ライフサイエンスコーパス: NO2

1 NO2 concentrations, a marker of traffic-related air poll

2 NO2 exposure negatively affected beta-cell function, evi

3 NO2 was positively associated with colorectal cancer mor

4 NO2(-) may act by S-nitrosating protein thiols, thereby

5 NO2(-) observed in HAO activity assays is a nonenzymatic

6 NO2-OA inhibited TNFalpha-induced NF-kappaB transcriptio

7 NO2-OA reduced TNBC cell growth and viability in vitro,

8 NO2-PLs were generated by NO2BF4 in hydrophobic environm

9 PM10 (hazard ratio 1.07, 95% CI 1.06-1.08), NO2 (1.09, 1.08-1.10), and CO (1.09, 1.08-1.10).

10 in concentrations of PM10 (1.07, 1.05-1.08), NO2 (1.09, 1.08-1.11), and CO (1.10, 1.08-1.11).

11 in concentrations of PM10 (1.08, 1.07-1.09), NO2 (1.12, 1.10-1.13), and CO (1.09, 1.08-1.10).

12 10-NO2-OA has been extensively tested in animal models and

13 In particular, 10-nitro oleic acid (10-NO2-OA) potently induces Nrf2-dependent antioxidant gene

14 nd fecal pathways are the main routes for 10-NO2-OA excretion in rats, and allowed the identification

15 Bio-elimination pathways for 10-NO2-OA were evaluated in rats (30 mg/kg.day) and in huma

16 tools for the experimental evaluation of 10-NO2-OA levels in vivo, but importantly they also set the

17 in concentrations of PM10 (1.09, 1.06-1.12), NO2 (1.09, 1.06-1.12), and CO (1.05, 1.02-1.08).

18 riment with (15)NH4NO3, NH4(15)NO3, or Na(15)NO2 amendments, we found distinct (15)N abundances in NO

19 lated sunlight, and [(15)N2]DNAN yielded (15)NO2(-) in leaves.

20 K(+)) and acid radical ion ligands (CO3(2-), NO2(-), SO4(2-), PO4(3-)) that are most frequently seen

21 : 0.27 +/- SD 0.55; Rusheen: 2.46 +/- SD 3.4 NO2(-) microg(-1) g(-1) day(-1), P < 0.001) than in Clew

22 uction, is unreactive toward weakly acidic 4-NO2-phenol, and stronger acids cleave the metal-O bonds,

23 y contrast, LS-4DCHIm forms an adduct with 4-NO2-phenol, which includes an H-bond to the peroxo O-ato

24 n-withdrawing group, such as C(4)-Cl or C(5)-NO2, they rearrange to products 4 in the presence of aqu

25 The production of both 4-Me-5-NO2-L-Trp and 4-Me-7-NO2-L-Trp uncovered remarkable regi

26 ) and N-H BDE = 77.4 kcal mol(-1) for 3-CN,7-NO2-phenoxazine to kinh = 6.6 x 10(8) M(-1) s(-1) and N-

27 oduction of both 4-Me-5-NO2-L-Trp and 4-Me-7-NO2-L-Trp uncovered remarkable regio-promiscuity of nitr

28 MOF-905, MOF-905-Me2, MOF-905-Naph, MOF-905-NO2, and MOF-950.

29 Therefore, major sulfate oxidation through a NO2-mediated pathway is not likely in China, or any othe

30 Findings suggest that absolute NO2 exposure disparities by race-ethnicity decreased fro

31 Nitro-conjugated linoleic acid (NO2-CLA) is preferentially formed, constitutes the most

32 dogenous occurrence of nitro-linolenic acid (NO2-Ln) in Arabidopsis and the modulation of NO2-Ln leve

33 identification of 4-nitro-octanedioic acid (NO2-8:0-diCOOH) as the most abundant metabolite in rat u

34 anti-inflammatory agent 10-nitro-oleic acid (NO2-OA), a component of the Mediterranean diet, reduced

35 itro-octadec-9-enoic acid (nitro-oleic acid, NO2-OA), were investigated in multiple preclinical model

36 ive rise to electrophilic nitro-fatty acids (NO2 -FAs), such as nitro oleic acid (OA-NO2 ) and nitro

37 Typical MS/MS fragmentation pattern of all NO2-PL included a neutral loss of HNO2, product ions ari

38 in two-pollutant models including PM2.5 and NO2 and in three-pollutant models with O3.

