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1 badgers Meles meles naturally infected with Mycobacterium bovis.
2 culosis (TB) is a zoonotic disease caused by Mycobacterium bovis.
3 the most frequently recognized antigens from Mycobacterium bovis.
4 with a strain of M. tuberculosis rather than Mycobacterium bovis.
5 ly differentiate between M. tuberculosis and Mycobacterium bovis.
6 approach and applied it to gene deletions in Mycobacterium bovis.
7 f gammadelta T cells in cattle infected with Mycobacterium bovis.
8 hallenge with purified protein derivative of Mycobacterium bovis.
9 lowing experimental infection of cattle with Mycobacterium bovis.
10 and intractable disease of cattle caused by Mycobacterium bovis.
11 h as Leishmania major, Escherichia coli, and Mycobacterium bovis.
12 is operon is specific to M. tuberculosis and Mycobacterium bovis.
13 he principal sigma factor (RpoV) of virulent Mycobacterium bovis, a member of the Mycobacterium tuber
14 Mycobacterium africanum (subtypes I and II), Mycobacterium bovis (along with the attenuated M. bovis
15 ity was observed with some compounds against Mycobacterium bovis and also against hepatitis C virus i
16 and detection platform for the diagnosis of Mycobacterium bovis and Brucella abortus infection simul
17 ere used to distinguish M. tuberculosis from Mycobacterium bovis and determine phylogenetic lineage.
18 H37Rv isolate and showed 99% identity to the Mycobacterium bovis and M. bovis BCG isolate sequences.
20 ized in the same way that molecular types of Mycobacterium bovis are geographically localized in catt
22 ability of whole blood to restrict growth of Mycobacterium bovis bacille Calmette Guerin and Mycobact
24 uency and numbers of CD8 T cells specific to Mycobacterium bovis bacille Calmette-Guerin (BCG) in the
25 ial activity against Mycobacterium avium and Mycobacterium bovis Bacille Calmette-Guerin (BCG) in vit
26 tion can interact with T cells responding to Mycobacterium bovis bacille Calmette-Guerin (BCG) infect
27 cts of an experimental iron-enriched diet on Mycobacterium bovis bacille Calmette-Guerin (BCG) infect
28 lta T cells displayed major expansion during Mycobacterium bovis Bacille Calmette-Guerin (BCG) infect
30 e and often poor protection conferred by the Mycobacterium bovis bacille Calmette-Guerin (BCG) vaccin
31 immunity could be assessed using intradermal Mycobacterium bovis bacille Calmette-Guerin (BCG) vaccin
34 xhibited nanomolar in vitro activity against Mycobacterium bovis bacille Calmette-Guerin and virulent
37 uperinfecting Mycobacterium tuberculosis and Mycobacterium bovis bacille Calmette-Guerin similarly ho
38 mycobacteria, Mycobacterium tuberculosis and Mycobacterium bovis bacille Calmette-Guerin, release MVs
41 ed (TNF-knockout (KO)) mice with recombinant Mycobacterium bovis bacillus Calmette Guerin (BCG) expre
44 the protection afforded by immunization with Mycobacterium bovis bacillus Calmette-Guerin (BCG) admin
46 AS01B, and compared this to vaccination with Mycobacterium bovis bacillus Calmette-Guerin (BCG) alone
47 these questions, cattle were vaccinated with Mycobacterium bovis bacillus Calmette-Guerin (BCG) and w
48 was phosphorylated in vivo when expressed in Mycobacterium bovis bacillus Calmette-Guerin (BCG) but n
49 nature of the peripheral cell wall lipids of Mycobacterium bovis bacillus Calmette-Guerin (BCG) by co
52 rial antigens on the intracellular growth of Mycobacterium bovis bacillus Calmette-Guerin (BCG) in hu
54 o track expression of the cytokine following Mycobacterium bovis bacillus Calmette-Guerin (BCG) infec
55 thway of macrophage activation during murine Mycobacterium bovis bacillus Calmette-Guerin (BCG) infec
57 ty-one controls were neither vaccinated with Mycobacterium bovis bacillus Calmette-Guerin (BCG) nor t
58 s of TNF and TNF receptors in the control of Mycobacterium bovis bacillus Calmette-Guerin (BCG) pleur
59 e bone marrow-derived DCs infected with live Mycobacterium bovis Bacillus Calmette-Guerin (BCG) produ
61 on heterologous prime-boost protocols using Mycobacterium bovis bacillus Calmette-Guerin (BCG) to pr
62 n immunodeficiency virus, and the failure of Mycobacterium bovis bacillus Calmette-Guerin (BCG) to pr
64 d the ability of human antibodies induced by Mycobacterium bovis bacillus Calmette-Guerin (BCG) vacci
67 eprae, Mycobacterium tuberculosis (Mtb), and Mycobacterium bovis bacillus Calmette-Guerin (BCG) were
68 eleted from the genome of the vaccine strain Mycobacterium bovis bacillus Calmette-Guerin (BCG), and
69 ients, yet the current tuberculosis vaccine, Mycobacterium bovis bacillus Calmette-Guerin (BCG), is c
71 rating exudate macrophages to infection with Mycobacterium bovis bacillus Calmette-Guerin (BCG), peak
72 g a strain deleted for the KATmt ortholog in Mycobacterium bovis Bacillus Calmette-Guerin (BCG), we s
81 (Mphi) isolated from mice given heat-killed Mycobacterium bovis bacillus Calmette-Guerin (HK-BCG) i.
