ライフサイエンスコーパス: NTS

1 NTS disease showed a seasonal peak following the rainy s

2 NTS has become the predominant invasive pathogen as Haem

3 NTS has great potential for treatment assessment and pol

4 NTS meningitis in South Africa is highly associated with

5 NTS remains an important cause of bacteremia in children

6 NTS was isolated in 162 of 3639 (4.5%) children at Teule

7 NTS(HSD2) neurons respond to sodium deficiency with spon

8 NTS-CeA neurons received greater numbers of ST-related i

9 We analyzed a total of 192 NTS isolates (114 Typhimurium, 78 Enteritidis) from bloo

10 A total of 460 of 620 (74.2%) NTS isolates serotyped were Salmonella enterica subspeci

11 Among 351 NTS isolates serotyped, 160 (45.6%) were Salmonella Ente

12 Four serovars accounted for 87% of the 687 NTS isolates, including Salmonella Enteritidis (n = 244

13 -4.73) vs 4.20 (4.00-4.50), P = 0.008] about NTS.

14 ential mechanisms by which cocaine activates NTS GLP-1-expressing neurons, we microinjected corticost

15 In addition, NTS GLP-1R knockdown significantly increased self-admini

16 onventional residency training or additional NTS training in a 2-month curriculum.

17 transmission across solitary tract afferent-NTS neuron synapses.

18 9% in vehicle-treated Flox mice to 47% after NTS treatment), was lost in mice with hepatic deletion o

19 al imaging to demonstrate that virtually all NTS-->lPBN and lPBN-->CeA CGRP projections coexpress ves

20 Among NTS isolates, >/=80% (79.7% of Salmonella Enteritidis an

21 tion (Ct) (average of 40.4 Mg C ha(-1)); and NTS showed the highest contribution of post-anthesis dry

22 Conversely, Pod-ATTAC mice and NTS-treated mice showed hypercholesterolemia and a 7- to

23 nteraction between angiotensin signaling and NTS(HSD2) neurons provides a neuronal context for the lo

24 Archived sputum and NTS samples collected in 2008-2012 from hospitalized adu

25 RT-PCR was performed on "dunked" sputum and NTS samples for influenza A (Flu A), respiratory syncyti

26 Thus, SSS and NTS systems offer the best options for removing CO2 from

27 Salmonella Typhi and NTS are major causes of BSI in DRC; their antimicrobial

28 ed with NTS, the linkage between PPI use and NTS remained significant in the subgroup without these m

29 Neurotensin (NT; also known as NTS), a 13-amino-acid peptide predominantly localized in

30 The source of CSF GLP-1 may be NTS GLP-1-producing neurons, as, (1) approximately 30% o

31 r and 20 grew 2 Salmonella serovars, 1 being NTS.

32 om rosetting, which is primarily mediated by NTS-DBL1alpha interactions with complement receptor 1.

33 In rat brainstem slices containing caudal NTS, shocks to the solitary tract (ST) triggered synchro

34 of CCK(NTS) neurons to be mediated by a CCK(NTS)-->PVH pathway that also encodes positive valence.

35 NTS neurons containing cholecystokinin (CCK(NTS)) is responsive to nutritional state and that their

36 Optogenetic activation of CCK(NTS) axon terminals within the PVH reveal the satiating

37 identify the functional significance of CCK(NTS) neurons and reveal a sufficient and discrete NTS to

38 the PVH reveal the satiating function of CCK(NTS) neurons to be mediated by a CCK(NTS)-->PVH pathway

39 ecific anterograde tracing revealed that CCK(NTS) neurons provide a distinctive innervation of the pa

40 re significantly increased following chronic NTS GLP-1R knockdown.

41 spectively analyzed 48 345 culture-confirmed NTS infections that occurred in Israel 1995-2012.

42 inished when injected into the contralateral NTS.

43 e supporting the effectiveness of curricular NTS training, however, is lacking.

