended on TNF production by BM-derived
222 ic submucosal dissection than for endoscopic mucosal resection (EMR).
223                                   Endoscopic mucosal resection of two polyps from the cardia and duod
224                              Colonization of mucosal respiratory surfaces is a prerequisite for the h
225  LC-DG and its PB attenuate the inflammatory mucosal response in an ex-vivo organ culture model of PI
226                                         Lung mucosal responses in CB2-deficient (CB2(-/-) ) mice were
227                              The respiratory mucosal route for delivery of VV vaccine in Hu-mice, but
228 is a risk of Zika virus transmission via the mucosal route, but that the risk posed by oral secretion
229 e exposed white-tailed deer to CWD prions by mucosal routes and performed serial necropsies to assess
230 LC2 numbers significantly increased in nasal mucosal samples and decreased in blood at the time of CO
231                       We collected fecal and mucosal samples from adult patients who met the Rome III
232  type 17 immune response (IL17A and IL23) in mucosal samples from patients with UC compared with pati
233            In the present study, luminal and mucosal samples taken along the longitudinal axis of the
234  healthy subjects); 46 fecal samples, but no mucosal samples, were collected and analyzed.
235                         Existing respiratory mucosal sampling methods are flawed, particularly in a p
236 tivation of Vgamma9/Vdelta2 T cells promoted mucosal secretion of IL-22 and ICOSL/TNF-alpha-dependent
237 rd currents, 5-HT release and an increase in mucosal secretion.
238 lla typhimurium that passed through from the mucosal side to the serosal side of the tissue.
239 y colonizing the upper airways, at the human mucosal site of the disease.
240             The surface of the eye is also a mucosal site, but proof of a living, resident ocular mic
241 ymphocytes and T cell subsets in lymphoid or mucosal sites did not vary with clinical state, and was
242 es but developed persistent infection at the mucosal sites including those of the anogenital region a
243                                              Mucosal sites such as the intestine, oral cavity, nasoph
244 whereas it arises in internal body surfaces (mucosal sites) and on the hands and feet (acral sites) i
245 id arthritis (RA), immunological triggers at mucosal sites, such as the gut microbiota, may promote a
246 to play an important role in host defense at mucosal sites, where it prevents invasion of pathogens b
247 owed active infections at both cutaneous and mucosal sites.
248 may be to promote immune cell persistence at mucosal sites.Our study identifies a new regulator of IF
249 chenoid esophagitis may cause ulceration and mucosal sloughing severe enough to result in massive upp
250 rity of lesions were found in the pharyngeal mucosal space (n=16) with squamous cell carcinoma being
251                     Neutrophil breach of the mucosal surface is a common pathological consequence of
252 tionship between secretory IgA (SIgA) on the mucosal surface of small airways and parameters of infla
253  role in protective immunity to infection at mucosal surface, but also mediate pathology in certain a
254 ave long been implicated in virulence at the mucosal surface, including contributions to colonization
255 a protein vaccine formulation to the vaginal mucosal surface, we used a novel vaginal ring device com
256 nological and physiological processes at the mucosal surface.
257 iated system in the protection of intestinal mucosal surface.
258 defined by an increased ability to adhere to mucosal surfaces and a decreased ability to cause diseas
259 s way readily discriminate between different mucosal surfaces and display the ability to characterize
260 ical degradation in the harsh environment of mucosal surfaces and enzymes of host or microbial origin
261 lls migrate and proliferate to cover denuded mucosal surfaces and repair the barrier defect.
262 ns between inflammation and tissue repair at mucosal surfaces in early life remain unknown.
263           Commensal bacteria co-exist on the mucosal surfaces of all vertebrates.
264 h titers in bat flies but not in blood or on mucosal surfaces of host bats.
265 perates through an altered microbiome of the mucosal surfaces of the airways.
266 trillions of microorganisms inhabiting human mucosal surfaces participate intricately in local homeos
267  is important for homeostasis in organs with mucosal surfaces that interface with the external enviro
268 tal zoonosis acquired by contact of skin and mucosal surfaces with soil and water contaminated with i
269 mbers of the microbiomes found on nearly all mucosal surfaces, and interest is growing in understandi
270 establish tolerance, prevent inflammation at mucosal surfaces, and regulate immunopathology during in
271 ies to exert local immunosurveillance at the mucosal surfaces.
272 immune crosstalk in allergic inflammation at mucosal surfaces.
273 t of Tr1 cells in various organs, and in the mucosal system during parasitic and viral infections.
274         Our findings offer new insights into mucosal T cell biology and have important implications i
275                                   Detectible mucosal tenofovir was lower in non-Lactobacillus women,
276                       Mechanisms that elicit mucosal TH17 cell responses have been described, yet how
277 ntibody blocking of HIV-1 acquisition within mucosal tissue may prove critical to effective vaccine d
278 d replication across a range of cellular and mucosal tissue models.
279  respective study drug was injected into the mucosal tissue surrounding the surgical site prior to su
280                                     In human mucosal tissue, inflammation increased the secretion of
281 t distribution, maturation, and migration in mucosal tissues (lungs, intestines), associated lymph no
282  with decreased expression in peripheral and mucosal tissues and DNA hypermethylation in CD patients
283 ld (M) cells are epithelial cells present in mucosal tissues and specialized for the capture of lumin
284  knockout not only decreased Cdc42 levels in mucosal tissues, but it also inhibited repair of damaged
285  immunoregulatory lectin widely expressed in mucosal tissues, contributes to Y. enterocolitica pathog
286 n-specific CD8(+) T cells that accumulate in mucosal tissues, including the female genital and respir
287 present in the inner mucus layer and invaded mucosal tissues.
288 nd HbetaD2 from colonic epithelial cells and mucosal tissues.
289 l types to encounter virus in the peripheral mucosal tissues.
290 poorly protective against HIV-1 infection of mucosal tissues.
291 h a reduced gut microbiota diversity and low mucosal total IgA levels in infancy have been associated
292 tralizing antibodies (nnAbs) may also impact mucosal transmission events through Fc-gamma receptor (F
293 aureus can induce, under some circumstances, mucosal type 2 immune responses.
294 sease Activity Index (CDAI) of 220-450, with mucosal ulcers in the ileum or colon, or both, and a Cro
295     These findings highlight the efficacy of mucosal vaccination via this attenuated strain and will
296 mising strategy to modulate the magnitude of mucosal vaccine responses.
297 , epithelial renewal, barrier integrity, and mucosal vascularisation and innervation.
298  tertiary lymphoid organs present within the mucosal wall, along with other lymphatic diseases, such
299 jury and was required for optimal intestinal mucosal wound closure.
300 tions in linking innate immune activation to mucosal wound repair.

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