83 the human immune responses to infection with Mycobacterium bovis bacillus Calmette-Guerin and Mycobac
84 f mice made immune by prior vaccination with Mycobacterium bovis bacillus Calmette-Guerin compared wi
86 ncurrent Th1 responses to influenza virus or Mycobacterium bovis bacillus Calmette-Guerin had no effe
87 MHC-restricted T cells during infection with Mycobacterium bovis bacillus Calmette-Guerin in mice.
90 unt adaptive immune responses in response to Mycobacterium bovis bacillus Calmette-Guerin infections.
91 tion of M. leprae DNA enhanced nonpathogenic Mycobacterium bovis bacillus Calmette-Guerin intracellul
92 ined the set of genes required for growth of Mycobacterium bovis bacillus Calmette-Guerin on minimal
93 mediated immunity by live attenuated vaccine Mycobacterium bovis bacillus Calmette-Guerin or the adop
94 36-wk-old infants, was incubated with viable Mycobacterium bovis bacillus Calmette-Guerin or TLR liga
95 after aerosol immunization with recombinant Mycobacterium bovis bacillus Calmette-Guerin overexpress
96 ine model that involves immunizing mice with Mycobacterium bovis bacillus Calmette-Guerin to augment
97 genous Ag-specific CD4(+) T cells induced by Mycobacterium bovis bacillus Calmette-Guerin vaccination
98 ion of mice with the intracellular bacterium Mycobacterium bovis bacillus Calmette-Guerin, macrophage
99 cobacterium tuberculosis and by bcg_1279c in Mycobacterium bovis bacillus Calmette-Guerin, plays an i
104 ed with the current live attenuated vaccine, Mycobacterium bovis-bacillus Calmette-Guerin as well as
105 tiate between Mycobacterium tuberculosis and Mycobacterium bovis based on their relative virulence in
107 ll have to be evaluated against the existing Mycobacterium bovis BCG "gold standard." It is therefore
108 Here, we characterize the glycolipids of Mycobacterium bovis BCG (BCG) that are released into mur
110 inigenes through combinations of recombinant Mycobacterium bovis BCG (rBCG), electroporated recombina
111 aque models of simian immunodeficiency virus-Mycobacterium bovis BCG (SIV/BCG) coinfection were emplo
112 e subcutaneously immunized with DeltafbpA or Mycobacterium bovis BCG and challenged with M. tuberculo
113 ock-in [KI] mice) to determine resistance to Mycobacterium bovis BCG and M. tuberculosis infections a
115 m tuberculosis antigens that are absent from Mycobacterium bovis BCG and most environmental mycobacte
117 on in vivo, we evaluated the consequences of Mycobacterium bovis BCG and staphylococcal enterotoxin B
118 on of AFB smears, sputum samples spiked with Mycobacterium bovis BCG at 5 x 10(8) CFU/ml produced 16
120 The phiRv1 element is not only absent from Mycobacterium bovis BCG but is in different locations wi
121 s infection, mice were immunized with viable Mycobacterium bovis BCG by the aerosol or intravenous ro
123 ciency virus (SIVmac) can develop persistent Mycobacterium bovis BCG coinfection and a fatal SIV-rela
124 of simian immunodeficiency virus (SIV(mac))-Mycobacterium bovis BCG coinfection were employed to exp
127 the mycobacterial antigen 85A (rAd85A), with Mycobacterium bovis BCG followed by rAd85A heterologous
128 rs among ~200 binding regions throughout the Mycobacterium bovis BCG genome, were identified using Ch
129 t of exogenous cAMP on protein expression in Mycobacterium bovis BCG grown under hypoxic versus ambie
132 k of macroscopic pathology, and a history of Mycobacterium bovis BCG immunization was associated with
133 ly inhibit intracellular bacterial growth of Mycobacterium bovis BCG in macrophages (MPhi) in cocultu
135 In this study, we examined the growth of Mycobacterium bovis BCG in the lungs under experimental
136 gen-specific Vgamma2Vdelta2(+) T cells after Mycobacterium bovis BCG infection and BCG reinfection, r
137 In this study we examined the effect of Mycobacterium bovis BCG infection on the L-arginine-depe
140 letion of the ClpP1P2 level in a conditional Mycobacterium bovis BCG mutant enhanced killing by ADEP
