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1 tic plasmids from the opportunistic pathogen Mycobacterium avium.
2 was identified in the opportunistic pathogen Mycobacterium avium.
3 of TLR2 expression following infection with Mycobacterium avium.
4 variety of intracellular pathogens including Mycobacterium avium.
5 s tested, including Mycobacterium leprae and Mycobacterium avium.
6 ase mediated by the intramacrophage pathogen Mycobacterium avium.
7 TLR) 2 mRNA was induced after infection with Mycobacterium avium.
8 s virus stimulated by in vivo infection with Mycobacterium avium.
9 llowing infection of murine macrophages with Mycobacterium avium.
10 rium bovis bacillus Calmette-Guerin (BCG) or Mycobacterium avium.
11 It is about 100-fold less effective against Mycobacterium avium.
12 nt, phenol treatment, and contamination with Mycobacterium avium.
14 ently were: Mycobacterium mucogenicum (52%), Mycobacterium avium (30%), and Mycobacterium gordonae (2
16 smegmatis and to a lesser extent pathogenic Mycobacterium avium, activate Ca(2+)-dependent calmoduli
17 restored antimycobacterial activity against Mycobacterium avium and Mycobacterium bovis Bacille Calm
18 thin 30 min and 30 to 60% after 3 h, whereas Mycobacterium avium and Mycobacterium fortuitum isolates
20 The clinical significance and prevalence of Mycobacterium avium and Mycobacterium intracellulare wer
22 olipids (GPLs), a major surface component of Mycobacterium avium and other non-tuberculosis mycobacte
23 s complex and to have homology with DNA from Mycobacterium avium and other nontuberculous mycobacteri
24 R2-dependent responses were seen using whole Mycobacterium avium and Staphylococcus aureus, demonstra
25 ples yielded cultures of Candida lusitaniae, Mycobacterium avium, and a filamentous fungus, Trichophy
26 lass II promoter (hu10Tg) were infected with Mycobacterium avium, and bacterial burdens and immune re
27 c infections caused by Pneumocystis carinii, Mycobacterium avium, and Campylobacter coli that require
28 /bg(-) mice were infected intravenously with Mycobacterium avium, and cultures of blood and brain as
29 7(-/-) and wild-type mice were infected with Mycobacterium avium, and host responses were analyzed.
30 er, Pneumocystis carinii, Toxoplasma gondii, Mycobacterium avium, and Lactobacillus casei) and showed
33 rtunistic pathogens (Legionella pneumophila, Mycobacterium avium, and Pseudomonas aeruginosa), broade
36 ag2(-/-)gammac(-/-) mice are as resistant to Mycobacterium avium as Rag2(-/-) mice, whereas Rag2(-/-)
37 duced in response to live M. tuberculosis or Mycobacterium avium as well as certain mycobacterial pro
39 ues from both Mycobacterium tuberculosis and Mycobacterium avium can complement lpqM donor mutants, s
44 rophages following infection with pathogenic Mycobacterium avium compared to the activation following
45 rophages infected with pathogenic strains of Mycobacterium avium compared with infections with the fa
48 (100%), CD4 cell count less than 200 (84%), Mycobacterium avium complex (73%), and Pneumocystis cari
49 We report a case of recurrent disseminated Mycobacterium avium complex (DMAC) disease with anti-gam
51 e cases, 122 (64%) were culture-positive for Mycobacterium avium complex (MAC) and 69 (36%) for M. ab
54 f 2,4-diamino-5-deazapteridine inhibitors of Mycobacterium avium complex (MAC) and human dihydrofolat
58 type 1-infected persons with newly diagnosed Mycobacterium avium complex (MAC) bacteremia were enroll
60 y virus type 1 (HIV-1)-infected persons with Mycobacterium avium complex (MAC) bacteremia, the levels
61 reproducibility of susceptibility testing of Mycobacterium avium complex (MAC) by broth microdilution
