戻る
「早戻しボタン」を押すと検索画面に戻ります。

今後説明を表示しない

[OK]

コーパス検索結果 (left1)

通し番号をクリックするとPubMedの該当ページを表示します
1                                              NAIP CIIA HET-E and TP1 (NACHT) family proteins are invo
2                                              NAIPs (NLR family, apoptosis inhibitory proteins) are NL
3 f the human IAP family, c-IAP-1, c-IAP-2 and NAIP.
4 e activation in response to NLRP3, AIM2, and NAIP triggers when expressed in living cells, highlighti
5 d standard (n-nonadecane) for both NACME and NAIP esters were identical.
6 4/NOD1, NOD2/CARD15, CIAS1, CARD7/NALP1, and NAIP, in more detail.
7 ed the possibility of alterations in SMN and NAIP in 154 patients with ALS (135 sporadic cases, 17 fa
8 calized the two SMA candidate genes, SMN and NAIP, to the Lgn1 critical region, making these two gene
9 ally have functional copies of both SMNT and NAIP.
10 ession of catalytically inactive JNK1 blocks NAIP and ML-IAP protection against ICE- and TNF-alpha-in
11 le proteins (PrgI and homologs), detected by NAIP and the NLRC4 inflammasome.
12           We analyzed a panel of 43 chimeric NAIPs, allowing us to map the NAIP domain responsible fo
13                                 In contrast, NAIP failed to bind tightly to any of these proteases.
14          Here we show in mice that different NAIP paralogues determine the specificity of the NLRC4 i
15 ted with SMA, it is not clear to what extent NAIP or other genes influence the SMA phenotype, or whet
16 rt that two other members of the IAP family, NAIP and ML-IAP, both activate JNK1.
17 ene products and CED-4) and NACHT (named for NAIP, CIIA, HET-E, and TEP1) subfamilies of the STAND NT
18 cal mechanism underlying the requirement for NAIP proteins by use of a reconstituted NLRC4 inflammaso
19  contrast, humans encode a single functional NAIP, raising the question of whether human NAIP senses
20 cription regulators) and NACHT NTPases (e.g. NAIP, TLP1, Het-E-1) that have been studied extensively
21 e neuronal apoptosis inhibitory protein gene NAIP, of the survival motor neuron gene SMN, and of a fu
22 ultiple isoforms encoded by the single human NAIP gene.
23     Furthermore, we show that a single human NAIP isoform is capable of sensing the T3SS inner rod, n
24 erial ligands is conferred by a single human NAIP.
25                     Here, we show that human NAIP also senses the Salmonella Typhimurium T3SS inner r
26            Previous studies found that human NAIP detects both flagellin and the T3SS needle protein
27 ine NAIP1 is functionally analogous to human NAIP.
28  NAIP, raising the question of whether human NAIP senses one or multiple bacterial ligands.
29                         Our results identify NAIPs as immune sensor proteins and provide biochemical
30 pite strong biochemical evidence implicating NAIPs in specific detection of bacterial ligands, geneti
31 eal the mechanism of signal amplification in NAIP-NLRC4 inflammasomes.
32             Wildfire-associated increases in NAIP and the EPC0 persisted 6 and 7 years after wildfire
33 of apoptosis (IAP) family members, including NAIP, cIAP-1, cIAP-2, XIAP/hILP, survivin, and BRUCE, bl
34       The N-alkylated indanylidenepyrroline (NAIP) Schiff base 3 is an unnatural alpha-amino acid pre
35 e converted to NACME and N-acetyl-isopropyl (NAIP) esters; the latter established derivative was empl
36 ificantly different (KIE(NACME) = 1.036; KIE(NAIP) = 1.038) and were shown in both cases to be reprod
37         We generated knock-in mice that lack NAIP of MeCP2 and found that they performed better in hi
38 indings indicate that, in contrast to murine NAIPs, promiscuous recognition of multiple bacterial lig
39 n mouse and human is the Lgn1 candidate Naip/NAIP.