142 Treatment of mice with heat-killed (HK) Mycobacterium bovis BCG or 1- to 10-microm chitin partic
143 d iniC (Rv 0341 and Rv 0343) by treatment of Mycobacterium bovis BCG or M. tuberculosis with INH or E
144 mycobacterial strains (Mycobacterium avium, Mycobacterium bovis BCG or Mtb), were exposed to encapsu
147 s of published data for two species; namely, Mycobacterium bovis BCG Pasteur and Mycobacterium smegma
148 n in a PE_PGRS gene (1818(PE_PGRS)) found in Mycobacterium bovis BCG Pasteur, which is the BCG homolo
153 this study we observe that strain AS-1, the Mycobacterium bovis BCG strain lacking the Rv0522 gene,
154 phages, attenuated M. tuberculosis H37Ra and Mycobacterium bovis BCG strongly induce THP-1 apoptosis,
155 fected with low or high doses of recombinant Mycobacterium bovis BCG that secreted murine TNF-alpha (
157 culosis promoter-lacZ reporter constructs in Mycobacterium bovis BCG under conditions of ambient air
158 5% carbon dioxide was required for growth of Mycobacterium bovis BCG under microaerophilic (1.3% O(2)
159 tensively in assessing novel vaccines, since Mycobacterium bovis BCG vaccination effectively prolongs
161 The World Health Organization recommends Mycobacterium bovis BCG vaccination in areas of high tub
163 her primary nor memory immunity conferred by Mycobacterium bovis BCG vaccination was affected in mice
166 ctive capacity as a potential adjunct to the Mycobacterium bovis BCG vaccine in the mouse and guinea
169 Beijing lineage strains) may be resistant to Mycobacterium bovis BCG vaccine-induced antituberculosis
174 The phosphatase activity in whole cells of Mycobacterium bovis BCG was significantly less than that
175 the TPA combination, the Ub combination, and Mycobacterium bovis BCG were able to limit the growth of
176 isolated PAS-resistant transposon mutants of Mycobacterium bovis BCG with insertions in the thymidyla
177 prepared and evaluated for activity against Mycobacterium bovis BCG with the thiourea-containing iso
178 tions, the highly related but non-pathogenic Mycobacterium bovis BCG yields partially ( approximately
180 ficient lysis of Mycobacterium tuberculosis, Mycobacterium bovis BCG, and Mycobacterium marinum.
181 atory strains of Mycobacterium tuberculosis, Mycobacterium bovis BCG, and Mycobacterium smegmatis.
182 an macrophages could survive infection, kill Mycobacterium bovis BCG, and severely limit the replicat
183 be propagated in Mycobacterium smegmatis and Mycobacterium bovis BCG, and their compatibility with ot
184 dimycocerosyl phthiocerol, were cloned from Mycobacterium bovis BCG, and their promoters were analyz
185 from virulent M. tuberculosis and attenuated Mycobacterium bovis BCG, bacteria were grown in broth cu
188 accine against tuberculosis, live attenuated Mycobacterium bovis BCG, has variable efficacy, but deve
189 ine cocktail or with the current TB vaccine, Mycobacterium bovis BCG, induced considerable antituberc
190 gues exhibited balanced profiles of potency (Mycobacterium bovis BCG, M tuberculosis H37Rv), selectiv
191 g to the Corynebacterineae suborder, namely, Mycobacterium bovis BCG, Mycobacterium smegmatis, and Co
193 , metabolome profiling in the Mtb surrogate, Mycobacterium bovis BCG, reveals significant changes in
194 st everyone is vaccinated early in life with Mycobacterium bovis BCG, the currently available vaccine
195 4(+) T cells to suppress T cell responses to Mycobacterium bovis BCG, the live vaccine that provides
197 ied a new cAMP-associated regulon in Mtb and Mycobacterium bovis BCG, which is distinct from the prev
199 studied granuloma formation after 6 weeks in Mycobacterium bovis BCG-infected CD28- and CD40 ligand (
200 tudy we analyzed humoral immune responses in Mycobacterium bovis BCG-vaccinated and control cattle (i
201 le of splenocytes restimulated in vitro from Mycobacterium bovis BCG-vaccinated and naive animals.
