ライフサイエンスコーパス: Neisseria

1 glL, the general O-OTase first discovered in Neisseria.

2 also a strictly conserved trait in commensal Neisseria.

3 hesis (FabI) as a therapeutic target against Neisseria.

4 reptococcus (18.4%), Haemophilus (12.7%) and Neisseria (6.8%).

5 nhibition of fatty acid synthesis in growing Neisseria, a delayed inhibition of growth phenotype, and

6 ][G-4], Pseudoramibacter, Streptococcus, and Neisseria and fewer in Actinomyces, Selenomonas, Veillon

7 rly in life, followed by later enrichment of Neisseria and Prevotella spp.

8 a strong correlation between prp-containing Neisseria and propionic acid generating bacteria from th

9 found that depletion of the commensal genus Neisseria and the species Streptococcus pneumoniae was a

10 rns of diversity for the genera Escherichia, Neisseria, and Borrelia are generally indistinguishable

11 nd smokers in species such as Porphyromonas, Neisseria, and Gemella, but lung bacterial populations d

12 We identified Haemophilus, Streptococcus, Neisseria, and Veillonella spp. to be more abundant in c

13 o these respiratory electron carriers within Neisseria are concordant with major transitions in the m

14 Neisseria are naturally competent and acquire genetic ma

15 Additionally, the Neisseria are naturally competent for DNA transformation

16 e cloning and heterologous expression of the Neisseria bacilliformis class III RNR and show that it c

17 tered bacterial abundance profile, with more Neisseria, Bacteroides, and Rothia species and less Sphi

18 The solved structures of Neisseria BamD and BamE share overall folds with Escheri

19 that is broadly distributed among commensal Neisseria, but absent in the pathogenic species.

20 a chemical biology tool to determine whether Neisseria can bypass the inhibition of fatty acid synthe

21 Here, we show that the commensal species Neisseria cinerea expresses functional fHbp on its surfa

22 allow studies of the molecular mechanisms of Neisseria colonization, transmission, persistence, and h

23 The genus Neisseria contains two pathogenic species (N. meningitid

24 The PubMLST Neisseria database was queried to extract the 53 ribosom

25 lar species names, and stored on the PubMLST Neisseria database, providing a curated electronic resou

26 to an RpoD-dependent mechanism as pathogenic Neisseria diverged from commensals during evolution.

27 Using Neisseria elongata as a model, we show that Sigma factor

28 Neisseria encodes a functional FabI that was potently in

29 fatty acid labeling experiments showed that Neisseria encodes the ability to incorporate exogenous f

30 Rothia mucilaginosa trending to increase and Neisseria flavescens (P < 0.01) increased after nitrate

31 uely enriched in members of the Haemophilus, Neisseria, Fusobacterium, and Porphyromonas species and

32 Streptococcus genus probe 4 and Neisseria genus probe 2 were the most frequently detecte

33 -negative species Neisseria meningitidis and Neisseria gonorrheae and improved activity against the G

34 Neisseria gonorrheae bacteria are the causative agent of

35 y loss of capsule and gene conversion of the Neisseria gonorrheae norB-aniA cassette promoting anaero

36 ible for the receptor-mediated engulfment of Neisseria gonorrheae or Neisseria meningitidis by human

37 The three species Neisseria meningitidis, Neisseria gonorrheae, and Neisseria lactamica are often

38 men who have receptive anal intercourse for Neisseria gonorrhoeae (GC) and Chlamydia trachomatis (CT

39 tal Chlamydia trachomatis (CT) and/or rectal Neisseria gonorrhoeae (GC).

40 rectal Chlamydia trachomatis (chlamydia) and Neisseria gonorrhoeae (gonorrhea) infections in women.

41 ous detection of Chlamydia trachomatis (CT), Neisseria gonorrhoeae (NG), and an internal control in t

42 h a network-based mathematical model of HIV, Neisseria gonorrhoeae (NG), and Chlamydia trachomatis (C

43 ic compound extrusion transporter, NorM from Neisseria gonorrhoeae (NorM_NG).

