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1                                              NTD deletion reduced the cytokine-induced expression of
2                                              NTD vaccines face unique challenges with respect to thei
3                                              NTDs are associated with hyperglycemia-induced protein m
4                                              NTDs have been included within the Sustainable Developme
5 ural and biological properties of the TDP-43 NTD, indicating that the NTD must be stably folded for T
6  temperature conditions, we show that TDP-43(NTD) is thermodynamically stable, well-folded and underg
7 of BRCA2 that exhibits DNA binding activity (NTD) and provide evidence for NTD promoting RAD51-mediat
8 e light of the renewed global action against NTDs and the global efforts to achieve the sustainable d
9  women remain suboptimally protected against NTDs.
10 esembled human PrP(c) The requirement for an NTD, but not for the specific human sequence, suggests t
11 alog from the same organism, we show that an NTD-CTD heterodimer forms when the domains are expressed
12 rganelle stress and apoptosis, leading to an NTD reduction.
13 his study establishes that all four Anabaena NTD-like proteins can bind a carotenoid and the differen
14 d CITED2 are critical for ER homeostasis and NTD formation in the developing embryo.
15 racellular portion, comprised of the LBD and NTD, is loosely arranged, mediating complex allosteric r
16 cholesterol transfer tunnel between NPC2 and NTD cholesterol binding pockets, supporting the "hydroph
17 reduction of cellular stress, apoptosis, and NTDs in their embryos.
18 ); however, the relationship between NKH and NTDs is unclear.
19 nduced CITED2 downregulation, ER stress, and NTDs in cultured embryos.
20 he pathway of multiple births, while the ART-NTD association was explained by the multiple-births pat
21 ace new colleagues not traditionally seen as NTD implementers.
22 h HBc rescued NTD assembly in RRL as well as NTD expression and assembly in mammalian cells, resultin
23 eracts directly with the membrane-associated NTD, which serves as both a membrane anchor and an allos
24 PSK H23C mutant, SynAPSK fused to the AtAPSK NTD, and the fusion protein with the H23C mutation showe
25 reviously determined Euprosthenops australis NTD structures reveals subtle conformational alterations
26 , but with a difference in the angle between NTD/CTD at the RNA hinge region.
27 S1 protein kinase, which phosphorylated both NTD and CTD.
28 h replacement of the human NTD by the bovine NTD resembled human PrP(c) The requirement for an NTD, b
29 the presence of white-tailed deer and bovine NTDs hindered seeded conversion of PrP(c), but human and
30  and the assessment of receptor occupancy by NTD modulators.
31 ns support the conclusion that the HTLV-1 CA NTD can functionally replace the HIV-1 CA CTD, but the H
32 y replace the HIV-1 CA CTD, but the HIV-1 CA NTD cannot replace the HTLV-1 CA CTD, indicating that th
33 highlighting the importance of the HTLV-1 CA NTD in HTLV-1 immature particle morphology.
34 re, chimeric Gag proteins with the HTLV-1 CA NTD produced particles phenotypically similar to HTLV-1
35 d that Gag proteins with a chimeric HIV-1 CA NTD/HTLV-1 CA CTD did not result in Gag oligomerization
36 The N-terminal and C-terminal domains of CA (NTD and CTD, respectively) engage in both homotypic and
37 NTDs, suggesting that Gldc deficiency causes NTDs through limiting supply of one-carbon units from mi
38                                        CDC73-NTD contains an extended hydrophobic groove on its surfa
39 ome are located in the region encoding CDC73-NTD.
40 the interactions observed at the N. clavipes NTD dimer interface.
41                Comparison of our N. clavipes NTD structure with previously determined Euprosthenops a
42 est that relatively little of the clomiphene-NTD association is mediated through the pathway of multi
43 o the functional regulatory role of the ClpB NTD in protein disaggregation.
44 ermore, treatment with PBA in vivo decreases NTD rate in the embryos of diabetic mice, as well as Cas
45  in diabetic pregnant mice in vivo decreases NTD rate in the embryos.
46 g at a rate of 11.3% for neural tube defect (NTD) formation, whereas no embryos in the control groups
47  axial midline, neural tube closure defects (NTDs) arose and dorsal extension was compromised.
48 c acid prevents neural tube closure defects (NTDs), but the causal metabolic pathways have not been e
49                         Neural tube defects (NTDs) are the most severe congenital malformations of th
50 rly pregnancy can cause neural tube defects (NTDs) in embryos by perturbing protein activity, causing
51 fully penetrant cranial neural tube defects (NTDs) in mice.
