1               If clinical VT is inducible at NIPS, repeat ablation may be considered because recurren

2 nine patients (44.7%) had no VT inducible at NIPS; 49 (37.1%) had inducible nonclinical VT only; and

3       Patients with inducible clinical VT at NIPS had markedly decreased 1-year VT-free survival comp

4 (3) receptors were readily alkylated by both NIPS (IC(50)=40 nM) and EEDQ (IC(50)=12 microM).

5 identified non-TCRbeta sequences that elicit NIPS.

6  We identified TCRbeta sequences crucial for NIPS but found that NIPS is not exclusively a property o

7                        Four underwent repeat NIPS after sedation; 3 then had induced VT terminated wi

8 s nonsense codon-induced partitioning shift (NIPS) response is not the result of cytoplasmic NMD but

9 with N-(p-isothiocyanatophenethyl)spiperone (NIPS), a selective D(2)-like receptor alkylating agent,

10 ility of noninvasive programmed stimulation (NIPS) after ventricular tachycardia (VT) ablation to ide

11 erformed noninvasive programmed stimulation (NIPS) in patients with defibrillators and ventricular ta

12 ble to undergo final programmed stimulation, NIPS should be considered in the following days to furth

13 ta sequences crucial for NIPS but found that NIPS is not exclusively a property of TCRbeta transcript

14  RNA interference experiments indicated that NIPS depends on the NMD factors UPF1 and eIF4AIII but no

15                              We propose that NIPS collaborates with NMD to retain and degrade a subse

16 Of the remaining 178 patients, 132 underwent NIPS through their implantable cardioverter-defibrillato

17                  Eighteen patients underwent NIPS in the resting-awake state (nonsedated).