ライフサイエンスコーパス: Mycobacterium smegmatis

1 nd Ms1712 lipoproteins of the model organism Mycobacterium smegmatis.

2 the poles in Mycobacterium tuberculosis and Mycobacterium smegmatis.

3 e the functional role of the CtpD protein of Mycobacterium smegmatis.

4 rium tuberculosis and its avirulent relative Mycobacterium smegmatis.

5 is of the mbt gene cluster ortholog found in Mycobacterium smegmatis.

6 membrane, midcell, and cell pole regions in Mycobacterium smegmatis.

7 but is important for proper cell division in Mycobacterium smegmatis.

8 conjugal DNA transfer process that occurs in Mycobacterium smegmatis.

9 longer restricts growth of the non-pathogen Mycobacterium smegmatis.

10 459c from M. tuberculosis and MSMEG3120 from Mycobacterium smegmatis.

11 ue of Rv3792 and a gene upstream of embC) in Mycobacterium smegmatis.

12 cidal activity in a whole-cell assay against Mycobacterium smegmatis.

13 ied an IS6110-related element in a strain of Mycobacterium smegmatis.

14 f genes involved in phosphate acquisition in Mycobacterium smegmatis.

15 and Omega, two related mycobacteriophages of Mycobacterium smegmatis.

16 thogenic, saprophytic mycobacterial species, Mycobacterium smegmatis.

17 eria, including the nonpathogenic saprophyte Mycobacterium smegmatis.

18 doglycan from Mycobacterium tuberculosis and Mycobacterium smegmatis.

19 ompared to cells infected with nonpathogenic Mycobacterium smegmatis.

20 carrier protein synthases, KasA and KasB, in Mycobacterium smegmatis.

21 cillus subtilis, Bacillus thuringiensis, and Mycobacterium smegmatis.

22 ects of a chromosomal DNA transfer system in Mycobacterium smegmatis.

23 ach of both Mycobacterium intracellulare and Mycobacterium smegmatis.

24 t occurs in the model mycobacterial organism Mycobacterium smegmatis.

25 m tuberculosis, Mycobacterium bovis BCG, and Mycobacterium smegmatis.

26 ation following infection with nonpathogenic Mycobacterium smegmatis.

27 taphylococcus aureus, Bacillus subtilis, and Mycobacterium smegmatis.

28 transfer occurs by an atypical mechanism in Mycobacterium smegmatis.

29 is a temperate phage that forms lysogens in Mycobacterium smegmatis.

30 ds indicated that the species was similar to Mycobacterium smegmatis.

31 prior evidence for alkaline phosphatases in Mycobacterium smegmatis.

32 fied to be the second (p)ppGpp synthetase in Mycobacterium smegmatis.

33 expression in both M. tuberculosis H37Rv and Mycobacterium smegmatis.

34 molecules in Mycobacterium tuberculosis and Mycobacterium smegmatis.

35 hole cells of Mycobacterium tuberculosis and Mycobacterium smegmatis.

36 ied and characterized the MceG orthologue of Mycobacterium smegmatis.

37 erial species, including the closely related Mycobacterium smegmatis.

38 lated, sequenced, and compared 627 phages of Mycobacterium smegmatis.

39 not found in its ortholog from nonpathogenic Mycobacterium smegmatis.

40 selected and expressed in the surrogate host Mycobacterium smegmatis.

41 ies, which include the fast-growing organism Mycobacterium smegmatis.

42 expressing them in both Escherichia coli and Mycobacterium smegmatis.

43 t form of M. leprae SodC (rSodC) produced in Mycobacterium smegmatis.

44 hogen Mycobacterium tuberculosis, but not in Mycobacterium smegmatis.

45 mation system in the fast-growing surrogate, Mycobacterium smegmatis.

46 e cell wall and the inner membrane lipids of Mycobacterium smegmatis.

47 RNA in M. tuberculosis, Escherichia coli and Mycobacterium smegmatis.

48 bility to suppress beta-lactam resistance in Mycobacterium smegmatis.

49 rium tuberculosis and its avirulent relative Mycobacterium smegmatis.

50 e degradation of lipids in the microorganism Mycobacterium smegmatis.