52 d to be associated with neural tube defects (NTDs) in offspring.
53 ls of folate-responsive neural tube defects (NTDs) indicate that impaired de novo thymidylate (dTMP)
54 educe the prevalence of neural tube defects (NTDs), although just how folates benefit the developing
55  birth defects, such as neural tube defects (NTDs), known as diabetic embryopathy.
56 agy deficiency leads to neural tube defects (NTDs), similar to those in diabetic pregnancy.
57 n prevents up to 70% of neural tube defects (NTDs), which result from a failure of neural tube closur
58 irth defects, including neural tube defects (NTDs).
59 vo and in vitro induces neural tube defects (NTDs).
60  for protection against neural tube defects (NTDs); among nonconsumers of folic acid, only 16% attain
61 tified in patients with neural tube defects (NTDs); however, the relationship between NKH and NTDs is
62  distinct lumenal domains A (also designated NTD), C, and I.
63 s no embryos in the control groups developed NTDs.
64 s was developed, using a needle trap device (NTD) in conjunction with thermal-desorption photoionizat
65 well established neglected tropical disease (NTD) programs have seen great progress towards disease c
66 histosomiasis, a neglected tropical disease (NTD) that affects more than 110 million people.
67              The neglected tropical disease (NTD) visceral leishmaniasis (VL) has been targeted by th
68 entions against neglected tropical diseases (NTD), including lymphatic filariasis (LF), scaled up dra
69            Many neglected tropical diseases (NTDs) affect the skin, causing considerable disability,
70                 Neglected tropical diseases (NTDs) are a group of viral, bacterial, and eukaryotic pa
71                 Neglected tropical diseases (NTDs) caused by helminths constitute some of the most co
72  The concept of neglected tropical diseases (NTDs) emerged more than a decade ago and has been recogn
73 iene (WASH) and neglected tropical diseases (NTDs) encourages integration, whilst maintaining existin
74 malaria and the neglected tropical diseases (NTDs) kill more than 800,000 people annually, while crea
75 s one of the 17 neglected tropical diseases (NTDs) recognized by the World Health Organization.
76 isk mapping for neglected tropical diseases (NTDs), particularly to scale up preventive chemotherapy,
77 ctions known as neglected tropical diseases (NTDs).
78                In conclusion, the disordered NTD of HSPB6 helps regulate the size and stability of he
79 domain (CTD), an N-terminal effector domain (NTD) and a ketocarotenoid; the chromophore spans the two
80 te (NMDA) receptor GluN2B N-terminal domain (NTD) aims for the treatment of various neurologic diseas
81 sequentially folded in an N-terminal domain (NTD) and a C-terminal domain (CTD) separated by a centra
82 e bond formed between the N-terminal domain (NTD) and a cysteine on the core scaffold.
83 nding site located at the N-terminal domain (NTD) and accelerating proteasomal degradation through dy
84 ble linker connecting the N-terminal domain (NTD) and C-terminal domain (CTD) of AhpF suggests that t
85 oles of the HTLV-1 CA amino-terminal domain (NTD) and CA CTD in particle biogenesis, we generated and
86 ts of the membrane-distal N-terminal domain (NTD) and ligand-binding domain (LBD), which is fused to
87 t the junction of the amino-terminal domain (NTD) and the carboxy-terminal domain (CTD) was found to
88  poorly understood Hsp104 N-terminal domain (NTD) enables this operational plasticity.
89                       The N-terminal domain (NTD) flexible-loop (FL) region is one such example: expo
90 highly conserved spidroin N-terminal domain (NTD) is a pH-driven self-assembly device that connects s
91 erization of the spidroin N-terminal domain (NTD) is crucial to this process.
92                  TDP-43's N-terminal domain (NTD) is important for these activities and dysfunctions;
93 periments showed that the N-terminal domain (NTD) is intrinsically disordered and binds presequence n
94                       Its N-terminal domain (NTD) is responsible for interaction with the antenna and
95 , we show that the LCMV Z N-terminal domain (NTD) mediates the inhibition of macrophage activation an
96 r(64), and Tyr(86) in the N-terminal domain (NTD) of beta-catenin.
97 s the conformation of the N-terminal domain (NTD) of DnaB to impair the ability of this DNA helicase
98                       The N-terminal domain (NTD) of HBc is sufficient for capsid assembly, in the ab
99 nteraction of EAF and the N-terminal domain (NTD) of MED26.
