ライフサイエンスコーパス: NAT

1 NAT gene regions also exhibit higher levels of H3K36me3,

2 NAT was measured at the level of the C5 vertebral body,

3 NAT(+) samples were confirmed by real-time polymerase ch

4 acetylation and internal modifications in a NAT substrate-specific manner.

5 ORE), a circadian-regulated lncRNA that is a NAT of CDF5.

6 tion at convergent loci is also found when a NAT to hygromycin resistance gene is driven off the endo

7 curonide and ethyl sulfate, N-acetyltaurine (NAT) was identified as a urinary metabolite that is high

8 homologous to arylamine N-acetyltransferase (NAT) and has been identified in Fusarium infecting cerea

9 ity of pineal serotonin N-acetyltransferase (NAT) exhibits a large circadian rhythm that is dependent

10 unconventional N-terminal acetyltransferase (NAT) because it localizes to organelles, in particular t

11 mutation in an N-terminal acetyltransferase (NAT) gene.

12 The human N-terminal acetyltransferase (NAT) Naa50p (NAT5/SAN) acetylates the alpha-amino group

13 ncharacterized N-terminal acetyltransferase (NAT) specificities, and emerging evidence of posttransla

14 he major human N-terminal acetyltransferase (NAT).

15 y a family of N-terminal acetyltransferases (NAT).

16 activity of arylamine N-acetyltransferases (NATs) in excised liver samples was examined using eighte

17 Arylamine N-acetyltransferases (NATs), a class of xenobiotic-metabolizing enzymes, catal

18 arried out by N-terminal acetyltransferases (NATs), is a conserved and primary modification of nascen

19 ukaryotic amino-terminal acetyltransferases (NATs), which are differentiated from one another on the

20 by different N-terminal acetyltransferases (NATs).

21 ut by six amino-terminal acetyltransferases (NATs).

22 biotics is powered by the N-acyltransferase (NAT) Orf11*/Dbv8 through N-acylation on glucosamine at t

23 st was followed <6 months by >/=1 additional NAT(s), or ALT, AST, and platelets <90 days, or any test

24 All NATs contain at least one catalytic subunit, and some co

25 rong interaction between water ice and alpha-NAT was found, which explains the experimental spectra a

26 d phase, alpha-nitric acid trihydrate (alpha-NAT), is presented.

27 nalyses reveal altered pathways shared among NATs across tissue types.

28 es and is believed to represent an ancestral NAT variant from which the eukaryotic NAT machinery evol

29 entrations of COT, COT-OH, ANATA, ANABA, and NAT were 5200, 2600, 30, 10, and 0.6 ng/L, respectively,

30 in this period were observed between DOX and NAT (33.10%) and DOX and BC (32.43%).

31 al differences were observed between DOX and NAT (34.61%) and DOX and BC (23.11%).

32 Much more lincRNA and NAT genes were derived from conserved genomic regions be

33 ors, and to conduct HIV antibody testing and NAT as close to the time of donation as possible to prev

34 d the X-ray crystal structure of an archaeal NAT from Sulfolobus solfataricus (ssNAT).

35 active and to acetylate prototypic arylamine NAT substrates.

36 However, a putative atypical NAT harboring a catalytic triad Glu residue was recently

37 functional characterization of this atypical NAT.

38 VAb testing in fingerstick blood followed by NAT in venipuncture blood yields relatively lower viremi

39 VAb testing in fingerstick blood followed by NAT in venipuncture blood.

40 The number of cases identified by NAT ranges between 1 : 1000 and 1 : 50 000, depending on

41 HBV blood safety is improved by NAT for HBV DNA when applied to individual donations.

42 ve five other samples which were negative by NAT.

43 sense and antisense transcripts produced by NAT pairs is significantly correlated, particularly unde

44 he mechanisms of epigenetic modifications by NATs and their emerging role as master regulators of chr

45 Cis-NAT promoters also show highly tissue-specific patterns

46 cis-NAT-derived siRNAs (nat-siRNAs) are present in plants, a

47 Among them, 16 cis-NAT pairs from Arabidopsis and 34 from rice gave rise to

48 We found 209 cis-NAT pairs that have opposite expression levels in neighb

49 Out of 1,439 and 767 cis-NAT pairs identified in Arabidopsis and rice, respective

50 We found that a cis-NAT positively regulates the level of a protein critical

51 Active cis-NAT promoters are enriched with activating histone modif

52 -NAT pairs and identified 918 additional cis-NAT pairs.

