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1 orrelia burgdorferi, Treponema pallidum, and Mycoplasma fermentans.
2 luding 750-kb Ureaplasma urealyticum, 1.2-Mb Mycoplasma fermentans, 2.3-Mb Streptococcus pneumoniae,
3 al phase variation is shown here to occur in Mycoplasma fermentans, a chronic human infectious agent
4 orrelia burgdorferi, Treponema pallidum, and Mycoplasma fermentans activated cells heterologously exp
5 ting the abundant P29 surface lipoprotein of Mycoplasma fermentans, binds human HeLa cells and inhibi
6 logs of S. kunkelii, Mycoplasma pulmonis and Mycoplasma fermentans cluster together and are more simi
7   The mature MALP-404 surface lipoprotein of Mycoplasma fermentans comprises a membrane-anchored N-te
8                                              Mycoplasma fermentans had previously been isolated from
9                                   Since 1970 Mycoplasma fermentans has been suspected of being associ
10                                              Mycoplasma fermentans incognitus has been isolated from
11                       We examined effects of Mycoplasma fermentans infection on the continuing surviv
12             A new insertion sequence (IS) of Mycoplasma fermentans is described.
13                             The malp gene of Mycoplasma fermentans is shown to occur in single copy b
14                          In comparison, live Mycoplasma fermentans or M. penetrans infection for 4 to
15 ably detect <5 copies of Mycoplasma hominis, Mycoplasma fermentans, or a molecular mimic control in s
16 atures that is present in four copies in the Mycoplasma fermentans PG18 chromosome, accounting for ap
17        The approximately 16 kb genome of the Mycoplasma fermentans phiMFV1 prophage is described, and
18 9, a major lipid-modified surface protein of Mycoplasma fermentans, reveal phase variation of surface
19 s found between the expression of a specific Mycoplasma fermentans surface antigen (Pra, proteinase-r

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