ライフサイエンスコーパス: N-glycosylation

1 the benzoxazinoid breakdown product MBOA by N-glycosylation.

2 vely charged residues such as Arg suppressed N-glycosylation.

3 PMM2 encodes a key enzyme in N-glycosylation.

4 e often poorly expressed and showed immature N-glycosylation.

5 the second disulfide bond and is affected by N-glycosylation.

6 sed as a model to study haloarchaeal protein N-glycosylation.

7 ne of nascent proteins during the process of N-glycosylation.

8 cilitate detection of micro-heterogeneity of N-glycosylation.

9 transferase complex, is directly involved in N-glycosylation.

10 ndependent role of HRD1 in the regulation of N-glycosylation.

11 for a better understanding of OST-catalyzed N-glycosylation.

12 icant alteration of the protein structure by N-glycosylation.

13 b at the surface, compared with high mannose N-glycosylation.

14 ocalizes extracellularly, where it undergoes N-glycosylation.

15 d metal binding as the first step of protein N-glycosylation.

16 all encode misfolded proteins with abnormal N-glycosylation.

17 ties with partially overlapping functions in N-glycosylation.

18 and inexpensive solution to achieve rapid de-N-glycosylation.

19 lfide-linked dimer, which is strengthened by N-glycosylation.

20 stringently modulated by ubiquitination and N-glycosylation.

21 y of mannose precursors required for protein N-glycosylation.

22 To date, 19 different genetic defects in N-glycosylation, 17 in O-glycosylation, and 21 in multip

23 he introduction, in BCR variable regions, of N-glycosylation acceptor sites harboring unusual high-ma

24 d plasma membrane localization of FLA4, with N-glycosylation acting at the level of ER-exit and O-gly

25 We showed here a post-translational N-glycosylation affected by the HRD1 E3 ubiquitin ligase

26 Thus, while unique N-glycosylation affected structural features of Env invo

27 Immunoglobulin G (IgG) Fc N-glycosylation affects antibody-mediated effector funct

28 Therefore, we explored how N-glycosylation alters the protein homeostasis or proteo

29 Xenograft studies reveal that blocking SCAP N-glycosylation ameliorates EGFRvIII-driven glioblastoma

30 Additionally, glycopeptide enrichment and N-glycosylation analysis identified 73 glycosylation sit

31 .1:p.Arg308Cys), which resulted in perturbed N-glycosylation and aberrant localization to the cell su

32 Bacterial protein N-glycosylation and its application to engineering recom

33 eleterious by a mechanism involving abnormal N-glycosylation and misfolding of the IFN-gammaR2 protei

34 Defects in N-glycosylation and N-glycan processing frequently cause

35 Therefore, N-glycosylation and specific ERQC components are essenti

36 We quantified the importance of protein N-glycosylation and synthesis of the DPAGT1 encoded enzy

37 In this study, we report the impact of N-glycosylation and the membrane on the secondary struct

38 interplay between EL-3 disulfide bonding and N-glycosylation and their roles in EL-3 topological fold

39 glycan substrates, 3-4 d to engineer the IgG N-glycosylation, and 2-5 d to synthesize the small-molec

40 iKO and ZIP8-LSKO mice had defective protein N-glycosylation, and humans homozygous for the minor all

41 ibit MICA018 surface expression by affecting N-glycosylation, and the retention was rescued by T24A s

42 the i+1 position increased the likelihood of N-glycosylation, and Thr is better than Ser at the i+2 p

43 rus infection and inflammation alter protein N-glycosylation, and we have previously shown that chang

44 O-Mannosylation and N-glycosylation are essential protein modifications that

45 ein translocation across the ER membrane and N-glycosylation are highly coordinated processes that ta

46 ve previously shown that changes in cellular N-glycosylation are involved in regulation of NKG2D liga

47 arides (mDLOs) needed for eukaryotic protein N-glycosylation are synthesized by a multistep pathway i

48 Our study reveals N-glycosylation as an allele-specific regulatory mechani

49 These findings identify N-glycosylation as critical for JAM-A's many functions.

50 osylation is an important feature of protein N-glycosylation as it has been reported to influence the

51 ' cells is enhanced by chemical modifiers of N-glycosylation, as previously shown for patients with g

52 This approach may identify N-glycosylation-associated biomarkers for other autoimmu

53 ful information for enhancing or suppressing N-glycosylation at a site of interest and valuable data