53 ol II binding show peaks centered around cis-NAT transcriptional start sites, and the levels of activ

54 s of activating histone modifications at cis-NAT promoters are positively correlated with cis-NAT exp

55 is-NATs, suggesting a connection between cis-NAT transcription and chromatin modification in plants.

56 Compared with other genes, cis-NAT-encoding genes have enriched low-nucleosome-density

57 d RNA Pol II binding properties of human cis-NAT promoters genome-wide.

58 Newly identified cis-NAT pairs are supported by polyadenylation data, alterna

59 is libraries confirmed most of the known cis-NAT pairs and identified 918 additional cis-NAT pairs.

60 ing data showed that approximately 4% of cis-NAT pairs produce putative cis-NAT-induced siRNAs.

61 data were used to identify thousands of cis-NAT promoters, and profiles of nine histone modification

62 found in the 5' regulatory sequences of cis-NAT-encoding genes.

63 We also broadly extend annotations of cis-NAT-siRNA loci, identifying ones with common expression

64 ely 4% of cis-NAT pairs produce putative cis-NAT-induced siRNAs.

65 and occupied by RNA Pol II, whereas weak cis-NAT promoters are depleted for both activating modificat

66 promoters are positively correlated with cis-NAT expression levels.

67 s (Arabidopsis thaliana), protein-coding cis-NATs are also characterized by high abundance, high coex

68 ations of new technologies for detecting cis-NATs, including direct RNA sequencing and strand-specifi

69 s, implying cell-type-specific roles for cis-NATs.

70 ural antisense small RNA production from cis-NATs is limited.

71 NATs and 19%-29% of the siRNA-generating cis-NATs in plants give rise to siRNAs only in their overlap

72 These results suggest that human cis-NATs are actively transcribed by the RNA Pol II and that

73 To identify cis-NATs using ssRNA-seq, we developed a new computational m

74 of siRNAs in the overlapping regions of cis-NATs and 19%-29% of the siRNA-generating cis-NATs in pla

75 entified a unique chromatin signature of cis-NATs, suggesting a connection between cis-NAT transcript

76 r multifaceted regulatory roles of plant cis-NATs.

77 sms for regulating the expression of the cis-NATs.

78 The extent to which these cis-NATs are actively regulated and ultimately functionally

79 luding dcl1, dcl2, dcl3, and rdr6 map to cis-NATs as frequently as small RNAs sequenced from wild-typ

80 an 17,000 unique siRNAs corresponding to cis-NATs from biotic and abiotic stress-challenged Arabidops

81 cis-natural antisense transcripts (cis-NATs) are widespread in plants and are often associated

82 alled cis-natural antisense transcripts (cis-NATs), and they play key roles in the regulation of gene

83 cis-natural sense antisense transcripts (cis-NATs).

84 erous cis-natural antisense transcripts (cis-NATs).

85 ncode cis-natural antisense transcripts (cis-NATs).

86 ved: BC (untreated, filled with blood clot), NAT (natrosol gel alone), and DOX (10% doxycycline gel).

87 te congenital infection and surveillance CMV NAT at 5 additional intervals between birth and 90 days,

88 The infants underwent serum and urine CMV NAT at birth to evaluate congenital infection and survei

89 We compared NAT compartments across lean, overweight, and obese grou

90 Conclusion NAT followed by resection has a significant survival ben

91 d multivariate regression models correlating NAT with CVD risk factors.

92 effects on 626 concordant and 766 discordant NAT pairs.

93 We show that the newly discovered NAT negatively regulates Nos1 gene expression.

94 w projections of dengue incidence from donor NAT yield data and vice versa, and suggest that viremic

95 So far, no eukaryotic NAT has been mechanistically investigated.

96 implications for the evolution of eukaryotic NAT enzymes and the substrate specificities therein.

97 estral NAT variant from which the eukaryotic NAT machinery evolved.

98 catalysis in both prokaryotic and eukaryotic NATs.

99 To date, two eukaryotic NATs, NatA and NatE, have been structurally characterize

100 In tumor xenografts that did not express NAT, intratumoral or intravenous injection of HSV1716/NA

101 Finally, NAT pairs in 368 diverse maize inbreds and 19 segregatin

102 We propose that the CDF5/FLORE NAT pair constitutes an additional circadian regulatory

103 loying a rapid, point-of-care assay) and for NAT (whether done by reflex or using separately drawn bl

104 in order to decipher the molecular basis for NAT's functionality.