44 patients are particularly at risk to develop NTS bacteremia.

45 neurons and reveal a sufficient and discrete NTS to hypothalamus circuit controlling appetite.

46 12-IFNgamma axis are at risk of disseminated NTS infection.

47 y to increase susceptibility to disseminated NTS infections.

48 ecorded neuron pairs: one dye positive (i.e. NTS-CeA) and a second unlabelled neighbour.

49 to the NTS to examine the role of endogenous NTS GLP-1R signaling in energy balance control.

50 vely, these data demonstrate that endogenous NTS GLP-1R signaling is required for the control of food

51 s found in the mammalian PVN (CCK, CRH, ENK, NTS, SS, VIP, OXT, AVP), we provide the first 3D arrange

52 wborn mice that specifically lack excitatory NTS neurons, we show that they are both mute and unable

53 , dopamine beta-hydroxylase (DBH)-expressing NTS neurons as two separate populations that directly ex

54 Here we investigate the basis for NTS(HSD2) neuron activation, identify the circuit by whi

55 e Manhica District Hospital and cultured for NTS; isolates were then characterized.

56 ore, and well-known predisposing factors for NTS, including autoimmune diseases, acquired immunodefic

57 g strategies, indicating a bright future for NTS.

58 208 (48.6%) and 70 (13.1%) were positive for NTS in Lwak and Kibera, respectively.

59 osis that can serve as a human reservoir for NTS infections.

60 Collectively, data demonstrate a role for NTS astrocytic GLP-1R signaling in energy balance contro

61 Four NTS serovars (Mbandaka, Bredeney, Infantis and Virchow)

62 and specificity for differentiating TS from NTS among patients without PPI or on PPI treatment.

63 bial subtyping data to assign cases of human NTS infection to different animal populations and foods

64 Importantly, NTS cells were more competent at discriminating taste +

65 Importantly, NTS(HSD2) neurons stimulate appetite via projections to

66 excitatory postsynaptic currents (EPSCs) in NTS neurons mediated by both AMPA- and NMDA-type glutama

67 an showing a clear role for host genetics in NTS susceptibility, there are no published studies inves

68 blunted the astrocytic inhibition of IKA in NTS neurons and increased the synaptic transmission in r

69 Chemogenetic activation of GLP-1 neurons in NTS similarly decreased nicotine intake.

70 er at this first primary afferent synapse in NTS, we determined whether vesicular glutamate transport

71 and body weight-suppressive effects of intra-NTS GLP-1R activation.

72 Invasive NTS disease occurred in 94 (5.5%) children, with an inci

73 Invasive NTS was isolated from 670 (1.6%) specimens collected fro

74 Risk factors for invasive NTS disease were human immunodeficiency virus infection,

75 sible as a susceptibility locus for invasive NTS disease.

76 ntimicrobial resistance patterns in invasive NTS infections among children aged 6 weeks to 5 years pa

77 e malaria transmission was intense, invasive NTS was common and Salmonella Typhi was uncommon, wherea

78 e sought to determine the burden of invasive NTS disease in a rural and urban setting in Kenya.

79 bserved an extremely high burden of invasive NTS disease in a rural area of Kenya and a lesser, but s

80 Incidence of invasive NTS disease was high in this area of high malaria and HI

81 Overall, the adjusted incidence of invasive NTS disease was higher in Lwak (839.4 per 100,000 person

82 2 weeks occurred in 18.8% (3/16) of invasive NTS episodes in HIV-infected and 66.2% (53/80) in HIV-un

83 protection against the majority of invasive NTS infections in sub-Saharan Africa.

84 In Kilifi, the incidence of invasive NTS was high, especially among newborn infants, but typh

85 s declined during the study period, invasive NTS infections remain an important cause of morbidity in

86 In both sites, invasive NTS disease incidence generally declined over the study

87 Of 553 patients with invasive NTS from whom 1 of the 4 predominant serovars was isolat

88 of vagal afferent endings in the ipsilateral NTS, abolished MTII-induced synapsin I phosphorylation i

89 ous disease problem in sub-Saharan Africa is NTS in children and immunocompromised adults.