54 Here, we report that N-glycosylation at the Asn(211) residue plays a unique r

55 2, and mutation of Asn-594 to Ala to disrupt N-glycosylation at the C terminus of COX-2 reduced the i

56 Taken together, these data suggest that N-glycosylation at the highly conserved (211)NDS motif e

57 of the mammalian rLILRA3 revealed canonical N-glycosylation at the predicted Asn(140), Asn(281), Asn

58 N-glycosylation at two sites, T87 and S97, were found to

59 ith peptide:N-glycosidase F, consistent with N-glycosylation being the principal form of post-transla

60 rucei RFT1 (TbRFT1) not only affects protein N-glycosylation but also glycosylphosphatidylinositol (G

61 protein RFT1 is essential for normal protein N-glycosylation, but its precise function is not known.

62 Disruption of N-glycosylation by site-directed mutagenesis or tunicamy

63 Cotranslational N-glycosylation by the STT3A isoform of the OST, which l

64 Because N-glycosylation can affect protein folding, stability, a

65 N-glycosylations can regulate the adhesive function of i

66 ransferase, NGTs constitute a novel class of N-glycosylation catalyzing enzymes.

67 on, and Mgat1 nulls are the most compromised N-glycosylation condition that survive long enough to pe

68 ed and lyso forms of GPI-APs is dependent on N-glycosylation, confirming a general role of N-glycans

69 Mutation of the single N-glycosylation consensus sequence at Asn-988 (Trpm4b-N9

70 ns 4 highly conserved cysteines and multiple N-glycosylation consensus sites.

71 N-glycosylation consists of the addition, and subsequent

72 owever, accumulating evidence indicates that N-glycosylation could also possibly occur at other atypi

73 ngly, glutamine deprivation or inhibition of N-glycosylation decreased M2 polarization and production

74 Wild-type and N-glycosylation-deficient TRPP2 is degraded in lysosomes

75 ular domain of MICA018 was essential for the N-glycosylation dependence, whereas the intracellular do

76 we pinpoint the residues essential for this N-glycosylation dependence.

77 The N-glycosylation dependent autoepitopes that emerge on er

78 Ectopic expression of CRK4 induced STT3a/N-glycosylation-dependent cell death in Arabidopsis and

79 te the repair cycle of photosystem II, while N-glycosylation determines enzyme activity of chloroplas

80 Our study reveals that N-glycosylation does not induce significant changes in p

81 Complex N-glycosylation doubled the percentage of Trpm4b at the

82 igate the influence of sequence variation on N-glycosylation efficiency in the context of a five-resi

83 We devised an algorithm for prediction of N-glycosylation efficiency using the SAS software, emplo

84 d side chains at the i-1 position had higher N-glycosylation efficiency, and Cys, in particular, comp

85 aining residues at the i-2 position improved N-glycosylation efficiency, while positively charged res

86 The lipid carrier specificity of the protein N-glycosylation enzyme C. jejuni PglB was tested using a

87 ependent on its extracellular domain and its N-glycosylation, especially at N117.

88 In mammals, most organ-specific N-glycosylation events occur in the brain.

89 s study indicates a crucial importance of HA N-glycosylation for immunogenicity.

90 alse discovery rate of 0 was achieved on the N-glycosylation-free Escherichia coli data set.

91 Removing N-glycosylation from the CD16 protein core by tunicamyci

92 the requirement of a consensus sequence for N-glycosylation further limits the number of possible de

93 -glycan knockouts on the CD demonstrate that N-glycosylation has little impact on cellulose conversio

94 cans at each of the three wild-type sites of N glycosylation, has been accomplished in our laboratory

95 st-translational modifications, specifically N-glycosylation, has only recently become a target of in

96 s and its posttranslational modifications by N-glycosylation have been described to induce a deleteri

97 ous studies on the sequence requirements for N-glycosylation have yielded the Asn-X-Ser/Thr (NXS/T) s

98 is crucial for the transport activity; (ii) N-glycosylation impacts membrane targeting and is conser

99 l immunogen and point to a potential role of N-glycosylation in increasing recombinant protein yields

100 Examining GABAAR subunit N-glycosylation in matched pairs of schizophrenia (N=14)

101 To characterize the process of cytoplasmic N-glycosylation in more detail, we studied the glycosyla

102 about the nature, function and regulation of N-glycosylation in neurons.