105 s, we propose a new three-step mechanism for NAT formation in high-altitude ice clouds.

106 sociated factor as a potential repressor for NAT abundance.

107 gy requiring the tested person to return for NAT.

108 The strategy that requires returning for NAT is even less viremia sensitive (<0.9000) because of

109 sive account of NATs and supports a role for NATs' regulation of tumor suppressors and oncogenes in c

110 The lessons from NAT are translatable to clinical oncology and may help t

111 d are similar to those present in functional NATs.

112 ing OncoNAT we identified several functional NATs, including NKX2-1-AS1 that regulates the NKX2-1 onc

113 We propose that fungal NAT isoenzymes may have evolved to perform diverse funct

114 Further, NATs are significantly hypomethylated and include fewer

115 rticipants recommended that HIV, HBV and HCV NAT should not be required for live donor evaluation; th

116 OPOs performed prospective HIV, HBV and HCV NAT, respectively.

117 mediated amplification assay for HIV and HCV NAT, respectively.

118 IV NAT and 55 respondents that performed HCV NAT, 39 tested all donors.

119 ntly infected" if they had >/=1 positive HCV NAT; "in care" if a positive RNA test was followed <6 mo

120 ively analyze the transcriptomes of healthy, NAT, and tumor tissues in 6506 samples across eight tiss

121 ingly, co-depletion of NatA, a heterodimeric NAT complex that physically interacts with Naa50, rescue

122 te indicated that the kidney has the highest NAT synthase activity among the tested organs, whereas t

123 Of the 56 respondents that performed HIV NAT and 55 respondents that performed HCV NAT, 39 tested

124 risk behaviors, and 55% never performed HIV NAT.

125 oradrenaline transporter (NAT) gene (HSV1716/NAT), whose expression endows infected cells with the ca

126 Administration of HSV1716/NAT and (131)I-MIBG resulted in decreased tumor growth a

127 atumoral or intravenous injection of HSV1716/NAT induced the capacity for active uptake of (131)I-MIB

128 We used HSV1716/NAT as a dual cell lysis-gene delivery vehicle for targe

129 nin tautomers and radical species into human NAT crystal structures supported the hypothesis that thi

130 e catalytic subunit of NatA, the major human NAT involved in the co-translational acetylation of prot

131 The involvement of human NAT enzymes in different pathological conditions, such a

132 The resulting HXT-NAT reporter strains exhibited a strict growth dependenc

133 0 (also named NatF) is a recently identified NAT found only in multicellular eukaryotes.

134 serology negative donors to 70.8% (17/24) if NAT was available and the donor had no increased activit

135 espondents were more likely to accept IRD if NAT was available.

136 Thus, many of our processes are, in NAT terms, interactively complex and tightly coupled wit

137 the hypothesis that thiol functionalities in NAT enzymes may be crucial in apocynin binding.

138 18 genes that are specifically activated in NATs.

139 cies was at least as effective in increasing NAT activity as seen with control nerves.

140 Expression of the spliced form of LEF1 NAT in trans prevents the action of unspliced NAT by com

141 Unspliced LEF1 NAT interacts with LEF1 promoter and facilitates PRC2 bi

142 pport the use of more sensitive assays, like NAT, in HIV screening of populations with a high prevale

143 cant (p < 0.001) reduction of in vitro liver NAT activity up to 93% as compared with untreated rats (

144 the opposite strand of a coding gene locus, NATs are proving to be a heterogeneous group with high p

145 Here we investigated a long NAT (antiNOS-2 RNA) associated with the regulation of ni

146 Here we report that a long NAT (Mm-antiNos1 RNA) complementary to mRNA encoding the

147 Although recent studies indicate that long NATs are engaged in the regulation of gene expression, t

148 in SK-N-BE(2c) (neuroblastoma) and UVW/NAT (NAT-transfected glioma) cells.

149 B (AmB), voriconazole (VCZ), and natamycin (NAT) was determined using the CLSI-M38-A2 method and XTT

150 h acute HIV infection (HIV antibody negative/NAT positive) were identified; the HIV Combo assay detec

151 s is silent in epithelial cells, and neither NAT transcript nor LEF1 mRNA are expressed, in cell line

152 -nitrosoanabasine (NAB), N-nitrosoanatabine (NAT), N-nitrosonornicotine (NNN), 4-(methylnitrosamino)-

153 the CSTs were crushed bilaterally, nocturnal NAT was decreased by 99%.