90 ices by recording from retrogradely labelled NTS projection neurons.

91 suggest structural circuitry between leptin, NTS, and hypocretinergic or dopaminergic neurons and est

92 Thus, the hypothalamic leptin-NTS-HCRT neuronal network orchestrates key homeostatic o

93 , Bio-Rad Laboratories, CA, USA) in Malawian NTS cases (n=106) during acute disease was correlated wi

94 As in mammals, NTS and HCRT neurons are localized adjacently in the hyp

95 In mammals, NTS-producing neurons that express leptin receptor (LepR

96 -carboxamide (ANA-12) into the dorsal medial NTS (dmNTS) of male Sprague-Dawley rats with coronary ar

97 istochemistry for VGLUT1 or VGLUT2 in medial NTS and evaluated with confocal microscopy.

98 elinated vagal axons and terminals in medial NTS, whereas IB4 was found only in unmyelinated afferent

99 Confocal analyses of medial NTS following injections of both CTb and IB4 into the sa

100 in the brainstem, particularly in the medial NTS by the endogenous peptide alpha-MSH, modulates gastr

101 When compared with control treated mice, NTS-challenged mice treated prophylactically with BI-BTK

102 ompared them with unlabelled, near-neighboor NTS neurons.

103 Neurotensin (NTS) is a 13 amino acid neuropeptide that is expressed i

104 ion will facilitate the development of novel NTS control measures.

105 tic terminals at the solitary tract nucleus (NTS).

106 ducing neurons, as, (1) approximately 30% of NTS GLP-1 neurons colocalized with the retrograde tracer

107 resistance was present in 23.9% (84/351) of NTS isolates and 46.2% (12/26) of Salmonella Typhi isola

108 Moreover, the ability of NTS treatment to increase the percentage of low-density

109 csk9 (5% in both the presence and absence of NTS).

110 ndings indicate that increased activation of NTS GLP-1-expressing neurons by corticosterone may repre

111 n Africa; however, few data on the burden of NTS bacteremia are available.

112 There were 392 cases of NTS meningitis and 9 cases of Salmonella Typhi meningiti

113 from 2000 to 2010 was searched for cases of NTS, defined by the International Classification of Dise

114 nd limited serovars involved make control of NTS disease by vaccines epidemiologically feasible, if p

115 knowledge, of a host genetic determinant of NTS disease in African children, and of a STAT4 variant

116 etween use of oral PPIs and the diagnosis of NTS.

117 knockout of Pcsk9ameliorated the effects of NTS on plasma lipids.

118 of NTS, with a focus on the epidemiology of NTS in Africa.

119 SH increased the intrinsic excitability of NTS neurons.

120 We assessed the function of NTS-associated variants in an expression quantitative tr

121 The incidence of NTS in children aged <5 years was 36.6 per 100 000 perso

122 The overall incidence of NTS in children varied markedly by location and declined

123 Finally, pharmacological inhibition of NTS astrocytes attenuates the anorectic and body weight-

124 additionally demonstrated that inhibition of NTS-->lPBN neurons attenuated cisplatin-induced anorexia

125 Transgenesis and double labeling of NTS and HCRT neurons showed that NTS axons project towar

126 ssium current (IKA), was observed in most of NTS neurons from control rats.

127 l PPIs was associated with the occurrence of NTS.

128 utative sources and transmission pathways of NTS, with a focus on the epidemiology of NTS in Africa.

129 Thus, in the presence of NTS, mice lacking hepatic Pcsk9 showed a 40% to 50% decr

130 ack the development and axonal projection of NTS neurons, the NTS promoter was isolated.

131 ata suggest the pharmacological relevance of NTS astrocytic GLP-1R activation for food intake and bod

132 isms of the circuitry and the unique role of NTS-expressing neurons remain unclear.