62 r antimycobacterial therapy for disseminated Mycobacterium avium complex (MAC) could be withdrawn fro
63 n alone and in combination for prevention of Mycobacterium avium complex (MAC) disease were compared
64 in is a major drug used for the treatment of Mycobacterium avium complex (MAC) disease, but standard
75 robiologic cure of AIDS-related disseminated Mycobacterium avium complex (MAC) is possible in patient
78 features and outcome of macrolide-resistant Mycobacterium avium complex (MAC) lung disease are not k
79 in prospective macrolide treatment trials of Mycobacterium avium complex (MAC) lung disease were asse
80 household water sources for 36 patients with Mycobacterium avium complex (MAC) lung disease were eval
81 cs and to evaluate relapses in patients with Mycobacterium avium complex (MAC) lung disease, but the
82 atment of noncavitary nodular bronchiectatic Mycobacterium avium complex (MAC) lung disease, supporti
85 The clinical significance of recovery of Mycobacterium avium complex (MAC) organisms from respira
87 Species identification of isolates of the Mycobacterium avium complex (MAC) remains a difficult ta
90 actors that contribute to protection against Mycobacterium avium complex (MAC), cytokine production b
91 genetically similar to other members of the Mycobacterium avium complex (MAC), some of which are non
92 y (HAART) on cell-mediated immunity (CMI) to Mycobacterium avium complex (MAC), we measured immune re
96 mocystis carinii (n = 26), bacteria (n = 3), Mycobacterium avium complex (n = 2), Nocardia sp. (n = 1
97 l disease (mean number of organs infected by Mycobacterium avium complex 4.1 [SD 0.8] vs 2.0 [1.1], p
98 tory pulmonary nontuberculous mycobacterial (Mycobacterium avium complex [MAC] or Mycobacterium absce
102 ttributable to cytomegalovirus retinitis and Mycobacterium avium complex declined over time (p=0.0058
103 Calif.) to detect Mycobacterium gordonae and Mycobacterium avium complex directly in liquid medium fl
104 Several agents are effective in preventing Mycobacterium avium complex disease in patients with adv
106 g the pseudocording, or loose aggregation of Mycobacterium avium complex from M. tuberculosis and the
108 yelitis (patient 1) and disseminated CMV and Mycobacterium avium complex infection (patient 2), respe
109 IS event in HIV-infected patients, unmasking Mycobacterium avium complex infection after starting ant
113 were evaluated for susceptibility testing of Mycobacterium avium complex isolates against clarithromy
114 ribosomal internal transcribed spacer of 56 Mycobacterium avium complex isolates from pediatric pati
115 g activities seem not to be risk factors for Mycobacterium avium complex lung disease in HIV-negative
118 ia, esophageal candidiasis, and disseminated Mycobacterium avium complex or Mycobacterium kansasii in
120 hose previously treated for tuberculosis and Mycobacterium avium complex predominated (27.7% [95% CI:
122 new therapy for Candida esophagitis, whereas Mycobacterium avium complex therapy may be discontinued
126 Nearly 60% of positive cultures were for Mycobacterium avium complex, although this ranged by sta
127 , 14% (1/7) for Cryptococcus, 10% (1/10) for Mycobacterium avium complex, and 4% (3/72) for PCP.
128 ifferent Mycobacterium tuberculosis complex, Mycobacterium avium complex, and Mycobacterium spp. dire
129 neumocystis jeroveci pneumonia, disseminated Mycobacterium avium complex, lymphoid interstitial pneum
131 tis carinii pneumonia [PCP] and disseminated Mycobacterium avium-complex [MAC] infection) in persons
133 ents and household water/biofilm isolates of Mycobacterium avium could be matched by DNA fingerprinti
136 hat represent 46 genes that are expressed by Mycobacterium avium during growth in human macrophages.
