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1 st to control repetitive selfish elements in Neurospora crassa.
2 lue light responses in the filamentous fungi Neurospora crassa.
3 lass of small RNAs in the filamentous fungus Neurospora crassa.
4 protein complexes in the filamentous fungus Neurospora crassa.
5 in the adaptation of blue-light responses in Neurospora crassa.
6 egulator protein from the filamentous fungus Neurospora crassa.
7 f the RNAi pathway in the filamentous fungus Neurospora crassa.
8 idial germination in the filamentous fungus, Neurospora crassa.
9 derived from cosmid libraries of the fungus Neurospora crassa.
10 for most of the light-mediated processes in Neurospora crassa.
11 ement of genes encoding the core histones of Neurospora crassa.
12 sex pheromones of the heterothallic species Neurospora crassa.
13 o the RT of the Mauriceville retroplasmid of Neurospora crassa.
14 ing the sre gene from the filamentous fungus Neurospora crassa.
15 quired for all known blue-light responses in Neurospora crassa.
16 ing regions and to predict gene structure in Neurospora crassa.
17 of 110 kDa) from Saccharomyces cerevisiae or Neurospora crassa.
18 little metabolized in the filamentous fungus Neurospora crassa.
19 nsible for all known cytosine methylation in Neurospora crassa.
20 e gene encoding subunit A of the V-ATPase of Neurospora crassa.
21 evisiae, Candida albicans, Mucor rouxii, and Neurospora crassa.
22 subunit, gna-3, from the filamentous fungus Neurospora crassa.
23 t-induced point mutation (RIP) in the fungus Neurospora crassa.
24 of the nuclear distribution protein RO11 of Neurospora crassa.
25 op-1, from the eukaryotic filamentous fungus Neurospora crassa.
26 recognize consensus GATA elements, exist in Neurospora crassa.
27 it sarcoplasmic reticulum and H+-ATPase from Neurospora crassa.
28 antage of genes controlled by methylation in Neurospora crassa.
29 ine kinase Nik-1 from the filamentous fungus Neurospora crassa.
30 the anx14 gene of the filamentous ascomycete Neurospora crassa.
31 metabolism is a highly regulated process in Neurospora crassa.
32 ein alpha subunits in the filamentous fungus Neurospora crassa.
33 hymidine kinase gene ( tk ) was expressed in Neurospora crassa.
34 ed as a component of the circadian system in Neurospora crassa.
35 and maintenance of regular hyphal growth in Neurospora crassa.
36 uring colony initiation in the fungal model, Neurospora crassa.
37 on selection in the model filamentous fungus Neurospora crassa.
38 rmal hyphal growth in the filamentous fungus Neurospora crassa.
39 PAF26 has been characterized in detail using Neurospora crassa.
40 xual sporulation in the multicellular fungus Neurospora crassa.
41 sexual sporulation in the filamentous fungus Neurospora crassa.
42 he deprotonation of nitroethane catalyzed by Neurospora crassa 2-nitropropane dioxygenase was investi
46 In contrast, the histone modifications in Neurospora crassa, a convenient model organism for multi
48 f the roughly 100 dispersed 5S rRNA genes in Neurospora crassa, a methylated 5S rRNA pseudogene, Psi6
49 cell communication in the filamentous fungus Neurospora crassa, a simple and experimentally amenable
53 of the homolog in the filamentous ascomycete Neurospora crassa affects the circadian clock output, yi
55 ubjected to repeat-induced point mutation in Neurospora crassa and A:T-rich repeated sequences in het
58 ased models were constructed to describe the Neurospora crassa and Drosophila melanogaster circadian
59 rgillus nidulans, Aspergillus fumigatus, and Neurospora crassa and expressed the genes as secreted pr
60 yed stage-specific expression and editing in Neurospora crassa and F. verticillioides Furthermore,F.
