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1 cleaves chemotactic peptide f-MLF to release N-formylmethionine (f-Met) and dipeptide leucylphenylala
2 Uk and H4 modulate the level of circulating N-formylmethionine (fMet), which initiates mitochondrial
4 ns were extracted from cells with or without N-formylmethionine leucyl-phenylalanine (fMLP) stimulati
5 ceptor (ALX), with only a modest response to N-formylmethionine-leucyl-phenylalanine (fMLP), we did r
6 ol conditions (no stimulant, anti-IgE, fMLP (N-formylmethionine-leucyl-phenylalanine), and anti-Fceps
8 ty, including both alpha-N-acetyl- and alpha-N-formylmethionine that exhibit higher activity than alp