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1 tic plasmids from the opportunistic pathogen Mycobacterium avium.
2 was identified in the opportunistic pathogen Mycobacterium avium.
3  of TLR2 expression following infection with Mycobacterium avium.
4 variety of intracellular pathogens including Mycobacterium avium.
5 s tested, including Mycobacterium leprae and Mycobacterium avium.
6 ase mediated by the intramacrophage pathogen Mycobacterium avium.
7 TLR) 2 mRNA was induced after infection with Mycobacterium avium.
8 s virus stimulated by in vivo infection with Mycobacterium avium.
9 llowing infection of murine macrophages with Mycobacterium avium.
10 rium bovis bacillus Calmette-Guerin (BCG) or Mycobacterium avium.
11  It is about 100-fold less effective against Mycobacterium avium.
12 nt, phenol treatment, and contamination with Mycobacterium avium.
13 virulent (SmD) or virulent (SmT) variants of Mycobacterium avium 2-151.
14 ently were: Mycobacterium mucogenicum (52%), Mycobacterium avium (30%), and Mycobacterium gordonae (2
15      Infection of HIV-1-transgenic mice with Mycobacterium avium, a common opportunistic pathogen in
16  smegmatis and to a lesser extent pathogenic Mycobacterium avium, activate Ca(2+)-dependent calmoduli
17  restored antimycobacterial activity against Mycobacterium avium and Mycobacterium bovis Bacille Calm
18 thin 30 min and 30 to 60% after 3 h, whereas Mycobacterium avium and Mycobacterium fortuitum isolates
19                                              Mycobacterium avium and Mycobacterium intracellulare are
20  The clinical significance and prevalence of Mycobacterium avium and Mycobacterium intracellulare wer
21                                              Mycobacterium avium and Mycobacterium tuberculosis are h
22 olipids (GPLs), a major surface component of Mycobacterium avium and other non-tuberculosis mycobacte
23 s complex and to have homology with DNA from Mycobacterium avium and other nontuberculous mycobacteri
24 R2-dependent responses were seen using whole Mycobacterium avium and Staphylococcus aureus, demonstra
25 ples yielded cultures of Candida lusitaniae, Mycobacterium avium, and a filamentous fungus, Trichophy
26 lass II promoter (hu10Tg) were infected with Mycobacterium avium, and bacterial burdens and immune re
27 c infections caused by Pneumocystis carinii, Mycobacterium avium, and Campylobacter coli that require
28 /bg(-) mice were infected intravenously with Mycobacterium avium, and cultures of blood and brain as
29 7(-/-) and wild-type mice were infected with Mycobacterium avium, and host responses were analyzed.
30 er, Pneumocystis carinii, Toxoplasma gondii, Mycobacterium avium, and Lactobacillus casei) and showed
31 rol organisms included Mycobacterium simiae, Mycobacterium avium, and Mycobacterium xenopi.
32                      Legionella pneumophila, Mycobacterium avium, and Pseudomonas aeruginosa are oppo
33 rtunistic pathogens (Legionella pneumophila, Mycobacterium avium, and Pseudomonas aeruginosa), broade
34 rom Pneumocystis carinii, Toxoplasma gondii, Mycobacterium avium, and rat liver.
35               Mycobacterium tuberculosis and Mycobacterium avium are facultative intracellular pathog
36 ag2(-/-)gammac(-/-) mice are as resistant to Mycobacterium avium as Rag2(-/-) mice, whereas Rag2(-/-)
37 duced in response to live M. tuberculosis or Mycobacterium avium as well as certain mycobacterial pro
38            CD43-transfected HeLa cells bound Mycobacterium avium, but not Salmonella typhimurium or S
39 ues from both Mycobacterium tuberculosis and Mycobacterium avium can complement lpqM donor mutants, s
40      Previous studies have demonstrated that Mycobacterium avium can invade intestinal epithelial cel
41                                              Mycobacterium avium causes disseminated disease in human
42                                              Mycobacterium avium causes disseminated infection in pat
43 against DHFR from both Toxoplasma gondii and Mycobacterium avium compared to mammalian DHFR.
