ライフサイエンスコーパス: Neisseria meningitidis

1 nic Neisseria spp. (Neisseria gonorrheae and Neisseria meningitidis).

2 an unexpectedly versatile Cas9 protein from Neisseria meningitidis.

3 nse of primary human meningothelial cells to Neisseria meningitidis.

4 key virulence factor and vaccine antigen of Neisseria meningitidis.

5 rm kinetic studies on PglL, the O-OTase from Neisseria meningitidis.

6 ream infection caused by the human pathogen, Neisseria meningitidis.

7 s catalysed by three DsbA oxidoreductases in Neisseria meningitidis.

8 ond most prevalent outer membrane protein in Neisseria meningitidis.

9 genetic island (GGI), as do a few strains of Neisseria meningitidis.

10 y to polymyxin B, as reported previously for Neisseria meningitidis.

11 nt mechanism of such phase variation (PV) in Neisseria meningitidis.

12 infection with the important human pathogen Neisseria meningitidis.

13 noclonal antibodies (MAbs) with encapsulated Neisseria meningitidis.

14 ival of infections with certain serotypes of Neisseria meningitidis.

15 ndonuclease paralogues in the human pathogen Neisseria meningitidis.

16 onent systems of the obligate human pathogen Neisseria meningitidis.

17 ide to the outer membrane protein complex of Neisseria meningitidis.

18 been described for the pathogenic bacterium Neisseria meningitidis.

19 embrane porin proteins of the human pathogen Neisseria meningitidis.

20 low rates of meningococcal disease caused by Neisseria meningitidis.

21 are it to the 1.50 A structure of nm-HO from Neisseria meningitidis.

22 erica serovar Typhi CT18, and two strains of Neisseria meningitidis.

23 plexes observed for Campylobacter jejuni and Neisseria meningitidis.

24 ecifically inhibit the CRISPR-Cas9 system of Neisseria meningitidis.

25 s with N. lactamica prevents colonization by Neisseria meningitidis.

26 , influenza, Mycobacterium tuberculosis, and Neisseria meningitidis.

27 sing a dynamic transmission model of group A Neisseria meningitidis.

28 cillus anthracis, Neisseria gonorrhoeae, and Neisseria meningitidis.

29 l investigations frequently fail to identify Neisseria meningitidis.

30 ningococcal disease caused by infection with Neisseria meningitidis.

31 ; for Listeria monocytogenes, 0.12/0.12; for Neisseria meningitidis, 0.12/0.25; for Haemophilus spp.,

32 % (3790/6286) of bacterial meningitis cases: Neisseria meningitidis (1350 cases, 22%), Streptococcus

33 le dynamic regulation mechanism observed for Neisseria meningitidis 3-deoxy-d-arabino-heptulosonate 7

34 Neisseria meningitidis, a causative agent of bacterial m

35 allelic profiles to characterize strains of Neisseria meningitidis, a major cause of bacterial menin

36 identified genes required for competence of Neisseria meningitidis, a naturally transformable human

37 of human umbilical vein endothelial cells or Neisseria meningitidis after incubation with human serum

38 e Control and Prevention for the analysis of Neisseria meningitidis and Bordetella bronchiseptica gen

39 clinically relevant bacterial CNS pathogens, Neisseria meningitidis and Borrelia burgdorferi.

40 cillin-tazobactam, cefepime, and gentamicin, Neisseria meningitidis and ceftriaxone, and Haemophilus

41 ownregulates complement activation, binds to Neisseria meningitidis and increases resistance to serum

42 eficiency and compares it to studies done on Neisseria meningitidis and Moraxella catarrhalis; the tw

43 hown that the lipooligosaccharide (LOS) from Neisseria meningitidis and N. gonorrhoeae engages the TL

44 erdin in AP activation on diverse strains of Neisseria meningitidis and N. gonorrhoeae specifically u

