ライフサイエンスコーパス: NADPH diaphorase

1 the nodose also were stained positively for NADPH-diaphorase.

2 immunoreactive cells were also positive for NADPH-diaphorase.

3 f this study was to determine the changes in NADPH-diaphorase (a commonly used marker for neuronal NO

4 ere used for dual localization of Phox2a and NADPH diaphorase, a marker of nitric oxide-containing ne

5 e Fos-immunoreactive neurons did not contain NADPH-diaphorase, a marker for nitric oxide synthase.

6 is study demonstrates the co-localisation of NADPH diaphorase activity and GFAP immunoreactivity in n

7 kocytes contained little iNOS antigen and no NADPH diaphorase activity and were minimally able to con

8 n nNOS to basal and injury-induced levels of NADPH diaphorase activity in MNs.

9 on of cyclic guanosine monophosphate (cGMP), NADPH diaphorase activity, and nitrotyrosine occurred 3

10 nthase (eNOS)--these cells also co-localised NADPH diaphorase activity.

11 hydroxylase, somatostatin, substance P, and NADPH diaphorase activity.

12 nicotinamide adenine dinucleotide phosphate (NADPH) diaphorase activity.

13 cally, the development of neurons expressing NADPH-diaphorase activity (an early marker found in inhi

14 NADPH-diaphorase activity also was observed in the later

15 Similar to iNOSFL, iNOS8(-)9(-) exhibited NADPH-diaphorase activity and contained tightly bound ca

16 Throughout this period, the optic lobes show NADPH-diaphorase activity and stain with an antibody to

17 n the literature relating to the presence of NADPH-diaphorase activity in hippocampal principal cells

18 The intensity of NADPH-diaphorase activity in pyramidal cells located in

19 If NADPH-diaphorase activity is an indicator of NOS, then o

20 critical determinants of CA1 pyramidal cell NADPH-diaphorase activity is shown to be incubation of b

21 The restrained-induced NADPH-diaphorase activity was significantly higher in th

22 d reductions in ischemia-induced BH4 levels, NADPH-diaphorase activity, and caspase-3 gene expression

23 rom guinea pig hearts stained positively for NADPH-diaphorase activity, suggesting that these cells d

24 ere also found to contain NOS and to possess NADPH-diaphorase activity.

25 4% of LES-projecting neurons also contained NADPH-diaphorase activity; however, TH immunoreactivity

26 n of the ventromedial nucleus contained both NADPH diaphorase and brain nitric oxide synthase as demo

27 That NADPH diaphorase and brain nitric oxide synthase were fo

28 inding cells was shown by co-localization of NADPH diaphorase and estrogen receptor and brain nitric

29 Mitochondria in mSOD1 mouse MNs accumulate NADPH diaphorase and inducible nitric oxide synthase (iN

30 The distribution of the enzymes NADPH diaphorase and nitric oxide synthase in the ventro

31 nicotinamide adenine dinucleotide phosphate (NADPH)-diaphorase and Fos.

32 anglia were extremely rich in VIP, nNOS, and NADPH-diaphorase and moderate in CHAT.

33 d plates were also demonstrated in SM, using NADPH-diaphorase and NOS immunoreactivity, indicating ni

34 small vessels exhibited strong staining for NADPH-diaphorase but no nNOS immunoreactivity.

35 the entire ventromedial nucleus showed that NADPH diaphorase cellular staining was localized primari

36 ed a high density of nNOS immunopositive and NADPH-diaphorase containing neurons and fibers at the ro

37 Combined techniques for NADPH-diaphorase enzyme histochemistry and huntingtin im

38 tive, affecting principally parvalbumin- and NADPH diaphorase-expressing interneurons.

39 n influences neuronal process projection for NADPH diaphorase-expressing, but not acetylcholinesteras

40 NADPH diaphorase histochemical and NOS I immunohistochem

41 ine acetyltransferase immunocytochemical and NADPH diaphorase histochemical preparations at ages (pos

42 We used a modified form of NADPH diaphorase histochemistry to compare the neuroanat

43 cholinergic (NANC) relaxation, NOS activity, NADPH diaphorase histochemistry, NOS immunohistochemistr

44 nicotinamide adenine dinucleotide phosphate (NADPH)-diaphorase histochemistry and in situ hybridizati

45 udies used nicotinamide adenine diphosphate (NADPH)-diaphorase histochemistry as an indicator of nitr

46 nicotinamide adenine dinucleotide phosphate (NADPH)-diaphorase histochemistry to identify populations

47 nicotinamide adenine dinucleotide phosphate (NADPH)-diaphorase histochemistry to investigate nitric o

48 nicotinamide adenine dinucleotide phosphate (NADPH)-diaphorase histochemistry.