133 llectively demonstrate the critical roles of NTS SRC-1 in mediating E2's actions on food intake and a

134 activation (live cell calcium signaling) of NTS astrocytes and neurons; these effects are also atten

135 Poultry is the primary source of NTS outbreaks, as well as the fastest growing meat secto

136 We reveal that a subset of NTS neurons containing cholecystokinin (CCK(NTS)) is res

137 Moreover, another target of NTS neurons is ntsr-expressing dopaminergeric neurons.

138 excitability and excitatory transmission of NTS neurons, which may contribute to the observed increa

139 tudy (SNP Array 6.0, Affymetrix, CA, USA) of NTS bacteraemia in Kenyan children, with replication in

140 nificantly increased the diagnostic yield of NTS alone (302/965 [31%] versus 197/965 [20%]; P = 0.000

141 Solitary tract (ST) afferents converged onto NTS-CeA second-order sensory neurons in greater numbers,

142 whether the (TG)2 sequence was on the TS or NTS of the reporter.

143 transcribed or non-transcribed strand (TS or NTS, respectively) of a reporter placed in both orientat

144 litude was higher in identified second-order NTS neurons from control than in SH rats.

145 The second-order NTS neurons were identified by previous application of f

146 xtraintestinal infections, compared to other NTS serovars.

147 l loads in sputa may increase detection over NTS testing alone.

148 In total, 1047 cases of persistent NTS infections, comprising 2.2% of all reported cases of

149 ways (93%) triggered EPSCs at CeA projecting NTS neurons.

150 eactivity was found throughout the adult rat NTS.

151 The remaining NTS-CeA neurons received viscerosensory input only via p

152 Remarkably, NTS(HSD2) neurons are necessary for sodium appetite, and

153 removed (NT0), no-till with straw retention (NTS), subsoiling with straw removed (SS0), and subsoilin

154 Nontyphoidal Salmonella (NTS) accounted for 10.8% and 5.8% of bacteremia cases in

155 human infections by nontyphoidal Salmonella (NTS) are poorly characterized.

156 Typhi and invasive nontyphoidal Salmonella (NTS) differs, and prevalence of these pathogens among ch

157 Invasive nontyphoidal Salmonella (NTS) infections constitute a major health problem among

158 Nontyphoidal Salmonella (NTS) is a frequent cause of diarrhea around the world, y

159 ough June 2014, 687 nontyphoidal Salmonella (NTS) isolates were obtained from 667 children; 667 yield

160 ive infections with nontyphoidal Salmonella (NTS) lead to bacteremia in children and adults and are a

161 characteristics of nontyphoidal Salmonella (NTS) meningitis in South Africa, where human immunodefic

162 ccasions (1.2%) and nontyphoidal Salmonella (NTS) serovars were isolated 10,139 times (6.1%), of whic

163 the association of nontyphoidal Salmonella (NTS) serovars with invasive infections, 48,345 Salmonell

164 o control foodborne nontyphoidal Salmonella (NTS), infections have not declined in decades.

165 Nontyphoidal Salmonella (NTS), mainly serotypes Typhimurium and Enteritidis, caus

166 Nontyphoidal Salmonella (NTS), particularly Salmonella enterica serovars Typhimur

167 Non-typhoidal Salmonella (NTS) causes invasive and frequently fatal disease in Afr

168 ed infections with non-typhoidal Salmonella (NTS) in sub-Saharan Africa.

169 ity-acquired BSI are nontyphoid salmonellae (NTS), Streptococcus pneumoniae, Escherichia coli, and St

170 coccus pneumoniae, nontyphoidal salmonellae (NTS), and Hib were the most frequently isolated pathogen

171 ibitors (PPIs) and nontyphoid salmonellosis (NTS) continues to be debated.