45 These would include the beta-proteobacteria Neisseria meningitidis and Neisseria gonnorhoeae, in whi

46 t activity against the Gram-negative species Neisseria meningitidis and Neisseria gonorrheae and impr

47 Both Neisseria meningitidis and Neisseria gonorrhoeae recruit

48 NrrF, a trans-acting sRNA in Neisseria meningitidis and Neisseria gonorrhoeae, has be

49 outer membrane of Neisseria gonorrhoeae and Neisseria meningitidis and sialylates surface lipooligos

50 coccus pneumoniae compared with responses to Neisseria meningitidis and that in each case, the bacter

51 such as the nitric oxide reductase (NorB) of Neisseria meningitidis and the flavohemoglobins (Hmp) of

52 e use the method to analyze the porB gene of Neisseria meningitidis and verify the inferences using p

53 des two pathogens, Neisseria gonorrhoeae and Neisseria meningitidis, and at least 13 species of comme

54 e in vitro against Streptococcus pneumoniae, Neisseria meningitidis, and H. influenzae.

55 ry tract pathogens Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae, but

56 rocesses of pathogenic Neisseria gonorrheae, Neisseria meningitidis, and Haemophilus influenzae.

57 igen) and qPCR for Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae.

58 negative pathogens, including P. aeruginosa, Neisseria meningitidis, and Helicobacter pylori.

59 Vaccines against Streptococcus pneumoniae, Neisseria meningitidis, and Hemophilus influenzae type b

60 ophilus influenzae type b (Hib), serogroup C Neisseria meningitidis, and multiple capsular serotypes

61 2 IgA1 proteases of Haemophilus influenzae, Neisseria meningitidis, and Neisseria gonorrhoeae.

62 ampylobacter jejuni, Haemophilus influenzae, Neisseria meningitidis, and Pasteurella multocida.

63 t human pathogens, Streptococcus pneumoniae, Neisseria meningitidis, and Staphylococcus aureus, can b

64 -MS assay identified Pseudomonas aeruginosa, Neisseria meningitidis, and Staphylococcus aureus; these

65 eported to occur in Lactobacillus plantarum, Neisseria meningitidis, and Streptococcus agalactiae, an

66 Infection with Haemophilus influenzae, Neisseria meningitidis, and Streptococcus pneumoniae cau

67 y species, including Haemophilus influenzae, Neisseria meningitidis, and Streptococcus pyogenes.

68 ee surface-exposed proteins from serogroup B Neisseria meningitidis (App, NhhA, and NadA) identified

69 Neisseria gonorrhoeae and Neisseria meningitidis are closely related organisms tha

70 methods for detecting pharyngeal carriage of Neisseria meningitidis are complex.

71 The HrpAB proteins of Neisseria meningitidis are members of a two-partner secr

72 Several outer membrane proteins of Neisseria meningitidis are subject to phase variation du

73 Proteins secreted by Neisseria meningitidis are thought to play important rol

74 the responses of human tonsil-derived DC to Neisseria meningitidis as a model organism.

75 previously identified lipooligosaccharide on Neisseria meningitidis as an acceptor for complement C4b

76 cterizing bacteria, using the human pathogen Neisseria meningitidis as an example.

77 that initiate complement-mediated killing of Neisseria meningitidis as they enter the bloodstream fro

78 hilus influenzae, Neisseria gonorrhoeae, and Neisseria meningitidis, as well as Moraxella catarrhalis

79 ucture of a bacterial homologue of ASBT from Neisseria meningitidis (ASBT(NM)) at 2.2 A.