51 terium glutamicum (5.20 +/- 0.20 kV/cm), and Mycobacterium smegmatis (5.56 +/- 0.08 kV/cm) have been

52 Expression of the lprI gene in Mycobacterium smegmatis (8-10-fold) protected its growth

53 this goal by creating a DeltamurI strain of Mycobacterium smegmatis, a close relative of Mycobacteri

54 e mechanism of protein pupylation by PafA in Mycobacterium smegmatis, a model mycobacterial organism.

55 with the observation that a mutant strain of Mycobacterium smegmatis, a nonpathogenic relative of M.

56 elE, relG, and relK induced growth arrest in Mycobacterium smegmatis; a phenotype that was completely

57 at an obligate aerobe, the soil actinomycete Mycobacterium smegmatis, adopts an anaerobe-type strateg

58 We show here that the Cpn60.2 homologue from Mycobacterium smegmatis also fails to oligomerize under

59 The present study describes a system using Mycobacterium smegmatis, an avirulent mycobacterium, to

60 Overexpression of Rv3802 orthologs in Mycobacterium smegmatis and Corynebacterium glutamicum i

61 richia coli, or recombinant LprG produced in Mycobacterium smegmatis and digested by alpha-mannosidas

62 annosylation in the nonpathogenic saprophyte Mycobacterium smegmatis and in the human pathogen Mtb by

63 MspA is the major porin of Mycobacterium smegmatis and is important for diffusion o

64 ncentration of myo-[14C]inositol is rapid in Mycobacterium smegmatis and leads to production of radio

65 ow cross-species activity against M. luteus, Mycobacterium smegmatis and M. bovis (BCG).

66 Deletion of pepD or mprAB in Mycobacterium smegmatis and M. tuberculosis alters the s

67 Here we show that Mycobacterium smegmatis and M. tuberculosis strains lack

68 stably integrated recombinant genes in both Mycobacterium smegmatis and M. tuberculosis, with maxima

69 ten called persisters, within populations of Mycobacterium smegmatis and M. tuberculosis.

70 e regulation over two orders of magnitude in Mycobacterium smegmatis and M.tuberculosis.

71 MspA is the major porin of Mycobacterium smegmatis and mediates diffusion of small

72 Analysis of Mycobacterium smegmatis and Mycobacterium bovis bacille

73 er repression of reporter gene activities in Mycobacterium smegmatis and Mycobacterium bovis BCG.

74 Mycobacterium smegmatis and Mycobacterium kansasii were

75 ections with the fast-growing, nonpathogenic Mycobacterium smegmatis and Mycobacterium phlei.

76 igated primarily with the readily cultivable Mycobacterium smegmatis and Mycobacterium tuberculosis a

77 ave reported that overexpression of NAT from Mycobacterium smegmatis and Mycobacterium tuberculosis m

78 les and Ms6 attB sites in the chromosomes of Mycobacterium smegmatis and Mycobacterium tuberculosis y

79 e used as a counterselectable marker in both Mycobacterium smegmatis and Mycobacterium tuberculosis.

80 binase of transposon gammadelta is active in Mycobacterium smegmatis and Mycobacterium tuberculosis.

81 Ac is acetyl) is the major thiol produced by Mycobacterium smegmatis and other actinomycetes.

82 IC 47 microM) but showed growth promotion of Mycobacterium smegmatis and other bacteria.

83 We report here that the genomes of Mycobacterium smegmatis and other soil mycobacteria cont

84 Further assays using membranes from Mycobacterium smegmatis and purified PimA and PimB' indi

85 ive integral membrane proteins, MSMEG4250 in Mycobacterium smegmatis and Rv2181 in M. tuberculosis, w

86 The GlgE orthologues from Mycobacterium smegmatis and Streptomyces coelicolor were

87 rent mycobacterial species, the fast-growing Mycobacterium smegmatis and the slow-growing M. bovis M.

88 We previously showed that nonpathogenic Mycobacterium smegmatis and to a lesser extent pathogeni

89 ty was purified approximately 2400-fold from Mycobacterium smegmatis and two proteins of slightly dif

90 les were unable to mediate LAM production in Mycobacterium smegmatis and were unable to complement an

91 ifferent stop codon usage (Escherichia coli, Mycobacterium smegmatis, and Bacillus subtilis), we show

92 DnaK is essential in Mycobacterium smegmatis, and ClpB is involved in asymmet

93 e suborder, namely, Mycobacterium bovis BCG, Mycobacterium smegmatis, and Corynebacterium glutamicum,

94 E-glnA2 cluster, was expressed separately in Mycobacterium smegmatis, and its gene product was charac

95 expression was measured in Escherichia coli, Mycobacterium smegmatis, and M. bovis BCG.

