ライフサイエンスコーパス: Na retention

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1 out mice, suggesting a facilitation in renal Na+ retention.

2 olume therefore appears to be the signal for Na+ retention.

3 How overexpression of WNK1 leads to Na(+) retention and hypertension is not entirely clear.

4 an important element of aldosterone-induced Na(+) retention and hypertension.

5 Renal AT2R activation with C-21 prevented Na(+) retention and lowered blood pressure in the angiot

6 Intrarenal AT2R activation prevents Na(+) retention and lowers blood pressure in angiotensin

7 rats, Ang II infusion increased both sodium (Na(+)) retention and BP on day 1, and BP remained elevat

8 ffective as spironolactone in overcoming the Na+ retention and lowering blood pressure.

9 sion, peripheral vasodilatation (PVD) causes Na+ retention as a result of vascular underfilling.

10 r Na+ retention ceases, it may contribute to Na+ retention by decreasing central blood volume.

11 tal peripheral resistance remains decreased, Na+ retention ceases after central blood volume is norma

12 Although PVD persists after Na+ retention ceases, it may contribute to Na+ retention

13 on 4A (the first exon for KS-WNK1) exhibited Na(+) retention, elevated blood pressure on a high-Na(+)

14 rmal or high-Na diet and prevented excessive Na retention in salt-loaded CD ET-1 KO mice.

15 tential for DCT hypertrophy to contribute to Na(+) retention in a hypertensive condition.

16 etermine the role of central blood volume in Na+ retention in portal hypertensive rats.

17 ine whether escape from aldosterone-mediated Na retention is associated with decreased abundance of o

18 ease or response may still contribute to the Na+ retention of low-renin hypertension.

19 nistration of C-21 prevented Ang II-mediated Na(+) retention on day 1, induced continuously negative

20 )-exchanger-3 and Na(+)/K(+)ATPase, prevents Na(+) retention resulting in negative cumulative Na(+) b

21 HKalpha(1)(-/-)) resulted in greater urinary Na(+) retention than observed in either wild-type mice o

22 ion of central blood volume and cessation of Na+ retention were observed despite persistent PVD.

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