44 rophages following infection with pathogenic Mycobacterium avium compared to the activation following
45 rophages infected with pathogenic strains of Mycobacterium avium compared with infections with the fa
46       The most common species recovered were Mycobacterium avium complex (69 isolates) and Mycobacter
47                                              Mycobacterium avium complex (72%) and Mycobacterium absc
48  (100%), CD4 cell count less than 200 (84%), Mycobacterium avium complex (73%), and Pneumocystis cari
49   We report a case of recurrent disseminated Mycobacterium avium complex (DMAC) disease with anti-gam
50                       Whether infection with Mycobacterium avium complex (MAC) among patients with ac
51 e cases, 122 (64%) were culture-positive for Mycobacterium avium complex (MAC) and 69 (36%) for M. ab
52         Of 600 sets tested, 85 (14%) yielded Mycobacterium avium complex (MAC) and 9 (2%) yielded oth
53          In lymphoid tissues coinfected with Mycobacterium avium complex (MAC) and HIV-1, increased v
54 f 2,4-diamino-5-deazapteridine inhibitors of Mycobacterium avium complex (MAC) and human dihydrofolat
55                               Members of the Mycobacterium avium complex (MAC) are important environm
56                                              Mycobacterium avium complex (MAC) are opportunistic resp
57        To determine the relationship between Mycobacterium avium complex (MAC) bacteremia and HIV RNA
58 type 1-infected persons with newly diagnosed Mycobacterium avium complex (MAC) bacteremia were enroll
59                     Eight AIDS patients with Mycobacterium avium complex (MAC) bacteremia were random
60 y virus type 1 (HIV-1)-infected persons with Mycobacterium avium complex (MAC) bacteremia, the levels
61 reproducibility of susceptibility testing of Mycobacterium avium complex (MAC) by broth microdilution
62 r antimycobacterial therapy for disseminated Mycobacterium avium complex (MAC) could be withdrawn fro
63 n alone and in combination for prevention of Mycobacterium avium complex (MAC) disease were compared
64 in is a major drug used for the treatment of Mycobacterium avium complex (MAC) disease, but standard
65        Among MSM, the most frequent OIs were Mycobacterium avium complex (MAC) disease, Pneumocystis
66 effective drug regimens for the treatment of Mycobacterium avium complex (MAC) disease.
67     Although opportunistic infections due to Mycobacterium avium complex (MAC) have been less common
68                         Persistent growth of Mycobacterium avium complex (MAC) in the lungs indicates
69                                              Mycobacterium avium complex (MAC) infection is the most
70                     The relationship between Mycobacterium avium complex (MAC) infection of blood and
71                                          The Mycobacterium avium complex (MAC) is an important cause
72                                          The Mycobacterium avium complex (MAC) is an important group
73                       Lung disease caused by Mycobacterium avium complex (MAC) is increasing in preva
74                      Rapid identification of Mycobacterium avium complex (MAC) is possible by use of
75 robiologic cure of AIDS-related disseminated Mycobacterium avium complex (MAC) is possible in patient
76                                              Mycobacterium avium complex (MAC) is the most common dis
77                                              Mycobacterium avium complex (MAC) isolates among patient
78  features and outcome of macrolide-resistant Mycobacterium avium complex (MAC) lung disease are not k
79 in prospective macrolide treatment trials of Mycobacterium avium complex (MAC) lung disease were asse
80 household water sources for 36 patients with Mycobacterium avium complex (MAC) lung disease were eval
81 cs and to evaluate relapses in patients with Mycobacterium avium complex (MAC) lung disease, but the
82 atment of noncavitary nodular bronchiectatic Mycobacterium avium complex (MAC) lung disease, supporti
83                         Organisms within the Mycobacterium avium complex (MAC) may have differential
84                                              Mycobacterium avium complex (MAC) organisms cause dissem
85     The clinical significance of recovery of Mycobacterium avium complex (MAC) organisms from respira
86                   The numbers of isolates of Mycobacterium avium complex (MAC) recovered were 172 (10
87    Species identification of isolates of the Mycobacterium avium complex (MAC) remains a difficult ta
88                                 Adherence of Mycobacterium avium complex (MAC) to human respiratory e
89                                              Mycobacterium avium complex (MAC) within macrophages und
90 actors that contribute to protection against Mycobacterium avium complex (MAC), cytokine production b
91  genetically similar to other members of the Mycobacterium avium complex (MAC), some of which are non
92 y (HAART) on cell-mediated immunity (CMI) to Mycobacterium avium complex (MAC), we measured immune re
93                             Organisms in the Mycobacterium avium complex (MAC; M. avium, M. intracell
94 ow-growing species, including members of the Mycobacterium avium complex (MAVC).
