ライフサイエンスコーパス: Muller cell

1 Muller cells from diabetic mice with CD40(+) Muller cells.

2 -dependent signaling pathways in bipolar and Muller cells.

3 HIF-1alpha and ANGPTL4 localize to ischemic Muller cells.

4 0eembedded shRNA against VEGFA that targeted Muller cells.

5 nt to promote vessel permeability by hypoxic Muller cells.

6 patial organization similar to those seen in Muller cells.

7 tem cells (ESMVs) have on retinal progenitor Muller cells.

8 27a1-/- retina was hypoxic and had activated Muller cells.

9 olocalized with glutamine synthetase-labeled Muller cells.

10 s were present in treated but not in control Muller cells.

11 asal levels of endogenous human Oct4 mRNA in Muller cells.

12 o gene expression by STAT3 activation in rat Muller cells.

13 lar adhesion molecule 1 and VEGF proteins in Muller cells.

14 s in comparison with unregulated promoter in Muller cells.

15 l significantly reduced death of the retinal Muller cells.

16 o cones by using 11-cis-retinol generated in Muller cells.

17 na, and are expressed by retinal neurons and Muller cells.

18 ular location of FRalpha and PCFT in primary Muller cells.

19 ERG components and showed immunostaining of Muller cells.

20 ferative diabetic retinopathy can arise from Muller cells.

21 induce GAPDH nuclear accumulation in retinal Muller cells.

22 ere assessed by Western blot analysis in rat Muller cells.

23 ocation and subsequent cell death in retinal Muller cells.

24 Instead, they found that Ad5/F37 transduced Muller cells.

25 as predominantly localized in the nucleus of Muller cells.

26 um entry that replenishes ER stores in mouse Muller cells.

27 l mediator of glutamine transport in retinal Muller cells.

28 -1beta-induced GAPDH nuclear accumulation in Muller cells.

29 data, and open probability in +/+ versus -/- Muller cells.

30 zed in salamander, where it was localized in Muller cells.

31 es was predominant on glial cells, primarily Muller cells.

32 horylated ERK1/2 isoforms) were localized in Muller cells.

33 on the level of eGFP transgene expression in Muller cells.

34 Ang II and Ang-(1-7) colocalized in retinal Muller cells.

35 aldehyde binding protein (CRALBP) present in Muller cells.

36 -retinal from the RPE, possibly imported via Muller cells.

37 emokine and cytokine expression in activated Muller cells.

38 s mediated by the 11-cis selective CRALBP in Muller cells.

39 not cause TNF-alpha or IL-1beta secretion in Muller cells.

40 aquaporin 4 (AQP4) water channel in retinal Muller cells.

41 loss of adhesion between photoreceptors and Muller cells.

42 Further, we show that MFAT is expressed in Muller cells.

43 tina and retinal endothelial cells (REC) and Muller cells.

44 an 11-cis-specific retinyl-ester synthase in Muller cells.

45 a of the mouse retina such as astrocytes and Muller cells.

46 sEH-dependent generation of a diol of DHA in Muller cells.

47 y TRPV4 antagonists and absent in TRPV4(-/-) Muller cells.

48 tional deletion of Crb2 in photoreceptors or Muller cells.

49 Pathogenesis involves toxicity to retinal Muller cells.

50 with the use of 11-cis-retinol supplied from Muller cells.

51 ehyde binding protein 1 gene for conditional Muller cell ablation and the consequences of primary Mul

52 ted retinal ganglion cell axon regeneration, Muller cell activation and CNTF production in the retina

53 Retinal vasculature and Muller cell activation were analyzed by quantifying acel

54 vasculature degeneration, and inhibition of Muller cell activation were demonstrated in the treated

55 adherent area, stiffness, and propagation of Muller cells all are affected by matrix stiffness, but t

56 The Wnt-responsive Muller cells also exhibited long-term survival and, in s

57 ced nuclear accumulation of GAPDH in retinal Muller cells, an event considered crucial for the induct

58 so we could specifically quantify changes in Muller cell anatomy between control mice (C57Bl/6) and t

59 marker Pax2 but inconsistently expressed the Muller cell and occasional astrocyte marker CRALBP.

60 leted in colorectal carcinoma), was found in Muller cells and astrocytes.