103 These findings suggest that disruptions of N-glycosylation in schizophrenia are not exclusive to gl

104 se glucosamine significantly decreased Glut1 N-glycosylation in Th1-polarized cells.

105 uned interplay between disulfide bonding and N-glycosylation in the membrane processed NBCe1-A dimer.

106 were associated with altered integrin-beta1 N-glycosylation, in particular with higher levels of bet

107 steady-state levels of mDLO and significant N-glycosylation, indicating robust M5-DLO flippase activ

108 In N-glycosylation inhibition experiments, we find that tre

109 ferent inhibitors of IL-10 signaling and the N-glycosylation inhibitor tunicamycin in cultured human

110 and DEX suppressed ER stress induced by the N-glycosylation inhibitor, tunicamycin (Tm).

111 N-glycosylation involves the attachment of an oligosacch

112 especially for characterizing site-specific N-glycosylation involving complex N-glycans.

113 We find that N-glycosylation is a critical determinant of EGFR confor

114 N-glycosylation is a fundamental modification of protein

115 N-glycosylation is a major modification of glycoproteins

116 N-Glycosylation is a post-translational modification com

117 N-Glycosylation is an important co- and/or post-translat

118 Thus, N-glycosylation is essential for processing, localizatio

119 A hallmark of protein N-glycosylation is extensive heterogeneity associated wi

120 We report that while N-glycosylation is generally required for dendritic deve

121 N-glycosylation is important for the function and regula

122 We show here that N-glycosylation is not a prerequisite for proper targeti

123 We conclude that N-glycosylation is not required for surface expression o

124 However, whether N-glycosylation is required for the assembly and/or func

125 The specific mode of regulation through N-glycosylation is, however, unknown.

126 We show that IgG N-glycosylation loci are strongly enriched for genes exp

127 thod for glycoconjugate synthesis using PglB N-glycosylation machinery and varied chemically synthesi

128 Further mutational studies, N-glycosylation mapping, and plasma membrane targeting s

129 We show that differential HA N-glycosylation markedly affected T cell activation and

130 It has been reported that influenza virus HA N-glycosylation markedly depends on the host cell line u

131 Targeting SCAP N-glycosylation may provide a promising means of treatin

132 it shares some biochemical features, such as N-glycosylation, membranous location, and reactivity wit

133 Carcinoma of the Prostate (LNCaP) cells, the N-glycosylation model identified and quantified glycan s

134 this limitation, we developed a quantitative N-glycosylation model that interprets and integrates mas

135 , we identified STT3a, a protein involved in N-glycosylation modification, as an important regulator

136 ophysiologically relevant changes in surface N-glycosylation modified hERG channel function.

137 egrin alpha3 subunit, thereby introducing an N-glycosylation motif at amino acid position 349.

138 ruses in conjunction with each amino acid or N-glycosylation motif, and performs Fisher's exact test

139 utations that disrupt the conserved (211)NDS N-glycosylation motif, but not other N-glycosylation sit

140 rticular, we evaluated the role of potential N-glycosylation motifs acquired by somatic hypermutation

141 he N-glycosylation state of TRPV5, since the N-glycosylation mutant (TRPV5(N358Q)) was activated to t

142 2014 is developed to automatically construct N-glycosylation networks in MATLAB with the involvement

143 ycosylphosphatidylinositol (GPI) anchors and N-glycosylation, O-fucosylation has been recently report

144 sing this method, we determined 117 absolute N-glycosylation occupancies in OVCAR-3 cells.

145 inhibits STT3B-dependent post-translational N-glycosylation of ABCG8.

146 Although N-glycosylation of alpha1, beta1, and beta2 were all cha

147 direct evidence for the first time that the N-glycosylation of archaeal flagellins is critical for m

148 We conclude that N-glycosylation of Asn-594 of COX-2 occurs in the ER, le

149 sis of CaValpha2delta1, and furthermore that N-glycosylation of CaValpha2delta1 is essential to produ

150 ts indicate that glucosamine interferes with N-glycosylation of CD25, and thereby attenuates IL-2 dow

151 enetic and molecular analysis indicated that N-glycosylation of CKX1 was not affected by the lack of

152 ammalian cells use two mechanisms to achieve N-glycosylation of cysteine proximal acceptor sites.