80 crystal structure of an ASBT homologue from Neisseria meningitidis (ASBT(NM)) in detergent was repor

81 hat increased phase variation frequencies in Neisseria meningitidis augment transmissibility and inva

82 d O-antigen capsular polysaccharide (CPS) of Neisseria meningitidis B (NmB) have been investigated by

83 Administration to respond to an outbreak of Neisseria meningitidis B at a U.S. university.

84 domonas aeruginosa, Acinetobacter baumannii, Neisseria meningitidis, Bacteroides fragilis, Bacillus a

85 Neisseria meningitidis binds factor H (fH), a key regula

86 Neisseria meningitidis binds the complement downregulati

87 Neisseria gonorrhoeae and Neisseria meningitidis both express the lacto-N-neotetra

88 diated engulfment of Neisseria gonorrheae or Neisseria meningitidis by human cells and can offer deep

89 Invasive bacterial meningitis caused by Neisseria meningitidis can be prevented by active immuni

90 Neisseria meningitidis can be transmitted via asymptomat

91 , we show that the Gram-negative diplococcus Neisseria meningitidis can trigger rapid redistribution

92 h intact, heat-killed cells of Gram-negative Neisseria meningitidis, capsular serogroup C (MenC) or G

93 Cas9 orthologs (including Neisseria meningitidis Cas9 [NmeCas9]) have also been ad

94 Neisseria meningitidis causes 500 000 cases of septicemi

95 Neisseria meningitidis causes bacterial meningitis and s

96 The pathogen Neisseria meningitidis causes disease amongst infants an

97 Neisseria meningitidis changes its capsular phenotype th

98 Among 25 serogroup B Neisseria meningitidis clinical isolates, we identified

99 Native outer membrane vesicles (NOMV) of Neisseria meningitidis consist of intact outer membrane

100 lP, an autotransporter of spherically shaped Neisseria meningitidis contains the molecular informatio

101 lipid A phosphoethanolamine transferase from Neisseria meningitidis, determined to 2.75-A resolution.

102 ainment strategies for outbreaks of invasive Neisseria meningitidis disease are informed by serogroup

103 e (SPR) based biosensor for the detection of Neisseria meningitidis DNA employing Kretschmann configu

104 Neisseria meningitidis employs redundant heme acquisitio

105 Neisseria meningitidis encodes up to five TpsA proteins

106 Lipooligosaccharide (LOS) from Neisseria meningitidis enhances the respiratory burst in

107 ts sporadic nature and the high diversity of Neisseria meningitidis, epidemiological surveillance inc

108 ding patients with Streptococcus pneumoniae, Neisseria meningitidis, Escherichia coli, and Pseudomona

109 m, and understanding the mechanisms by which Neisseria meningitidis evades host innate and acquired i

110 Neisseria meningitidis expresses a two-component TonB-de

111 antibodies raised against sheaths presenting Neisseria meningitidis factor H binding protein (fHbp) a

112 with a radiolabeled lipooligosaccharide from Neisseria meningitidis for binding to LBP or to the clos

113 In the human pathogen Neisseria meningitidis for example, 23 proteins are dedi

114 admission to hospital and identification of Neisseria meningitidis from a sterile site.

115 aim of accurately identifying the bacterium Neisseria meningitidis from species within this genus an

116 ce diversity in the Campylobacter jejuni and Neisseria meningitidis genomes encoded hypothetical prot

117 d duplicate copies in the N. gonorrhoeae and Neisseria meningitidis genomes, respectively.

118 ta that the class III Fic protein NmFic from Neisseria meningitidis gets autoadenylylated in cis, the

119 ced meningitis incidence and carriage due to Neisseria meningitidis group A (MenA).

120 ampaign was launched using a newly developed Neisseria meningitidis group A (NmA) polysaccharide-teta

121 The capsular polysaccharide of Neisseria meningitidis group B (MBPS) is a polymer of al

122 ine containing the N-propionyl derivative of Neisseria meningitidis group B (MenB) capsular polysacch

123 S) or with LPS as a noncovalent complex with Neisseria meningitidis group B outer membrane protein (L

124 xed with the outer membrane protein (OMP) of Neisseria meningitidis group B, induces anti-core glycol

125 l meningitis (Haemophilus influenzae type b, Neisseria meningitidis group C and seven serotypes of St

