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2 Treatment of MALME-3M cells with 10 microM N1,N11-diethylnorspermine caused an increase in hypophos
5 The clinically relevant polyamine analogue N(1),N(11)-diethylnorspermine (DENSPM) inhibits cell gro
7 1210 murine leukemia cell growth than either N(1),N(11)-diethylnorspermine (DENSPM) or (2R,10R)-N(1),
8 g which most potently induces SSAT activity, N1, N11-diethylnorspermine (DENSPM), increases SSAT mRNA
9 rmacokinetic parameters of i.v.-administered N1,N11-diethylnorspermine (DENSPM) are evaluated in Cebu
10 hat the clinically relevant polyamine analog N1,N11-diethylnorspermine (DENSPM) causes rapid apoptosi
13 ecifically target polyamine metabolism, i.e. N1,N11-diethylnorspermine (DENSPM) or alpha-difluorometh
14 eeded in the presence of the spermine analog N1,N11-diethylnorspermine (DENSPM), cell proliferation c
15 ach, the impact of four polyamine analogues, N1,N11-diethylnorspermine (DENSPM), N1,N14-diethylhomosp
16 ensitive to the growth inhibitory effects of N1,N11-diethylnorspermine (DENSPM)-a polyamine analogue
17 an that of unhydroxylated parent drug (e.g., N(1),N(11)-diethylnorspermine [DENSPM]) at 1 microM; how
18 oxycycline deprivation before treatment with N(1), N(11)-diethylnorspermine markedly increased analog
19 The linear molecules include norspermine, N1, N11-diethylnorspermine, N1,N12-bis(2,2,2-trifluoroet
20 this observation, we examined the effects of N1,N11-diethylnorspermine on cell cycle regulatory prote
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