ライフサイエンスコーパス: N-glycosylation

1 the benzoxazinoid breakdown product MBOA by N-glycosylation.

2 for a better understanding of OST-catalyzed N-glycosylation.

3 vely charged residues such as Arg suppressed N-glycosylation.

4 PMM2 encodes a key enzyme in N-glycosylation.

5 cilitate detection of micro-heterogeneity of N-glycosylation.

6 lfide-linked dimer, which is strengthened by N-glycosylation.

7 stringently modulated by ubiquitination and N-glycosylation.

8 y of mannose precursors required for protein N-glycosylation.

9 bined with the use of tunicamycin to prevent N-glycosylation.

10 e often poorly expressed and showed immature N-glycosylation.

11 the second disulfide bond and is affected by N-glycosylation.

12 sed as a model to study haloarchaeal protein N-glycosylation.

13 ne of nascent proteins during the process of N-glycosylation.

14 transferase complex, is directly involved in N-glycosylation.

15 ndependent role of HRD1 in the regulation of N-glycosylation.

16 ng of proteoforms with regard to heavy chain N-glycosylation.

17 ase st3gal2, two enzymes involved in protein N-glycosylation.

18 rders of glycosylation (CDGs) with defective N-glycosylation.

19 between protein O-mannosylation and protein N-glycosylation.

20 harmacological inhibition of cotranslational N-glycosylation.

21 proBDNF by tunicamycin-induced inhibition of N-glycosylation.

22 For N-glycosylation, a mouse-trained model performs equally

23 In eukaryotic protein N-glycosylation, a series of glycosyltransferases cataly

24 he introduction, in BCR variable regions, of N-glycosylation acceptor sites harboring unusual high-ma

25 d plasma membrane localization of FLA4, with N-glycosylation acting at the level of ER-exit and O-gly

26 We showed here a post-translational N-glycosylation affected by the HRD1 E3 ubiquitin ligase

27 Immunoglobulin G (IgG) Fc N-glycosylation affects antibody-mediated effector funct

28 Therefore, we explored how N-glycosylation alters the protein homeostasis or proteo

29 Xenograft studies reveal that blocking SCAP N-glycosylation ameliorates EGFRvIII-driven glioblastoma

30 Additionally, glycopeptide enrichment and N-glycosylation analysis identified 73 glycosylation sit

31 owerful technique for protein sequencing and N-glycosylation analysis.

32 ring the relationship between hybrid/complex N-glycosylation and cardiac function and disease.

33 sociated large membrane machines for protein N-glycosylation and protein O-mannosylation, respectivel

34 ed lymphocytes with MAGT1 mRNA restored both N-glycosylation and receptor function.

35 Therefore, N-glycosylation and specific ERQC components are essenti

36 We quantified the importance of protein N-glycosylation and synthesis of the DPAGT1 encoded enzy

37 In this study, we report the impact of N-glycosylation and the membrane on the secondary struct

38 interplay between EL-3 disulfide bonding and N-glycosylation and their roles in EL-3 topological fold

39 glycan substrates, 3-4 d to engineer the IgG N-glycosylation, and 2-5 d to synthesize the small-molec

40 iKO and ZIP8-LSKO mice had defective protein N-glycosylation, and humans homozygous for the minor all

41 esses such as N-terminal protein maturation, N-glycosylation, and metabolism.

42 ibit MICA018 surface expression by affecting N-glycosylation, and the retention was rescued by T24A s

43 the i+1 position increased the likelihood of N-glycosylation, and Thr is better than Ser at the i+2 p

44 rus infection and inflammation alter protein N-glycosylation, and we have previously shown that chang

45 contents; the gamma/gamma' chains ratio; the N-glycosylation; and the post-translational modification

46 O-Mannosylation and N-glycosylation are essential protein modifications that

47 ein translocation across the ER membrane and N-glycosylation are highly coordinated processes that ta

48 ve previously shown that changes in cellular N-glycosylation are involved in regulation of NKG2D liga

49 vant changes in cardiomyocyte hybrid/complex N-glycosylation are sufficient to cause DCM and early de

50 Our study reveals N-glycosylation as an allele-specific regulatory mechani

51 Our study establishes N-glycosylation as an important regulator of LDL metabol

52 These findings identify N-glycosylation as critical for JAM-A's many functions.

53 osylation is an important feature of protein N-glycosylation as it has been reported to influence the

54 This approach may identify N-glycosylation-associated biomarkers for other autoimmu

55 ful information for enhancing or suppressing N-glycosylation at a site of interest and valuable data

56 nts, upon HGF stimulation, is due to loss of N-glycosylation at the Asn150 or Asn261 site, respective

57 Taken together, these data suggest that N-glycosylation at the highly conserved (211)NDS motif e

58 Loss of N-glycosylation at this site may remove steric hindrance

59 rucei RFT1 (TbRFT1) not only affects protein N-glycosylation but also glycosylphosphatidylinositol (G

60 protein RFT1 is essential for normal protein N-glycosylation, but its precise function is not known.

