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1 n otherwise identical fibroblasts expressing Nek1.
2 cribed sequences from this region identified Nek1, a NIMA (never in mitosis A)-related kinase as a ca
7 d ubiquitination and that the interaction of Nek1 and TAZ maintain PC2 at the level needed for proper
8 drome Majewski type to identify mutations in NEK1 as an underlying cause of this lethal osteochondrod
9 ated with ALS (MATR3, CHCHD10, TBK1, TUBA4A, NEK1, C21orf2, and CCNF), all of which were identified b
15 activity is increased within 4 minutes, and Nek1 expression is up-regulated shortly thereafter and s
22 the structure of the kinase domain of human NEK1 in its apo- and ATP-mimetic inhibitor bound forms.
23 s identify a previously unsuspected role for Nek1 in the kidney and open a new avenue for studying cy
25 induced by ionizing radiation (IR) and that Nek1 is important for cells to repair and recover from D
29 mary or transformed cells are exposed to IR, Nek1 kinase activity is increased within 4 minutes, and
34 ed community in the Netherlands identified a NEK1 p.Arg261His variant as a candidate risk factor.
35 report that the nimA-related protein kinase Nek1 phosphorylates TAZ at a site essential for the ubiq
36 hus, G2-specific phosphorylation of Rad54 by Nek1 promotes Rad51 chromatin removal during HR in G2 ph
37 e homozygous mutant animals suggest that the NEK1 protein participates in different signaling pathway
38 its kinase activity is highest, a portion of Nek1 redistributes in cells from cytoplasm to discrete n
42 found that absence of functional full-length NEK1 severely reduces cilia number and alters ciliar mor
46 at inhibiting the set of four kinases AURKB, NEK1, TTK, and WEE1 causes simulated HeLa cells to accum
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