172 wian children with NTS bacteraemia, the same NTS risk allele was associated with lower serum concentr

173 Nontarget screening (NTS) based on high resolution mass spectrometry (HRMS) h

174 brane potential show that CO2/H(+)-sensitive NTS neurons are activated by focal ATP application; howe

175 paratyphoid fever), nontyphoidal septicemia (NTS), and gastroenteritis in humans and other animals wo

176 laria and nontyphoidal Salmonella serotypes (NTS) can cause life-threatening bacteremia in humans.

177 LN in which mice receive nephrotoxic serum (NTS) containing anti-glomerular antibodies.

178 ce) and mice treated with nephrotoxic serum (NTS), which triggers immune-mediated podocyte damage.

179 sing protamine sulfate or nephrotoxic serum (NTS).

180 , despite multifibre convergence, all single NTS-CeA neurons received inputs derived from only unmyel

181 Nontechnical skills (NTS) have been identified as critical competencies of su

182 The nucleus tractus solitarii (NTS) integrates inputs from cardiovascular afferents and

183 ic relay from the nucleus tractus solitarii (NTS) to corticotropin releasing hormone neurons in the p

184 al tissues to the nucleus tractus solitarii (NTS).

185 rent pathway (the nucleus tractus solitarii, NTS; nodose ganglion, NG).

186 ht the hindbrain nucleus tractus solitarius (NTS) as a brain region important for GLP-1R-mediated eff

187 lex (DVC; nucleus of the tractus solitarius (NTS) dorsal motor nucleus of the vagus (DMV) and area po

188 at the hindbrain nucleus tractus solitarius (NTS) is essential for vocalization in mice.

189 nsmission in the nucleus tractus solitarius (NTS) neurons, the first synaptic station of afferents of

190 ntestines and in nucleus tractus solitarius (NTS) neurons.

191 platin activates nucleus tractus solitarius (NTS) projections to the lateral parabrachial nucleus (lP

192 d neurons in the nucleus tractus solitarius (NTS), a hindbrain nucleus critical for energy balance co

193 g neurons in the nucleus tractus solitarius (NTS), a hindbrain nucleus that projects monosynaptically

194 b connecting the nucleus tractus solitarius (NTS), the major central source of GLP-1, with the other

195 synapses in the nucleus tractus solitarius (NTS), where afferent endings from arterial chemoreceptor

196 clei such as the nucleus tractus solitarius (NTS).

197 ) neurons in the nucleus tractus solitarius (NTS).

198 d by 26.3, 19.0, 16.5, and 9.4 for NT0, SS0, NTS, and SSS, respectively.

199 ssential in maintaining throughput across ST-NTS synapses.

200 antly enriched on the nontranscribed strand (NTS) of yeast genes, particularly in BER-deficient strai

201 ptase PCR (RT-PCR) of nose and throat swabs (NTS) is useful for patient care by informing antiviral u

202 symptoms (TS) and non-troublesome symptoms (NTS).

203 The results indicated that NTS and SSS can enhance translocation of photosynthates

204 labeling of NTS and HCRT neurons showed that NTS axons project toward HCRT neurons, some of which exp

205 The NTS, located in the dorsal brainstem, receives constant

206 The NTS-associated variant was an eQTL for STAT4 expression

207 -stimulated monocytes (p=9.59 x 10(-6)), the NTS risk allele being associated with lower STAT4 expres

208 interaction of odorants and tastants at the NTS underscores its role as the initial node in the neur

209 modulate GABAergic transmission between the NTS and DMV, resulting in the engagement of an otherwise

210 tite, and uncover an interaction between the NTS(HSD2) circuit and ATII signaling.

211 This translocation can be inhibited by the NTS-derived peptide (EPE) that blocks the ERK1/2-importi

212 zebrafish and cloned the genes encoding the NTS neuropeptide and receptor (NTSR).