61 Cotranslational N-glycosylation by the STT3A isoform of the OST, which l

62 These results reveal that N-glycosylation can modulate GPCR function by altering r

63 Ablation of post-translational N-glycosylation can therefore make HSV-1 infectivity, an

64 N-glycosylations can regulate the adhesive function of i

65 ransferase, NGTs constitute a novel class of N-glycosylation catalyzing enzymes.

66 Reduced myocyte complex N-glycosylation causes dilated cardiomyopathy.

67 lulose biosynthesis, provides a link between N-glycosylation, cell wall biosynthesis, and abiotic str

68 ns 4 highly conserved cysteines and multiple N-glycosylation consensus sites.

69 data demonstrate that reduced hybrid/complex N-glycosylation contributes to aberrant cardiac function

70 owever, accumulating evidence indicates that N-glycosylation could also possibly occur at other atypi

71 ering of mRNA, protein, phosphorylation, and N-glycosylation data identified four subtypes of diffuse

72 ere identified mainly by phosphorylation and N-glycosylation data.

73 ngly, glutamine deprivation or inhibition of N-glycosylation decreased M2 polarization and production

74 All three patients have an N-glycosylation defect, as was shown by the study of dif

75 Wild-type and N-glycosylation-deficient TRPP2 is degraded in lysosomes

76 ular domain of MICA018 was essential for the N-glycosylation dependence, whereas the intracellular do

77 we pinpoint the residues essential for this N-glycosylation dependence.

78 The N-glycosylation dependent autoepitopes that emerge on er

79 Ectopic expression of CRK4 induced STT3a/N-glycosylation-dependent cell death in Arabidopsis and

80 te the repair cycle of photosystem II, while N-glycosylation determines enzyme activity of chloroplas

81 Our study reveals that N-glycosylation does not induce significant changes in p

82 igate the influence of sequence variation on N-glycosylation efficiency in the context of a five-resi

83 We devised an algorithm for prediction of N-glycosylation efficiency using the SAS software, emplo

84 d side chains at the i-1 position had higher N-glycosylation efficiency, and Cys, in particular, comp

85 aining residues at the i-2 position improved N-glycosylation efficiency, while positively charged res

86 The lipid carrier specificity of the protein N-glycosylation enzyme C. jejuni PglB was tested using a

87 ependent on its extracellular domain and its N-glycosylation, especially at N117.

88 In mammals, most organ-specific N-glycosylation events occur in the brain.

89 alse discovery rate of 0 was achieved on the N-glycosylation-free Escherichia coli data set.

90 Removing N-glycosylation from the CD16 protein core by tunicamyci

91 the requirement of a consensus sequence for N-glycosylation further limits the number of possible de

92 -glycan knockouts on the CD demonstrate that N-glycosylation has little impact on cellulose conversio

93 s and its posttranslational modifications by N-glycosylation have been described to induce a deleteri

94 ous studies on the sequence requirements for N-glycosylation have yielded the Asn-X-Ser/Thr (NXS/T) s

95 ndings underline the functional relevance of N-glycosylation in biogenesis and membrane trafficking o

96 for investigating the role of hybrid/complex N-glycosylation in cardiac pathogenesis.

97 udy suggest that mutations and variations in N-glycosylation in HA caused antigenic variations in H1N

98 The status and biological relevance of ORF2 N-glycosylation in HEV lifecycle remain to be elucidated

99 l immunogen and point to a potential role of N-glycosylation in increasing recombinant protein yields

100 ertebrates and confirm the high diversity of N-glycosylation in lower organisms.

101 Examining GABAAR subunit N-glycosylation in matched pairs of schizophrenia (N=14)

102 To characterize the process of cytoplasmic N-glycosylation in more detail, we studied the glycosyla

103 about the nature, function and regulation of N-glycosylation in neurons.