39 lled vehicles to emissions of both PM2.5 and NO2 emphasises the importance of further controls on emi

40 ression to associate physical activities and NO2 (high/medium/low) with asthma and COPD, and then int

41 tion term between each physical activity and NO2.

42 of the transition state in which the Ar and NO2 groups are anti to each other.

43 mical cell potential for 2 with R = F, H and NO2.

44 osynthetic oxidation of Fe(II), H2 S, H2 and NO2-.

45 At warm temperatures, the HO2 and NO2 come to a rapid steady state with pernitric acid (HO

46 ation of HO2 from observations of HO2NO2 and NO2 in metropolitan Atlanta, US, in winter 2014 and summ

47 th varying concentrations of O2, NH4(+), and NO2(-).

48 space analysis measurements show that NO and NO2 gases are formed within minutes on heating catalytic

49 ter exposure for up to 10 h, although NO and NO2 may have exhibited oxidizing effects (e.g., exposure

50 The results showed that SO2, NO, and NO2 are unlikely to have adsorbed on the surface vanadiu

51 uces levels of gaseous nitrous acid, NO, and NO2 within the model and measurement uncertainty.

52 vidence of interaction between nutrition and NO2 exposure during pregnancy.

53 effects of input uncertainties on ozone and NO2 output, to allow robust global sensitivity analysis

54 Exposure to PM and NO2 air pollution was associated with the prevalence of

55 arting complexes with the release of SO2 and NO2.

56 ups of increasing polarity (-CH3, -CHO, and -NO2) on the imidazole linkers provide control over the d

57 lar headgroup and HNO2, [NO2-FA + H](+) and [NO2-FA - H](-) product ions, and cleavages on the fatty

58 Increased mean annual NO2 exposure was associated with increased risk of atopy

59 ing oxidative addition of LPd(0) into the Ar-NO2 bond.

60 y NO2-OA in TNBC cells were multifaceted, as NO2-OA (a) inhibited the inhibitor of NF-kappaB subunit

61 tes to live in a block group with an average NO2 concentration above the WHO annual guideline in 2010

62 We combined annual average NO2 concentration estimates from a temporal land use reg

63 Estimated annual average NO2 concentrations decreased from 2000 to 2010 for all o

64 without universal kriging on annual average NO2 measurements (1990-2012, contiguous U.S. EPA sites).

65 dels to estimate neighborhood annual average NO2.

66 umber (PN) (over 5.6 nm); black carbon (BC); NO2; benzene, toluene, ethylbenzene, and xylene (BTEX);

67 face and not from chemical reactions between NO2 and the dielectric/semiconductor components.

68 nally, human serum albumin was found to bind NO2-CLA both non-covalently and to form covalent adducts

69 ikely an important fate for NO3 indoors, but NO2 photolysis could be an important source of indoor O3

70 re, steric obstruction of Cys-119/Cys-162 by NO2-OA pretreatment in Langendorff-perfused murine heart

71 unting for NF-kappaB signaling inhibition by NO2-OA in TNBC cells were multifaceted, as NO2-OA (a) in

72 However, the examined nitration by NO2(+) is supported by (1) the absence of 3M6NC in the a

73 k of S-nitrosation during anoxia alone or by NO2(-) during normoxia places constraints on how S-nitro

74 ndition that promotes rapid SO2 oxidation by NO2 to form aerosol sulfate concentration consistent wit

75 ification at low O2 levels was stimulated by NO2(-) at levels as low as 0.2 mM.

76 egiochemistry of the nitration of toluene by NO2(+)BF4(-) in dichloromethane is accurately predicted

77 Concentrations of black carbon, NO2, PM10, PM2.5, and ultrafine particles were higher on

78 nt substituents on the fused benzo ring (Cl, NO2, NH2, CF3, ureido, amido, heterocycles), were synthe

79 Classroom NO2 was collected by means of passive sampling for 1-wee

80 In children with asthma, indoor classroom NO2 levels can be associated with increased airflow obst

81 to determine the effect of indoor classroom NO2 on lung function and symptoms in inner-city school c

82 nships with the temporally nearest classroom NO2 level.

83 We compiled NO2 data from fixed-site monitors, chemical transport mo

84 count for inorganic nutrient concentrations (NO2/3, NH4, PO4) in each respective DOM addition.