213 Removing polySia from the NTS had functional consequences.

214 termine if CO2/H(+)-sensitive neurons in the NTS and medullary raphe respond to ATP, and whether puri

215 -gamma and -alpha (PPAR-gamma/-alpha) in the NTS and NG in HFD rats were markedly reversed by chronic

216 with Akt-eNOS-NO signaling activation in the NTS and NG induced by acute intravenous rhFGF21 administ

217 e ultrastructural location of polySia in the NTS and the functional effects of enzymatic removal of p

218 radely labeled vagal afferent endings in the NTS and thus are aptly positioned to activate vagal affe

219 excitatory neurotransmitter released in the NTS by vagal afferents, which arrive there via the solit

220 r hypothesis that glutamate receptors in the NTS contribute to plasticity in the HVR with CSH.

221 sults reveal that CCK and DBH neurons in the NTS directly engage CGRP(PBN) neurons to promote anorexi

222 ns (range 12-50 nm) could be detected in the NTS in anaesthetized rats in response to electrical stim

223 that BDNF/TrkB signalling is impaired in the NTS in the CHF state.

224 phosphorylated NR1 and GluR1 proteins in the NTS increased 35% and 70%, respectively, relative to tha

225 icate that endogenous BDNF signalling in the NTS is integral for the maintenance of BRS and that BDNF

226 In the NTS model, we observed a lack of foot process spreading

227 Removal of polySia in the NTS of anesthetized rats increased sympathetic nerve act

228 Here we report that in the NTS of high-fat diet-induced obese (DIO) rats, the apoA-

229 r kinase B (TrkB) receptor signalling in the NTS on baroreflex control both in healthy and CHF rats.

230 ata show that GLP-1-producing neurons in the NTS project to the LDTg, providing anatomical evidence o

231 hat SRC-1 gene knockdown specifically in the NTS significantly diminished E2's anorectic action, lead

232 fect of E2 on apoA-IV gene expression in the NTS was significantly attenuated in SRC-1 knockdown rats

233 of vagal afferent fibers and synapses in the NTS were significantly reduced 10 days following vagotom

234 in both male and female PNS offspring in the NTS, and in the PVN in males.

235 ha-hydroxysteroid dehydrogenase mRNAs in the NTS, and this normalized hyperactive HPA axis responses

236 e expressed by vagal afferent endings in the NTS, but it is not known whether these endings participa

237 d from baroreceptor afferent synapses in the NTS, the influence of other neurotransmitters and neurom

238 g of central vagal afferent terminals in the NTS.

239 In vivo, ATP injection into the NTS increased cardiorespiratory activity; however, injec

240 ood intake following MTII injection into the NTS ipsilateral to nodose ganglion removal was significa

241 gy balance, AAV-GLP-1R was injected into the NTS to examine the role of endogenous NTS GLP-1R signali

242 e receptor agonists and antagonists into the NTS.

243 nt and axonal projection of NTS neurons, the NTS promoter was isolated.

244 colocalized with apoA-IV in the cells of the NTS and E2 treatment enhances the recruitment of ERalpha

245 We also show that neurons of the NTS directly connect to and entrain the activity of spin

246 tudy period; the pattern of dominance of the NTS serotypes also shifted from Salmonella Enteritidis t

247 Half of the NTS-CeA neurons received at least one primary afferent i

248 -klotho (klb) significantly expressed on the NTS and NG.

249 rains, due to higher damage formation on the NTS and transcription-coupled repair of the transcribed

250 rs of magnitude higher when (TG)2 was on the NTS.

251 We studied the NTS neuronal networks in zebrafish and cloned the genes

252 In summary, our work demonstrates that the NTS is an obligatory component of the neuronal circuitry

253 lasmon resonance experiments showed that the NTS-DBLalpha and DBL2gamma domains bind strongly to hepa

254 rmal transmission of information through the NTS and that changes in its expression alter sympathetic

255 radrenaline applied by microinjection to the NTS.

256 de novel evidence that astrocytes within the NTS are relevant for energy balance control by GLP-1 sig

257 rents and their central terminals within the NTS is a common obstacle for evaluating functional group

258 in part, as a volume transmitter within the NTS.