153 N-glycosylation of eukaryotic proteins is widespread and

154 Dolichol plays an indispensable role in the N-glycosylation of eukaryotic proteins.

155 he oligosacharyltransferase (AglB)-dependent N-glycosylation of flagellins is required for flagella a

156 N-glycosylation of JAM-A is required for the protein's a

157 Finally, we show that N-glycosylation of JAM-A regulates leukocyte adhesion an

158 N-glycosylation of Lhs1 is largely Ost3-independent and

159 N-glycosylation of molecules associated with glutamaterg

160 ral defects, a paucity of granules, aberrant N-glycosylation of multiple proteins and increased incid

161 Signal peptide cleavage and N-glycosylation of proteins are co-translational process

162 N-glycosylation of proteins in endoplasmic reticulum is

163 N-glycosylation of proteins is now routinely characteriz

164 s of DC2 causes a defect in co-translational N-glycosylation of proteins that mimics an STT3A(-/-) ph

165 cuss the recent advancements in the field of N-glycosylation of recombinant therapeutic proteins in E

166 N-glycosylation of Sil1 is predominantly Ost3-dependent

167 Here, we show that N-glycosylation of Sil1, but not of Lhs1, is diminished

168 stress in the ER inhibits the Ost3-dependent N-glycosylation of Sil1, which regulates specific BiP fu

169 ling, by increasing glucose uptake, promotes N-glycosylation of sterol regulatory element-binding pro

170 glycans on the alpha1 subunit, more immature N-glycosylation of the 49-kDa beta1 subunit isoform, and

171 n normal human cortex, we found evidence for N-glycosylation of the alpha1, beta1, and beta2 gamma-am

172 osmogs roots were reduced due to incomplete N-glycosylation of the B subfamily of ATP-binding casset

173 Measures of altered N-glycosylation of the beta1 and beta2 subunits were con

174 kDa beta1 subunit isoform, and altered total N-glycosylation of the beta2 GABAAR subunit in schizophr

175 Here we investigated whether direct N-glycosylation of the NKG2D ligand MICA itself is criti

176 acetate gives products resulting from O- and N-glycosylation of the pyrimidine ring.

177 We examined the role of N-glycosylation of transient receptor potential melastat

178 we discuss the subcellular localization and N-glycosylation of two commercially-relevant recombinant

179 her, our results highlight the importance of N-glycosylation on BCMA in the regulation of ligand bind

180 We then investigated the effect of N-glycosylation on the function of BCMA and found that t

181 The impact of N-glycosylation on the function of glycoproteins is gene

182 Cells deficient in N-glycosylation or the synthesis of specific ganglioside

183 Mutations associated with the N-glycosylation pathway and in the family of serine pept

184 Genetic defects in the N-glycosylation pathway cause >35 inherited human disord

185 The successful functional transfer of an N-glycosylation pathway from Campylobacter jejuni to Esc

186 ration determine the metabolic flux into the N-glycosylation pathway.

187 in the native host, and exploitation of the N-glycosylation pathways to create novel vaccines and di

188 ding the characterization of novel bacterial N-glycosylation pathways, examination of pathway enzymes

189 nonical rules developed for other eukaryotic N-glycosylation pathways, raising questions as to the ba

190 This integrin-beta1 N-glycosylation pattern was correlated with higher level

191 Upon further analysis of the resulting N-glycosylation pattern, we discovered that the enhanced

192 protein was characterized by determining the N-glycosylation patterns of the ectodomain.

193 investigated the impact of the differential N-glycosylation patterns of two influenza A virus PR/8/3

194 e authors conduct a multivariate GWAS on IgG N-glycosylation phenotypes and identify 5 novel loci enr

195 ciation studies of 23 immunoglobulin G (IgG) N-glycosylation phenotypes.

196 ession or channel function, but that complex N-glycosylation plays a crucial role in stabilizing surf

197 N-Glycosylation plays a fundamental role in many biologi

198 its motility, supporting our hypothesis that N-glycosylation plays an important role in regulating th

199 Protein N-glycosylation (PNG) is crucial for protein folding and

200 The N-glycosylation probes were analyzed by mass spectrometr

201 isingly facile and highly diastereoselective N-glycosylation process.

202 hways are improving our understanding of the N-glycosylation process.