137 tosporidium parvum, Enterocytozoon bieneusi, Mycobacterium avium, Entamoeba histolytica, Balantidium
138 imensional gel electrophoresis revealed that Mycobacterium avium expresses several proteins unique to
139 n binding motif of the previously identified Mycobacterium avium fibronectin attachment protein (FAP-
148 In this study, 30 AIDS patients without Mycobacterium avium infection were randomized to receive
149 ceptor (TLR) signaling in host resistance to Mycobacterium avium infection, mice deficient in the TLR
151 ere we show, using an in vivo mouse model of Mycobacterium avium infection, that an increased proport
158 that most resemble Mycobacterium simiae and Mycobacterium avium-intracellulare complex but which pos
159 These included a psoas abscess secondary to Mycobacterium avium-intracellulare, septic wrist, bacter
161 Invasion of intestinal mucosa of the host by Mycobacterium avium is a critical step in pathogenesis a
169 terium tuberculosis, Mycobacterium leprae or Mycobacterium avium is correlated with strong inflammato
170 The cell wall of the environmental pathogen Mycobacterium avium is important to its virulence and in
175 pj), Toxoplasma gondii (T. gondii, tg), and Mycobacterium avium (M. avium, ma) are the principal cau
176 vide a tool to better understand the role of Mycobacterium avium-M. intracellulare complex isolates i
179 cystis carinii (Pc), Toxoplasma gondii (Tg), Mycobacterium avium (Ma), and rat liver in comparison wi
180 is carinii (Pc), Toxoplasma gondii (Tg), and Mycobacterium avium (Ma), three life-threatening pathoge
181 is carinii (Pc), Toxoplasma gondii (Tg), and Mycobacterium avium (Ma), three of the opportunistic org
182 is carinii (Pc), Toxoplasma gondii (Tg), and Mycobacterium avium (Ma), three of the opportunistic pat
184 arinii (pcDHFR), Toxoplasma gondii (tgDHFR), Mycobacterium avium (maDHFR), and rat liver (rlDHFR).
185 fected with different mycobacterial strains (Mycobacterium avium, Mycobacterium bovis BCG or Mtb), we
186 erium smegmatis, Mycobacterium tuberculosis, Mycobacterium avium, Mycobacterium gadium and Mycobacter
187 PP (Legionella spp., Legionella pneumophila, Mycobacterium avium, Mycobacterium intracellulare, Pseud
190 lpha) production in macrophage infected with Mycobacterium avium or M smegmatis is dependent on myelo
191 erived macrophages were infected with either Mycobacterium avium or Mycobacterium tuberculosis and th
194 drome, and opportunistic infections, such as Mycobacterium avium or Pneumocystis carinii infections.
195 sease in humans have led to speculation that Mycobacterium avium paratuberculosis (MAP) might be a ca
199 hat primary murine macrophages infected with Mycobacterium avium produced lower levels of tumor necro
201 primary murine macrophages had no effect on Mycobacterium avium retention in an early endosomal comp
202 human macrophages respond to infection with Mycobacterium avium, serovar 4, by producing tumor necro
203 olipids (GPLs), a major surface component of Mycobacterium avium, showed limited acidification and de
204 cal survey of distribution in Great Britain, Mycobacterium avium ssp. paratuberculosis (MAP) was dete
205 time in macrophages infected with pathogenic Mycobacterium avium strains relative to infections with
206 say that uses a unique element (ISMap02) for Mycobacterium avium sub0:36 PMparatuberculosis that is p
210 obacterium avium subsp. paratuberculosis and Mycobacterium avium subsp. avium are antigenically and g
211 obacterium avium subsp. paratuberculosis and Mycobacterium avium subsp. avium are antigenically and g
213 of Mycobacterium avium subsp. silvaticum and Mycobacterium avium subsp. avium, and a single isolate e
217 cosa by M. avium subsp. paratuberculosis and Mycobacterium avium subsp. hominissuis, a pathogen known
218 egration loci of IS900 (loci L1 and L9), one Mycobacterium avium subsp. paratuberculosis (M. paratube