95 s with Emberger syndrome, monocytopenia with Mycobacterium avium complex (MonoMAC), and MDS.
96 mocystis carinii (n = 26), bacteria (n = 3), Mycobacterium avium complex (n = 2), Nocardia sp. (n = 1
97 l disease (mean number of organs infected by Mycobacterium avium complex 4.1 [SD 0.8] vs 2.0 [1.1], p
98 tory pulmonary nontuberculous mycobacterial (Mycobacterium avium complex [MAC] or Mycobacterium absce
99                      One hundred isolates of Mycobacterium avium complex and eight M. simiae isolates
100                          Prophylaxis against Mycobacterium avium complex can safely be withdrawn or w
101                                              Mycobacterium avium complex cultures, CD4(+) cell counts
102 ttributable to cytomegalovirus retinitis and Mycobacterium avium complex declined over time (p=0.0058
103 Calif.) to detect Mycobacterium gordonae and Mycobacterium avium complex directly in liquid medium fl
104   Several agents are effective in preventing Mycobacterium avium complex disease in patients with adv
105 phylaxis versus withdrawal for prevention of Mycobacterium avium complex disease.
106 g the pseudocording, or loose aggregation of Mycobacterium avium complex from M. tuberculosis and the
107  safety of discontinuing prophylaxis against Mycobacterium avium complex has been uncertain.
108 yelitis (patient 1) and disseminated CMV and Mycobacterium avium complex infection (patient 2), respe
109 IS event in HIV-infected patients, unmasking Mycobacterium avium complex infection after starting ant
110                                          The Mycobacterium avium complex is a source of disseminated
111                               Infection with Mycobacterium avium complex is acquired from the environ
112              A major phenotypic trait of the Mycobacterium avium complex is the ability to produce ro
113 were evaluated for susceptibility testing of Mycobacterium avium complex isolates against clarithromy
114  ribosomal internal transcribed spacer of 56 Mycobacterium avium complex isolates from pediatric pati
115 g activities seem not to be risk factors for Mycobacterium avium complex lung disease in HIV-negative
116                                              Mycobacterium avium complex lung disease is an increasin
117 omponent of multidrug treatment regimens for Mycobacterium avium complex lung disease.
118 ia, esophageal candidiasis, and disseminated Mycobacterium avium complex or Mycobacterium kansasii in
119                Ten of 639 MGIT cultures grew Mycobacterium avium complex or Mycobacterium kansasii, h
120 hose previously treated for tuberculosis and Mycobacterium avium complex predominated (27.7% [95% CI:
121                                              Mycobacterium avium complex pulmonary disease (MAC-PD) i
122 new therapy for Candida esophagitis, whereas Mycobacterium avium complex therapy may be discontinued
123                                              Mycobacterium avium complex was concomitantly isolated i
124                                              Mycobacterium avium complex was seen in 75% of NTM-posit
125                              Isolates of the Mycobacterium avium complex were examined for hemolysin
126     Nearly 60% of positive cultures were for Mycobacterium avium complex, although this ranged by sta
127 , 14% (1/7) for Cryptococcus, 10% (1/10) for Mycobacterium avium complex, and 4% (3/72) for PCP.
128 ifferent Mycobacterium tuberculosis complex, Mycobacterium avium complex, and Mycobacterium spp. dire
129 neumocystis jeroveci pneumonia, disseminated Mycobacterium avium complex, lymphoid interstitial pneum
130 e closely related organisms and comprise the Mycobacterium avium complex.