100 -CoV) in group A uses the N-terminal domain (NTD) of S protein to bind to its receptor, whereas the b
101                       The N-terminal domain (NTD) of STAT3 performs multiple functions, such as coope
102  studied, the role of the N-terminal domain (NTD) of STING remains an important subject of investigat
103  sequence homology to the N-terminal domain (NTD) of the Orange Carotenoid Protein (OCP), and are ref
104 es are located within the N-terminal domain (NTD) of the Z protein and the N-terminal CARD domains of
105  crystal structure of the N-terminal domain (NTD) of YabA solved at 2.7 A resolution reveals an exten
106 te the impact of the PHF1 N-terminal domain (NTD) on the Tudor domain interaction with the nucleosome
107 idence that the conserved N-terminal domain (NTD) plays a significant role in the binding of Ltn1 to
108 Here, we show that Cet1's N-terminal domain (NTD) promotes the recruitment of FACT (facilitates chrom
109 eaminates cytidine, and a N-terminal domain (NTD) that binds to ssDNA.
110       This protein has an N-terminal domain (NTD) that has been shown to bind to several viral protei
111 o the tyrosines, the ClpB N-terminal domain (NTD) was suggested to provide a second substrate-binding
112 equilibrium involving the N-terminal domain (NTD) with implications for the binding of certain adapto
113  by interactions with the N-terminal domain (NTD), masking an RNA polymerase binding site until a spe
114 ed an Mcm10 mutant at the N-terminal Domain (NTD), Mcm10-4A, defective for self-interaction.
115 Sirtuins with an extended N-terminal domain (NTD), represented by yeast Sir2 and human SIRT1, harbor
116 t the extracellular AMPAR N-terminal domain (NTD), which projects midway into the synaptic cleft, pla
117 glutamate, and the distal N-terminal domain (NTD), whose function is the least clear.
118 he functionally important N-terminal domain (NTD).
119 ers cholesterol to NPC1's N-terminal domain (NTD).
120  by point mutation in the N-terminal domain (NTD).
121 s to an integral-membrane N-terminal domain (NTD); however, how the NTD activates the C-terminal cata
122 c) amino-terminal domain (N-terminal domain [NTD], amino acids [aa] 23 to 90) in cross-species conver
123 motif (MIM), both N- and C-terminal domains (NTD and CTD) of MAD1 also contribute to mitotic checkpoi
124 more pronounced when the N-terminal domains (NTDs) of both RelA and p50 were present, even though the
125 ies established that the N-terminal domains (NTDs) of cMyBP-C (e.g., C0, C1, M, and C2) can bind to a
126 have shown that the sHSP N-terminal domains (NTDs), which have a high degree of intrinsic disorder, a
127 a critical role for eIF3 requiring the eIF3a NTD, in stabilizing mRNA interactions at the exit channe
128 ysis to assess whether the observed elevated NTD risk was mediated through multiple births.
129 tigate mechanisms underlying the established NTD allostery in NMDA-type iGluRs, as well as the fold-r
130                            The stably folded NTD also promotes dimerization, which is pertinent to th
131 rongly support the idea that a stably folded NTD is essential for correct TDP-43 function.
132 ding activity (NTD) and provide evidence for NTD promoting RAD51-mediated HR.
133 xity to the pH-sensitive relay mechanism for NTD dimerization.
134 nt receptor occupancy of eliprodil, a GluN2B NTD modulator.
135 dues form a globularly folded domain (hCDC73-NTD).
136 ave determined a crystal structure of hCDC73-NTD at 1.02 A resolution, which reveals a novel protein
137 he folding of the hydrophobic core of hCDC73-NTD, while others such as the K34Q mutant reduce its the
138                 We establish that the Hsp104 NTD enables cooperative substrate translocation, which i
139      A chimera with replacement of the human NTD by the bovine NTD resembled human PrP(c) The require
140 M that are known to be associated with human NTDs.
141 Here, we briefly review recent work on iGluR NTD structure and dynamics, and further explore the allo
142 r schistosomiasis, one of the most important NTDs.
143 on of Pax3, Alx1 and Alx3 genes was found in NTD SR-BI(-/-) embryos.
144 this article, we identify novel mutations in NTD of Sting of the MOLF strain in response to HSV and L
145 ds to move toward the midline and results in NTD.