259 These NTS neurons then excite brain regions known to mediate f

260 When these NTS neurons are activated using optogenetic or chemogene

261 Alterations to NTS signalling in CHF remain particularly undefined.

262 l loads in dunked sputum samples compared to NTS samples for Flu A, RSV, and HMPV (P = 0.0001, P = 0.

263 no- and polysynaptic ST afferent pathways to NTS-CeA neurons were organized exclusively as either tra

264 investigating host genetic susceptibility to NTS in African populations.

265 en implicated in increased susceptibility to NTS, it is not known whether malaria affects resistance

266 eurons in the nucleus of the solitary tract (NTS(HSD2) neurons) were shown to drive sodium appetite.

267 ocated in the nucleus of the solitary tract (NTS) and projecting to numerous brain regions, is ideall

268 The nucleus of the solitary tract (NTS) is a key gateway for meal-related signals entering

269 eA-projecting nucleus of the solitary tract (NTS) neurons for synaptic characterization and compared

270 ession in the nucleus of the solitary tract (NTS) of lean ovariectomized (OVX) rodents.

271 TII) into the nucleus of the solitary tract (NTS) produces rapid and sustained reduction of food inta

272 ns within the nucleus of the solitary tract (NTS) receive vagal afferent innervations that initiate g

273 eurons in the nucleus of the solitary tract (NTS) via the vagus nerve.

274 l such as the nucleus of the solitary tract (NTS), a site that receives chemosensory afferents, and t

275 eurons in the nucleus of the solitary tract (NTS), but their exact role remains unclear.

276 including the nucleus of the solitary tract (NTS), medullary raphe and retrotrapezoid nucleus (RTN).

277 first in the nucleus of the solitary tract (NTS).

278 rtex, and the nucleus of the solitary tract (NTS).

279 regions: the nucleus of the solitary tract (NTS, 6% of CTB-ir neurons), area postrema (AP, 8%), caud

280 teract in the nucleus of the solitary tract (NTS; the first neural relay in the central gustatory pat

281 g strategies to prevent, diagnose, and treat NTS disease are inadequate.

282 ate (NDS) and 5 mM naphthalene trisulfonate (NTS) in 0.4 M formic acid, pH 2.0 is developed for detec

283 f mfd globally shifts the distribution of TS/NTS ratios downward by a factor of about 2 on average fo

284 hing, slightly pushed the distribution of TS/NTS ratios to higher ratios.

285 transcribed strand/nontranscribed strand (TS/NTS) repair ratio demonstrated that deletion of mfd glob

286 Of the 102 typed NTS isolates, 40% (41) were Salmonella enterica serovar

287 of ST-related inputs compared to unlabelled NTS neurons, indicating that highly convergent viscerose

288 inal regions of the IT4var09 PfEMP1 variant (NTS-DBL1alpha and DBL2gamma domains) specifically inhibi

289 on activation, identify the circuit by which NTS(HSD2) neurons drive appetite, and uncover an interac

290 With NTS excluded, the prevalence of bacteremia at Teule was

291 -1 axons form close synaptic apposition with NTS astrocytes.

292 within the 30 days were also associated with NTS, the linkage between PPI use and NTS remained signif

293 In Malawian children with NTS bacteraemia, the same NTS risk allele was associated

294 s resistance to intestinal colonization with NTS.

295 s resistance to intestinal colonization with NTS.

296 STAT4 was associated (recessive model) with NTS in both Kenyan and Malawian children (Kenya p=5.6 x

297 Persons with NTS had a higher rate of using oral PPIs within the prio

298 hepatic deletion of Pcsk9 were treated with NTS to determine the contribution of PCSK9 to the dyslip

299 nct subpopulations of vagal afferents within NTS, we injected cholera toxin B subunit (CTb) and isole

300 -1R agonists activate and internalize within NTS astrocytes, while behavioral data suggest the pharma