44 Neisseria gonorrhoeae (the gonococcus, Gc) triggers a po

45 s demonstrated with the analysis of clinical Neisseria gonorrhoeae 16S rRNA to show its potential val

46 cillus crispatus, Gardnerella vaginalis, and Neisseria gonorrhoeae All vaginal microbiota and N. gono

47 Neisseria gonorrhoeae and Chlamydia trachomatis are well

48 lis by vaginal swabs; NAATs for detection of Neisseria gonorrhoeae and Chlamydia trachomatis from pha

49 ed amplification (TMA) enhances detection of Neisseria gonorrhoeae and Chlamydia trachomatis from rec

50 , and urethral/first-void urine samples) for Neisseria gonorrhoeae and Chlamydia trachomatis using nu

51 e BAM complex, from two species of bacteria: Neisseria gonorrhoeae and Haemophilus ducreyi.

52 Neisseria gonorrhoeae and Neisseria meningitidis are clo

53 an-adapted Neisseria includes two pathogens, Neisseria gonorrhoeae and Neisseria meningitidis, and at

54 ction of an intense inflammatory response by Neisseria gonorrhoeae and the persistence of this pathog

55 irth outcome (OR, 0.24; 95% 0.09, 0.66), and Neisseria gonorrhoeae and/or Chlamydia trachomatis had 9

56 In this study, we predicted Neisseria gonorrhoeae Ape1a to be an SGNH hydrolase with

57 to test the hypothesis that multiple pili of Neisseria gonorrhoeae are coordinated through a tug-of-w

58 Infections by Neisseria gonorrhoeae are increasingly common, are often

59 pic pregnancy, and Chlamydia trachomatis and Neisseria gonorrhoeae are recognized microbial causes.

60 s) can block complement-dependent killing of Neisseria gonorrhoeae by otherwise bactericidal Abs.

61 enome assemblies, we analyzed 25 isolates of Neisseria gonorrhoeae by using a high-resolution single

62 Neisseria gonorrhoeae causes gonorrhea, a sexually trans

63 ting, this study examined the persistence of Neisseria gonorrhoeae DNA following treatment for pharyn

64 The human-restricted pathogen Neisseria gonorrhoeae encodes a single N-acetylmuramyl-l

65 in female mice to study mechanisms by which Neisseria gonorrhoeae evades host-derived antimicrobial

66 um and the homologous NgoAVII RM system from Neisseria gonorrhoeae FA1090 are composed of three genes

67 Neisseria gonorrhoeae has developed resistance to each o

68 mical analyses of Kingella denitrificans and Neisseria gonorrhoeae HpuA mutants, although validating

69 conditions, as well as in the human pathogen Neisseria gonorrhoeae identified HemN as a copper toxici

70 -sensitive and cefixime-resistant strains of Neisseria gonorrhoeae in MSM in England, which was appli

71 ed a point-of-care test for the detection of Neisseria gonorrhoeae in patients attending a public hea

72 n is associated with bacterial burden during Neisseria gonorrhoeae infection and alters the infection

73 point-of-care Gram stain testing to diagnose Neisseria gonorrhoeae infection and nongonococcal urethr

74 he main consequences of sexually transmitted Neisseria gonorrhoeae infection and probably involve an

75 Treatment of Neisseria gonorrhoeae infection is empirical and based o

76 acid amplification tests (NAATs) to diagnose Neisseria gonorrhoeae infections complicates the perform

77 osporins are the cornerstone of treatment of Neisseria gonorrhoeae infections, cefixime is the only o

78 g efficacious exposures for the treatment of Neisseria gonorrhoeae infections.

79 Multidrug-resistant Neisseria gonorrhoeae is a top threat to public health.

80 The major outer membrane porin (PorB) of Neisseria gonorrhoeae is an essential protein that media

81 Antimicrobial resistance (AMR) by Neisseria gonorrhoeae is considered a serious global thr

82 f the most frequent infectious diseases, and Neisseria gonorrhoeae is emerging as resistant to most a

83 ce and spread of antimicrobial resistance in Neisseria gonorrhoeae is globally recognised.