146 prisingly, uridine supplementation increased NTD incidence, independent of embryo genotype and dietar
147 nown and the contribution of each individual NTD to TF activation/inhibition is unclear.
148 s only is necessary and sufficient to induce NTDs.
149 hagy and ameliorating hyperglycaemia-induced NTDs.
150 n by methotrexate during neurulation induces NTDs by inhibiting folate interaction with its uptake sy
151 TD activated the t-SNARE complex to initiate NTD zippering with the v-SNARE, a mechanism likely share
152 bunits as adapters, at least in part via its NTD, to target stalled NSPs for ubiquitylation.
153  beta-CoV in group A, uses the galectin-like NTD in its spike protein to bind its receptor protein, w
154  we report the crystal structure of the Ltn1 NTD at 2.4-A resolution.
155                                         MAD1(NTD) and MAD1(CTD) also interacted with each other and w
156     Our results have uncovered that the MAD1(NTD) and MAD1(CTD) directly interact with each other and
157 of the B vitamin folic acid can prevent many NTDs.
158           Our results suggest that the MED26-NTD functions as a molecular switch in the exchange of T
159 between AJ dynamics and EMT by Jak3-mediated NTD phosphorylation of beta-catenin.
160 Vangl2, Prickle, Wnt11) rescued Gsc-mediated NTDs.
161 d even though it is exposed in the monomeric NTD.
162 egion 4, the N-terminal domain of MotA [MotA(NTD)], and the C-terminal domain of MotA [MotA(CTD)]), m
163  that were more pronounced when the NFkappaB NTDs were present.
164                                           No NTDs were observed in litters from dams fed the folate-d
165 mbryos were detected in SR-BI(-/-) with NTD (NTD SR-BI(-/-)).
166 ic CTD can bind carotenoid in the absence of NTD, and name this truncated variant the C-terminal doma
167 bal efforts to accelerate the development of NTD vaccines and some of the hurdles to ensuring their a
168                           The development of NTD vaccines, including those for hookworm infection, sc
169 sults showed that energetics and kinetics of NTD self-assembly are highly conserved across spider spe
170 t provide new insights into the mechanism of NTD dimerization.
171 s autophagy in the forebrain and midbrain of NTD embryos.
172 psid surface that (i) permits the packing of NTD domains in the outer layer of the Gag shell, (ii) di
173 per group) were examined for the presence of NTDs.
174  interactions when present at high ratios of NTDs to F-actin.
175 ridine with and without folate deficiency on NTD incidence in the Shmt1 mouse model.
176  healthy, nonsmoking subjects was sampled on NTDs.
177 s and the experience of integration of other NTDs, strengthen existing health systems, and contribute
178   Mutant embryos develop partially penetrant NTDs while surviving mice exhibit post-natal features of
179 urring Nos2 inhibitor, quercetin, to prevent NTDs in the embryos of diabetic mice.
180 tocopherol dietary supplementation prevented NTD almost completely (from 54% to 2%, p < 0.001) in SR-
181 by folate and lead to new ways of preventing NTDs.
182 file, restores embryonic growth and prevents NTDs, suggesting that Gldc deficiency causes NTDs throug
183                       The partially purified NTD did not show intrinsic deamination activity and did
184                                        PyoS2(NTD) induces unfolding by TonB1 of a force-labile portio
185 Mimicry leads to fluorescently labeled pyoS2(NTD) being imported into FpvAI-expressing P. aeruginosa
186 ure of the N-terminal domain of pyoS2 (pyoS2(NTD)) bound to FpvAI (Kd = 240 pM) reveals that the pyoc
187 ( approximately 10 mum) between human LCN2-R-NTD and apoNGAL suggests that the N terminus on its own
188                        However, human LCN2-R-NTD could be involved in the fine-tuning of the interact
189 eriod of organogenesis significantly reduced NTDs and cell apoptosis in the embryos, compared with th
190 with positively charged residues in the RelA NTD.
191  Coexpression of the full-length HBc rescued NTD assembly in RRL as well as NTD expression and assemb
192 a model to investigate folic acid-responsive NTDs wherein disruption of de novo thymidylate synthesis
193 airments and mechanisms of folate-responsive NTDs resulting from decreased Shmt1 expression.
194 expression is causative in folate-responsive NTDs.