104 These findings suggest that disruptions of N-glycosylation in schizophrenia are not exclusive to gl

105 se glucosamine significantly decreased Glut1 N-glycosylation in Th1-polarized cells.

106 -mediated cholesterol clearance by targeting N-glycosylation in the LDL pathway may represent a novel

107 uned interplay between disulfide bonding and N-glycosylation in the membrane processed NBCe1-A dimer.

108 were associated with altered integrin-beta1 N-glycosylation, in particular with higher levels of bet

109 These changes due to N-glycosylation increased the ligand on-rate and decreas

110 In N-glycosylation inhibition experiments, we find that tre

111 We further demonstrate that ManN and two N-glycosylation inhibitors stimulate EC proliferation vi

112 N-glycosylation involves the attachment of an oligosacch

113 especially for characterizing site-specific N-glycosylation involving complex N-glycans.

114 We find that N-glycosylation is a critical determinant of EGFR confor

115 N-glycosylation is a fundamental modification of protein

116 N-glycosylation is a major modification of glycoproteins

117 N-Glycosylation is a post-translational modification com

118 N-Glycosylation is an important co- and/or post-translat

119 Thus, N-glycosylation is essential for processing, localizatio

120 A hallmark of protein N-glycosylation is extensive heterogeneity associated wi

121 We report that while N-glycosylation is generally required for dendritic deve

122 IL-22-mediated host N-glycosylation is likely impaired in patients with ulce

123 However, whether N-glycosylation is required for the assembly and/or func

124 The specific mode of regulation through N-glycosylation is, however, unknown.

125 . aeruginosa twitching motility involves its N-glycosylation, its pili-binding capacity is insufficie

126 We show that IgG N-glycosylation loci are strongly enriched for genes exp

127 By altering the N-glycosylation machinery in the endoplasmic reticulum a

128 Further mutational studies, N-glycosylation mapping, and plasma membrane targeting s

129 review discusses how changes in endothelial N-glycosylation may impact vascular and monocytic inflam

130 Targeting SCAP N-glycosylation may provide a promising means of treatin

131 it shares some biochemical features, such as N-glycosylation, membranous location, and reactivity wit

132 , we identified STT3a, a protein involved in N-glycosylation modification, as an important regulator

133 ruses in conjunction with each amino acid or N-glycosylation motif, and performs Fisher's exact test

134 rticular, we evaluated the role of potential N-glycosylation motifs acquired by somatic hypermutation

135 We previously identified that an EBV gL N-glycosylation mutant (gL-N(69)L/S(71)V) was hyperfusog

136 clones (VH/VL region reverted into GL), and N-glycosylation mutants (N->Q) and analyzed for anti-NET

137 2014 is developed to automatically construct N-glycosylation networks in MATLAB with the involvement

138 and thus solely dependent upon STT3A-OST for N-glycosylation, NGI-1 treatment resulted in HSV-1 havin

139 ycosylphosphatidylinositol (GPI) anchors and N-glycosylation, O-fucosylation has been recently report

140 sing this method, we determined 117 absolute N-glycosylation occupancies in OVCAR-3 cells.

141 N-glycosylation of 2 representative envelope proteins (g

142 inhibits STT3B-dependent post-translational N-glycosylation of ABCG8.

143 We conclude that N-glycosylation of Asn-594 of COX-2 occurs in the ER, le

144 N-glycosylation of asparagine 130 in its extracellular d

145 post-translational modifications, including N-glycosylation of asparagine residues.

146 Impairment in N-glycosylation of CaV2.1 promotes gain-of-function effe

147 sis of CaValpha2delta1, and furthermore that N-glycosylation of CaValpha2delta1 is essential to produ

148 ts indicate that glucosamine interferes with N-glycosylation of CD25, and thereby attenuates IL-2 dow

149 enetic and molecular analysis indicated that N-glycosylation of CKX1 was not affected by the lack of

150 ammalian cells use two mechanisms to achieve N-glycosylation of cysteine proximal acceptor sites.