61 FRalpha and PCFT are expressed in retinal Muller cells and colocalize in the endosomal compartment

62 OPL and HFL, indicating junctions in between Muller cells and cone axons.

63 equence of loss of control with depletion of Muller cells and exposure of the remaining retinal vesse

64 old increase in promoter activity in primary Muller cells and human Muller cell lines, respectively.

65 y reduced the reactive expression of GFAP in Muller cells and improved the morphology of astrocytes i

66 immunoreactivity was observed in rat retinal Muller cells and in the RGC layer and blood vessels, res

67 understand better the mechanosensitivity of Muller cells and its association with vitreoretinal diso

68 Muller cells and macrophages/microglia are likely import

69 s for two weeks resulted in proliferation of Muller cells and maintenance of some rod and cone photor

70 nced retinal disease highlighting a role for Muller cells and may inform future therapeutic strategie

71 and activity were also increased in cultured Muller cells and microglia treated with LPS.

72 glaucoma, upregulates TNFalpha production by Muller cells and microglia.

73 del, we show that TNFalpha is upregulated by Muller cells and microglia/macrophages soon after induct

74 Retinal astrocytes/Muller cells and retinal ganglion cells were the source

75 ptor in angiogenic cell behaviors of retinal Muller cells and retinal microvascular endothelial cells

76 ear layer; they are present in primary mouse Muller cells and rMC-1 cells.

77 ribution of Jam-C in the apical membranes of Muller cells and RPE suggests that Jam-C has a novel fun

78 s in the regulation of potassium channels in Muller cells and subsequently in the promotion of glutam

79 n mouse, about the anatomical arrangement of Muller cells and their arbors, and how these features ar

80 ine cells and their processes, as well as in Muller cells and their processes that extended radially

81 wed macrophages, granulocytes (neutrophils), Muller cells, and microglial cells as TNF-alpha sources.

82 targets were ganglion cells, bipolar cells, Muller cells, and photoreceptors.

83 meshwork, ciliary body, ciliary epithelium, Muller cells, and retinal ganglion cells.

84 photoreceptors, bipolar cells, mitochondria, Muller cells, and retinal pigment epithelium (RPE) cells

85 neural retina and RPE/eyecup, primary mouse Muller cells, and rMC-1 cells for the three known folate

86 viding cells, with anti-vimentin to identify Muller cells, and with the isolectin B4 to identify micr

87 bution of glutamine synthetase, expressed by Muller cells, and zonula occludens-1, a tight-junction p

88 embranes of photoreceptor inner segments and Muller cell apical processes in the zebrafish retina.

89 the putative all-trans retinol isomerase in Muller cells, appears to be 9-cis retinol.

90 Positively identified Muller cells are present in diabetic epiretinal tissues

91 Muller cells are principal glial cells in rat retina and

92 Muller cells are subtly different in the GFAP(-/-)vim(-/

93 Muller cells are the major glia of the retina that serve

94 A subpopulation of mammalian Muller cells are Wnt responsive and, when Wnt signaling

95 toskeletal integrity, and gene regulation in Muller cells as a function of the stiffness of the subst

96 uorescent protein expression only in retinal Muller cells at P17.

97 P = 0.03, Student's t test), and gliosis in Muller cells (at 6 mo, using SPION-glial fibrillary acid

98 n of glutamine and glutamate was observed in Muller cells before established gliosis markers.

99 Gliosis, characterized by translocation of Muller cell bodies to the outer retina and thickening of

100 The retinal area occupied by the total Muller cell body (soma and processes) was significantly

101 ls within the retinal pigment epithelium and Muller cells, but may limit iron transport into the reti

102 oration of CRALBP expression specifically in Muller cells, but not retinal pigment epithelial (RPE) c

103 Swelling and [Ca(2+)]i elevations evoked in Muller cells by hypotonic stimulation were antagonized b

104 Knockdown of VEGFA in rat Muller cells by lentivector VEGFA-shRNA significantly re

105 Muller cell changes in culture included loss of glutamin

106 c rodents and by hyperglycemic conditions in Muller cells concomitant with increased VEGF expression.