85 Studies confirm that cyclopalladated NO2 complexes are rapidly oxidized to the corresponding

86 jor roads within 100 m and satellite-derived NO2 were consistently the strongest predictors.

87 The sensor is able to detect NO2(-) in a linear response ranging from 1 to 20 muM wit

88 A new type of nitrogen dioxide (NO2 ) gas sensor based on copper phthalocyanine (CuPc) t

89 icrom (PM2.5) (1999-2004), nitrogen dioxide (NO2) (2006), and ozone (O3) (2002-2004) concentrations w

90 xposure to traffic related nitrogen dioxide (NO2) air pollution is associated with adverse health out

91 ent channels, forming: (1) nitrogen dioxide (NO2) and hydroxyl radical ((*)OH), (2) nitrite (NO2(-))

92 tolysis rate constants for nitrogen dioxide (NO2) and nitrate radicals (NO3) in the presence of the d

93 elevated concentrations of nitrogen dioxide (NO2) and particulate matter with aerodynamic diameter <2

94 bient and home exposure to nitrogen dioxide (NO2) causes asthma symptoms and decreased lung function

95 nd PM10, respectively) and nitrogen dioxide (NO2) concentrations at place of residence using logistic

96 , ultrafine particles, and nitrogen dioxide (NO2) concentrations were measured.

97 Mean annual residential nitrogen dioxide (NO2) exposure was estimated for current residential addr

98 e and rapidly converted to nitrogen dioxide (NO2) in the presence of ozone, making it impossible to d

99 imates we created a global nitrogen dioxide (NO2) land use regression model for 2011 using annual mea

100 urements of urban form and nitrogen dioxide (NO2) to explore relationships between urban form and air

101 .5), black carbon (BC), or nitrogen dioxide (NO2) variations that were independent of year, month, an

102 in aerodynamic diameter), nitrogen dioxide (NO2), and carbon monoxide (CO) and risk of incident chro

103 tween maternal exposure to nitrogen dioxide (NO2), dietary intake of methyl nutrients, and the odds o

104 ion-related air pollutant, nitrogen dioxide (NO2), in the United States.

105 e-specific measurements of nitrogen dioxide (NO2), sulfur dioxide (SO2) and volatile organic compound

106 s of carbon monoxide (CO), nitrogen dioxide (NO2), sulfur dioxide (SO2), ozone (O3), and particulate

107 meters or less (PM2.5) and nitrogen dioxide (NO2)] predictors, we conducted a nonlinear extension of

108 0.6 ppm), toluene (1 ppm), nitrogen dioxide (NO2; 2 ppm) and sulfur dioxide (SO2; 5.6 ppm), in a whol

109 ting O-O cleavage, giving nitrogen dioxide (.NO2) plus a ferryl compound [(P(Im))Fe(IV) horizontal li

110 exposures (represented by nitrogen dioxide; NO2) from a dispersion model, using residential addresse

111 nsitivities, with NO and isoprene emissions, NO2 photolysis, ozone BCs, and deposition velocity being

112 there is considerable interest in employing NO2 -FAs and other EFAs for the prevention and treatment

113 Electrochemically reduced HA enhanced NO2-to-HONO conversion by a factor of 2 relative to non-

114 h models are a valid approach for estimating NO2 exposures in Australian cities.

115 , particularly in countries without existing NO2 monitoring data or models.

116 m 0.088 to 0.029 and from 0.115 to 0.076 for NO2.

117 ts, a cavity phase shift analyzer (CAPS) for NO2 measurements, and a UV ozone analyzer.

118 In contrast, for NO2, estimates were higher among ever [1.81 (95% CI: 1.1

119 re already in breach of EU safety limits for NO2, this phenomenon does not seem to be only restricted

120 to be 37% lower for PM2.5 and 63% lower for NO2 than at the residential address.

121 ensitivity is approximately 50x greater for [NO2 ] = 1 ppm with the UVO-treated TFTs, and a limit of

122 treatment is approximately 400x greater for [NO2 ] = 30 ppm than for those without UVO treatment.

123 the efficiency of the first channel, to form NO2, is well documented, a large range of values have be

124 surrounding rural areas are seeing frequent NO2 concentration violations.

125 and their proclivity to react with generated NO2, which also increases hydrophilicity.

126 The model captured 54% of global NO2 variation, with a mean absolute error of 3.7 ppb.

127 rom Tyrosine (Tyr) by adding a nitro group (-NO2) with nitrating agents.