202 macrophages harvested from nonvaccinated and Mycobacterium bovis BCG-vaccinated guinea pigs were infe
205 sentially equivalent to the survival time of Mycobacterium bovis BCG-vaccinated mice (294 +/- 15 days
220 cterium tuberculosis and its close relative, Mycobacterium bovis (BCG) contain five genes whose predi
221 and is not functional in the closely related Mycobacterium bovis because of an inactivating frameshif
224 icans hyphae and extracellular aggregates of Mycobacterium bovis, but not in response to small yeast
225 to explore opportunities for transmission of Mycobacterium bovis [causal agent of bovine tuberculosis
228 f BCG against Mycobacterium tuberculosis and Mycobacterium bovis challenge in animal models, for effi
231 test is compromised by vaccination with the Mycobacterium bovis-derived vaccine strain bacille Calme
232 , and their assessment as typing tools in 47 Mycobacterium bovis field isolates and nine MTBC strains
234 notyping techniques that have differentiated Mycobacterium bovis from Mycobacterium tuberculosis sinc
235 mental models of protective immunity against Mycobacterium bovis: (i) vaccination with M. bovis BCG a
237 orescence for rapid, definitive detection of Mycobacterium bovis in lymph node specimens from 38 catt
240 s adenylate cyclase (CyaA) are recognized by Mycobacterium bovis-infected cattle more effectively tha
242 ngly recognized by cattle with early primary Mycobacterium bovis infection and by healthy MTB-sensiti
243 ods capable of detecting and differentiating Mycobacterium bovis infection from other pathogenic and
244 ategy in countries where there is persistent Mycobacterium bovis infection in wildlife and in develop
251 he surface of Mycobacterium tuberculosis and Mycobacterium bovis, initiates responses that can lead b
257 The bacillus Calmette-Guerin (BCG) strain of Mycobacterium bovis is used in many parts of the world a
259 Calmette-Guerin (BCG), an attenuated form of Mycobacterium bovis, is associated with persistent activ
260 lmette-Guerin (BCG), an attenuated strain of Mycobacterium bovis, is widely used as adjunctive therap
262 dy set of 180 Mycobacterium tuberculosis and Mycobacterium bovis isolates having low copy numbers of
263 the fundamental in vitro characteristics of Mycobacterium bovis--its requirement for pyruvate in gly
265 duced greater IL-22 and IL-17A production in Mycobacterium bovis (M. bovis)-infected cattle compared
266 Although the bovine tuberculosis (TB) agent, Mycobacterium bovis, may infect humans and cause disease
267 etween 72 and 96 h), in cattle infected with Mycobacterium bovis (n = 22) and animals sensitized by e
268 s to determine the minimum infective dose of Mycobacterium bovis necessary to stimulate specific immu
270 ma formation in response to bead-immobilized Mycobacterium bovis-purified protein derivative in aged
271 e to reproducibly generate cavities by using Mycobacterium bovis Ravenel, M. bovis AF2122, M. bovis B
272 .C.13, H37Rv, and H37Ra) and two isolates of Mycobacterium bovis (Ravenel and BCG) to reactive oxygen
274 ty, and both the incidence and prevalence of Mycobacterium bovis show marked variation in space.
275 advantages, we investigated the potential of Mycobacterium bovis-specific antigens to stimulate delay
276 ular composition and bacterial protection in Mycobacterium bovis strain bacille Calmette-Guerin (BCG)
277 both acute and chronic granulomas induced by Mycobacterium bovis strain bacillus Calmette-Guerin (BCG
279 4 as a selective agent, transposon-generated Mycobacterium bovis strain BCG (M. bovis) mutants that c
281 ct 10 colony-forming units of the attenuated Mycobacterium bovis strain BCG in human sputum in the pr
284 city of rough morphology M. tuberculosis and Mycobacterium bovis strains was greater than smooth "M.
285 creasingly recognized MtbC groupings include Mycobacterium bovis subsp. caprae and "Mycobacterium tub
286 ette-Guerin (BCG) is an attenuated strain of Mycobacterium bovis that is used widely as a vaccine for
287 rculosis vaccine, is an attenuated mutant of Mycobacterium bovis that was isolated after serial subcu
289 (Meles meles) are implicated in transmitting Mycobacterium bovis, the causative agent of bovine tuber
290 a series of attempts to limit the spread of Mycobacterium bovis, the causative agent of bovine tuber
295 tantial evidence suggests that the burden of Mycobacterium bovis, the cause of bovine tuberculosis, m
297 vage pathway of the bovine tubercle bacillus Mycobacterium bovis was reported defective due to a muta
298 us, 1758) population naturally infected with Mycobacterium bovis, we built an integrated population m
299 fected with either M. avium subsp. avium and Mycobacterium bovis were exposed to the array to identif
300 ore than 50 million cattle are infected with Mycobacterium bovis worldwide, resulting in severe econo
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