84 In the United States, 19.2% of Neisseria gonorrhoeae isolates are resistant to ciproflo

85 We identified a cluster of Neisseria gonorrhoeae isolates with high-level azithromy

86 rmed by laboratory isolation or detection of Neisseria gonorrhoeae only from a clinical specimen, and

87 jor outer membrane porin (PorB) expressed by Neisseria gonorrhoeae plays multiple roles during infect

88 BD ProbeTec Chlamydia trachomatis Q(x) (CTQ)/Neisseria gonorrhoeae Q(x) (GCQ), Hologic Aptima Combo 2

89 rol groups were immunized i.m. and s.c. with Neisseria gonorrhoeae recombinant porin B (Ng-rPorB) or

90 Neisseria gonorrhoeae releases peptidoglycan (PG) fragme

91 The closely related pathogen Neisseria gonorrhoeae releases PG fragments during norma

92 During infection, Neisseria gonorrhoeae senses and responds to stress; suc

93 Previous studies have demonstrated that Neisseria gonorrhoeae sialylates the terminal N-acetylla

94 etect Chlamydia trachomatis AC2 also detects Neisseria gonorrhoeae Storage and temperature conditions

95 All Neisseria gonorrhoeae strains whose DNA sequences have b

96 Neisseria gonorrhoeae successfully overcomes host strate

97 of ceftriaxone, cefixime, and cefpodoxime in Neisseria gonorrhoeae surveillance.

98 e A (gyrA) genotypic assay for prediction of Neisseria gonorrhoeae susceptibility to ciprofloxacin.

99 against multidrug-resistant bacteria such as Neisseria gonorrhoeae The first structure of BamA, the c

100 reasing azithromycin usage and resistance in Neisseria gonorrhoeae threatens current dual treatment.

101 ferrin-binding proteins TbpA and TbpB enable Neisseria gonorrhoeae to obtain iron from human transfer

102 In this study, we used the type IV pilus of Neisseria gonorrhoeae to test whether variation of surfa

103 We characterized the inhibition of Neisseria gonorrhoeae type II topoisomerases gyrase and

104 Neisseria gonorrhoeae uses a type IV secretion system (T

105 Neisseria gonorrhoeae uses a type IV secretion system (T

106 1 (ORF1) of the glutamine synthetase gene of Neisseria gonorrhoeae was able to tolerate urea concentr

107 lamydia trachomatis was detected in 8.7% and Neisseria gonorrhoeae was detected in 6.6%.

108 on antimicrobial resistance determinants in Neisseria gonorrhoeae was developed and is publicly acce

109 he force-dependent velocity of DNA uptake by Neisseria gonorrhoeae We found that the DNA uptake veloc

110 nts such as human immunodeficiency virus and Neisseria gonorrhoeae with concurrent T. vaginalis infec

111 The emergence of Neisseria gonorrhoeae with decreased susceptibility to e

112 icated for evaluating UTI (E. coli) and STD (Neisseria gonorrhoeae) from human urine samples.

113 We previously demonstrated that Neisseria gonorrhoeae, a Gram-negative pathogen responsi

114 s, type-specific human papillomavirus (HPV), Neisseria gonorrhoeae, and HIV antibody.

115 on effect on incident Chlamydia trachomatis, Neisseria gonorrhoeae, and Mycoplasma genitalium infecti

116 g Staphylococcus aureus, Bacillus anthracis, Neisseria gonorrhoeae, and Neisseria meningitidis.