128 oval through the nitrite pathway (NH4(+) --> NO2(-) --> N2) is favorable for wastewater treatment pla

129 Higher NO2 and PM2.5 exposures over follow-up were also associa

130 Higher NO2 and PM2.5 were associated with a faster decline in S

131 exposure disparities persisted, with higher NO2 concentrations for nonwhites than whites in 2010.

132 ty was also inversely associated with higher NO2 exposure (beta = -22.8; 95% CI, -36.0 to -9.7; P = .

133 gh methionine intake, women with the highest NO2 exposure and lowest methionine intake had the greate

134 reaction of BH4(-) with a large excess of HN(NO2 )2 .

135 eaction of BH4(-) with two equivalents of HN(NO2 )2 afforded the di-substituted borate anion consisti

136 he BH4(-) anion with equimolar amounts of HN(NO2 )2 or of BH3 THF with K[N(NO2 )2 ](-) produced a mon

137 combined loss of polar headgroup and HNO2, [NO2-FA + H](+) and [NO2-FA - H](-) product ions, and cle

138 rnal boundary layer and the enhanced [HONO]/[NO2] ratios observed during dust storms in urban areas.

139 the presence of two electrophilic centers in NO2-CLA located on the beta- and delta-carbons with resp

140 In 2000 and 2010, disparities in NO2 concentrations were larger by race-ethnicity than by

141 between and within-city spatial gradients in NO2 concentrations.

142 2-1.28 per 1 interquartile range increase in NO2 [2.2 ppb]) and current wheeze (aOR, 1.14; 1.02-1.28)

143 ends suggested that there was an increase in NO2 columns in most areas from 2005 to around 2011 which

144 struction, such that each 10-ppb increase in NO2 level was associated with a 5% decrease in FEV1/forc

145 pect to capturing the spatial variability in NO2 and O3 and are very convenient to use, having great

146 hydroxyl radical ([HO2]/[OH]) and increased [NO2]/[NO] with higher levels of RBS and/or RIS.

147 ves steady-state NO conversion but increases NO2 surface reactivity and hydrophilicity.

148 ytic air pretreatment transforms NO gas into NO2 gas and thereby enhances the absorption of NOx in th

149 mospheric photolysis rate constants [e.g., J(NO2) and J(O3)] by attenuating solar flux.

150 ve sites on soil surfaces drive ground-level NO2-to-nitrite conversion in the atmospheric boundary la

151 Relative to women with the lowest NO2 exposure and high methionine intake, women with the

152 b in 2000 to 2.9 ppb in 2010, estimated mean NO2 concentrations remained 37% higher for nonwhites tha

153 The mean NO2 value was 11.1 ppb (range, 4.3-29.7 ppb).

154 Although the national nonwhite-white mean NO2 concentration disparity decreased from a difference

155 Moreover, NO2-OA reduced the dephosphorylation of p38alpha by hema

156 2 )2 ](-) produced a mono-substituted [BH3 N(NO2 )2 ](-) anion, which contains a B-N connected dinitr

157 amounts of HN(NO2 )2 or of BH3 THF with K[N(NO2 )2 ](-) produced a mono-substituted [BH3 N(NO2 )2 ](

158 Nitrite accumulation (N-NO2(-)/N-NOX) of 92% was maintained.

159 The salts [H2 N-NO2 H][AsF6 ], [H3 N-NO2 ][SbF6 ], [MeHNNO2 H][SbF6 ], a

160 The salts [H2 N-NO2 H][AsF6 ], [H3 N-NO2 ][SbF6 ], [MeHNNO2 H][SbF6 ], and [Me2 NNO2 H][SbF6

161 to derive improved estimates of neighborhood NO2 concentrations by blending measurements with modeled

162 We identified nine NO2-PCs and one NO2-PE species.

163 Nitrite (NO2(-)) and nitroso compounds (E-NO, E = RS, RO, and R2N

164 Nitrite (NO2(-)) supplementation limits hypoxia-induced oxidative

165 ) and hydroxyl radical ((*)OH), (2) nitrite (NO2(-)) and oxygen atom (O((3)P)), and (3) peroxynitrite

