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10 cult, is now accepted management for Crigler-Najjar type I disease but remains controversial for the
11 rubinemia in humans that suffer from Crigler-Najjar type I disease results from lesions in the UGT1A1
12 r type II patients and, recently, in Crigler-Najjar type I patients treated with human hepatocyte cel
14 lirubinemia in the Gunn rat model of Crigler-Najjar syndrome can be achieved with one injection of HD
15 perbilirubinemic Gunn rats (model of Crigler-Najjar syndrome type 1) resulted in hUGT1A1 expression i
18 both alleles, whereas patients with Crigler-Najjar syndrome are homozygous for a defect that yields
19 report describes a young adult with Crigler-Najjar syndrome type II in whom kernicterus developed af
20 nduced levels noted in patients with Crigler-Najjar type 1 disease, is fatal in neonatal Ugt1(-/-) mi
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