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1 NaI-treated and mutant photosystem I complexes were used
2 bulin autoantibodies when they receive 0.05% NaI in their drinking water beginning at 8 wk of age.
3 utoimmune thyroiditis (SAT) when given 0.05% NaI in their drinking water, whereas B cell-deficient NO
11 difluorocarbene [generated from (CF3)2Hg and NaI] gave a diastereomeric mixture of the 3',4'-difluoro
12 ifluorocarbene (generated from PhHgCF(3) and NaI) gave diastereomeric mixtures of the 2,2-difluorospi
13 al planar gamma-camera images and whole-body NaI probe counts were obtained to estimate (131)I-antibo
14 lving Rh-catalyzed aziridination followed by NaI-promoted rearrangement to an isomeric cyclic sulfami
15 e extracted from the Paracoccus membranes by NaI or alkaline treatment, unlike the peripheral subunit
16 e extracted from the Paracoccus membranes by NaI or alkaline treatment, which is consistent with the
17 cation of the recently introduced chaotropic NaI method, reducing our estimate of the level of steady
23 on of iodine species such as HIO and I2 from NaI aerosolized solutions exposed to 50 ppbv O3 can occu
25 AT), all TGF-beta transgenic (Tg) mice given NaI water for 2-7 mo developed thyroid lesions character
26 using pairs of spherical ions (NaCl > NaBr > NaI) correlates to experimental observations (NaI > NaCl
27 ced in the following order: NaSCN > NaClO4 > NaI > NaNO3 approximately NaBr > NaCl > pure water appro
29 of (125)I-DCIBzL, 111 MBq (3 mCi) of (125)I-NaI, an equivalent amount of nonradiolabeled DCIBzL, or
30 the labeling of arylstannanes using [(125)I]NaI, where products were isolated in high specific activ
32 ation is eliminated using the sodium iodide (NaI) isolation method and that the level of oxo8dG in nu
33 mma(-/-) NOD.H-2h4 mice given sodium iodide (NaI)-supplemented water develop a slow onset autoimmune
34 I compare to different alkali metal iodides: NaI, RbI, CsI; also investigation of different potassium
35 were examined in the presence of 3 and 25 mM NaI, corresponding to I(-)-activated and I(-)-inhibited
36 ent also confirms that the loss of a neutral NaI unit, instead of an Na(+) ion, occurs during the dis
37 the dissolution processes, loss of a neutral NaI unit, occurs when six or more water molecules have b
42 associated with contact ion-pair binding of NaI (alpha = 1300, DeltaGalpha = -18 kJ mol(-1)) and NaC
43 ed ex vivo from cervical lymph node cells of NaI-fed or control mice, suggesting that the iodide-rich
45 complexes with a different concentration of NaI showed that the mutations decreased affinity between
47 ) mice develop severe TEC H/P, and 2-3 wk of NaI is sufficient for optimal development of severe TEC
48 and 9 wk after injection using spectrometric NaI(Tl) and HPGe detectors, and imaging between 5 and 10
49 nominal counting rates were obtained for the NaI systems, and the bismuth germanate (BGO) systems wer
51 Using difluorocarbene, generated from TMSCF3/NaI, these spirocycles were produced in yields up to 97%
52 Treatments with chaotropic reagents (urea, NaI, or NaBr) or with alkaline buffer (pH 10-12) resulte
53 aotropic technique of DNA isolation by using NaI produced the lowest and least variable oxo8dG values
55 and ATP-induced processing were blocked when NaI was substituted for NaCl within the medium; substitu
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