151 Enzymatic de-N-glycosylation of DMBT1, but not de-O-glycosylation, re

152 Dolichol plays an indispensable role in the N-glycosylation of eukaryotic proteins.

153 he oligosacharyltransferase (AglB)-dependent N-glycosylation of flagellins is required for flagella a

154 N-glycosylation of JAM-A is required for the protein's a

155 Finally, we show that N-glycosylation of JAM-A regulates leukocyte adhesion an

156 They also showed that MAGT1 is required for N-glycosylation of key T cell and NK cell receptors that

157 N-glycosylation of Lhs1 is largely Ost3-independent and

158 ral defects, a paucity of granules, aberrant N-glycosylation of multiple proteins and increased incid

159 We showed that N-glycosylation of ORF2 protein does not play any role i

160 N-glycosylation of proteins in endoplasmic reticulum is

161 N-glycosylation of proteins is now routinely characteriz

162 s of DC2 causes a defect in co-translational N-glycosylation of proteins that mimics an STT3A(-/-) ph

163 cuss the recent advancements in the field of N-glycosylation of recombinant therapeutic proteins in E

164 N-glycosylation of Sil1 is predominantly Ost3-dependent

165 Here, we show that N-glycosylation of Sil1, but not of Lhs1, is diminished

166 stress in the ER inhibits the Ost3-dependent N-glycosylation of Sil1, which regulates specific BiP fu

167 ling, by increasing glucose uptake, promotes N-glycosylation of sterol regulatory element-binding pro

168 glycans on the alpha1 subunit, more immature N-glycosylation of the 49-kDa beta1 subunit isoform, and

169 osmogs roots were reduced due to incomplete N-glycosylation of the B subfamily of ATP-binding casset

170 kDa beta1 subunit isoform, and altered total N-glycosylation of the beta2 GABAAR subunit in schizophr

171 N-Glycosylation of the major structural proteins (archae

172 Here we investigated whether direct N-glycosylation of the NKG2D ligand MICA itself is criti

173 acetate gives products resulting from O- and N-glycosylation of the pyrimidine ring.

174 Here, we analyzed the N-glycosylation of the receptor-binding domain (RBD) of

175 N-Glycosylation of the TBEV envelope (E) glycoprotein is

176 In addition to requiring N-glycosylation of TpoR, mutant CALRs require a hydropho

177 Cells deficient in N-glycosylation or the synthesis of specific ganglioside

178 The successful functional transfer of an N-glycosylation pathway from Campylobacter jejuni to Esc

179 ration determine the metabolic flux into the N-glycosylation pathway.

180 same heptameric glycan, suggesting a common N-glycosylation pathway.

181 nonical rules developed for other eukaryotic N-glycosylation pathways, raising questions as to the ba

182 proinflammatory IgG fraction crystallizable N-glycosylation pattern and provide a mechanistic link t

183 This integrin-beta1 N-glycosylation pattern was correlated with higher level

184 Upon further analysis of the resulting N-glycosylation pattern, we discovered that the enhanced

185 en revealed that abnormal changes of protein N-glycosylation patterns are associated with many diseas

186 e authors conduct a multivariate GWAS on IgG N-glycosylation phenotypes and identify 5 novel loci enr

187 ciation studies of 23 immunoglobulin G (IgG) N-glycosylation phenotypes.

188 N-Glycosylation plays a fundamental role in many biologi

189 its motility, supporting our hypothesis that N-glycosylation plays an important role in regulating th

190 ransmembrane proteins and suggest that TRPC6 N-glycosylation plays multiple roles in modulating chann

191 Protein N-glycosylation (PNG) is crucial for protein folding and

192 The N-glycosylation probes were analyzed by mass spectrometr

193 H1N1 virus vaccine from chicken eggs by the N-glycosylation process inhibitor kifunensine and the en

194 is of patient sera also revealed an abnormal N-glycosylation profile for transferrin, a clinical diag

195 host and suggests that the length and/or the N-glycosylation profile of the V1/V2 domain influences t

196 Our data reveal that N-glycosylation profiles can differ between subcellular

197 rehensive mass spectrometric analysis of the N-glycosylation profiles of the SARS-CoV-2 spike protein

198 Full automation to enable high throughput N-glycosylation profiling and sequencing with good repro

199 ty, and reveal how varying the efficiency of N-glycosylation provides a mechanism to modulate antibod

200 In conclusion, site-specific CBG N-glycosylation regulates the bioavailability of cortiso

201 oth Sil1 and Lhs1 are glycoproteins, but how N-glycosylation regulates their function is not known.

202 tenin signaling pathway, whose target is the N-glycosylation-regulating gene, DPAGT1.

203 ful tool for IgG Fc (fragment cystallizable) N-glycosylation remodeling.

204 alyze cotranslational and post-translational N-glycosylation, respectively.