40 ined NOD-like receptors (NLRs): NODs, NALPs, NAIP and IPAF.
41 ctivate the best-studied NLRs (NLRP3, NLRC4, NAIP, and NLRP1) and in uncovering inflammasome NLRs.
42 letion of NAIP, with a homozygous absence of NAIP exon 5.
43 ingle patient revealed a partial deletion of NAIP, with a homozygous absence of NAIP exon 5.
44 of spinal muscular atrophy while the loss of NAIP and perhaps other genes primarily affects the sever
45             These data indicate that loss of NAIP may affect disease severity and further, that the m
46  bystander apoptosis, while up-regulation of NAIP and cIAP2 mRNA suggest that EBOV has evolved additi
47 red for the subsequent co-oligomerization of NAIPs with the downstream signaling adaptor NLR family,
48 an macrophages are known to express only one NAIP gene, which detects the needle protein, but not rod
49 l either provide a novel function for SMN or NAIP or reveal the existence of another, yet uncharacter
50 higher levels of bioavailable particulate P (NAIP) - these effects were also observed downstream at l
51 tional roles of inflammasomes - particularly NAIP/NLRC4, NLRP6, and noncanonical caspase-4 (caspase-1
52 t neuronal activity-induced phosphorylation (NAIP) of methyl CpG-binding protein 2 (MeCP2) precedes i
53 r the neuronal apoptosis inhibitory protein (NAIP) gene and a somewhat lesser fraction for the basal
54 f the neuronal apoptosis inhibitory protein (NAIP) gene are preferentially deleted in patients with s
55 gnition by NLR apoptosis inhibitory protein (NAIP) inflammasomes.
56 , the neuronal apoptosis inhibitory protein (NAIP).
57 SMN); Neuronal Apoptosis Inhibitory Protein (NAIP); and p44, a subunit of transcription factor II H (
58 ining family, apoptosis inhibitory proteins (NAIPs) activate the nucleotide-binding domain, leucine-r
59 he NLR family apoptosis inhibitory proteins (NAIPs) bind conserved bacterial ligands, such as the bac
60         Mice express multiple highly related NAIP paralogs that recognize distinct bacterial proteins
61  is one of the rare carriers of SMA who show NAIP deletions, a further explanation is that the NAIP d
62 sults provide genetic evidence that specific NAIP proteins function to detect specific bacterial prot
63 mean STDV(NACME) = 0.3 per thousand and STDV(NAIP) = 0.4 per thousand).
64                                We found that NAIP proteins control ligand-dependent oligomerization o
65                     Our results suggest that NAIP of MeCP2 is required for modulating dynamic functio
66                                          The NAIP/NLRC4 inflammasomes activate caspase-1 in response
67 tivation of other inflammasomes, such as the NAIP (NLR family, apoptosis inhibitory protein)/NLRC4 in
68 of 43 chimeric NAIPs, allowing us to map the NAIP domain responsible for specific ligand detection.
69 , a previously uncharacterized member of the NAIP gene family, whereas NAIP5 and NAIP6 activate NLRC4
70  receptor-ligand model for activation of the NAIP-NLRC4 inflammasomes.
71 pyroptosis by a mechanism independent of the NAIP/NLRC4 and caspase-1 axis.
72 deletions, a further explanation is that the NAIP deletion is in some way contributing to the ALS phe
73 KCdelta, ligand binding proteins such as the NAIPs, and caspase-11 and caspase-8 in addition to caspa
74 regions of the oligomerization domain of the NAIPs, rather than within the leucine-rich repeats, as w
75  death proteases, caspases-3 and -7, whereas NAIP presumably inhibits apoptosis via other targets.
76  with estimated Kis of <=0.1 microM, whereas NAIP did not.
77 poptosis (IAP) family (HIAP-1, HIAP-2, XIAP, NAIP, Survivin, and Livin).
78                           In addition, XIAP, NAIP, and JNK1 bind to TAK1.

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。