117 fluoroquinolone-resistant Campylobacter spp, Neisseria gonorrhoeae, and Salmonella typhi were include

118 ycoplasma genitalium, Chlamydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vaginalis in liqu

119 M. genitalium and for Chlamydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vaginalis Sequenc

120 e urine specimens for Chlamydia trachomatis, Neisseria gonorrhoeae, and Trichomonas vaginalis via com

121 Candida albicans, Gardnerella vaginalis, and Neisseria gonorrhoeae, as well as to toxic shock syndrom

122 d method to detect Chlamydia trachomatis and Neisseria gonorrhoeae, but no commercial tests are clear

123 , and nucleic acid amplification testing for Neisseria gonorrhoeae, Chlamydia trachomatis, and Tricho

124 All participants were tested for Neisseria gonorrhoeae, Chlamydia trachomatis, Treponema

125 The MtrCDE multidrug pump, from Neisseria gonorrhoeae, is assembled from the inner and o

126 ng the concurrence of Chlamydia trachomatis, Neisseria gonorrhoeae, Mycoplasma genitalium, and Tricho

127 In Neisseria gonorrhoeae, one of the first bacteria for whi

128 d to be infected with Chlamydia trachomatis, Neisseria gonorrhoeae, or herpes simplex virus type 2.

129 experiments conducted with Escherichia coli, Neisseria gonorrhoeae, Salmonella enterica, Streptococcu

130 s the gonococcal type IV pilus (GC-T4P) from Neisseria gonorrhoeae, the causative agent of gonorrhea.

131 Neisseria gonorrhoeae, the causative agent of the sexual

132 Neisseria gonorrhoeae, the causative agent of the sexual

133 parallel those of Chlamydia trachomatis and Neisseria gonorrhoeae, the mechanisms by which this path

134 In the beta-proteobacterium Neisseria gonorrhoeae, the native PilQ secretin ring emb

135 other STI organisms (Chlamydia trachomatis, Neisseria gonorrhoeae, Trichomonas vaginalis) and the E6

136 Tests for Chlamydia trachomatis and Neisseria gonorrhoeae, which can provide results rapidly

137 Neisseria gonorrhoeae, which causes gonorrhea, is partic

138 llows it to modulate biofilm accumulation by Neisseria gonorrhoeae.

139 ce and global spread of antibiotic-resistant Neisseria gonorrhoeae.

140 c inflammation that is insufficient to clear Neisseria gonorrhoeae.

141 l with substantial in vitro activity against Neisseria gonorrhoeae.

142 to date has examined the effects of CrgA in Neisseria gonorrhoeae.

143 r survival and establishment of infection by Neisseria gonorrhoeae.

144 ases (LDHs) from the obligate human pathogen Neisseria gonorrhoeae.

145 ons due to suspected cephalosporin-resistant Neisseria gonorrhoeae.

146 o the fiber model of the type IVa pilin from Neisseria gonorrhoeae.

147 he more common sexually transmitted pathogen Neisseria gonorrhoeae.

148 terial infections: Chlamydia trachomatis and Neisseria gonorrhoeae.

149 r changes or no changes in the prevalence of Neisseria, Haemophilus, Gemella, Leptotrichia, Solobacte

150 eveloped a rhesus macaque model for studying Neisseria-host interactions using Neisseria species indi

151 Here, investigations of the genus Neisseria illustrated the gene-by-gene conceptual approa

152 studies of lactate metabolism in pathogenic Neisseria in the postgenomic era.

153 Human-adapted Neisseria includes two pathogens, Neisseria gonorrhoeae

154 Neisseria is a Gram-negative pathogen with phospholipids

155 Neisseria isolated from the rhesus macaque recolonize an

156 In Neisseria, it has now been shown that loss of the 4' pho

157 ge for Neisseria meningitidis (P < 0.05) and Neisseria lactamica (P < 0.002) (2-sided Fisher's exact

158 ssociation between colonization by commensal Neisseria lactamica and meningococcal disease, we invest

159 observed in four individuals cocolonized by Neisseria lactamica and Neisseria meningitidis One HGT e

160 eria meningitidis, Neisseria gonorrheae, and Neisseria lactamica are often regarded as highly recombi

161 Neisseria lactamica or its components could be a novel b

162 e relative of N. meningitidis, the commensal Neisseria lactamica, which chiefly colonizes infants not

163 tal structures of alanyl aminopeptidase from Neisseria meningitides (the etiological agent of meningi

164 FA) class II-IV disease, vaccination against Neisseria meningitides, and previous treatment with at l

165 % (3790/6286) of bacterial meningitis cases: Neisseria meningitidis (1350 cases, 22%), Streptococcus

166 crystal structure of an ASBT homologue from Neisseria meningitidis (ASBT(NM)) in detergent was repor

167 nation with glyco-conjugate capsular group C Neisseria meningitidis (Men C) vaccines in infancy.