126 Vaccines against Neisseria meningitidis group C are based on its alpha-2,

127 ) responses to polysaccharides (PS), such as Neisseria meningitidis group C PS (MCPS), are characteri

128 Neisseria meningitidis groups A (GAM) and W135 capsular

129 l with the ability of each ligand to support Neisseria meningitidis growth, with higher affinities ex

130 ncluding pathogens such as Escherichia coli, Neisseria meningitidis, Haemophilus influenzae, and Past

131 Neisseria meningitidis, Haemophilus influenzae, and Stre

132 d with 3 dilutions of Neisseria gonorrhoeae, Neisseria meningitidis, Haemophilus influenzae, and Stre

133 terial meningitis, which is caused mainly by Neisseria meningitidis, Haemophilus influenzae, and Stre

134 terial infections (Streptococcus pneumoniae, Neisseria meningitidis, Haemophilus influenzae, S suis)

135 Group A Neisseria meningitidis has been a major cause of bacteri

136 Neisseria meningitidis has been recognized as a cause of

137 e substrate complexes of heme oxygenase from Neisseria meningitidis has been systematically perturbed

138 Neisseria meningitidis has evolved the ability to contro

139 Neisseria meningitidis has several strategies to evade c

140 ative bacteria, including the human pathogen Neisseria meningitidis, have evolved means to preferenti

141 droxide complex of the protohemin complex of Neisseria meningitidis heme oxygenase (NmHO) have been a

142 identified small-molecule inhibitors of the Neisseria meningitidis HO (nm-HO).

143 Recombinant forms of Neisseria meningitidis human factor H binding protein (f

144 H binding protein (fHbp) is a lipoprotein of Neisseria meningitidis important for the survival of the

145 pneumoniae, GBS, Listeria monocytogenes, or Neisseria meningitidis in cerebrospinal fluid or other n

146 ge infectivity potentiator (rMIP) protein of Neisseria meningitidis induces significant serum bacteri

147 The incidence of Neisseria meningitidis infection decreased from 0.721 pe

148 sential to understanding the epidemiology of Neisseria meningitidis infection.

149 ts used for either therapy or prophylaxis of Neisseria meningitidis infections.

150 ations by using isolates from an outbreak of Neisseria meningitidis infections.

151 Neisseria meningitidis inhibits the alternative pathway

152 We previously reported that Neisseria meningitidis internalization into human brain

153 Neisseria meningitidis is a cause of fatal sepsis and ep

154 Neisseria meningitidis is a commensal microbe that colon

155 Neisseria meningitidis is a commensal of humans that can

156 Neisseria meningitidis is a frequent colonizer of the hu

157 Neisseria meningitidis is a human commensal that can als

158 Neisseria meningitidis is a human pathogen causing bacte

159 Neisseria meningitidis is a human-specific bacterium tha

160 Neisseria meningitidis is a human-specific pathogen and

161 GNA2091 of Neisseria meningitidis is a lipoprotein of unknown funct

162 Neisseria meningitidis is a major causative agent of bac

163 Neisseria meningitidis is a major cause of bacterial men

164 Neisseria meningitidis is a major cause of bacterial men

165 The bacterium Neisseria meningitidis is a major cause of meningitis an

166 Neisseria meningitidis is a major global pathogen causin

167 Neisseria meningitidis is a strict human pathogen that c

168 We show that the closely related bacterium Neisseria meningitidis is also polyploid, while the comm

169 Neisseria meningitidis is an encapsulated pathogen, and

170 Neisseria meningitidis is an important cause of invasive

171 Neisseria meningitidis is an important cause of septicae

172 Neisseria meningitidis is an important cause of septicem

173 Neisseria meningitidis is an important human pathogen th

174 Neisseria meningitidis is an obligate human nasopharynge

175 nt inhibitor factor H (fH) to the surface of Neisseria meningitidis is critical for evasion of innate