96 lmark of all TA toxins, in Escherichia coli, Mycobacterium smegmatis, and M. tuberculosis.

97 Bacillus subtilis, Streptomyces coelicolor, Mycobacterium smegmatis, and Pseudomonas aeruginosa.

98 1), C16:0 (sn-2) in the natural product from Mycobacterium smegmatis, and the structural assignment o

99 product lost its antibiotic activity against Mycobacterium smegmatis, and this loss in bioactivity is

100 GPLs from Mycobacterium smegmatis are composed of a lipopeptide co

101 hia coli; recombinant DesA3 was expressed in Mycobacterium smegmatis as a catalytically active membra

102 tration (MIC) by broth microdilution against Mycobacterium smegmatis as a function of the initial cel

103 d study bacterial cell wall structures using Mycobacterium smegmatis as a model organism.

104 Using Mycobacterium smegmatis as an expression host, we purifi

105 ftsEX and ripC caused similar phenotypes in Mycobacterium smegmatis, as expected for genes in a sing

106 We report the first apo-MbtA structure from Mycobacterium smegmatis at 2.3 A.

107 d protein EspB from both M. tuberculosis and Mycobacterium smegmatis at a homologous cut site in vitr

108 th the rapidly growing mycobacterial species Mycobacterium smegmatis bearing an intact esx-3 locus we

109 esistance to the common biocide triclosan in Mycobacterium smegmatis, binding to form the initial EI

110 Here we show that the FM in Mycobacterium smegmatis biofilms is produced through the

111 Full development of Mycobacterium smegmatis biofilms requires addition of su

112 Growth of Mycobacterium smegmatis biofilms requires multiple facto

113 e in phagosomes of macrophages infected with Mycobacterium smegmatis but increased in those infected

114 ding enzyme from the closely related species Mycobacterium smegmatis but not by the enzyme from Esche

115 functional for this purpose in fast-growing Mycobacterium smegmatis but not in slow-growing mycobact

116 that repression of pyr operon expression in Mycobacterium smegmatis by exogenous uracil requires the

117 not AG, ceases after inactivation of embC in Mycobacterium smegmatis by insertional mutagenesis.

118 ported that a genetically modified strain of Mycobacterium smegmatis called IKEPLUS is a promising TB

119 We describe here a mutant strain of Mycobacterium smegmatis, called DL1215, which demonstrat

120 arate or oxygen become available, suggesting Mycobacterium smegmatis can switch between fermentation,

121 not phosphorylation-defective PknK(Mtb), in Mycobacterium smegmatis cause significant retardation of

122 Conversely, expression of ppgS in Mycobacterium smegmatis conferred upon this species othe

123 Here we report that cydAB and cydDC in Mycobacterium smegmatis constitute two separate operons

124 active membrane and cell wall fractions from Mycobacterium smegmatis containing the overexpressed Rv3

125 By applying cell-free extracts of Mycobacterium smegmatis, containing cell wall and membra

126 Mycobacterium smegmatis contains 6 homologous mce (mamma

127 a TAT signal sequence, we demonstrated that Mycobacterium smegmatis contains a TAT system.

128 The outer membrane of the saprophytic Mycobacterium smegmatis contains the Msp family of porin

129 Mycobacterium smegmatis contains three hemerythrin-like

130 Mycobacterium smegmatis contains three putative TA syste

131 Deletion of M. tuberculosis or Mycobacterium smegmatis ctpC leads to cytosolic Mn(2+) a

132 homeostasis of Co(2+) and Fe(2+) Mutation of Mycobacterium smegmatis ctpJ affects the homeostasis of

133 rved a 40-fold increase in light output from Mycobacterium smegmatis cultures 2 h after adding 20 ng

134 e was used to define the M. tuberculosis and Mycobacterium smegmatis CysH enzymes as APS reductases.