168 ulation-level protection against serogroup A Neisseria meningitidis (MenA) are unknown.

169 Serogroup B Neisseria meningitidis (MenB) is a major cause of severe

170 Neisseria meningitidis (N. meningitidis), Streptococcus

171 ion of the main meningitis-causing bacteria, Neisseria meningitidis (N. meningitidis).

172 Neisseria meningitidis (Nm) clonal complex 11 (cc11) lin

173 Neisseria meningitidis (Nm) is a Gram-negative diplococc

174 The human pathogen Neisseria meningitidis (Nm) is a leading cause of bacter

175 Neisseria meningitidis (Nm) is a leading cause of bacter

176 The human pathogen Neisseria meningitidis (Nm) is known to possess several

177 Neisseria meningitidis (Nm) strains infecting these pati

178 k in 2009 due to a large epidemic of group A Neisseria meningitidis (NmA) meningitis.

179 The conjugate vaccine against serogroup A Neisseria meningitidis (NmA), MenAfriVac, was first intr

180 Carriage for Neisseria meningitidis (P < 0.05) and Neisseria lactamic

181 le dynamic regulation mechanism observed for Neisseria meningitidis 3-deoxy-d-arabino-heptulosonate 7

182 of human umbilical vein endothelial cells or Neisseria meningitidis after incubation with human serum

183 eficiency and compares it to studies done on Neisseria meningitidis and Moraxella catarrhalis; the tw

184 t activity against the Gram-negative species Neisseria meningitidis and Neisseria gonorrheae and impr

185 Neisseria gonorrhoeae and Neisseria meningitidis are closely related organisms tha

186 methods for detecting pharyngeal carriage of Neisseria meningitidis are complex.

187 Several outer membrane proteins of Neisseria meningitidis are subject to phase variation du

188 the responses of human tonsil-derived DC to Neisseria meningitidis as a model organism.

189 Administration to respond to an outbreak of Neisseria meningitidis B at a U.S. university.

190 diated engulfment of Neisseria gonorrheae or Neisseria meningitidis by human cells and can offer deep

191 Invasive bacterial meningitis caused by Neisseria meningitidis can be prevented by active immuni

192 Neisseria meningitidis can be transmitted via asymptomat

193 Cas9 orthologs (including Neisseria meningitidis Cas9 [NmeCas9]) have also been ad

194 Neisseria meningitidis causes 500 000 cases of septicemi

195 Neisseria meningitidis causes bacterial meningitis and s

196 The pathogen Neisseria meningitidis causes disease amongst infants an

197 Among 25 serogroup B Neisseria meningitidis clinical isolates, we identified

198 e (SPR) based biosensor for the detection of Neisseria meningitidis DNA employing Kretschmann configu

199 Neisseria meningitidis encodes up to five TpsA proteins

200 antibodies raised against sheaths presenting Neisseria meningitidis factor H binding protein (fHbp) a

201 admission to hospital and identification of Neisseria meningitidis from a sterile site.

202 ce diversity in the Campylobacter jejuni and Neisseria meningitidis genomes encoded hypothetical prot

203 ta that the class III Fic protein NmFic from Neisseria meningitidis gets autoadenylylated in cis, the

204 ced meningitis incidence and carriage due to Neisseria meningitidis group A (MenA).