195                            Absence of Cet1's NTD decreases FACT targeting to ADH1 and consequently re
196 , our results support the notion that Cet1's NTD promotes FACT targeting to the active gene independe
197 stinctive domains, the N-terminal domain (S1-NTD) and the C-terminal domain (S1-CTD), both of which c
198                                           S1-NTDs and S1-CTDs from three major coronavirus genera rec
199                     Moreover, coronavirus S1-NTDs can recognize either protein or sugar receptors.
200 he Nephila clavipes major ampullate spidroin NTD dimer.
201 rt the comparative investigation of spidroin NTDs originating from the major ampullate glands of the
202 ructural template with which to design STAT3 NTD inhibitors with potential therapeutic value.
203 etermined the crystal structure of the STAT3 NTD and identified a dimer interface responsible for coo
204 tion, whilst maintaining existing structured NTD investments, and acceleration towards Sustainable De
205                             Here, we studied NTD mutations designed to destabilize its structure util
206  controlled by interactions of the substrate NTD with the rest of the substrate PrP(c) molecule.
207 harged groove formed between the N-terminal (NTD) and C-terminal (CTD) domains of its successive N pr
208 g of Ltn1 to 60S ribosomal subunits and that NTD mutations causing defective 60S binding also lead to
209                                          The NTD interaction destabilizes client proteins, priming th
210                                          The NTD zippering then dramatically stabilized the CTD, faci
211  triggered upon binding to DNA, allowing the NTD to interact with RNA polymerase to facilitate transc
212 ositions have been identified that alter the NTD equilibrium; these lie in specific regions that loca
213  interaction between the AMC peptide and the NTD of SIRT1.
214 sids containing both full-length HBc and the NTD.
215 ormational selectivity and plasticity at the NTD dimer interface play a role in the pH-dependent tran
216 d that direct interactions occur between the NTD and the CTD.
217 s that localize to the interface between the NTD and the D1 nucleotide-binding domain of the complex.
218 interaction of client proteins with both the NTD and the pore-loop tyrosines in the 580-kDa ClpB hexa
219 amshell motions intrinsically favored by the NTD bilobate fold are coupled to dimeric and higher-orde
220              Hexameric turrets formed by the NTD decorate the majority of the capsid surface.
221 rain was blocked in vitro and in vivo by the NTD modulators Ro-25-6981 and eliprodil.
222 AR anchoring mechanism that is driven by the NTD.
223 to H3K36me3, is dramatically enhanced by the NTD.
224 ns of Jak3 interacted with beta-catenin, the NTD domain of beta-catenin facilitated its interactions
225 scopy study to structurally characterize the NTD-substrate interaction.
226                      In marked contrast, the NTD of the spike glycoprotein of human respiratory beta-
227           This phosphorylation decreased the NTD:CTD interaction and also CTD's interaction with MPS1
228 mild L28A mutation severely destabilized the NTD, drastically reducing TDP-43's in vitro splicing act
229 her explore the allosteric potential for the NTD in AMPA-type iGluRs using coarse-grained simulations
230 ity of HCPs have functions distinct from the NTD of the OCP.
231                             Furthermore, the NTD-REMPI-TOFMS setup was tested for breath gas taken fr
232 ne N-terminal domain (NTD); however, how the NTD activates the C-terminal catalytic domain is unclear
233 rative deletion mapping to elucidate how the NTD of HSPB6 influences its preferential association wit
234                             Importantly, the NTD alone also failed to accumulate in mammalian cells,
235        Pathological mutations mapping in the NTD alter the domain structure, and decrease catalytic a
236 ted in breast cancer patients located in the NTD impairs HR stimulation on dsDNA/ssDNA junction conta
237  previously named the helical hairpin in the NTD of DnaB altered the conformation of the helical hair
238 at Ile-85 is located at the interface in the NTD of DnaB that contacts primase.
239                             Mutations in the NTD substrate-binding groove are shown to have a dramati
240 nct from the cholesterol-binding site in the NTD.
241 ground state and translocates fully into the NTD upon illumination.
242 pose that a concerted movement involving the NTD, C-terminal NADH, and FAD domains, and the flexible
243                           Hsp104 lacking the NTD (Hsp104(DeltaN)) dissolves disordered aggregates but
244                        Receptors lacking the NTD exhibit increased mobility in synapses, depress syna
245 all structure of the enzyme, which lacks the NTD found in homologs from mosses and plants.
246    The juxtamembrane region, which links the NTD and the catalytic domain, is necessary and sufficien
247 hich stably associated only with C-MAD2, the NTD and CTD in MAD1 surprisingly bound both O- and C-MAD
248 23C mutation showed that the addition of the NTD and cysteines recapitulated thiol-based regulation.