176 Although the opportunistic pathogen Neisseria meningitidis is enriched for colonization in t

177 Neisseria meningitidis is infrequently reported as a lab

178 However, the human pathogen Neisseria meningitidis is intrinsically highly resistant

179 Although Neisseria meningitidis is naturally competent and porB g

180 Neisseria meningitidis is one of the leading causes of b

181 Neisseria meningitidis is one of the main agents of bact

182 ron-regulated FetA outer membrane protein of Neisseria meningitidis is one of various outer membrane

183 riation systems of Neisseria gonorrhoeae and Neisseria meningitidis is presented.

184 The human pathogen Neisseria meningitidis is the major causative agent of b

185 The LTTR, CrgA, from the human pathogen Neisseria meningitidis, is upregulated during bacterial-

186 An apparently rare Neisseria meningitidis isolate containing one copy of a

187 Almost all invasive Neisseria meningitidis isolates express capsular polysac

188 ication of the beta-chain heptose residue of Neisseria meningitidis lipo-oligosaccharide.

189 Neisseria meningitidis lipooligosaccharide (LOS) was a p

190 beta-chain heptose (HepII) of the inner-core Neisseria meningitidis lipopolysaccharide (LPS), but it

191 Our whole-genome microarray studies of Neisseria meningitidis MC58 previously identified a set

192 a genomic DNA sequence of lpt3, derived from Neisseria meningitidis MC58, to search the genomic seque

193 nation with glyco-conjugate capsular group C Neisseria meningitidis (Men C) vaccines in infancy.

194 ulation-level protection against serogroup A Neisseria meningitidis (MenA) are unknown.

195 Serogroup B Neisseria meningitidis (MenB) is a major cause of severe

196 absence of an effective vaccine, serogroup B Neisseria meningitidis (MenB) remains a major cause of i

197 Neisseria meningitidis (meningococcus) is a symbiont of

198 ce identity, PilE is structurally similar to Neisseria meningitidis minor pilins PilXNm and PilVNm, r

199 lation of lipopolysaccharide-null mutants in Neisseria meningitidis, Moraxella catarrhalis, and most

200 tococcus pneumoniae, Haemophilus influenzae, Neisseria meningitidis, Mycoplasma pneumoniae, Mycobacte

201 Neisseria meningitidis (N. meningitidis), Streptococcus

202 ion of the main meningitis-causing bacteria, Neisseria meningitidis (N. meningitidis).

203 The human-specific pathogens Neisseria meningitidis, N. gonorrhoea, Haemophilus influ

204 s present within the obligate human pathogen Neisseria meningitidis, NApe and NExo, are important for

205 The three species Neisseria meningitidis, Neisseria gonorrheae, and Neisse

206 nships among three closely related bacteria, Neisseria meningitidis, Neisseria gonorrhoeae and Neisse

207 Neisseria meningitidis (Nm) clonal complex 11 (cc11) lin

208 evasion is an important survival strategy of Neisseria meningitidis (Nm) during colonization and infe

209 Neisseria meningitidis (Nm) is a Gram-negative diplococc

210 Neisseria meningitidis (Nm) is a leading cause of bacter

211 The human pathogen Neisseria meningitidis (Nm) is a leading cause of bacter

212 The human pathogen Neisseria meningitidis (Nm) is known to possess several

213 Neisseria meningitidis (Nm) strains infecting these pati

214 k in 2009 due to a large epidemic of group A Neisseria meningitidis (NmA) meningitis.

215 The conjugate vaccine against serogroup A Neisseria meningitidis (NmA), MenAfriVac, was first intr

216 ing of O2 and CO by the heme oxygenases from Neisseria meningitidis (nmHO) and Pseudomonas aeruginosa

217 The HO from the pathogenic bacterium Neisseria meningitidis (NmHO) possesses a crystallograph

218 terohemin complex of the heme oxygenase from Neisseria meningitidis (NmHO) with respect to the mode o

219 The HO from the pathogenic bacterium Neisseria meningitidis, NmHO, possesses C-terminal His20

220 neither inactivated, unencapsulated, intact Neisseria meningitidis nor Streptococcus agalactiae inhi

221 observed in this loop when compared with the Neisseria meningitidis NT domain structure.