205 ampaign was launched using a newly developed Neisseria meningitidis group A (NmA) polysaccharide-teta

206 Group A Neisseria meningitidis has been a major cause of bacteri

207 Neisseria meningitidis has several strategies to evade c

208 H binding protein (fHbp) is a lipoprotein of Neisseria meningitidis important for the survival of the

209 ge infectivity potentiator (rMIP) protein of Neisseria meningitidis induces significant serum bacteri

210 The incidence of Neisseria meningitidis infection decreased from 0.721 pe

211 sential to understanding the epidemiology of Neisseria meningitidis infection.

212 We previously reported that Neisseria meningitidis internalization into human brain

213 Neisseria meningitidis is a commensal microbe that colon

214 Neisseria meningitidis is a commensal of humans that can

215 Neisseria meningitidis is a frequent colonizer of the hu

216 Neisseria meningitidis is a human commensal that can als

217 Neisseria meningitidis is a human pathogen causing bacte

218 Neisseria meningitidis is a human-specific bacterium tha

219 Neisseria meningitidis is a human-specific pathogen and

220 GNA2091 of Neisseria meningitidis is a lipoprotein of unknown funct

221 Neisseria meningitidis is a major cause of bacterial men

222 Neisseria meningitidis is a major cause of bacterial men

223 Neisseria meningitidis is an encapsulated pathogen, and

224 Neisseria meningitidis is an important cause of invasive

225 Neisseria meningitidis is an important human pathogen th

226 Although the opportunistic pathogen Neisseria meningitidis is enriched for colonization in t

227 Although Neisseria meningitidis is naturally competent and porB g

228 Almost all invasive Neisseria meningitidis isolates express capsular polysac

229 ce identity, PilE is structurally similar to Neisseria meningitidis minor pilins PilXNm and PilVNm, r

230 neither inactivated, unencapsulated, intact Neisseria meningitidis nor Streptococcus agalactiae inhi

231 lonization of the upper respiratory tract by Neisseria meningitidis occurs despite elicitation of ada

232 duals cocolonized by Neisseria lactamica and Neisseria meningitidis One HGT event resulted in the acq

233 Neisseria meningitidis phasevarions regulate genes inclu

234 asmodium falciparum infections, and virulent Neisseria meningitidis samples.

235 To combat Neisseria meningitidis serogroup A epidemics in the meni

236 Neisseria meningitidis serogroup B (MnB) is a leading ca

237 Use of recently licensed vaccines against Neisseria meningitidis serogroup B (NmB) will depend par

238 tococcus pneumoniae, Listeria monocytogenes, Neisseria meningitidis serogroup B, Candida albicans, an

239 vasive meningococcal disease (IMD) caused by Neisseria meningitidis serogroup Y has increased in Euro

240 des of recombinant capsular polymerases from Neisseria meningitidis serogroups A (CsaB) and X (CsxA)

241 Neisseria meningitidis serogroups A and X are among the

242 al virulence determinants of disease causing Neisseria meningitidis species are their extracellular p

243 Neisseria meningitidis strain H44/76 was modified by exp

244 age prevention against antigenically diverse Neisseria meningitidis strains and to compare this prote

245 erized a TE6 thioesterase from the bacterium Neisseria meningitidis Structural analysis with X-ray cr

246 ned H influenzae type b and capsular group C Neisseria meningitidis tetanus toxoid conjugate vaccine

247 The ability of the human bacterial pathogen Neisseria meningitidis to cause invasive disease depends

248 Neisseria meningitidis use Type IV pili (T4P) to adhere

249 Neisseria meningitidis utilizes capsular polysaccharide,

250 egative pathogens Haemophilus influenzae and Neisseria meningitidis We hypothesized that activation o

251 ve isolates and 25 isolates from carriers of Neisseria meningitidis without disease.

252 des two pathogens, Neisseria gonorrhoeae and Neisseria meningitidis, and at least 13 species of comme

253 e in vitro against Streptococcus pneumoniae, Neisseria meningitidis, and H. influenzae.

254 igen) and qPCR for Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae.

255 Infection with Haemophilus influenzae, Neisseria meningitidis, and Streptococcus pneumoniae cau

256 h intact, heat-killed cells of Gram-negative Neisseria meningitidis, capsular serogroup C (MenC) or G

257 lipid A phosphoethanolamine transferase from Neisseria meningitidis, determined to 2.75-A resolution.