107 ostaglandin E(2) (PGE(2)) and VEGF levels in Muller cell-conditioned medium and in retinal tissue sam

108 ed hypoxia-induced PGE(2) and VEGF levels in Muller cell-conditioned medium by 68.6% (P < 0.001) and

109 ely expressed by ganglion cells, and perhaps Muller cells, cone photoreceptor terminals, and horizont

110 The previously reported increases in Muller cell contraction-promoting activity detected in h

111 iated with significant increases in vitreous Muller cell contraction-promoting activity that are pres

112 diabetic swine were evaluated for changes in Muller cell contraction-promoting activity, the presence

113 Our results identify Muller cell CRALBP as a key component of the retinal vis

114 ype 2 diabetic rat model, as well as REC and Muller cells cultured in normoglycemia and hyperglycemic

115 ion of insulin receptor signaling in retinal Muller cells cultured under hyperglycemic conditions and

116 Similar analyses were performed in Muller cell cultures exposed to hyperglycemic conditions

117 ow levels in the retina, and none in primary Muller cell cultures, indicating the presence of Isomera

118 induce neurogenesis in vivo and in purified Muller cell cultures.

119 minoadipate were also determined in purified Muller cell cultures.

120 lipoproteins may be implicated in apoptotic Muller cell death, acting at least partially via enhance

121 cusing on its ability to influence mammalian Muller cell dedifferentiation, proliferation, and neurog

122 ngiogenic therapies may be required to treat Muller cell deficiency in retinal diseases and in other

123 In contrast, Muller cells demonstrated robust extracellular matrix co

124 x2 in the mouse postnatal retina, leading to Muller cell depletion and retinal degeneration.

125 Muller cells derived from COX-2-null mice were treated w

126 l role in retinal inflammatory signaling and Muller cell-derived inflammatory cytokine production in

127 ription factor 4 (ATF4) in the regulation of Muller cell-derived inflammatory mediators in diabetic r

128 ntial, was similar between rd/rd and control Muller cells during cone degeneration.

129 ell ablation and the consequences of primary Muller cell dysfunction have been studied in adult mice.

130 nd vascular pathogenic pathways secondary to Muller cell dysfunction, the cause of which remains obsc

131 f VEGFA by lentivector VEGFA-shRNAetargeting Muller cells efficiently reduced 50/10 OIR up-regulated

132 Muller cell electrophysiology, particularly K(+) current

133 ESMVs transferred to Muller cells embryonic stem cell (ESC) mRNAs involved in

134 inner segments, and pedicles, as well as to Muller cell endfeet.

135 ImM10 Muller cells express numerous genes associated with neur

136 In this article, we show that Muller cells express TLR2, a key sensor implicated in re

137 Retinal endothelial and Muller cells expressed CD40.

138 entiviral-delivered shRNAs that knocked down Muller cell-expressed VEGF in the retinopathy of prematu

139 Muller cell fates were analyzed in retinal sections by u

140 Immunohistochemistry confirmed that Muller cells followed a radial distribution around the f

141 stmortem histologic artifact of eosinophilic Muller cell foot process swelling that mimics a nerve fi

142 A focal artifact of Muller cell foot process swelling was identified in most

143 iew demonstrates an artifact of eosinophilic Muller cell foot processes swelling in postmortem examin

144 han 3 months, demonstrated diffusely swollen Muller cell foot processes with intensely eosinophilic c

145 After retinal detachment, Muller cells formed subretinal glial scars in the wt mic

146 parable Kir4.1 protein expression pattern in Muller cells from +/+ and -/- retinas, with greatest exp

147 TNF-alpha was not detected in Muller cells from diabetic mice with CD40(+) Muller cell

148 thread in many retinal diseases is reactive Muller cell gliosis, an untreatable condition that leads

149 s, migration, and formation of new synapses; Muller cell gliosis, migration, and scarring; blood vess

150 In cultured Muller cells, high glucose induced a time-dependent incr

151 pigment epithelial cells (HRPEs), and human Muller cells (HMCs) under elevated glucose conditions wa

152 Muller cell line (rMC-1) and isolated human Muller cells (HMCs), the authors examined the effect of

153 In Muller cells, hyperglycemic conditions attenuated global

154 kening of OPLHenle in older eyes may reflect Muller cell hypertrophy associated with rod loss.

155 In Muller cells, hypoxia increased the phosphorylation of c

156 rhombencephalon at the level of the isthmic Muller cell I1 close to sulcus limitans of His.

157 ntly increased GAPDH nuclear accumulation in Muller cells in a concentration-dependent manner within

158 the modulation of inflammatory cytokines in Muller cells in a hyperglycemic environment.