135 of a mutant strain (designated MSMEG4245) of Mycobacterium smegmatis, defective in a broadly conserve

136 compounds identified as lipid I and II from Mycobacterium smegmatis demonstrated that the lipid moie

137 the comparable C-terminal sequence from the Mycobacterium smegmatis DesA3 homolog Msmeg_1886 also co

138 on with lipopolysachharide or infection with Mycobacterium smegmatis diminished expression of both en

139 This study unveils Mycobacterium smegmatis DinB2 as the founder of a clade

140 Mycobacterium smegmatis DinB2 is the founder of a clade

141 By contrast, the Mycobacterium smegmatis DNA transfer system is chromosom

142 The mshA::Tn5 mutant of Mycobacterium smegmatis does not produce mycothiol (MSH)

143 tate cluster at the pore interface unique to Mycobacterium smegmatis Dps protein, MsDps2.

144 In contrast to E. coli, Bacillus brevis and Mycobacterium smegmatis Dps:DNA complexes, in which DNA

145 ulties associated with the overexpression of Mycobacterium smegmatis EgtE protein, the proposed EgtE

146 Mycobacterium smegmatis encodes four RNase H enzymes: Rn

147 sts including Mycobacterium tuberculosis and Mycobacterium smegmatis, encompass substantial genetic d

148 yrA-G81C variant of Escherichia coli and its Mycobacterium smegmatis equivalent (GyrA-G89C).

149 oteins from M. tuberculosis (EsxG and EsxH), Mycobacterium smegmatis (EsxA and EsxB), and Corynebacte

150 The model mycobacterium Mycobacterium smegmatis executes homologous recombinatio

151 say of phosphoribosyltransferase activity in Mycobacterium smegmatis expressing recombinant Rv3242c (

152 and purified highly active Mtb NDH-2 using a Mycobacterium smegmatis expression system, and the stead

153 ycobacteria, we executed a genetic screen in Mycobacterium smegmatis for biotin auxotrophs and identi

154 itecture of the enzyme (MsAcT) isolated from Mycobacterium smegmatis forms the mechanistic basis for

155 The Mycobacterium tuberculosis and Mycobacterium smegmatis genomes contain open reading fra

156 Pseudomonas fluorescens, gi 70731221 ; anti, Mycobacterium smegmatis, gi 118470554 ) document that th

157 Inactivation of the Mycobacterium smegmatis groEL1 gene by phage Bxb1 integr

158 Glu(83) of the (75)DPSDVARVE(83) element of Mycobacterium smegmatis GTP-dependent phosphoenolpyruvat

159 hydroxylation reaction and demonstrated that Mycobacterium smegmatis has an enzyme activity that can

160 We show that Mycobacterium smegmatis has an enzyme catalyzing transfe

161 ucing phosphoinositol-capped LAM [PILAM]) in Mycobacterium smegmatis has been implicated in various f

162 The porin MspA of Mycobacterium smegmatis has remarkable stability against

163 Here we demonstrate that Mycobacterium smegmatis has three DSB repair pathway opt

164 ve recombinant HBHA vaccines in fast-growing Mycobacterium smegmatis have been unsuccessful so far, w

165 sents the structure of MsAcg (MSMEG_5246), a Mycobacterium smegmatis homologue of Mycobacterium tuber

166 whmD, the Mycobacterium smegmatis homologue of Streptomyces coelic

167 tional derepression in the heterologous host Mycobacterium smegmatis in a way that requires metal sit

168 s efficient repression of lacZ expression in Mycobacterium smegmatis in the presence but not the abse

169 ort here the crystal structures of PatA from Mycobacterium smegmatis in the presence of its naturally

170 Phage RedRock forms stable lysogens in Mycobacterium smegmatis in which the prophage replicates

171 o be potentially expressed on the surface of Mycobacterium smegmatis incubated with HEp-2 cells and,

172 pE gene is lethal to Helicobacter pylori and Mycobacterium smegmatis, indicating that DapE's are esse

173 e show that nonvirulent mycobacteria such as Mycobacterium smegmatis induce AIM2 inflammasome activat

174 The fast-growing bacterium Mycobacterium smegmatis is a model mycobacterial system,

175 The MS of Mycobacterium smegmatis is cytoplasmic but the M. tb MS

176 A deletion of the homologous operon in Mycobacterium smegmatis is more susceptible to ethidium

177 an ABC transporter encoded by the genome of Mycobacterium smegmatis is stabilized by d-threitol.