154 ansposable element sequences relative to non-NAT genes.

155 ly higher proportion of NATs relative to non-NATs are specifically expressed under water stress (WS).

156 information necessary for directing a normal NAT rhythm and thus normal pineal function.

157 eporter strains in which the nourseothricin (NAT) resistance gene is expressed under the control of t

158 udy, we identified and characterized a novel NAT, AS-IL1alpha, which is partially complementary to IL

159 were performed to examine the association of NAT compartments with metabolic syndrome.

160 The results suggest that availability of NAT would increase IRD utilization.

161 th Carolina, we have applied the concepts of NAT to our practice to better understand our systems' be

162 e first molecular scaffold for the design of NAT-specific small molecule inhibitors with possible the

163 we investigate functional diversification of NAT enzymes in crop-compromising species of Fusarium and

164 logy (NAT) yield to estimate the duration of NAT-detectable viremia and compared with reported clinic

165 bacteria against the antibacterial effect of NAT-26.

166 To gain insight into the evolution of NAT enzymes, we determined the X-ray crystal structure o

167 ocynin led to a dose-dependent inhibition of NAT activity with IC50 = 0.69 +/- 0.02 mM.

168 gested that acetate is the main precursor of NAT, which was further confirmed by the stable isotope l

169 is known about the transcriptomic profile of NAT, how it is influenced by the tumor, and how the prof

170 histone modifications have in regulation of NAT expression, and the significance of NATs in response

171 A set of NAT genes also showed expression correlation with their

172 ans, whereas the cytosol is the main site of NAT biosynthesis inside the cell.

173 Among three possible substrates of NAT biosynthesis (taurine, acetyl-CoA, and acetate), the

174 These findings support the use of NAT, particularly as a patient selection tool, in the ma

175 is study provides a comprehensive account of NATs and supports a role for NATs' regulation of tumor s

176 with light-responsive expression changes of NATs.

177 Understanding the broad range of effects of NATs will shed light on the complex mechanisms that regu

178 further study of the evolutionary history of NATs and the functional significance of the predominant

179 We also show that inhibition of NATs leads to increases in glial-derived neurotrophic fa

180 nificance of the subcellular localization of NATs and their biological functions remain to be defined

181 A significantly higher proportion of NATs relative to non-NATs are specifically expressed und

182 We found an unexpected large proportion of NATs with protein coding potential, as estimated by ribo

183 were enriched in the overlapping regions of NATs and exhibited either site-specific or distributed p

184 tion of cognate sense genes, and the role of NATs in cancer remain poorly understood.

185 To understand the role of NATs in the clock, we put the frq antisense transcript q

186 n of NAT expression, and the significance of NATs in response to WS.

187 The functional significance of NATs is poorly understood, but their prevalence in the C

188 Although thousands of NATs are encoded by mammalian genomes, their functions i

189 his modification is less prevalent, only one NAT enzyme has been identified.

190 or NAT (n = 87; P = 0.003); N+ without VR or NAT (n = 50; P = 0.004).

191 atectomy (n = 117; P = 0.049); without VR or NAT (n = 87; P = 0.003); N+ without VR or NAT (n = 50; P

192 d 1769 sense and antisense transcript pairs (NAT pairs) in two maize inbreds with different sensitivi

193 egion of natural antisense transcript pairs (NAT) (nat-siRNAs).

194 und evidence for cytoplasmically partitioned NATs.

195 agments containing the antimicrobial peptide NAT-26.

196 Since 2008, the number of OPOs performing NAT has increased and more OPOs are testing all donors.

197 a considerable promise for low-cost and POC NAT in remote areas.

198 in insects: typical insect aaNAT, polyamine NAT-like aaNAT, and mosquito unique putative aaNAT (paaN

199 aaNAT5b, a protein from polyamine NAT -like aaNAT cluster, uses hydrazine and histamine as

200 ed confirmatory testing for all HCV-positive NAT results.

201 ed confirmatory testing for all HIV-positive NAT results, and 15 (27%) OPOs performed confirmatory te

202 %, all strategies when adopting quantitative NAT vary little in cost (range, $29.50-$30.70) and are h

203 of-care assay, when followed by quantitative NAT done reflexively or in separately drawn blood, are c

204 dopting qualitative rather than quantitative NAT are slightly cheaper (range, $28.90-$29.99), similar

205 Only Cs- and Rb-NAT reveal a phase separation into a dense form (P2 phas

206 The results point to FAAH-regulated NAT signaling as an unprecedented lipid-based mechanism

207 3000 IU/mL and have the potential to replace NAT in settings with high HCV prevalence.