222 lonization of the upper respiratory tract by Neisseria meningitidis occurs despite elicitation of ada

223 membrane (OM) protein that is an ortholog of Neisseria meningitidis Omp85.

224 duals cocolonized by Neisseria lactamica and Neisseria meningitidis One HGT event resulted in the acq

225 al disease caused by ciprofloxacin-resistant Neisseria meningitidis, one in North Dakota and two in M

226 th or without holotoxin, or proteosomes from Neisseria meningitidis outer membrane proteins complexed

227 Carriage for Neisseria meningitidis (P < 0.05) and Neisseria lactamic

228 Neisseria meningitidis phasevarions regulate genes inclu

229 rs of bacterial pathogenic strains including Neisseria meningitidis, Pseudomonas aeruginosa and Esche

230 ion of a purified polysialyltransferase from Neisseria meningitidis (PST(Nm)) to the extracellular en

231 t infectious diseases over the past century, Neisseria meningitidis remains a major causative agent o

232 Neisseria meningitidis represents a pathogen of great pu

233 Inactivation of the misR/misS system in Neisseria meningitidis results in the loss of phosphoryl

234 f our data with those recently published for Neisseria meningitidis revealed that only a small portio

235 asmodium falciparum infections, and virulent Neisseria meningitidis samples.

236 Respiratory pathogens, such as Neisseria meningitidis, secrete site-specific proteases

237 serological correlates of protection against Neisseria meningitidis serogroup A (NmA) in Burkina Faso

238 Neisseria meningitidis serogroup A capsular polysacchari

239 To combat Neisseria meningitidis serogroup A epidemics in the meni

240 An affordable, highly immunogenic Neisseria meningitidis serogroup A meningococcal conjuga

241 The gene from Neisseria meningitidis serogroup A, encoding a putative,

242 belt undergoes recurrent epidemics caused by Neisseria meningitidis serogroup A.

243 The prediction of efficacy of Neisseria meningitidis serogroup B (MenB) vaccines is cu

244 Neisseria meningitidis serogroup B (MnB) is a leading ca

245 Use of recently licensed vaccines against Neisseria meningitidis serogroup B (NmB) will depend par

246 the outer-membrane protein complex (OMPC) of Neisseria meningitidis serogroup B and adsorption to alu

247 The capsular polysaccharide of the pathogens Neisseria meningitidis serogroup B and of Escherichia co

248 this study, we used an acetate auxotroph of Neisseria meningitidis serogroup B to facilitate metabol

249 Candidate Neisseria meningitidis serogroup B vaccines that are bas

250 abeled ([(14)C]-acetate) blebs purified from Neisseria meningitidis serogroup B with either human mon

251 tococcus pneumoniae, Listeria monocytogenes, Neisseria meningitidis serogroup B, Candida albicans, an

252 We characterized five Neisseria meningitidis serogroup C isolates from a Chica

253 (at 2, 3, and 4 months or 2 and 4 months) or Neisseria meningitidis serogroup C monovalent meningococ

254 outbreak of meningococcal disease caused by Neisseria meningitidis serogroup W-135 was identified am

255 d States, South Africa, and Israel caused by Neisseria meningitidis serogroup Y (NmY) was greater tha

256 vasive meningococcal disease (IMD) caused by Neisseria meningitidis serogroup Y has increased in Euro

257 esponse to i.p.-injected intact, heat-killed Neisseria meningitidis, serogroup C (MenC), a gram-negat

258 des of recombinant capsular polymerases from Neisseria meningitidis serogroups A (CsaB) and X (CsxA)