205 and order-specific aspects of this species' N-glycosylation reveal both invertebrate- and vertebrate

206 surprisingly does not possess the canonical N-glycosylation sequence N-X-S/T.

207 ProBDNF carries a single N-glycosylation sequon (Asn-127) that remains virtually

208 N-X-S/T (X not equal P), a motif known as an N-glycosylation'sequon'.

209 10 epitopes, as well as addition of putative N-glycosylation sequons.

210 sted differences in glycosylation, including N-glycosylation, sialylation and fucosylation, were obse

211 No N-glycosylation signals were detected in SLP-8348 and SL

212 ubiquitinated CTB polypeptides revealed that N-glycosylation significantly relieved ER stress and hyp

213 We found that a single N-glycosylation site (Asn(8)) was important for MICA018

214 d a knockin mouse expressing RDS without the N-glycosylation site (N229S).

215 toward a differential selection pressure of N-glycosylation site acquisition during affinity maturat

216 esigns of an immunogenic region to add a new N-glycosylation site and mask it from antibody binding.

217 similarity with beta-hLH, including a common N-glycosylation site at the N-terminus but differs mainl

218 ation CLRN1(N48K), which affects a conserved N-glycosylation site in hCLRN1, is a common causative US

219 finity for EphA2, indicating that the EBV gL N-glycosylation site might be responsible for inhibiting

220 The N-glycosylation site occupancy and disulfide pattern, th

221 ehensive mass spectrometry-based analysis of N-glycosylation site occupancy in pmtDelta mutants.

222 ize how hemophilia mutations near the unused N-glycosylation site of the A2 domain (N582) of FVIII af

223 n sites, a third novel, albeit low abundant, N-glycosylation site on C9 is identified, which surprisi

224 IgG antibodies contain a conserved N-glycosylation site on the Fc domain to which a complex

225 creted protein was due not to the loss of an N-glycosylation site, but rather an SNP-specific targeti

226 overall selection bias against acquiring an N-glycosylation site, except for the CDR3 of the H chain

227 er to decipher the function of its potential N-glycosylation site, we produced pro-KLK2 in Leishmania

228 the N38S amino acid change and a loss of an N-glycosylation site.

229 domain, was not affected by mutation of this N-glycosylation site.

230 results demonstrate a critical role for the N glycosylation sites and cysteines for the structure an

231 be heavily modified as it harbors 5 putative N-glycosylation sites (10 in the mature dimer).

232 Next, we showed that the cluster of N-glycosylation sites (Asn-467, Asn-473, and Asn-494) wa

233 ce contains three highly conserved potential N-glycosylation sites (N1, N2 and N3).

234 IgA2m(1), the engineered molecule lacked two N-glycosylation sites (N166 and N337), two free cysteine

235 To understand how individual N-glycosylation sites (NGS) coordinate to form a dynamic

236 a single N-acetylglucosamine at each of the N-glycosylation sites [monoglycosylated HA (HA(mg))] can

237 In contrast, mutations of NKCC2 N-glycosylation sites abolished the effects of OS9, indi

238 However, mutating the two TRPC6 N-glycosylation sites abrogated the cytotoxicity of muta

239 ethod allowed for the identification of 2172 N-glycosylation sites and 1047 surface glycoproteins.

240 Furthermore, it included two additional N-glycosylation sites and a pair of cysteines suggestive

241 N-glycosylation sites are clustered around CDRs and the

242 tions in both the number and distribution of N-glycosylation sites are found in the 18 alpha and 8 be

243 We recently reported that the N-glycosylation sites Asn-168, Asn-538, and Asn-745 in r

244 SHM introduces DNA sequences encoding N-glycosylation sites asparagine-X-serine/threonine (N-g

245 subunits of hK(2P)17.1 harbor two functional N-glycosylation sites at positions N65 and N94.

246 identification and orthogonal validation of N-glycosylation sites based on alternating sequential sa

247 ssed as rmAb, 35 out of 38 (92%) nongermline N-glycosylation sites became occupied.

248 assess the relative occupancies of numerous N-glycosylation sites by endoglycosidase H-resistant N-g

249 Genetically introducing aspartates at these N-glycosylation sites bypasses the requirement for PNG-1

250 an location and heterogeneity of surrounding N-glycosylation sites can be altered, resulting in expos

251 e of FL-BCRs is the acquisition of potential N-glycosylation sites during somatic hypermutation.