49 al nucleus of the trapezoid body (MNTB) with NADPH-diaphorase histochemistry and in situ hybridizatio

50 fied with either ChAT immunocytochemistry or NADPH-diaphorase histochemistry and they appeared to be

51 NADPH-diaphorase histochemistry has been shown to stain

52 + neurons and fibers were also identified by NADPH-diaphorase histochemistry in sections and whole-mo

53 ic oxide synthase activity (visualized using NADPH-diaphorase histochemistry) was undetectable in the

54 The brainstem nuclei were also visualized by NADPH-diaphorase histochemistry, a marker of nNOS activi

55 Using NADPH-diaphorase histochemistry, neuronal nitric oxide s

56 Furthermore, NADPH-diaphorase immunohistochemical staining of neurons

57 Our results suggest that NADPH diaphorase in aldehyde-fixed tissues is not enzyma

58 Generally, the staining pattern of nNOS and NADPH-diaphorase in the NTS was similar.

59 The localization of NADPH-diaphorase in these efferents indicated that they

60 iety of identified neurons were positive for NADPH-diaphorase in various central ganglia, including t

61 NADPH diaphorase is also present in a subpopulation of t

62 NADPH diaphorase is used as a histochemical marker of ni

63 We conclude that (i) fixative-resistant NADPH-diaphorase is a characteristic marker of 12-15% of

64 NOS I, as defined mainly by the presence of NADPH diaphorase, is present in a subpopulation of both

65 At the cellular level the NADPH-diaphorase marker for nNOS revealed a significant

66 the presence and subcellular distribution of NADPH diaphorase (NADPH-d)/nitric oxide synthase (NOS) i

67 Expression of specific, fixative-resistant NADPH-diaphorase (NADPH-d) activity, characteristic of N

68 Recent evidence indicates that NADPH-diaphorase (NADPH-d) and nitric oxide synthase (NO

69 usc Aplysia californica was studied by using NADPH-diaphorase (NADPH-d) histochemistry in the CNS and

70 xide synthase (NOS) immunohistochemistry and NADPH-diaphorase (NADPH-d) histochemistry, which yielded

71 means of specific antibodies against NOS and NADPH-diaphorase (NADPH-d) histochemistry, which, with t

72 eech, Hirudo medicinalis, were studied using NADPH-diaphorase (NADPH-d) histochemistry.

73 californica was studied histochemically via NADPH-diaphorase (NADPH-d) reduction of Nitro Blue Tetra

74 ined the normal distribution of constitutive NADPH-diaphorase (NADPH-d), a marker for nitric oxide sy

75 Formaldehyde-insensitive NADPH diaphorase (NADPHd) activity is used widely as a h

76 e synthase (nNOS) and enzymatic activity for NADPH diaphorase (NADPHd) are extensively colocalized in

77 labeling (IFL) methods, and each followed by NADPH diaphorase (NADPHd) histochemical staining in the

78 n parvalbumin (PV) and nitric oxide synthase NADPH diaphorase (NADPHd) is well documented within neur

79 nicotinamide adenine dinucleotide phosphate (NADPH)-diaphorase (NADPHd) and parvalbumin (PV)-positive

80 NADPH-diaphorase (NADPHd) has been determined biochemica

81 NADPH-diaphorase (NADPHd) histochemistry or NOS-immunost

82 gregates were present in almost all cortical NADPH-diaphorase neurons and in approximately 50% of the

83 were more prominent as nuclear inclusions in NADPH-diaphorase neurons, with less perikaryal and neuro

84 myenteric and submucosal plexus stained with NADPH diaphorase (neurons and neurites), anti-TuJ1 (neur

85 A few VIP+ and NADPH- diaphorase+ neurons were also observed in the cho

86 increase in the number of cells labeled for NADPH diaphorase or neuronal NOS in the lumbosacral spin

87 es, with little or no colocalization between NADPH-diaphorase or nitric oxide synthase neurons and hu

88 Axons labeled for VIP, nNOS, NADPH-diaphorase, or the 3A10 antigen could be traced fr

89 ic striatal neurones and the degeneration of NADPH-diaphorase positive interneurones within 24 h.