249 ease mutants by shifting the position of the NTD back to its wild-type location.
250 a role in the pH-dependent transition of the NTD from monomer to stably associated dimer as the spidr
251  These studies shed light on the role of the NTD in transcriptional regulation by STAT3 and provide a
252 primase by modifying the conformation of the NTD of DnaB.
253 s is likely the evolutionary ancestor of the NTD of the OCP, which arose following a domain fusion ev
254 d EcAhpF flexibility and the movement of the NTD relative to the CTD.
255 U1, the purified C domain, downstream of the NTD, could block HKU1 virus infection of human respirato
256 es predominantly from reconfiguration of the NTD-CTD and CTD trimer interfaces.
257 vide both a detailed characterization of the NTD-substrate complex and insight into the functional re
258 , is crucial for the swiveling motion of the NTD.
259  subsets of STAT3 target genes depend on the NTD for transcriptional regulation.
260 ting from clustering of point charges on the NTD surface required for function.
261  hot injector, the analytes collected on the NTD were thermally desorbed and directly transferred to
262 with truncated S protein containing only the NTD did not block infection.
263 logically low HBc concentrations in RRL, the NTD was insufficient for capsid assembly and the CTD was
264 e mutant involving an R155C substitution the NTD equilibrium can be shifted back to its wild-type pos
265                             We show that the NTD includes a substrate-binding groove that specificall
266 e specific human sequence, suggests that the NTD interacts with other regions of the human PrP(c) to
267                             We show that the NTD is partially ordered when it is natively attached to
268 rties of the TDP-43 NTD, indicating that the NTD must be stably folded for TDP-43's physiological fun
269         These data provide evidence that the NTD of STING affects DNA responses via control of traffi
270 carotenoid-binding proteins ancestral to the NTD and CTD.
271 ate state that builds up upon binding to the NTD and shares features from both folds, in agreement wi
272  paralogous genes coding for homologs to the NTD of the OCP.
273      Unlike contacts with the tyrosines, the NTD-substrate interaction is independent of the ClpB nuc
274 -OC43, another beta-CoV in group A, uses the NTD to bind to its sialic-acid containing receptor.
275 es to shuttle electrons from the CTD via the NTD to AhpC.
276  anchor Tim44 to the translocon, whereas the NTD is a dynamic arm, interacting with multiple componen
277 nding site; however, the manner in which the NTD recognizes and binds substrate proteins has remained
278                                          The NTDs represent the most common diseases of people living
279 ined, as well as the underlying cause of the NTDs in Grhl2 mutants.
280 rects the maturational rearrangements of the NTDs that yield a functional core structure, and (iii) s
281 efore their interaction with substrates, the NTDs block the translocation channel.
282 onal mutational studies, provides insight to NTD's role in binding stalled 60S subunits.
283  teratogenicity of hyperglycaemia leading to NTDs.
284 ) impairs neural tube formation and leads to NTDs.
285 he folate-deficient diet were susceptible to NTDs.
286                                      The two NTD-bound resveratrol molecules are principally responsi
287 emical perturbations after titration of USP7-NTD with vIRF1 (44)SPGEGPSGTG(53) peptide.
288            The crystal structure of the USP7-NTD.vIRF1 peptide complex revealed an identical mode of
289 binding as that of the EBNA1 peptide to USP7-NTD.
290 cts with the N-terminal domain of USP7 (USP7-NTD).
291  demonstrated that vIRF1 interacts with USP7-NTD via its EGPS motif.
292  that the vIRF1 peptide interacted with USP7-NTD with a Kd of 2.0 mum.
293 onversion of PrP(c), but human and bank vole NTDs did the opposite.
294         Accordingly, SDG-associated and WASH-NTD indicators have been developed, commencing important
295 pressing wild-type STAT3 or STAT3 from which NTD was deleted.
296 However, the molecular mechanism(s) by which NTDs modulate interaction of myosin with the TF remains
297 Trichuris trichiura) are the most widespread NTDs, but broad geographical analyses are scarce.
298 /-) embryos were detected in SR-BI(-/-) with NTD (NTD SR-BI(-/-)).
299         We demonstrate that the embryos with NTD failed to adequately methylate septin2, a key regula
300                       Swapping of the LCMV Z NTD into the nonpathogenic Pichinde virus (PICV) genome

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