131 tis carinii pneumonia [PCP] and disseminated Mycobacterium avium-complex [MAC] infection) in persons
132         HIV-infected patients with untreated Mycobacterium avium-complex diarrhea are associated with
133 ents and household water/biofilm isolates of Mycobacterium avium could be matched by DNA fingerprinti
134             Of the two common morphotypes of Mycobacterium avium, designated smooth transparent (SmT)
135 Pneumocystis carinii, Toxoplasma gondii, and Mycobacterium avium DHFR vs rat DHFR.
136 hat represent 46 genes that are expressed by Mycobacterium avium during growth in human macrophages.
137 tosporidium parvum, Enterocytozoon bieneusi, Mycobacterium avium, Entamoeba histolytica, Balantidium
138 imensional gel electrophoresis revealed that Mycobacterium avium expresses several proteins unique to
139 n binding motif of the previously identified Mycobacterium avium fibronectin attachment protein (FAP-
140 ate of MTB from a nonTB species of the genus Mycobacterium avium grown in liquid culture media.
141 ating inflammation induced by infection with Mycobacterium avium in human primary macrophages.
142 vation of Janus Kinase (JAK)-STAT pathway in Mycobacterium avium-infected macrophages.
143                           Here, we show that Mycobacterium avium-infected T cell-deficient mice injec
144                                 Disseminated Mycobacterium avium infection is common in AIDS patients
145                          Active disseminated Mycobacterium avium infection may significantly compromi
146                  We previously reported that Mycobacterium avium infection of mouse macrophages decre
147                                 Disseminated Mycobacterium avium infection persisted and the patient
148      In this study, 30 AIDS patients without Mycobacterium avium infection were randomized to receive
149 ceptor (TLR) signaling in host resistance to Mycobacterium avium infection, mice deficient in the TLR
150            Here, we report on a patient with Mycobacterium avium infection, recurrent Staphylococcus
151 ere we show, using an in vivo mouse model of Mycobacterium avium infection, that an increased proport
152                     Using the mouse model of Mycobacterium avium infection, we show in this study tha
153 d whether ISS-ODN could modify the course of Mycobacterium avium infection.
154 s evaluated in a mouse model of disseminated Mycobacterium avium infection.
155 sis pulmonary hypersensitive pneumonitis and Mycobacterium avium infections.
156                                  Isolates of Mycobacterium avium-intracellulare (MAI) form multiple c
157                                              Mycobacterium avium-intracellulare complex (MAC) is one
158  that most resemble Mycobacterium simiae and Mycobacterium avium-intracellulare complex but which pos