203 e I (GluI) is a key member of the eukaryotic N-glycosylation processing pathway, selectively catalyzi

204 host and suggests that the length and/or the N-glycosylation profile of the V1/V2 domain influences t

205 Full automation to enable high throughput N-glycosylation profiling and sequencing with good repro

206 ty, and reveal how varying the efficiency of N-glycosylation provides a mechanism to modulate antibod

207 and pathophysiologically relevant changes in N-glycosylation; reduced channel sialylation increases h

208 tress and UPR activation induced by blocking N-glycosylation, reducing ER Ca(2+) or depleting glucose

209 In conclusion, site-specific CBG N-glycosylation regulates the bioavailability of cortiso

210 oth Sil1 and Lhs1 are glycoproteins, but how N-glycosylation regulates their function is not known.

211 tenin signaling pathway, whose target is the N-glycosylation-regulating gene, DPAGT1.

212 Neural development requires N-glycosylation regulation of intercellular signaling, b

213 ful tool for IgG Fc (fragment cystallizable) N-glycosylation remodeling.

214 All complex and hybrid N-glycosylation requires MGAT1 (UDP-GlcNAc:alpha-3-D-man

215 dated for O-glycosylation (kappa casein) and N-glycosylation (ribonuclease B).

216 surprisingly does not possess the canonical N-glycosylation sequence N-X-S/T.

217 N-X-S/T (X not equal P), a motif known as an N-glycosylation'sequon'.

218 We conclude that MGAT1-dependent N-glycosylation shapes the synaptomatrix carbohydrate en

219 sted differences in glycosylation, including N-glycosylation, sialylation and fucosylation, were obse

220 ubiquitinated CTB polypeptides revealed that N-glycosylation significantly relieved ER stress and hyp

221 We found that a single N-glycosylation site (Asn(8)) was important for MICA018

222 d a knockin mouse expressing RDS without the N-glycosylation site (N229S).

223 The N-glycosylation site at Asn-644 in the LOX catalytic dom

224 similarity with beta-hLH, including a common N-glycosylation site at the N-terminus but differs mainl

225 ation CLRN1(N48K), which affects a conserved N-glycosylation site in hCLRN1, is a common causative US

226 e we demonstrate that the conserved putative N-glycosylation site in K(2P)3.1 and K(2P)9.1 is a glyca

227 The N-glycosylation site occupancy and disulfide pattern, th

228 To address N-glycosylation site occupancy and N-glycan composition

229 ehensive mass spectrometry-based analysis of N-glycosylation site occupancy in pmtDelta mutants.

230 ize how hemophilia mutations near the unused N-glycosylation site of the A2 domain (N582) of FVIII af

231 n sites, a third novel, albeit low abundant, N-glycosylation site on C9 is identified, which surprisi

232 IgG antibodies contain a conserved N-glycosylation site on the Fc domain to which a complex

233 vity assays on CDKAL1 constructs carrying an N-glycosylation site within the luminal domain, we furth

234 tified as a 73 amino acid protein having one N-glycosylation site, and a variant 74 residue non-glyco

235 xhibiting complex-type N-glycans at a single N-glycosylation site, asparagine 42.

236 creted protein was due not to the loss of an N-glycosylation site, but rather an SNP-specific targeti

237 equences into NCAM Ig5, including an "extra" N-glycosylation site, decreases or completely blocks NCA

238 er to decipher the function of its potential N-glycosylation site, we produced pro-KLK2 in Leishmania

239 the N38S amino acid change and a loss of an N-glycosylation site.

240 domain, was not affected by mutation of this N-glycosylation site.

241 results demonstrate a critical role for the N glycosylation sites and cysteines for the structure an

242 211)NDS N-glycosylation motif, but not other N-glycosylation sites (Asn(260), Asn(371), and Asn(394))

243 Next, we showed that the cluster of N-glycosylation sites (Asn-467, Asn-473, and Asn-494) wa

244 ulatory sequences established that all eight N-glycosylation sites (N1 to N8) are used in the wild ty

245 2-229) is needed for TC-Cbl binding, but the N-glycosylation sites (N126, N195, and N213) are of no i

246 IgA2m(1), the engineered molecule lacked two N-glycosylation sites (N166 and N337), two free cysteine

247 ophages transfected with FcgammaRIa in which N-glycosylation sites 1, 4, and 5 are mutated to alanine

248 In contrast, mutations of NKCC2 N-glycosylation sites abolished the effects of OS9, indi

249 tions in both the number and distribution of N-glycosylation sites are found in the 18 alpha and 8 be

250 plastidal reporter proteins with artificial N-glycosylation sites are indeed glycosylated during tra

251 l analysis revealed that all eight potential N-glycosylation sites are utilized.