259 Neisseria meningitidis serogroups A and X are among the

260 al virulence determinants of disease causing Neisseria meningitidis species are their extracellular p

261 o human pathogens, Neisseria gonorrhoeae and Neisseria meningitidis, stimulate PS/gammaS processing o

262 The native lipooligosaccharide (LOS) from Neisseria meningitidis strain 89I was analyzed by matrix

263 We prepared an OMV vaccine from a Neisseria meningitidis strain engineered to overexpress

264 Neisseria meningitidis strain H44/76 was modified by exp

265 age prevention against antigenically diverse Neisseria meningitidis strains and to compare this prote

266 Neisseria meningitidis strains are characterized with ML

267 membrane vesicle (OMV) vaccines from mutant Neisseria meningitidis strains engineered to overexpress

268 gainst nine genetically diverse encapsulated Neisseria meningitidis strains expressing subvariants of

269 Laboratory data on 4,735 Neisseria meningitidis strains was collected and reporte

270 mophilus influenzae, Listeria monocytogenes, Neisseria meningitidis, Streptococcus pneumoniae, Strept

271 s, and we do so for seven bacterial species (Neisseria meningitidis, Streptococcus pneumoniae, Strept

272 erized a TE6 thioesterase from the bacterium Neisseria meningitidis Structural analysis with X-ray cr

273 his reveals how the important human pathogen Neisseria meningitidis subverts immune responses by mimi

274 ned H influenzae type b and capsular group C Neisseria meningitidis tetanus toxoid conjugate vaccine

275 iochemical study of the AP endonuclease from Neisseria meningitidis that has allowed us to capture st

276 putative peptidoglycan binding protein from Neisseria meningitidis that has been shown to interact w

277 In Neisseria meningitidis the majority of iron-responsive g

278 OpcA from Neisseria meningitidis, the causative agent of meningoco

279 sm of action has been studied extensively in Neisseria meningitidis, the specific subset of genes tha

280 o the closely related opportunistic pathogen Neisseria meningitidis, there is an absence of adaptive

281 d from the surface of the bacterial pathogen Neisseria meningitidis; they play a key role in adhesion

282 The ability of the human bacterial pathogen Neisseria meningitidis to cause invasive disease depends

283 Neisseria meningitidis, typically a resident of the oro-

284 Neisseria meningitidis use Type IV pili (T4P) to adhere

285 Neisseria meningitidis utilizes capsular polysaccharide,

286 method for the direct quantification of two Neisseria meningitidis vaccine antigens, in mono- and mu

287 A 2.9-kilobase pair locus in Neisseria meningitidis was identified as containing tran

288 A model pathogen, Neisseria meningitidis, was used to validate the technol

289 egative pathogens Haemophilus influenzae and Neisseria meningitidis We hypothesized that activation o

290 ere, using a distinct CRISPR-Cas system from Neisseria meningitidis, we demonstrate efficient targeti

291 ausative agent of meningitis and septicemia, Neisseria meningitidis, we showed that the Pfs reaction

292 n antigenic variation (Av) of two strains of Neisseria meningitidis were determined using an unbiased

293 gosaccharide (LOS) and proteins and LOS from Neisseria meningitidis were examined for MBL binding by

294 known TNF (-308) genotype after exposure to Neisseria meningitidis were measured.

295 cord by a number of microorganisms including Neisseria meningitidis, which can lead to permanent neur

296 a family of outer membrane lipoproteins from Neisseria meningitidis, which elicits bactericidal antib

297 Hyperinvasive lineages of Neisseria meningitidis, which persist despite extensive

298 abeling of hexaacylated endotoxin (LOS) from Neisseria meningitidis with [(13)C]acetate allowed the u

299 we investigate the interaction of PorB from Neisseria meningitidis with TLR2 and describe the direct

300 ve isolates and 25 isolates from carriers of Neisseria meningitidis without disease.