159  These included a psoas abscess secondary to Mycobacterium avium-intracellulare, septic wrist, bacter
160                                              Mycobacterium avium is a common opportunistic infection
161 Invasion of intestinal mucosa of the host by Mycobacterium avium is a critical step in pathogenesis a
162                                              Mycobacterium avium is a facultative intracellular patho
163                                              Mycobacterium avium is a major opportunistic pathogen in
164                                              Mycobacterium avium is a major opportunistic pathogen of
165                                              Mycobacterium avium is abundant in the environment.
166                                              Mycobacterium avium is an intracellular pathogen that ha
167                                              Mycobacterium avium is an opportunistic pathogen in AIDS
168                                              Mycobacterium avium is capable of invading the intestina
169 terium tuberculosis, Mycobacterium leprae or Mycobacterium avium is correlated with strong inflammato
170  The cell wall of the environmental pathogen Mycobacterium avium is important to its virulence and in
171                                              Mycobacterium avium is the most frequent cause of dissem
172                                  The role of Mycobacterium avium isolates in modulating human immunod
173                          The LRF patterns of Mycobacterium avium isolates recovered from potable wate
174                                              Mycobacterium avium (M. avium) subspecies vary widely in
175  pj), Toxoplasma gondii (T. gondii, tg), and Mycobacterium avium (M. avium, ma) are the principal cau
176 vide a tool to better understand the role of Mycobacterium avium-M. intracellulare complex isolates i
177                                      The 159 Mycobacterium avium-M. intracellulare complex isolates w
178                Additional differentiation of Mycobacterium avium-M. intracellulare complex strains in
179 cystis carinii (Pc), Toxoplasma gondii (Tg), Mycobacterium avium (Ma), and rat liver in comparison wi
180 is carinii (Pc), Toxoplasma gondii (Tg), and Mycobacterium avium (Ma), three life-threatening pathoge
181 is carinii (Pc), Toxoplasma gondii (Tg), and Mycobacterium avium (Ma), three of the opportunistic org
182 is carinii (Pc), Toxoplasma gondii (Tg), and Mycobacterium avium (Ma), three of the opportunistic pat
183 r activity against M. tuberculosis (Mtb) and Mycobacterium avium (MAC) are reported.
184 arinii (pcDHFR), Toxoplasma gondii (tgDHFR), Mycobacterium avium (maDHFR), and rat liver (rlDHFR).
185 fected with different mycobacterial strains (Mycobacterium avium, Mycobacterium bovis BCG or Mtb), we
186 erium smegmatis, Mycobacterium tuberculosis, Mycobacterium avium, Mycobacterium gadium and Mycobacter
187 PP (Legionella spp., Legionella pneumophila, Mycobacterium avium, Mycobacterium intracellulare, Pseud
188                                              Mycobacterium avium, Mycobacterium tuberculosis, and Myc
189                       Infection of mice with Mycobacterium avium or immunization with a novel PE gene
190 lpha) production in macrophage infected with Mycobacterium avium or M smegmatis is dependent on myelo
191 erived macrophages were infected with either Mycobacterium avium or Mycobacterium tuberculosis and th
192                     Later, the mice received Mycobacterium avium or Mycobacterium tuberculosis I.T.
193 nse in the thymus following dissemination of Mycobacterium avium or Mycobacterium tuberculosis.
194 drome, and opportunistic infections, such as Mycobacterium avium or Pneumocystis carinii infections.
195 sease in humans have led to speculation that Mycobacterium avium paratuberculosis (MAP) might be a ca
196                          When tested against Mycobacterium avium, PCIH was more effective than INH at
197           A structural genomics protein from Mycobacterium avium (PDB ID 3q1t) has been reported to b
198              The role that colonization with Mycobacterium avium plays in the development of dissemin
199 hat primary murine macrophages infected with Mycobacterium avium produced lower levels of tumor necro
200              T cells from mice infected with Mycobacterium avium responded to FAP-A, suggesting a pos
201  primary murine macrophages had no effect on Mycobacterium avium retention in an early endosomal comp
202  human macrophages respond to infection with Mycobacterium avium, serovar 4, by producing tumor necro
203 olipids (GPLs), a major surface component of Mycobacterium avium, showed limited acidification and de
204 cal survey of distribution in Great Britain, Mycobacterium avium ssp. paratuberculosis (MAP) was dete
205 time in macrophages infected with pathogenic Mycobacterium avium strains relative to infections with
206 say that uses a unique element (ISMap02) for Mycobacterium avium sub0:36 PMparatuberculosis that is p
207                                              Mycobacterium avium subsp hominissuis is associated with
208               Johnes disease (JD), caused by Mycobacterium avium subsp paratuberculosis (MAP), occurs
209 evaluated for growth stimulating behavior in Mycobacterium avium subsp paratuberculosis.
210 obacterium avium subsp. paratuberculosis and Mycobacterium avium subsp. avium are antigenically and g
211 obacterium avium subsp. paratuberculosis and Mycobacterium avium subsp. avium are antigenically and g
212 tories has shown >98% sequence identity with Mycobacterium avium subsp. avium in some regions.