166 Nitrate (NO3(-)) and nitrite (NO2(-)) are known to be cardioprotective and to alter en

167 Excess nitrite (NO2(-)) concentrations in water supplies is considered d

168 eactions between nitric oxide (NO), nitrite (NO2-), and unsaturated fatty acids give rise to electrop

169 cal methods for conversion of NO to nitrite (NO2(-)) and nitrate (NO3(-)) have been proposed for (15)

170 etabolism involves NH3 oxidation to nitrite (NO2(-)) via a single obligate intermediate, hydroxylamin

171 otopic composition (delta(15)N) of NOx (NO + NO2) was measured during the fourth Fire Lab at Missoula

172 The global inventory of NOx (NOx = NO + NO2) emissions is poorly constrained, with a large porti

173 of nitrogen oxides (NOx identical with NO + NO2) decreases.

174 Resulting maps of annual daytime NO, NO2, and black carbon at 30 m-scale reveal stable, persi

175 fects of minor flue gas components (SO2, NO, NO2, H2O, and O2) on vanadium at 500-600 degrees C were

176 ated with high nitrogen oxide (NOX = [NO] + [NO2]) concentrations.

177 of Dhc 16S rRNA genes, CH4, Fe(2+), NO3(-), NO2(-), and SO4(2-) concentrations, total organic carbon

178 CO3(-), HCO2(-), CH3CO2(-), SO4(2-), NO3(-), NO2(-), BrO3(-), AsO4(-), F(-), Cl(-), and Br(-) and, to

179 with dechlorination potential, with NO3(-), NO2(-), and Fe(2+) concentrations exhibiting precedence

180 first time at the molecular level that the o-NO2 group contributes less compared to the p-NO2 group t

181 resulting in a very weak -M effect of the o-NO2 group, whereas the 2p orbitals of the N atom of the

182 l of the adjacent ring C atom to which the o-NO2 is attached are inclined at >30 degrees to each othe

183 om other interfering species (i.e., CO2, O2, NO2, NO, SO2, H2O, H2, and cyclohexane, tested at the sa

184 ids (NO2 -FAs), such as nitro oleic acid (OA-NO2 ) and nitro linoleic acid (LNO2 ).

185 studied the effects of nitro-oleic acid (OA-NO2) on the human endothelial cell transcriptome.

186 , we show that the overexpression of both OA-NO2 and Nrf2 substantially decreased and that Nrf2 silen

187 ects of endothelin-1 (ET-1) is induced by OA-NO2 Inasmuch as ET-1 is one of the key regulators of vas

188 Moreover, OA-NO2 is capable of enhancing regulatory T-cell activity.

189 these results support the development of OA-NO2 as a promising therapeutic for ACD and provides new

190 We evaluated the effect of OA-NO2 on allergic contact dermatitis (ACD) using an establ

191 e to examine in more detail the effect of OA-NO2 on endothelin signaling in human endothelial cells.

192 In vivo SC OA-NO2 significantly inhibits pathways that lead to inflamm

193 We found that subcutaneous (SC) OA-NO2 injections administered 18 h prior to sensitization

194 Based on these results, we propose that OA-NO2 and Nrf2 may alleviate the vasoconstrictive effects

195 mmunoprecipitation analysis revealed that OA-NO2 increased the binding of Nrf2 to an antioxidant resp

196 These data suggest that OA-NO2 modulates endothelin signaling by increasing Nrf2-de

197 Nrf2 was found to regulate the OA-NO2-induced transcription of ET-B in human and mouse end

198 Thus, OA-NO2 treatment results in anti-inflammatory effects capab

199 Thus, we sought to determine whether OA-NO2 , an exemplary nitro-fatty acid, has the capacity to

200 c acid levels are elevated in the airways of NO2-exposed mice, the powerful inhibitory effect of uric

201 There was no significant association of NO2 levels with percent predicted FEV1, fraction of exha

202 vide new insights into the chemical basis of NO2-CLA signaling actions.

203 th the addition of different concertation of NO2(-).

204 sponsible for the functional consequences of NO2(-) exposure.

205 Conversion of NO2 to HONO in the dark was found to be significant and

206 orm very well in photo-assisted detection of NO2 gas.

207 to the sensitive and selective detection of NO2(-) with increasing concentrations quantifiable by UV

208 ensor could be used for the determination of NO2(-) with a linear range from 0.1-1600microm and a det

209 However, the worldwide distribution of NO2 exposure and associated impacts on health is still l

210 It is shown that the effect of NO2 from road traffic upon premature mortality was ten-f

211 ly contributing to the beneficial effects of NO2(-) on mitochondrial metabolism.