178 octameric transmembrane channel protein from Mycobacterium smegmatis, is one of the most stable prote

179 These lysine acyltransferases from Mycobacterium smegmatis (KATms) and Mycobacterium tuberc

180 ncentrations (10 uM) are required to observe Mycobacterium smegmatis killing.

181 Colonies of Mycobacterium smegmatis LR222 on iron-limiting (0.1 micr

182 o long chain fatty acids synthesized using a Mycobacterium smegmatis lysate.

183 hromosome dynamics at a single-cell level in Mycobacterium smegmatis (M. smegmatis) and Mycobacterium

184 microbial activities were determined against Mycobacterium smegmatis (M. smegmatis).

185 The high G6P level in Mycobacterium smegmatis may result from 10-25-fold highe

186 When expressed in noninvasive Mycobacterium smegmatis, MBP-1 increased the ability of

187 In our present work, Mycobacterium smegmatis mc(2) 155 (WTMsm) was used as a

188 phages known to infect a single common host, Mycobacterium smegmatis mc(2) 155.

189 namely, Mycobacterium bovis BCG Pasteur and Mycobacterium smegmatis mc(2) 155.

190 GC measurements, that the soil actinomycete Mycobacterium smegmatis mc(2)155 constitutively oxidizes

191 arge set of phages infecting the common host Mycobacterium smegmatis mc(2)155 shows that they span co

192 Heterologous expression of the etnE gene in Mycobacterium smegmatis mc(2)155 using the pMV261 vector

193 Temperature-sensitive mutant 2-20/32 of Mycobacterium smegmatis mc(2)155 was isolated and geneti

194 ince an ortholog of MT1671 is not present in Mycobacterium smegmatis mc(2)155, a recombinant strain w

195 pulation of phages that can infect the host, Mycobacterium smegmatis mc(2)155.

196 myces coelicolor or Streptomyces lividans to Mycobacterium smegmatis mc2155 in plate crosses.

197 cetylation of Cys-GlcN-Ins to produce MSH in Mycobacterium smegmatis mc2155, and Cys-GlcN-Ins is main

198 We report the characterization of Mycobacterium smegmatis MmpL11.

199 the functionally related ESX-1 apparatus of Mycobacterium smegmatis (Ms) to show that fluorescently

200 , the orthologous ESAT-6 from non-pathogenic Mycobacterium smegmatis (MsESAT-6) was essentially inact

201 Recombinant Mycobacterium smegmatis MshC catalyzes the ATP-dependent

202 Mycobacterium smegmatis MshC catalyzes the ATP-dependent

203 -steady-state approaches, of the recombinant Mycobacterium smegmatis MshC.

204 vival (Eis) protein improves the survival of Mycobacterium smegmatis (Msm) in macrophages and functio

205 rt a 3.2 A-resolution crystal structure of a Mycobacterium smegmatis (Msm) open promoter complex (RPo

206 iratory polyketide quinones (PkQs) from both Mycobacterium smegmatis (Msmeg) and Mtb.

207 e, we show that exposure to an esterase from Mycobacterium smegmatis (Msmeg_1529), hydrolyzing the es

208 acteria, we deleted the Rv3574 orthologue in Mycobacterium smegmatis (MSMEG_6042) and used real-time

209 When overexpressed in Escherichia coli or Mycobacterium smegmatis, MtbFHb remained associated with

210 ivity to ampicillin and chloramphenicol of a Mycobacterium smegmatis mutant lacking the main porin Ms

211 We identified a Mycobacterium smegmatis mutant, named FUEL (which stands

212 We isolated 27 Mycobacterium smegmatis mutants that were hypersusceptib

213 The four Ub2-resistant Mycobacterium smegmatis mutants were also resistant to t

214 occus gordonii, Streptococcus parasanguinis, Mycobacterium smegmatis, Mycobacterium tuberculosis and

215 Here we report that Mycobacterium smegmatis NucS/EndoMS, a putative endonucl

216 Conjugal DNA transfer in Mycobacterium smegmatis occurs by a mechanism distinct f

217 ansfer of chromosomal DNA between strains of Mycobacterium smegmatis occurs by a novel mechanism.