213 of Mycobacterium avium subsp. silvaticum and Mycobacterium avium subsp. avium, and a single isolate e
214                                              Mycobacterium avium subsp. hominissuis is an opportunist
215                                             "Mycobacterium avium subsp. hominissuis" is a robust and
216                                             "Mycobacterium avium subsp. hominissuis" is an opportunis
217 cosa by M. avium subsp. paratuberculosis and Mycobacterium avium subsp. hominissuis, a pathogen known
218 egration loci of IS900 (loci L1 and L9), one Mycobacterium avium subsp. paratuberculosis (M. paratube
219 R) sequencing approach for the genotyping of Mycobacterium avium subsp. paratuberculosis (M. paratube
220                  Presently, no drugs against Mycobacterium avium subsp. paratuberculosis (M. paratube
221 ing affinity of Ag85A, Ag85B, and Ag85C from Mycobacterium avium subsp. paratuberculosis (MAP) (K(D)
222  protocol was optimized for the isolation of Mycobacterium avium subsp. paratuberculosis (MAP) from m
223              Available diagnostic assays for Mycobacterium avium subsp. paratuberculosis (MAP) have p
224 r regulatory mechanisms of host responses to Mycobacterium avium subsp. paratuberculosis (MAP) infect
225    To investigate the stochastic dynamics of Mycobacterium avium subsp. paratuberculosis (MAP) infect
226                 Johne's disease is caused by Mycobacterium avium subsp. paratuberculosis (MAP) infect
227                                              Mycobacterium avium subsp. paratuberculosis (MAP), a slo
228 = 128), Mycobacterium kansasii (n = 10), and Mycobacterium avium subsp. paratuberculosis (n = 10), ca
229 SOD], and 35-kDa protein) were purified from Mycobacterium avium subsp. paratuberculosis and evaluate
230     Little is known of protein expression in Mycobacterium avium subsp. paratuberculosis and how this
231 nce repeats (SSRs) of 211 and 56 isolates of Mycobacterium avium subsp. paratuberculosis and M. avium
232                                              Mycobacterium avium subsp. paratuberculosis and Mycobact
233                                              Mycobacterium avium subsp. paratuberculosis and Mycobact
234               The genetic similarity between Mycobacterium avium subsp. paratuberculosis and other my
235  made between PBMCs stimulated in vitro with Mycobacterium avium subsp. paratuberculosis and PBMCs st
236 = 39) were tested for the presence of viable Mycobacterium avium subsp. paratuberculosis by a novel p
237        Efficient attachment and ingestion of Mycobacterium avium subsp. paratuberculosis by cultured
238                  Attachment and ingestion of Mycobacterium avium subsp. paratuberculosis by two epith
239                                              Mycobacterium avium subsp. paratuberculosis causes an en
240                                              Mycobacterium avium subsp. paratuberculosis causes Johne
241                                              Mycobacterium avium subsp. paratuberculosis causes Johne
242                               Infection with Mycobacterium avium subsp. paratuberculosis causes Johne
243                  With the genome sequence of Mycobacterium avium subsp. paratuberculosis determined,
244 ism (AFLP) to characterize the genomes of 20 Mycobacterium avium subsp. paratuberculosis field isolat
245  the isolation, separation, and detection of Mycobacterium avium subsp. paratuberculosis from milk an
246                                              Mycobacterium avium subsp. paratuberculosis has been inc
247                     The infection biology of Mycobacterium avium subsp. paratuberculosis has recently
248          Interactions between epithelium and Mycobacterium avium subsp. paratuberculosis have not bee
249 orescent probes (molecular beacons) detected Mycobacterium avium subsp. paratuberculosis in bovine fe
250                                              Mycobacterium avium subsp. paratuberculosis infection of
251                                              Mycobacterium avium subsp. paratuberculosis infection of
252  sensitivity and reduce time to diagnosis of Mycobacterium avium subsp. paratuberculosis infection.
