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1 cally infected with the respiratory pathogen Mycoplasma pulmonis.
2 virulence of the murine respiratory pathogen Mycoplasma pulmonis.
3  were undertaken to examine gene transfer in Mycoplasma pulmonis.
4 /lpr, and B6-gld/gld mice were infected with Mycoplasma pulmonis.
5 iable R-M systems encoded by the hsd loci of Mycoplasma pulmonis.
6 lyze one of the membrane nuclease genes from Mycoplasma pulmonis.
7  with 10(5) or 10(7) colony-forming units of Mycoplasma pulmonis.
8 esponse in the airways of mice infected with Mycoplasma pulmonis.
9  were immunized i.m. with DNA for one or two Mycoplasma pulmonis Ags (A7-1, A8-1) beginning either 1
10 (a) chronic respiratory tract infection with Mycoplasma pulmonis and (b) adenoviral transduction of a
11 e ftsZ is present in two mycoplasma species, Mycoplasma pulmonis and Acholeplasma laidlawii, which ar
12 d (C57-SCID), and C57BL/6N (C57BL) mice with Mycoplasma pulmonis and at 14 and 21 days postinfection
13             We examined biofilm formation by Mycoplasma pulmonis and found it to be dependent on the
14 attempts to introduce transposon Tn4001 into Mycoplasma pulmonis and Mycoplasma arthritidis have not
15 etic analysis, TraE homologs of S. kunkelii, Mycoplasma pulmonis and Mycoplasma fermentans cluster to
16 d configuration in Mycoplasma pneumoniae and Mycoplasma pulmonis, and in both the d and l configurati
17  nasal or nasal-pulmonary immunizations with Mycoplasma pulmonis antigen alone or with cholera toxin
18 ens (Vsa) of the murine respiratory pathogen Mycoplasma pulmonis are associated with the virulence of
19                                              Mycoplasma pulmonis binds the lectin Griffonia simplicif
20               Genital infection of rats with Mycoplasma pulmonis causes adverse pregnancy outcome and
21 ovokes histamine release, we exposed mice to Mycoplasma pulmonis, comparing responses in wild-type an
22                                              Mycoplasma pulmonis contains a family of extensively stu
23                  To improve the detection of Mycoplasma pulmonis contamination of isolates of cilia-a
24 synthase(-/-) (iNOS(-/-)) mice infected with Mycoplasma pulmonis developed higher bacterial numbers a
25 of AM depletion on intrapulmonary killing of Mycoplasma pulmonis during the early phase of infection
26                             The hsd genes of Mycoplasma pulmonis encode restriction and modification
27                             The vsa genes of Mycoplasma pulmonis encode the V-1 lipoproteins.
28 ty determinant), have been identified in the Mycoplasma pulmonis genome.
29    Airways of LTbeta(-/-) mice infected with Mycoplasma pulmonis had significantly more lymphangiogen
30 addition to phase-variable surface proteins, Mycoplasma pulmonis has a family of phase-variable restr
31    Although the variation of V-1 antigens of Mycoplasma pulmonis has been correlated with variable ex
32 e tandem repeat region of the Vsa protein of Mycoplasma pulmonis has previously been shown to modulat
33 enic mice and chronic airway inflammation in Mycoplasma pulmonis-infected C3H/HeNCr mice.
34 ty and reversibility of vessel remodeling in Mycoplasma pulmonis-infected mice using immunohistochemi
35 remodeling in the respiratory tract by using Mycoplasma pulmonis infection as a model of sustained in
36        ANG2 drove vascular remodeling during Mycoplasma pulmonis infection by acting as a Tie2 antago
37 o these changes in airway inflammation after Mycoplasma pulmonis infection in mice.
38  ideal opportunity to study this process, as Mycoplasma pulmonis infection of mouse airways induces w
39 l of chronic airway inflammation produced by Mycoplasma pulmonis infection of the airways of mice or
40 in airway lymphatics at 14 and 28 days after Mycoplasma pulmonis infection of the respiratory tract.
41 t mice whose airways were inflamed by either Mycoplasma pulmonis infection or ovalbumin sensitization
42 use strains differ markedly in resistance to Mycoplasma pulmonis infection, and investigation of thes
43 rine respiratory mycoplasmosis (MRM), due to Mycoplasma pulmonis infection, is an excellent animal mo
44 r baseline conditions and 3 to 28 days after Mycoplasma pulmonis infection, using prospero heomeobox
45 f sustained airway inflammation in mice with Mycoplasma pulmonis infection, which is known to be acco
46  C3H/HeN) and resistant (C57BL/6) mice after Mycoplasma pulmonis infection.
47 urine Mycoplasma respiratory disease, due to Mycoplasma pulmonis infection.
48                   The infection of mice with Mycoplasma pulmonis is a model for studying chronic myco
49 flammation in mice infected by the bacterium Mycoplasma pulmonis (M. pulmonis), the segment of the mi
50                                   FtsZs from Mycoplasma pulmonis (MpuFtsZ) and Bacillus subtilis (BsF
51 uman synovial sections and sections from the Mycoplasma pulmonis MRL-lpr/lpr mouse arthritis model.
52  determined for 1700 members of a library of Mycoplasma pulmonis mutants.
53  7-day infection by the respiratory pathogen Mycoplasma pulmonis, or iv) leakage after bradykinin cha
54                                              Mycoplasma pulmonis possesses a cassette of genes that a
55 al and bronchial epithelial cell surfaces by Mycoplasma pulmonis, submucosal and intraluminal immune
56 way inflammation, produced by infection with Mycoplasma pulmonis, was compared in strains of mice kno
57 el of asthma and the natural murine pathogen Mycoplasma pulmonis, we provide a mechanistic explanatio
58                  C3H mice were infected with Mycoplasma pulmonis, which attaches to the airway epithe

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