159 retinal endothelial cells (REC) and retinal Muller cells in either 5 mM (normal) or 25 mM (high) glu

160 Furthermore, IKKbeta isolated from Muller cells in normal glucose medium was found to be gl

161 porting a potential mechanism of traction by Muller cells in the CB.

162 iated virus (AAV) variant, ShH10, transduces Muller cells in the Dp71-null mouse retina efficiently a

163 The end foot region of Muller cells in the GFAP(-/-)vim(-/-) mice often sheared

164 tetrahydrofolate ([(3)H]-MTF) was assayed in Muller cells in the presence/absence of thiamine pyropho

165 anatomical remodeling and gliosis of retinal Muller cells in the rd/rd mouse model of photoreceptor d

166 explores the nature of the cis retinol that Muller cells in the retina provide to cones for the rege

167 VEGF) expression were partially localized to Muller cells in the retina.

168 We found that the Muller cells in the salamander neural retina promote con

169 a cytokine up-regulated by HIF-1 in hypoxic Muller cells in vitro and the ischemic inner retina in v

170 ced expression of progenitor cell markers by Muller cells in vitro or stimulated Muller cell migratio

171 00 mum composed of densely packed cones (and Muller cells) in the central fovea.

172 gative CASP6 mutant activates astrocytes and Muller cells, increases CNTF levels in the retina and le

173 lation of hypoxia-inducible factor-1alpha in Muller cells induces the expression of VEGF, which, in t

174 duced release of inflammatory mediators from Muller cells, inhibited accumulation of mononuclear phag

175 Our studies indicate that CD40 in Muller cells is sufficient to upregulate retinal inflamm

176 e hybrid retinas and expressed abundantly in Muller cells, is the enzyme that drives this reaction.

177 ssociation with KCNJ10 mutations affecting a Muller cell K+ channel.

178 lar hypertension (COH) in rat down-regulated Muller cells Kir2.1, Kir4.1, TASK-1, GS and GLAST expres

179 functionally significant AQP4 modulation of Muller cell Kir4.1 K(+) channel function.

180 ive A2A AR antagonist SCH442416 up-regulated Muller cell Kir4.1, TASK-1, GS and GLAST expressions and

181 KO mice had reduced protection, although the Muller cell KO mice did not, thus gp130-induced protecti

182 r signal-regulated kinase (ERK) signaling in Muller cells, leading to Stat3 activation in photorecept

183 and specific expression of exogenous Dp71 in Muller cells leads to correct localization of Dp71 prote

184 We found that selective ablation of Muller cells led to photoreceptor apoptosis, vascular te

185 aled that sEH deletion decreased retinal and Muller cell levels of 19,20-dihydroxydocosapentaenoic ac

186 With use of the transformed rat Muller cell line (rMC-1) and isolated human Muller cells

187 A conditionally immortalized mouse Muller cell line was cultured on laminin-coated polyacry

188 In a human Muller cell line, MIO-M1, transfection of a miR-200b mim

189 r activity in primary Muller cells and human Muller cell lines, respectively.

190 The inner nuclear layer/Muller cell localization of the key proteins induced by

191 Additionally, mutant Muller cells lose apical processes and their perikarya t

192 evaluation was performed on freshly isolated Muller cells maintained in high- and low-glucose culture

193 he ganglion cell marker Thy 1.2 and with the Muller cell marker vimentin, confirming their presence i

194 Ras-MAPK) signaling plays a critical role in Muller cell maturation and function, which is necessary

195 Foveal Muller cells may play an integral role in the transmissi

196 g of RFC expression and activity in cultured Muller cells may reflect the upregulation of this protei

197 ent epithelial cells on Bruch's membrane and Muller cells (MCs) on the inner limiting membrane (ILM),

198 cells that provide a mechanical link at the Muller cell membrane by direct binding to actin and a tr

199 rkers by Muller cells in vitro or stimulated Muller cell migration to the outer nuclear layer (ONL) a

200 Cultured human retinal Muller cells (MIO-M1) were treated with highly oxidized

201 ected in rabbit eyes along with fibroblasts, Muller cells (MIO-M1), or Muller cells transfected to in

202 tive inhibitor of PVR in both fibroblast and Muller cell models of PVR.