253        In the present study, instillation of Mycobacterium avium subsp. paratuberculosis into the ton
254 and veterinary Johne's disease suggests that Mycobacterium avium subsp. paratuberculosis is a causati
255                               Infection with Mycobacterium avium subsp. paratuberculosis is associate
256                                              Mycobacterium avium subsp. paratuberculosis is genetical
257                                              Mycobacterium avium subsp. paratuberculosis is shed into
258                                              Mycobacterium avium subsp. paratuberculosis is the causa
259                                              Mycobacterium avium subsp. paratuberculosis is the causa
260                                              Mycobacterium avium subsp. paratuberculosis is the cause
261        Analysis of short sequence repeats of Mycobacterium avium subsp. paratuberculosis isolated fro
262 lar diversity of animal and human strains of Mycobacterium avium subsp. paratuberculosis isolated in
263  for the study of the genetic relatedness of Mycobacterium avium subsp. paratuberculosis isolates har
264 s, infection with the intracellular pathogen Mycobacterium avium subsp. paratuberculosis results in a
265 nd/or enrichment methods on the selection of Mycobacterium avium subsp. paratuberculosis subtypes.
266                                Attachment of Mycobacterium avium subsp. paratuberculosis to host tiss
267                   Five pigmented isolates of Mycobacterium avium subsp. paratuberculosis were examine
268 luate whether cows that were low shedders of Mycobacterium avium subsp. paratuberculosis were passive
269                                              Mycobacterium avium subsp. paratuberculosis, the agent o
270 omprised of crude whole-cell preparations of Mycobacterium avium subsp. paratuberculosis.
271 ease in cattle requires antigens specific to Mycobacterium avium subsp. paratuberculosis.
272 ntrol of mycobacterial infections, including Mycobacterium avium subsp. paratuberculosis.
273    The causative agent of Johne's disease is Mycobacterium avium subsp. paratuberculosis.
274 atuberculosis isolates, two isolates each of Mycobacterium avium subsp. silvaticum and Mycobacterium
275                                      MAP and Mycobacterium avium subspecies avium, a closely related
276                                  The role of Mycobacterium avium subspecies paratuberculosis (MAP) in
277 sheep, is caused by slow replicating bacilli Mycobacterium avium subspecies paratuberculosis (MAP) in
278 omplete genome sequence of a common clone of Mycobacterium avium subspecies paratuberculosis (Map) st
279                                              Mycobacterium avium subspecies paratuberculosis (MAP), t
280                   By the analysis of 14 SNPs Mycobacterium avium subspecies paratuberculosis isolates
281 uipment for the large-scale global typing of Mycobacterium avium subspecies paratuberculosis isolates
282 erformed using genome sequence data from 133 Mycobacterium avium subspecies paratuberculosis isolates
283 g-chain acyl-CoA carboxylase holoenzyme from Mycobacterium avium subspecies paratuberculosis revealed
284                                    Typing of Mycobacterium avium subspecies paratuberculosis strains
285   A number of intestinal pathogens including Mycobacterium avium subspecies paratuberculosis, adheren
286 ed possibilities for the characterization of Mycobacterium avium subspecies paratuberculosis, and who
287 acillary paratuberculosis are both caused by Mycobacterium avium subspecies paratuberculosis.
288 guinea pigs were infected with M. bovis BCG, Mycobacterium avium, the attenuated Mycobacterium tuberc
289                                              Mycobacterium avium, the most common opportunistic patho
290  however, even within a single subspecies of Mycobacterium avium these lipids can differ.
291 ed mouse macrophages to resist the growth of Mycobacterium avium via alpha(2)-adrenergic stimulation.
292 nce strain 104 of the opportunistic pathogen Mycobacterium avium was isolated from an adult AIDS pati
293  effect described in macrophages infected by Mycobacterium avium was not observed in our model, which
294                                        While Mycobacterium avium was once regarded as innocuous, its
295                                              Mycobacterium avium was recovered from 41 specimens; 36
296 f the fibrinolytic system and infectivity by Mycobacterium avium were analyzed.
297 pportunistic pathogens Toxoplasma gondii and Mycobacterium avium when administered via the i.p. or i.
298 e demonstrated improved growth inhibition of Mycobacterium avium when incubated with exogenous granul
299 s contrasted with Gram-positive bacteria and Mycobacterium avium, which activated cells via TLR2 but
300 valuated the effect of iron on the growth of Mycobacterium avium within macrophages as well as on the

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