61 ATPases) isolated from vacuolar membranes of Neurospora crassa and from chromaffin granule membranes
62 f4, inhibits growth of the ascomycete fungi, Neurospora crassa and Fusarium graminearum, at micromola
64 he ascomycete fungi Fusarium graminearum and Neurospora crassa and induces accumulation of reactive o
68 wealth of sequence information available for Neurospora crassa and other fungi has greatly facilitate
71 Full-length constructs of the proteins of Neurospora crassa and Saccharomyces cerevisiae (ncVDAC a
75 and dewA from Aspergillus nidulans, EAS from Neurospora crassa and ssgA from Metarhizium anisopliae)
76 e filamentous fungi Aspergillus nidulans and Neurospora crassa and the yeast Saccharomyces cerevisiae
77 ferase center (PTC) function was analyzed in Neurospora crassa and wheat germ translation extracts us
78 was initially inoculated with the mycelium (Neurospora crassa), and following the initial incubation
79 e TPP riboswitches in the filamentous fungus Neurospora crassa, and found that one activates and two
80 derstand the role of MAP kinase signaling in Neurospora crassa, and to identify downstream target gen
81 6p of Saccharomyces cerevisiae and CYT-19 of Neurospora crassa are ATP-dependent helicases that funct
82 ic MLEs and that of CMLE from the eukaryotic Neurospora crassa are completely different from that of
83 ons of the Saccharomyces cerevisiae CPA1 and Neurospora crassa arg-2 AAPs using translation extracts
84 frame (uORF) in the 5' leader segment of the Neurospora crassa arg-2 mRNA causes reduced initiation a
86 reading frame (uORF) in the 5'-leader of the Neurospora crassa arg-2 mRNA was reconstituted in a homo
88 ces cerevisiae GCN4, S. cerevisiae CPA1, and Neurospora crassa arg-2, regulation by uORFs controls ex
90 eatures using an in vivo tethering system in Neurospora crassa Artificial recruitment of the H3K9 met
95 ae, S. carlsbergensis, Kluyveromyces lactis, Neurospora crassa, Aspergillus nidulans, and A. flavus.
96 vels of aequorin expression were obtained in Neurospora crassa, Aspergillus niger and Aspergillus awa
99 lamentous fungi, such as the model eukaryote Neurospora crassa, but is absent from the genomes of bak
100 re essential for light-mediated responses in Neurospora crassa, but the molecular mechanisms underlyi
101 requently mutated during the sexual cycle in Neurospora crassa by a process named repeat-induced poin
102 nted these issues in the microbial eukaryote Neurospora crassa by using a "reverse-ecology" populatio
103 r the structure of the ring of c subunits in Neurospora crassa by using data from the crystal structu
109 present a comprehensive dynamic model of the Neurospora crassa circadian clock that incorporates its
114 O system conserved between S. cerevisiae and Neurospora crassa compared with that which has evolved i
115 ues in subunit c of the vacuolar ATPase from Neurospora crassa conferred strong resistance to bafilom
119 used to profile circadian gene expression in Neurospora crassa cultures grown in constant darkness.
123 s studies showed that one of these proteins, Neurospora crassa CYT-18, binds group I introns by using
125 ATPase, we have generated mutant strains of Neurospora crassa defective in six subunits, C, H, a, c,
126 he at the position equivalent to Phe(281) of Neurospora crassa DIM-5 or Phe(1205) of human G9a allows
127 ously shown that a DNA methylation mutant of Neurospora crassa, dim-5 (defective in methylation), has
128 ications is generally unknown, in the fungus Neurospora crassa, DNA methylation acts genetically down
129 d condensed chromatin, "heterochromatin." In Neurospora crassa, DNA methylation depends on trimethyla
131 of a Moco-free eukaryotic NR from the fungus Neurospora crassa, documenting that Moco is necessary an
132 Ornithine decarboxylase (ODC) of the fungus Neurospora crassa, encoded by the spe-1 gene, catalyzes
134 Here we show that in the model organism Neurospora crassa entrainment of the circadian clock, wh
135 Zymoseptoria tritici, Magnaporthe oryzae and Neurospora crassa, exhibited PAMP activity, inducing cel
136 tein purified from one of its natural hosts, Neurospora crassa, exists in a multimeric form and has t
137 erevisiae homologue, although it does have a Neurospora crassa expressed sequence tag homologue.