90 in activity was in accordance with decreased NADPH-diaphorase-positive cells and decreased staining o

91 In contrast, a similar loss of NADPH-diaphorase-positive cells was observed in the stri

92 NADPH-diaphorase-positive cells were also increased in t

93 sed by efferent fibers and to localize these NADPH-diaphorase-positive efferent cell bodies in the tu

94 ound the locus coeruleus corresponded to the NADPH-diaphorase-positive efferent cells in the avian is

95 n which P-CREB-lir was induced by light were NADPH-diaphorase-positive neurons of the SCN's retinorec

96 nNOS immunostaining and NADPH-diaphorase-positive neurons were significantly inc

97 At both dorsal and ventral levels, NADPH-diaphorase-positive subicular pyramidal cells and

98 ric ganglia, total neurons per ganglion, and NADPH diaphorase presumptive inhibitory neurons per gang

99 nicotinamide-adenine dinucleotide phosphate (NADPH)- diaphorase reaction was used as a marker for nit

100 nicotinamide adenine dinucleotide phosphate (NADPH)-diaphorase reaction, we determined that the organ

101 The NADPH-diaphorase reaction was also used as a marker for

102 r of studies have mapped the distribution of NADPH-diaphorase-reactive neurons in the hippocampal for

103 oint of controversy concerns the presence of NADPH-diaphorase-reactive pyramidal cells in the CA1 sub

104 results show that nNOS immunoreactivity and NADPH-diaphorase reactivity are consistently increased i

105 nNOS immunostaining and NADPH-diaphorase reactivity was neither altered in the g

106 rs exhibiting both nNOS immunoreactivity and NADPH-diaphorase reactivity was present in the central,

107 nNOS expression and NADPH-diaphorase reactivity were quantified by using a m

108 teric neurons, and whole-mount staining with NADPH-diaphorase showed that myenteric and submucosal ga

109 Diabetic mice had loss of NADPH diaphorase-stained myenteric neurons, delayed gast

110 NADPH diaphorase staining (NOS index) intensity was high

111 as demonstrated by in situ hybridization and NADPH diaphorase staining in rats treated with TGF-beta1

112 osition of S-nitrosothiols, markedly reduced NADPH diaphorase staining in tissue sections subsequentl

113 NADPH diaphorase staining was visible in both neuronal p

114 as 32 and 25, three defined bands of diffuse NADPH diaphorase staining were located in layer 2 and in

115 teral dorsal tegmental nucleus identified by NADPH diaphorase staining, as well as the cuneiform nucl

116 NADPH-diaphorase staining also was detected in blood ves

117 O-producing neurons using fixation-resistant NADPH-diaphorase staining and antisera that recognize a

118 ed, however, between nNOS immunostaining and NADPH-diaphorase staining in blood vessels in the brains

119 treated arteries showed a 2-fold increase in NADPH-diaphorase staining intensity relative to sham-inf

120 urons exhibiting NOS activity as assessed by NADPH-diaphorase staining was significantly greater in t

121 study, nitric oxide synthase (NOS) activity (NADPH-diaphorase staining), neuronal NOS (nNOS) protein,

122 independent of cell volume, correlates with NADPH-diaphorase staining, and appears to be a character

123 inhibitory neurotransmitter NO, as shown by NADPH-diaphorase staining, and the glial marker GFAP.

124 fibers exhibiting nNOS immunoreactivity and NADPH-diaphorase staining.

125 urons and in approximately 50% of the spared NADPH-diaphorase striatal neurons from early grade HD ca

126 n to be involved in learning and memory, the NADPH-diaphorase technique was used in conjunction with

127 rons was determined on sections stained with NADPH diaphorase to identify the cholinergic boundaries

128 cotinamide adenosine dinucleotide phosphate (NADPH)-diaphorase, tyrosine hydroxylase (TH), and dopami

129 n almost perfect co-localization of ChAT and NADPH-diaphorase was also observed.

130 ns and fibers that stained for both nNOS and NADPH-diaphorase was noted in the interstitial and ventr

131 ascular endothelial cells known to stain for NADPH diaphorase were rich in S-nitrosothiols, and (7) p

132 containing choline acetyltransferase and/or NADPH diaphorase were studied in E12.5-E17.5 reeler and

133 Nerve terminals positive for NADPH- diaphorase were colocalized with SM alpha-actin-p

134 ioside (P-Path), and the granule cell marker NADPH-diaphorase, were disrupted.