203 ofile of cavities around MHs correlates with Muller cell morphology and is consistent with the hypoth

204 Muller cell morphology was different and glutamine synth

205 Bergmann glia and retinal Muller cells, nonforebrain astrocytes that have not been

206 , we use ShH10 to restore Dp71 expression in Muller cells of Dp71 deficient mouse to study molecular

207 uclear layer corresponding to CRALBP-labeled Muller cells of pups in the 50/10 OIR model.

208 izing enzyme, is expressed by astrocytes and Muller cells of the retina, but little is known about it

209 A second visual cycle is present in Muller cells of the retina.

210 lacking RDH10 either in cone photoreceptors, Muller cells, or the entire retina.

211 e experimental diabetic retinopathy and that Muller cells orchestrate inflammatory responses in myelo

212 Conditioned medium from the Muller cells overexpressing Grx1 was added to fresh cult

213 versus low-glucose medium does not influence Muller cell phenotype changes.

214 prevented high glucose-induced cell death of Muller cells potentially by inhibiting p53 phosphorylati

215 hot spots of rod death and the remodeling of Muller cell process into zones of low density of photore

216 ha(2)delta(4) subunit is mainly localized to Muller cell processes and endfeet, photoreceptor termina

217 However, at older ages an increase in Muller cell processes was seen.

218 outer retina was considered, we found rd/rd Muller cell processes were dramatically reduced during t

219 synthetase shows that CB2R is restricted to Muller cell processes, extending from the internal limit

220 rophysiology followed by an active growth of Muller cell processes, glial seal formation, and attenua

221 At P10, weak GAT-1 immunostaining was in Muller cell processes.

222 rvative glial response involving the loss of Muller cells processes, hyperexpression of GFAP, and pre

223 Porcine Muller cells proliferate in response to PDGF, but not IG

224 he loss of the Wnt negative regulator Axin2, Muller cells proliferated after injury and adopted the e

225 his study was to characterize its effects on Muller cell proliferation and tractional force generatio

226 ombinant protein for the ability to modulate Muller cell proliferation and tractional force generatio

227 Palomid 529 is an effective suppressor of Muller cell proliferation, glial scar formation, and pho

228 hy, we provide evidence showing that hypoxic Muller cells promote vascular permeability by stabilizin

229 eceptor subtype 6), retinoschisin (Rs1), the Muller cell proteins glial fibrillary acidic protein (GF

230 bit the development of PVR in fibroblast and Muller cell rabbit models of PVR.

231 we show that the store-operated response in Muller cells, radial glia that perform key structural, s

232 uggest a critical role for these proteins in Muller cell reaction to retinal detachment and participa

233 g the ERG b-wave and oscillatory potentials, Muller cell reactive gliosis, and neuronal cell death, a

234 -characterized retina visual cycle, in which Muller cells recycle spent all-trans-retinol visual chro

235 Interestingly, Muller cells require HIF--but not VEGF--to promote vascu

236 TNF-alpha-only treatments of Muller cells resulted in significant decreases of tyrosi

237 Chromophore supply by the retinal Muller cells (retina visual cycle) supports the efficien

238 Drug toxicity toward Muller cells, retinal pigment epithelium, and cones was

239 NA microarrays of ESMV-treated vs. untreated Muller cells revealed the up-regulation of genes and miR

240 Also, incubation of rat retinal Muller cells (rMC-1) in normal glucose (5 mm) or diabeti

241 n VEGF and PEDF expression were evaluated in Muller cells (rMCs), primary mouse retinal pigment epith

242 f these deficiencies is that they impair the Muller cell's ability to metabolize Glc.

243 targeted knockdown of overexpressed VEGFA in Muller cells safely reduces IVNV in a relevant ROP model

244 emonstrate a complex interplay among hypoxic Muller cells, secreted angiogenic factors, and neighbori

245 , whereas retinas lacking CRB2 in developing Muller cells showed late onset retinal disorganization.

246 reticular neurons of the lamprey brainstem (Muller cells) showed ASP immunoreactivity in perikarya a

247 We found no change in the number of Muller cell somata between mice strains, indicating no c

248 At P10, Muller cell somata were observed in the middle of the IN

249 Retinal Muller cells span the retina and secrete several trophic

250 sEH was localized in Muller glia cells, and Muller cell-specific sEH deletion reproduced the sEH(-/-

251 othelial cells (ECs), retinal microglia, and Muller cells stimulated with TNF-alpha, VEGF, or LPS.