219 R) sequencing approach for the genotyping of Mycobacterium avium subsp. paratuberculosis (M. paratube
221 ing affinity of Ag85A, Ag85B, and Ag85C from Mycobacterium avium subsp. paratuberculosis (MAP) (K(D)
222 protocol was optimized for the isolation of Mycobacterium avium subsp. paratuberculosis (MAP) from m
224 r regulatory mechanisms of host responses to Mycobacterium avium subsp. paratuberculosis (MAP) infect
225 To investigate the stochastic dynamics of Mycobacterium avium subsp. paratuberculosis (MAP) infect
228 = 128), Mycobacterium kansasii (n = 10), and Mycobacterium avium subsp. paratuberculosis (n = 10), ca
229 SOD], and 35-kDa protein) were purified from Mycobacterium avium subsp. paratuberculosis and evaluate
230 Little is known of protein expression in Mycobacterium avium subsp. paratuberculosis and how this
231 nce repeats (SSRs) of 211 and 56 isolates of Mycobacterium avium subsp. paratuberculosis and M. avium
235 made between PBMCs stimulated in vitro with Mycobacterium avium subsp. paratuberculosis and PBMCs st
236 = 39) were tested for the presence of viable Mycobacterium avium subsp. paratuberculosis by a novel p
244 ism (AFLP) to characterize the genomes of 20 Mycobacterium avium subsp. paratuberculosis field isolat
245 the isolation, separation, and detection of Mycobacterium avium subsp. paratuberculosis from milk an
249 orescent probes (molecular beacons) detected Mycobacterium avium subsp. paratuberculosis in bovine fe
252 sensitivity and reduce time to diagnosis of Mycobacterium avium subsp. paratuberculosis infection.
254 and veterinary Johne's disease suggests that Mycobacterium avium subsp. paratuberculosis is a causati
262 lar diversity of animal and human strains of Mycobacterium avium subsp. paratuberculosis isolated in
263 for the study of the genetic relatedness of Mycobacterium avium subsp. paratuberculosis isolates har
264 s, infection with the intracellular pathogen Mycobacterium avium subsp. paratuberculosis results in a
265 nd/or enrichment methods on the selection of Mycobacterium avium subsp. paratuberculosis subtypes.
268 luate whether cows that were low shedders of Mycobacterium avium subsp. paratuberculosis were passive
274 atuberculosis isolates, two isolates each of Mycobacterium avium subsp. silvaticum and Mycobacterium
277 sheep, is caused by slow replicating bacilli Mycobacterium avium subspecies paratuberculosis (MAP) in
278 omplete genome sequence of a common clone of Mycobacterium avium subspecies paratuberculosis (Map) st
281 uipment for the large-scale global typing of Mycobacterium avium subspecies paratuberculosis isolates
282 erformed using genome sequence data from 133 Mycobacterium avium subspecies paratuberculosis isolates
283 g-chain acyl-CoA carboxylase holoenzyme from Mycobacterium avium subspecies paratuberculosis revealed
285 A number of intestinal pathogens including Mycobacterium avium subspecies paratuberculosis, adheren
286 ed possibilities for the characterization of Mycobacterium avium subspecies paratuberculosis, and who
288 guinea pigs were infected with M. bovis BCG, Mycobacterium avium, the attenuated Mycobacterium tuberc
291 ed mouse macrophages to resist the growth of Mycobacterium avium via alpha(2)-adrenergic stimulation.
292 nce strain 104 of the opportunistic pathogen Mycobacterium avium was isolated from an adult AIDS pati
293 effect described in macrophages infected by Mycobacterium avium was not observed in our model, which
297 pportunistic pathogens Toxoplasma gondii and Mycobacterium avium when administered via the i.p. or i.
298 e demonstrated improved growth inhibition of Mycobacterium avium when incubated with exogenous granul
299 s contrasted with Gram-positive bacteria and Mycobacterium avium, which activated cells via TLR2 but
300 valuated the effect of iron on the growth of Mycobacterium avium within macrophages as well as on the
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