208 s for a large number of the light-responsive NAT pairs are associated with histone modification peaks

209 iochemical and metabolomic analysis revealed NAT is a novel metabolite of ethanol and a potential bio

210 nt NatA and Naa50 do not affect each other's NAT activity in vitro Because NatA and Naa50 exhibit dis

211 tegies, we identify two long-chain saturated NATs-N-tetracosanoyl-taurine [NAT(24:0)] and N-eicosanoy

212 od to assess metabolic risk, cross-sectional NAT assessment provides further insight into fat accumul

213 r, has encouraged the research for selective NAT inhibitors in both humans and animal models with pos

214 iptome analysis of 10 genes that make up six NATs in humans from eight different cell lines suggests

215 Contrarily to the spliced NAT, this unspliced NAT down-regulates the main LEF1 pro

216 bility of an adrenergic agonist to stimulate NAT activity was reduced in rats with regenerated CSTs.

217 Most strikingly, NAT can catalyze formation of 2-N,6-O-diacylated or C6--

218 trate that local administration of synthetic NATs accelerates wound closure in mice and stimulates re

219 st that pharmacological approaches targeting NATs can confer locus-specific gene upregulation effects

220 aurine [NAT(24:0)] and N-eicosanoyl-taurine [NAT(20:0)]-as primary substrates for FAAH in mouse skin,

221 hain saturated NATs-N-tetracosanoyl-taurine [NAT(24:0)] and N-eicosanoyl-taurine [NAT(20:0)]-as prima

222 n fatty acids with taurine [N-acyl-taurines (NATs)].

223 ve to nucleic acid amplification technology (NAT) yield to estimate the duration of NAT-detectable vi

224 ody (HCVAb) followed by a nucleic acid test (NAT) for HCV RNA when the antibody test is positive, are

225 We compared HIV RNA nucleic acid test (NAT) results to the results of a 4th-generation Ag/Ab as

226 Although the majority of tested NATs were found to localize to the nucleus, we also foun

227 epatitis B virus (HBV) nucleic acid testing (NAT) for donor blood screening led to its implementation

228 cy requires the use of nucleic acid testing (NAT) for organ donor screening.

229 Cytomegalovirus nucleic acid testing (NAT) of transfused blood components and breast milk was

230 sing a high-throughput nucleic acid testing (NAT) platform.

231 NGS) with confirmatory nucleic acid testing (NAT) to routine clinical diagnostic testing in detection

232 plant centers used HIV nucleic acid testing (NAT) to screen either all donors or donors with recogniz

233 ) had access to timely nucleic acid testing (NAT), and respondents were more likely to accept IRD if

234 creen and confirmatory nucleic acid testing (NAT).

235 l use of nucleic acid amplification testing (NAT) for the screening of potential organ donors should

236 ther point-of-care (POC) nucleic acid tests (NAT), MTNT could assay both DNA and RNA directly from li

237 , HCV RNA, HCV genotype (nucleic acid tests [NAT]), liver function tests, and platelet counts; patien

238 antigen may become more cost-effective than NAT for the screening of potential organ donors.

239 sis using deuterated ethanol determined that NAT is a novel metabolite of ethanol.

240 ut lower levels of H3K27me3, indicating that NAT gene pairs generally exhibit an open chromatin confi

241 Our analysis shows that NAT presents a unique intermediate state between healthy

242 Recent findings have shown that NATs can exert their regulatory functions by acting as e

243 The NAT enzyme possesses enormous value in untapped applicat

244 The NAT group was associated with improved survival compared

245 The NAT hNaa50p preferentially modifies peptides starting wi

246 rapy for cancer using HSV1716 expressing the NAT transgene and targeted radionuclide therapy.

247 henotype presenting low LEF1 expression, the NAT is synthesized and remains unprocessed.

248 or VR; (2) R1-0 mm posterior-margin for the NAT group (P = 0.004).

249 From the NAT group, 2,005 patients (95%) were matched with 6,015

250 ysis-gene delivery vehicle for targeting the NAT transgene to human tumor xenografts in vivo.