212 ression, and greater MRP1-mediated efflux of NO2-OA-glutathione conjugates.

213 nity tag labeling, to quantify the extent of NO2(-)-dependent S-nitrosation of proteins thiols in viv

214 athways stimulated by low O2, independent of NO2(-) concentrations.

215 The magnitude of NO2-to-HONO conversion depends on the amount of Fe(2+) p

216 ctrochemical analyzer for the measurement of NO2(-) in hypoxia induced H9c2 cardiac cells using ARM m

217 e used coated-wall flow tube measurements of NO2 reactivity on environmentally relevant surfaces (Fe

218 The mechanism of NO2 reduction on soil surrogates composed of HA and clay

219 NO2-Ln) in Arabidopsis and the modulation of NO2-Ln levels throughout this plant's development by mas

220 e main mechanism is the photodissociation of NO2, which then recombines with the ground electronic st

221 At greater than a threshold of 8 ppb of NO2 and after adjusting for race and season (spirometry

222 he involvement of heterogeneous reactions of NO2 on surfaces as a source of HONO, mechanisms remain p

223 In this work, we examined the reactions of NO2-CLA with low molecular weight thiols (glutathione, c

224 tion of substrates to show that reduction of NO2 by Fe-bearing minerals in soil can be a more importa

225 Reduction of NO2 on ubiquitous Fe-bearing minerals in soil may explai

226 t there is energy barrier for the release of NO2 which prevent NO oxidation.

227 stepwise mechanisms with thiolate attack on NO2-CLA as rate-controlling step.

228 nd that the combined effect of urban form on NO2 is larger for small cities (beta x IQR = -0.46 for c

229 We identified nine NO2-PCs and one NO2-PE species.

230 between exposures to ambient PM10, PM2.5, or NO2 and PD risk.

231 -phenyl)pyridine-2,6-dicarboxamide, R = H or NO2, or N,N'-bis(2,6-diisopropylphenyl)-1-methylpiperidi

232 ignificant overall association between PM or NO2 exposures and PD risk.

233 r pollution exposure (represented by outdoor NO2 levels) at the birth address and performed a genome-

234 ctively); PM2.5 absorbance; nitrogen oxides (NO2 and NOx); traffic intensity; and elemental compositi

235 e derivatives were obtained for p-OCH3 and p-NO2 benzaldehyde.

236 othly afforded BFpC-, FpC-, DABCYLpC-, and p-NO2-PhpC-derived monomers suitable for standard oligonuc

237 ereas the 2p orbitals of the N atom of the p-NO2 group and the adjacent ring C atom are almost coplan

238 Hence, the p-NO2 group contributes largely toward the molecular react

239 NO2 group contributes less compared to the p-NO2 group toward the reactivity of 2,4-DNP-based probes.

240 The attachment of pendant NO2 and NEt2 groups at the opposite positions of the pi-

241 easure an average nitrite quantum yield (Phi(NO2(-))) of (1.1 +/- 0.2)% (313 nm, 50 muM nitrate, pH >

242 ls to evaluate the association between PM10, NO2, and CO concentrations and risk of incident estimate

243 notonic increasing association between PM10, NO2, and CO concentrations and risk of kidney outcomes.

244 l exposure to higher concentrations of PM10, NO2, and CO is associated with increased risk of inciden

245 M2.5 and 0.88; 95% CI: 0.79, 0.98 per 20 ppb NO2).

246 ences that include increased N2O production, NO2(-) toxicity, and shifts in phytoplankton community c

247 Humic acid (HA) is thought to promote NO2 conversion to nitrous acid (HONO) on soil surfaces d

248 e important source of HONO than the putative NO2 hydrolysis mechanism.

249 of the MeCN and EtCN solvates of 2 with R = NO2 have been determined.