258 ts sporadic nature and the high diversity of Neisseria meningitidis, epidemiological surveillance inc

259 ncluding pathogens such as Escherichia coli, Neisseria meningitidis, Haemophilus influenzae, and Past

260 terial meningitis, which is caused mainly by Neisseria meningitidis, Haemophilus influenzae, and Stre

261 terial infections (Streptococcus pneumoniae, Neisseria meningitidis, Haemophilus influenzae, S suis)

262 tococcus pneumoniae, Haemophilus influenzae, Neisseria meningitidis, Mycoplasma pneumoniae, Mycobacte

263 The three species Neisseria meningitidis, Neisseria gonorrheae, and Neisse

264 rs of bacterial pathogenic strains including Neisseria meningitidis, Pseudomonas aeruginosa and Esche

265 mophilus influenzae, Listeria monocytogenes, Neisseria meningitidis, Streptococcus pneumoniae, Strept

266 sm of action has been studied extensively in Neisseria meningitidis, the specific subset of genes tha

267 Neisseria meningitidis, typically a resident of the oro-

268 ere, using a distinct CRISPR-Cas system from Neisseria meningitidis, we demonstrate efficient targeti

269 Hyperinvasive lineages of Neisseria meningitidis, which persist despite extensive

270 ecifically inhibit the CRISPR-Cas9 system of Neisseria meningitidis.

271 s with N. lactamica prevents colonization by Neisseria meningitidis.

272 , influenza, Mycobacterium tuberculosis, and Neisseria meningitidis.

273 sing a dynamic transmission model of group A Neisseria meningitidis.

274 cillus anthracis, Neisseria gonorrhoeae, and Neisseria meningitidis.

275 l investigations frequently fail to identify Neisseria meningitidis.

276 ningococcal disease caused by infection with Neisseria meningitidis.

277 an unexpectedly versatile Cas9 protein from Neisseria meningitidis.

278 The outer membrane protein (OMP) from Neisseria meninigitidis, PorB, is a naturally occurring

279 cells and primary monocytes with pathogenic Neisseria or with purified lipooligosaccharides (LOS) af

280 al and gonococcal disease, and mechanisms of Neisseria pathogenicity.

281 australis, and Alloprevotella, Leptotrichia, Neisseria, Porphyromonas, and Prevotella.

282 le genomes has indicated that some commensal Neisseria species also contain genes that potentially en

283 outer membrane protein that is common among Neisseria species and is required for survival.

284 ity foregut community with a highly abundant Neisseria species associated with foregut lactate.

285 lore this question, we focused on pathogenic Neisseria species harboring a genomic island in their di

286 gations, the DNA uptake sequence receptor in Neisseria species has remained elusive.

287 r studying Neisseria-host interactions using Neisseria species indigenous to the animal.

288 haryngeal swab specimens were collected, and Neisseria species were identified by microbiological and

289 an alternative genetic target common to all Neisseria species which can be readily sequenced to prov

290 to the potential detection of nongonococcal Neisseria species.

291 e IgA1 protease that is unique to pathogenic Neisseria spp.

292 12 taxa associated with gum health including Neisseria spp. and a significant decrease in 10 taxa ass

293 ken (n = 112) to ascertain the prevalence of Neisseria spp. following the eighth case of invasive men

294 In the case of Neisseria spp., the importance of the lactate permease i

295 ast to previous studies in the T4P system of Neisseria spp., we found that components of the inner me

296 iptional regulator present in the pathogenic Neisseria that functions as both an activator and a repr

297 at provide insight into the evolution of the Neisseria, the epidemiology of meningococcal and gonococ

298 a concentrative nucleoside transporter from Neisseria wadsworthii.

299 252 remained an effective antibacterial when Neisseria were supplemented with exogenous fatty acids.

300 clinical situations in those genera, such as Neisseria, where some species are associated with diseas