252 he current study is to establish patterns of N-glycosylation sites in Ab V regions of naive and memor

253 We disrupted eight predicted N-glycosylation sites in HeV G by conservative mutations

254 mportance of the conserved cysteines and the N-glycosylation sites in NBCe1-A EL-3, we analyzed the p

255 t upon complex oligosaccharide processing of N-glycosylation sites in the amino-terminal domain and d

256 mphoma (FL), but not normal B cells, acquire N-glycosylation sites in the immunoglobulin variable reg

257 st likely to predominate at all the putative N-glycosylation sites in the parasite proteome.

258 In 7/80 RA-rmAb, SHM resulted in ex novo N-glycosylation sites in VH and/or VL regions.

259 , we have mutated each of the nine consensus N-glycosylation sites independently.

260 The N-glycosylation sites of both inhibitors were determined

261 her indicated that, among the four potential N-glycosylation sites of E-cadherin, Asn-554 is the key

262 le Asn-to-Ala substitutions at the predicted N-glycosylation sites of the M41-RBD were evaluated alon

263 cally triplicate experiments, on average 953 N-glycosylation sites on 393 surface glycoproteins per e

264 Disruption of both N-glycosylation sites results in loss of hK(2P)17.1 curr

265 complex-type biantennary N-glycans linked to N-glycosylation sites that emerge during somatic hypermu

266 In this study, we tried to identify atypical N-glycosylation sites using our recently developed solid

267 ptides derived from 56 glycoproteins with 83 N-glycosylation sites were identified from human serum a

268 ides derived from 129 glycoproteins with 157 N-glycosylation sites were identified from mouse liver t

269 a series of ORF2 mutants in which the three N-glycosylation sites were mutated individually or in co

270 The distribution and occurrence of N-glycosylation sites were systematically investigated,

271 ctrometry demonstrated that all 11 predicted N-glycosylation sites were utilized in both HEK293- and

272 analyzed the distribution and acquisition of N-glycosylation sites within Ab V regions of peripheral

273 rvous system function - contain two putative N-glycosylation sites within the large N-terminal domain

274 uired for histamine catabolism, has multiple N-glycosylation sites, but their roles, for example in D

275 extracellular domain of KOR1 contains eight N-glycosylation sites, N1 to N8, of which only N3 to N7

276 (AXL) as a model glycoprotein with multiple N-glycosylation sites, we show that those LCMS features

277 here was considerable variation in potential N-glycosylation sites, with GA2 and ON1 viruses showing

278 tase is glycosylated at all twelve potential N-glycosylation sites.

279 ly glycosylated and contains numerous O- and N-glycosylation sites.

280 We propose that N-glycosylation, specifically the capping of N-glycans a

281 de bond is spontaneous and unaffected by the N-glycosylation state of EL-3 or the first disulfide bon

282 Whereas, HRD1 had only slight effect on the N-glycosylation status of ABCG5; rather it accelerated A

283 of gamma-proteobacteria encode a cytoplasmic N-glycosylation system mediated by a soluble N-glycosylt

284 N-glycosylation - the sequential addition of complex sug

285 Asparagine-linked protein N-glycosylation, the most complex glycosylation, initiat

286 ggest that R345W F3, but not WT F3, requires N-glycosylation to acquire a stable, native-like structu

287 However, the re-introduction of N-glycosylation to its original or an alternative site s

288 hly divergent eukaryote has re-wired protein N-glycosylation to provide protein sequence-specific N-g

289 Here, we show that during evolution N-glycosylation triggered a dual selection pressure on s

290 scaped glycoproteins to bypass the classical N-glycosylation trimming pathway involving ER glucosidas

291 Therefore, we studied whether IgG Fc N-glycosylation varies in populations with different env

292 he secondary structure, which is enhanced by N-glycosylation via additional interactions between the

293 Genetic elimination of F3 N-glycosylation (via an N249Q mutation) caused R345W F3

294 IgG Fc N-glycosylation was analysed in serum/plasma of 700 scho

295 N-glycosylation was disrupted using N-glycosidase F and

296 ing of extractable metabolites revealed that N-glycosylation was the main transformation pathway of S

297 positive, breast cancer cells ADAM8 contains N-glycosylation, which is required for its correct proce

298 assical, oligosaccharyltransferase-catalyzed N-glycosylation, with NX(S/T) sequons being the optimal

299 The patterns of N-glycosylation within the recombinant ectodomain and S1

300 rily conserved metabolic pathways of protein N-glycosylation, Wnt/beta-catenin signaling pathway, and