250 A denitrifier method was used to reduce NO2(-) and NO3(-) in the trapping solution quantitativel

251 ty decreased from 2000 to 2010, but relative NO2 exposure disparities persisted, with higher NO2 conc

252 influenced model performance and residential NO2 exposure estimates for a cohort of 13679 participant

253 nce, it is their intrinsic ability to resist NO2 reduction reactions, compared to AC, that makes them

254 ent), our models suggest that Christchurch's NO2 concentrations would be approximately 60% higher tha

255 and competitively by N3(-), OCN(-), SCN(-), NO2(-), and NO3(-), whereas CO2 reduction is inhibited o

256 metry standardized by age, height, and sex), NO2 levels were associated highly with airflow obstructi

257 oxidation of atmospheric gases such as SO2, NO2, volatile organic compounds, organic and inorganic a

258 elevant toxic chemicals, including NH3, SO2, NO2, H2S, and some volatile organic compounds, with part

259 riging or satellite data to a well-specified NO2 land-use regression model each improves prediction.

260 ion is supported by experiments that studied NO2 reactivity on mineral surfaces coated with the model

261 n to NO2 in excess ozone (O3) and subsequent NO2 collection in a 20% triethanolamine (TEA) solution a

262 iency of NO conversion to NO2 and subsequent NO2 collection in the TEA solution is >98% under a varie

263 In animal systems, NO2-FAs are considered novel signaling mediators of cell

264 y and headspace GC-MS analysis indicate that NO2(*) is released upon photolysis of 4, also consistent

265 Bioinformatics analysis revealed that NO2-Ln was also involved in the response to oxidative st

266 thelial MCF-10A and MCF7 cells revealed that NO2-OA more selectively inhibited TNBC function.

267 The NO2 sensitivity of these TFTs with the dielectric surfac

268 The NO2 trends suggested that there was an increase in NO2 c

269 The NO2(-) spiked cultures with an initial COD:N = 11:1 accu

270 The NO2-PLs were then detected by electrospray ionization (E

271 r results indicate it is as important as the NO2 channel.

272 In comparison, the NO2(-) spiked cultures with COD:N = 4:1 showed significa

273 Thus, (NO2)L is aqueous soluble and a biologically acceptable p

274 ta for historical and recent N loadings (TN, NO2 + NO3), chl-a, floral composition, and net primary p

275 show that the efficiency of NO conversion to NO2 and subsequent NO2 collection in the TEA solution is

276 cts soil-emitted NO through NO conversion to NO2 in excess ozone (O3) and subsequent NO2 collection i

277 romium trioxide (CrO3), before conversion to NO2(-) and NO3(-) in an alkaline hydrogen peroxide (H2O2

278 NO3(-) action results from its conversion to NO2(-) by salivary bacteria, but the mechanism(s) by whi

279 increased in the airways of mice exposed to NO2 and that administration of uricase inhibited the dev

280 Exposure to NO2 also showed positive associations with wheeze and sh

281 However, exposure to NO2(-) in conjunction with ischemia led to extensive S-n

282 ded affinity tag), we found that exposure to NO2(-) under normoxic conditions or exposure to ischemia

283 the corresponding NO3 adducts on exposure to NO2(g).

284 with an increase in during-walk exposure to NO2, ultrafine particles and PM2.5, and an increase in P

285 yzes the four-electron oxidation of NH2OH to NO2(-) We provide evidence that HAO oxidizes NH2OH by on

286 ic features, bacteria able to reduce NO3- to NO2- were isolated from soil, N-oxide gas products were

287 chondrial inner membrane to HNO2, but not to NO2(-), combined with the lack of S-nitrosation during a

288 od in which NO is oxidized quantitatively to NO2 by chromium trioxide (CrO3), before conversion to NO

289 posite shows appropriate selectivity towards NO2(-) in the presence of potentially interfering metal

290 ons and satellite-retrieved CO, tropospheric NO2, and aerosol optical depth (AOD) (R(2) > 0.8).

291 stically significant relationship with urban NO2; their combined effect could be substantial.

292 e NH4(+) to N2 under anoxic conditions using NO2(-).

293 isited from 6 to 12 times, for 20 min, using NO2 and O3 sensors manufactured by Aeroqual.

294 Twice as much HONO was formed when NO2 reacted with HA that was photoreduced by irradiation

295 and Oceania (adjusted R(2) within 11%) where NO2 monitoring data are sparse.

296 vide the only likely set of conditions where NO2-mediated oxidation of SO2 outcompetes with other wel

297 vary bacteria, but the mechanism(s) by which NO2(-) affects metabolism remains obscure.

298 found for BC, and a weaker association with NO2.

299 When combined with NO2(-) shock, this prolonged the duration over which ins

300 increase in PWV and augmentation index with NO2 and ultrafine particles.