218 igated, using a GFP reporter system, whether Mycobacterium smegmatis OhrR has the ability to sense an

219 f phagocytic and nonphagocytic cell lines by Mycobacterium smegmatis or M. bovis BCG harboring a plas

220 ffect several reporter proteins in wild-type Mycobacterium smegmatis or Mycobacterium tuberculosis.

221 pare the RNA and DNA modifying activities of Mycobacterium smegmatis PNPase.

222 ngle DNA template strands through a modified Mycobacterium smegmatis porin A (M2MspA) nanopore under

223 otein nanopores such as alpha-haemolysin and Mycobacterium smegmatis porin A (MspA) can be used to se

224 e use the engineered biological protein pore Mycobacterium smegmatis porin A (MspA) to detect and map

225 tivity of a mutated form of the protein pore Mycobacterium smegmatis porin A (MspA) with phi29 DNA po

226 single-stranded DNA-NeutrAvidin complex in a Mycobacterium smegmatis porin A nanopore.

227 force and diffusion constant of DNA inside a Mycobacterium smegmatis porin A pore were determined to

228 hat the ionic current through the engineered Mycobacterium smegmatis porin A, MspA, has the ability t

229 not complement the permeability defects of a Mycobacterium smegmatis porin mutant to glucose, serine

230 Mycobacterium tuberculosis and Mycobacterium smegmatis possess three pathways for the s

231 Mycobacterium smegmatis possesses two such transporters,

232 overexpression of the Rv2629 191C allele in Mycobacterium smegmatis produced an eightfold increase i

233 Here we show that a Mycobacterium smegmatis protein homologous to eubacteria

234 enetic diversity of phages of a common host, Mycobacterium smegmatis, provides a higher resolution of

235 ed mechanisms of RMPs in both reactions with Mycobacterium smegmatis RecO (MsRecO) and demonstrated t

236 Disruption of the ortholog of Rv3789 in Mycobacterium smegmatis resulted in a reduction of the a

237 trated that deletion of MSMEG_6281 (Ami1) in Mycobacterium smegmatis resulted in the formation of cel

238 proteins, expression in a Deltatat mutant of Mycobacterium smegmatis revealed a defect in precursor p

239 Studies with a model Mycobacterium (Mycobacterium smegmatis) revealed that by removing salts

240 ive integral membrane proteins, MSMEG2785 in Mycobacterium smegmatis, Rv2673 in Mycobacterium tubercu

241 is and the nonpathogenic model mycobacterium Mycobacterium smegmatis, SecA1 is essential for protein

242 moautotrophicum, Archaeoglobus fulgidus, and Mycobacterium smegmatis showed that they contained only

243 A transposon mutant of Mycobacterium smegmatis shown to be GlcNAc-Ins and mycot

244 lectively access mycolates on the surface of Mycobacterium smegmatis spheroplasts, allowing us to mon

245 bacterial activity against Escherichia coli, Mycobacterium smegmatis, Staphylococcus aureus and Staph

246 Deletion of polD1, polD2, or both from a Mycobacterium smegmatis strain carrying an inactivating

247 ow that priming with a prototype recombinant Mycobacterium smegmatis strain expressing human immunode

248 plasmid-based NHEJ assay and a collection of Mycobacterium smegmatis strains bearing deletions or mut

249 and homologous recombination (HR)-deficient Mycobacterium smegmatis strains to probe the importance

250 showed that three different Actinobacteria (Mycobacterium smegmatis, Streptomyces lividans, and Rhod

251 Analysis of Mycobacterium smegmatis subcellular fractions and sphero

252 but not kasA mutants, could be generated in Mycobacterium smegmatis, suggesting that unlike kasB, ka

253 source dependent in Mtb and did not occur in Mycobacterium smegmatis, suggesting that V-58-mediated g

254 aneous mutants of DAP-auxotrophic strains of Mycobacterium smegmatis that can grow in the absence of

255 t strain was constructed in ManLAM-deficient Mycobacterium smegmatis that coexpressed Rv2181 and Rv16

256 ns in genes encoding ribosomal components in Mycobacterium smegmatis that confer resistance to severa

257 polymerase in Mycobacterium tuberculosis and Mycobacterium smegmatis that has evolved independently f

258 ne a LigD ligase-independent NHEJ pathway in Mycobacterium smegmatis that requires the ATP-dependent

259 en applied to bacteria (Escherichia coli and Mycobacterium smegmatis), the yeast Saccharomyces cerevi