252 validated the congruence of the potential HA N-glycosylation sites as well as the presence of the HA

253 We recently reported that the N-glycosylation sites Asn-168, Asn-538, and Asn-745 in r

254 assess the relative occupancies of numerous N-glycosylation sites by endoglycosidase H-resistant N-g

255 dual elimination of evolutionarily conserved N-glycosylation sites did not abolish proper KOR1 foldin

256 e of FL-BCRs is the acquisition of potential N-glycosylation sites during somatic hypermutation.

257 We disrupted eight predicted N-glycosylation sites in HeV G by conservative mutations

258 ta1,4-glucanase and contains eight potential N-glycosylation sites in its extracellular domain.

259 mportance of the conserved cysteines and the N-glycosylation sites in NBCe1-A EL-3, we analyzed the p

260 t upon complex oligosaccharide processing of N-glycosylation sites in the amino-terminal domain and d

261 mphoma (FL), but not normal B cells, acquire N-glycosylation sites in the immunoglobulin variable reg

262 st likely to predominate at all the putative N-glycosylation sites in the parasite proteome.

263 , we have mutated each of the nine consensus N-glycosylation sites independently.

264 ur data indicate that utilization of several N-glycosylation sites is important for KOR1 activity, wh

265 The N-glycosylation sites of both inhibitors were determined

266 her indicated that, among the four potential N-glycosylation sites of E-cadherin, Asn-554 is the key

267 To elucidate the functional significance of N-glycosylation sites of STIM1, we created different mut

268 complex-type biantennary N-glycans linked to N-glycosylation sites that emerge during somatic hypermu

269 In this study, we tried to identify atypical N-glycosylation sites using our recently developed solid

270 uired for histamine catabolism, has multiple N-glycosylation sites, but their roles, for example in D

271 extracellular domain of KOR1 contains eight N-glycosylation sites, N1 to N8, of which only N3 to N7

272 here was considerable variation in potential N-glycosylation sites, with GA2 and ON1 viruses showing

273 that of JR-FL or with mutations in putative N-glycosylation sites.

274 arenaviruses is glycosylated at 11 conserved N-glycosylation sites.

275 void of glycans at the first, second or both N-glycosylation sites.

276 mbinatorial mutagenesis of all six potential N-glycosylation sites.

277 tase is glycosylated at all twelve potential N-glycosylation sites.

278 de bond is spontaneous and unaffected by the N-glycosylation state of EL-3 or the first disulfide bon

279 However, this effect was independent of the N-glycosylation state of TRPV5, since the N-glycosylatio

280 ive protein in insect cells and analyzed its N-glycosylation state.

281 Whereas, HRD1 had only slight effect on the N-glycosylation status of ABCG5; rather it accelerated A

282 of gamma-proteobacteria encode a cytoplasmic N-glycosylation system mediated by a soluble N-glycosylt

283 ng on similarities and subtle differences in N-glycosylation that often reflect the subcellular traff

284 N-glycosylation - the sequential addition of complex sug

285 Asparagine-linked protein N-glycosylation, the most complex glycosylation, initiat

286 ggest that R345W F3, but not WT F3, requires N-glycosylation to acquire a stable, native-like structu

287 However, the re-introduction of N-glycosylation to its original or an alternative site s

288 hly divergent eukaryote has re-wired protein N-glycosylation to provide protein sequence-specific N-g

289 irect demonstration of the role of GOLPH3 in N-glycosylation to regulate cell biological functions.

290 Here, we show that during evolution N-glycosylation triggered a dual selection pressure on s

291 translational modification, including O- and N-glycosylation, ubiquitination, and phosphorylation as

292 Therefore, we studied whether IgG Fc N-glycosylation varies in populations with different env

293 he secondary structure, which is enhanced by N-glycosylation via additional interactions between the

294 Genetic elimination of F3 N-glycosylation (via an N249Q mutation) caused R345W F3

295 IgG Fc N-glycosylation was analysed in serum/plasma of 700 scho

296 contained high mannose, whereas complex type N-glycosylation was observed on Asn-436 and Asn-612 in t

297 positive, breast cancer cells ADAM8 contains N-glycosylation, which is required for its correct proce

298 lexible interaction of the distal portion of N-glycosylation with bulk water and biomolecular assembl

299 assical, oligosaccharyltransferase-catalyzed N-glycosylation, with NX(S/T) sequons being the optimal

300 rily conserved metabolic pathways of protein N-glycosylation, Wnt/beta-catenin signaling pathway, and