251 Thus, the NAT gene-infected cells are susceptible to targeted radi

252 rtebrates (e.g. fish) and mammals, where the NAT gene is selectively expressed in noradrenergic cells

253 The physiological functions of the NATs are unknown.

254 ower), concepts from Normal Accident Theory (NAT), a framework for analyzing failure potential within

255 n patients who received neoadjuvant therapy (NAT) followed by resection and those who received upfron

256 f UR patients who received adjuvant therapy, NAT patients had a better survival (adjusted hazard rati

257 by an antisense non-coding RNA and that this NAT function is regulated by the balance between its spl

258 ith the hypothesis that neck adipose tissue (NAT) accumulation preferentially involves specific compa

259 bjective was to measure neck adipose tissue (NAT) compartments and examine relations with CVD risk ma

260 tigen (HCVcAg) is a potential alternative to NAT.

261 A natural antisense transcript (NAT) is transcribed from a promoter present in the first

262 by acting as a natural antisense transcript (NAT) that regulates the neuronal mRNA levels of seizure

263 s of sense and natural antisense transcript (NAT).

264 proportion of natural antisense transcripts (NATs) and sense-antisense transcript pairs (SATs).

265 Natural antisense transcripts (NATs) are a class of long noncoding RNAs (lncRNAs) that

266 Natural antisense transcripts (NATs) are a prominent and complex class of regulatory RN

267 Natural antisense transcripts (NATs) are endogenous RNA molecules that are complementar

268 ese so-called natural antisense transcripts (NATs) are possibly co-regulated.

269 ng non-coding natural antisense transcripts (NATs) are widespread in eukaryotic species.

270 tory roles of natural antisense transcripts (NATs) arising from what was previously thought to be 'ju

271 egradation of natural antisense transcripts (NATs) by single-stranded oligonucleotides or siRNAs can

272 scovered that natural antisense transcripts (NATs) frequently have actively translated sORFs, includi

273 production of natural antisense transcripts (NATs) in the context of actively expressed genes.

274 ong noncoding natural antisense transcripts (NATs) originate from Period in mammals and frequency (fr

275 NAs) that are natural antisense transcripts (NATs) to transcripts encoding central oscillator compone

276 this period, natural antisense transcripts (NATs), which are transcribed from the opposite strand of

277 he extent of natural antisense transcripts' (NATs) expression, their regulation of cognate sense gene

278 The 20th Nantes Actualites Transplantation (NAT) meeting was held on June 11, 2015, and June 12, 201

279 ber of the nucleobase/ascorbate transporter (NAT) family of proteins, and is responsible for the prot

280 ubiquitous nucleobase-ascorbate transporter (NAT/NCS2) family includes more than 2,000 members, but o

281 716 to convey the noradrenaline transporter (NAT) gene (HSV1716/NAT), whose expression endows infecte

282 expression of the noradrenaline transporter (NAT) gene, and not the serotonin transporter genes, in d

283 oblastoma and UVW/noradrenaline transporter (NAT) glioma cells.

284 itive toxicity to noradrenaline transporter (NAT)-expressing cells and xenografts.

285 rty patients received neoadjuvant treatment (NAT = 20%); 41 had venous resection (VR = 28%), and 70%

286 gically normal tissue adjacent to the tumor (NAT) is commonly used as a control in cancer studies.

287 Interestingly, these two NATs use different catalytic strategies to accomplish su

288 Patients who underwent NAT followed by curative-intent resection were matched b

289 AT in trans prevents the action of unspliced NAT by competing for interaction with the promoter.

290 ontrarily to the spliced NAT, this unspliced NAT down-regulates the main LEF1 promoter activity and a

291 al CMV infection, detected by serum or urine NAT.

292 lysis in SK-N-BE(2c) (neuroblastoma) and UVW/NAT (NAT-transfected glioma) cells.

293 ly delayed the growth of SK-N-BE(2c) and UVW/NAT xenografts, compared with (131)I-MIBG/topotecan ther

294 inistration induced enhanced efficacy in UVW/NAT cells.

295 n epidemiology and assay sensitivity whether NAT is implemented in individual donations or pools of s

296 ory concentrations [MIC] >/=16 mug/mL, while NAT or VCZ MICs were 2-8 mug/mL).

297 Screening of our population with NAT cost more than screening with the HIV Combo assay bu

298 Small RNAs significantly accumulate within NAT pairs, with 21 nt smRNA particularly enriched in ove

299 N0 patients without NAT, N+ patients without NAT or VR; (2) R1-0 mm posterior-margin for the NAT grou

300 (upfront pancreatectomy, N0 patients without NAT, N+ patients without NAT or VR; (2) R1-0 mm posterio