260 In the nonpathogen Mycobacterium smegmatis, the ESX-1 T7SS plays a role in

261 noxia upregulates a homologue of HU (Hlp) in Mycobacterium smegmatis, the nonpathogenic model of Myco

262 However, when expressed in Mycobacterium smegmatis, the Rv0348 transcripts were sig

263 s lack activity against Escherichia coli and Mycobacterium smegmatis, they proved to be highly potent

264 eria, we disrupted the gene encoding MsrA in Mycobacterium smegmatis through homologous recombination

265 , MSMEG_5437, in the intrinsic resistance of Mycobacterium smegmatis to a variety of stresses includi

266 ant (secA2 K129R) of the model mycobacterium Mycobacterium smegmatis to better understand the pathway

267 in determining the spectrum of responses of Mycobacterium smegmatis to challenge with rifampicin.

268 TreS was purified from the cytosol of Mycobacterium smegmatis to give a single protein band on

269 umor activity required coadministration with Mycobacterium smegmatis to induce IL-1beta production an

270 Ablation of UvrD1 sensitizes Mycobacterium smegmatis to killing by ultraviolet and io

271 atase (TPP) was purified from the cytosol of Mycobacterium smegmatis to near homogeneity using a vari

272 Escherichia coli, Staphylococcus aureus, and Mycobacterium smegmatis to quinolone antibiotics.

273 essential for conjugal recipient activity in Mycobacterium smegmatis Transcription of esx4 genes in t

274 ate of reactive oxygen species production in Mycobacterium smegmatis treated with CFZ and a CFZ analo

275 ned the global protein turnover profiles for Mycobacterium smegmatis under acid shock and iron starva

276 tions and their impact on gene expression in Mycobacterium smegmatis under hypochlorite stress.

277 The biological sample model included two Mycobacterium smegmatis unlabeled cell cultures grown at

278 Q TLR2 polymorphism on macrophage sensing of Mycobacterium smegmatis Upon infection with M. smegmatis

279 By characterizing truncated versions of Mycobacterium smegmatis UvrD2, we show that whereas the

280 erminal domain, the embC knock-out mutant of Mycobacterium smegmatis was complemented with plasmids e

281 combination of monosodium urate crystals and Mycobacterium smegmatis was effective at delaying the gr

282 nonpathogenic, rapidly growing mycobacterium Mycobacterium smegmatis was engineered as a vector expre

283 Sulfite reduction in Mycobacterium smegmatis was investigated using a combina

284 The trehalose-phosphate synthase (TPS) of Mycobacterium smegmatis was previously purified to appar

285 A calmodulin-like protein (CAMLP) from Mycobacterium smegmatis was purified to homogeneity and

286 Phosphatase activity in whole cells of Mycobacterium smegmatis was significantly lower than tha

287 The enzyme Msd from Mycobacterium smegmatis was taken as a representative ca

288 To assess the physiological role of Ald in Mycobacterium smegmatis, we cloned the ald gene, identif

289 expression of inhA confers INH resistance in Mycobacterium smegmatis, we designed a promoter trap bas

290 screening of an M. avium genomic library in Mycobacterium smegmatis, we have identified a number of

291 en for antibiotic resistance determinants in Mycobacterium smegmatis, we identified a multidrug-sensi

292 actions from wild-type and DeltasecA2 mutant Mycobacterium smegmatis, we identified the Msmeg1712 and

293 e dynamics of 379 extracellular compounds of Mycobacterium smegmatis were deconvoluted with a genome-

294 ttempts to delete the NCgl2760 orthologue in Mycobacterium smegmatis were unsuccessful, consistent wi

295 ree different glycosyltransferase mutants of Mycobacterium smegmatis were used here to investigate th

296 exhibited impaired cell wall localization in Mycobacterium smegmatis, whereas mPDE-4A behaved similar

297 H37Rv enhances the intracellular survival of Mycobacterium smegmatis, which does not contain eis, wit

298 suite of compounds, inhibited the growth of Mycobacterium smegmatis with an MIC80 value of 2 mug/mL.

299 ed mutants of Mycobacterium tuberculosis and Mycobacterium smegmatis with deletions in the genes for

300 d to the NMOs from Aspergillus fumigatus and Mycobacterium smegmatis with K(d) values of 2.1 +/- 0.2