252 a2, or Ret was detected in photoreceptors or Muller cells, suggesting that these cells are not direct

253 Complementary in vitro studies in cultured Muller cells supported these findings and demonstrated t

254 nd accumulation by targeting siah-1 promotes Muller cell survival under hyperglycemic conditions.

255 RFC was expressed in mouse Muller cells that had been allowed to proliferate in cul

256 Edn2 has detrimental effects on the BRB and Muller cells that involve interactions with the renin-an

257 ne cytoskeletal protein, expressed mainly in Muller cells that provide a mechanical link at the Mulle

258 ogy, propagation, and expression of genes in Muller cells that were cultured on substrates of varying

259 decrease in the number of anti-BrdU-labeled Muller cells, the number and size of subretinal scars, a

260 Muller cells, the principal glia of the retina, play sev

261 eterogeneous cell population of ESMV-treated Muller cells, their expression of retinal cell markers w

262 ties of store-operated signaling pathways in Muller cells, these studies expand the current knowledge

263 in, which may exert its effects in cones and Muller cells through multiple ways of action.

264 lucose-induced GAPDH nuclear accumulation in Muller cells through production and autocrine stimulatio

265 he cycle, 11-cis-retinol is transported from Muller cells to cone inner segments, where it is oxidize

266 the endogenous Wnt signal causes a subset of Muller cells to proliferate and dedifferentiate into RPC

267 It appears that Muller cell tractional force generation in PDR is driven

268 Intact IGFBP-3 modulates Muller cell tractional force generation stimulated by IG

269 with fibroblasts, Muller cells (MIO-M1), or Muller cells transfected to increase their expression of

270 rs capable of highly efficient and sustained Muller cell transgene expression in healthy and diseased

271 Muller cells trasfected with plasmids or virus were comp

272 Wild-type mouse Muller cells treated with increasing concentrations of L

273 COX-2-null mouse Muller cells treated with increasing concentrations of P

274 These changes occurred in both REC and Muller cells treated with pioglitazone, suggesting that

275 er cells, we identified a mechanism by which Muller cells trigger proinflammatory cytokine expression

276 CD40 ligation in Muller cells triggered phospholipase C-dependent ATP rel

277 in which CD40 stimulation of endothelial and Muller cells triggers adhesion molecule up-regulation an

278 s of K2P channel expression are observed for Muller cells (TWIK-1, TASK-3, TRAAK, and TREK-2) and ret

279 Muller cells, unlike neurons, are spread across the reti

280 During diabetes, mice with CD40 expressed in Muller cells upregulated retinal tumor necrosis factor-a

281 Muller cell uptake of [(3)H]-MTF was robust at pH 5.0 to

282 ntify TRPV4 channels as transducers of mouse Muller cell volume increases into physiological response

283 lucose-induced GAPDH nuclear accumulation in Muller cells was mediated by IL-1beta.

284 Furthermore, ectopic expression of REDD1 in Muller cells was sufficient to promote both increased 4E

285 sing mice with CD40 expression restricted to Muller cells, we identified a mechanism by which Muller

286 Retinal Muller cells were cultured in normal (5 mM) or high (25

287 sion in cultures of freshly isolated porcine Muller cells were evaluated by indirect immunofluorescen

288 Cultured human Muller cells were exposed to mouse ESMVs every 48 hours

289 Muller cells were isolated from normal porcine retina.

290 IL-33(+) Muller cells were more abundant and IL-33 cytokine was e

291 Muller cells were stimulated to proliferate by serum and

292 tase) revealed that approximately 95% of the Muller cells were transduced in the region near the inje

293 In separate experiments, Muller cells were treated with 7-ketocholesterol (7-KC,

294 f the EP(4) agonist PGE(1)-OH, and wild-type Muller cells were treated with increasing concentrations

295 toreceptors and horizontal cells, as well as Muller cells, were immunonegative, whereas retinal gangl

296 Further, macroglial Muller cells, which influence the integrity of the BRB a

297 erentiation and pluripotency in their target Muller cells, which may turn on an early retinogenic pro

298 Muller cells, which normally elongate and thicken in res

299 al membranes contained positively identified Muller cells with vimentin, GFAP, and glutamine syntheta

300 dition, zonula occludens-1 co-localized with Muller cells within the complex of OPL and HFL, indicati