141 study explores the relative contributions of Neurospora crassa G alpha subunits, gna-1, gna-2, and gn
143 -cell communication and fusion in the fungus Neurospora crassa Genetically identical germinating spor
145 Prior to initiation of this project, the Neurospora crassa genome assembly contained only 3 of th
147 s to control selfish DNA, an analysis of the Neurospora crassa genome sequence reveals a complete abs
148 eport the analysis of a 36-kbp region of the Neurospora crassa genome, which contains homologs of two
153 rupts post-transcriptional gene silencing in Neurospora crassa has been found to affect the homologue
157 i in general other than the model ascomycete Neurospora crassa--has been neglected, leaving this type
159 ental mechanisms in Aspergillus nidulans and Neurospora crassa have been intensively studied, leading
160 e control and function of DNA methylation in Neurospora crassa have led to a greater understanding of
162 ccharomyces pombe and the filamentous fungus Neurospora crassa have served as important model systems
163 an RdRP component of the quelling pathway in Neurospora crassa, have rapidly diverged in evolution at
164 and disiRNA locus DNA methylation (DLDM) in Neurospora crassa Here we show that the conserved exonuc
168 H(+)-ATPases (V-ATPase), inhibited growth of Neurospora crassa in medium adjusted to alkaline pH.
169 tor of vacuolar ATPases, inhibited growth of Neurospora crassa in medium adjusted to pH 7 or above.
170 n X-ray crystal structures of an enzyme from Neurospora crassa in the resting state and of a copper(I
171 ng a genetic screen of the ascomycete fungus Neurospora crassa, in which dynein is nonessential.
175 laser scanning microscopy that a LPMO (from Neurospora crassa) introduces carboxyl groups primarily
185 at heterochromatin in the filamentous fungus Neurospora crassa is marked by cytosine methylation dire
189 n transcription/translation feedback loop in Neurospora crassa is the protein FREQUENCY (FRQ), shown
191 xamined secretion in a temperature-sensitive Neurospora crassa mcb mutant that shows a loss of growth
192 ent studies showed that the splicing of some Neurospora crassa mitochondrial group I introns addition
195 rystal structure of a C-terminally truncated Neurospora crassa mitochondrial tyrosyl-tRNA synthetase
199 hage T4 td intron to test the ability of the Neurospora crassa mitochondrial tyrosyl-tRNA synthetase
202 nsertional mutagenesis approach, a series of Neurospora crassa mutants affected in the ability to con
204 studies on the Schizosaccharomyces pombe and Neurospora crassa Nbp2p orthologues and the high conserv
205 with the corresponding dicer-like genes from Neurospora crassa [Ncdcl-1 (50.5%); Ncdcl-2 (38.0%)] and
207 is study, we have characterized an LPMO from Neurospora crassa (NcLPMO9C; also known as NCU02916 and
210 a conserved amino-terminal domain within the Neurospora crassa negative regulator sulfur controller-2
211 ning sensors, such as BarA and TorS; and the Neurospora crassa Nik-1 (Os-1) sensor that contains a ta
213 owed the highest degree of similarity to the Neurospora crassa nrc-1, Schizosaccharomyces pombe byr2
217 s that are expressed in mycelial cultures of Neurospora crassa over the course of the circadian day,
221 viously reported that the filamentous fungus Neurospora crassa possesses a Galpha protein, GNA-1, tha
223 Genomes with good genetic maps, such as Neurospora crassa, provide a means for reducing ambiguit
224 to RNA-directed RNA polymerase (RdRP) and to Neurospora crassa QDE-1, two proteins implicated in post
226 We show that the model cellulolytic fungus Neurospora crassa relies on a high-affinity cellodextrin
229 analysis of how the model filamentous fungus Neurospora crassa responds to the three main cell wall p
230 estigating the introns of the model organism Neurospora crassa revealed a different organization at t
231 he genome sequence of the filamentous fungus Neurospora crassa reveals a gene number very much higher
232 FEX KOs in three eukaryotic model organisms, Neurospora crassa, Saccharomyces cerevisiae, and Candida
233 tic studies of Schizosaccharomyces pombe and Neurospora crassa show that these types of enzymes are i
234 central component of the circadian clock in Neurospora crassa, shows daily cycles that are exquisite
235 ntly found that NOP-1, a membrane protein of Neurospora crassa, shows homology to haloarchaeal rhodop
237 cell extracts derived from MacroD-deficient Neurospora crassa strain exhibit a major reduction in th
238 e polymorphisms (SNPs) between the reference Neurospora crassa strain Oak Ridge and the Mauriceville
239 implement our algorithm on a real dataset of Neurospora crassa strains, using the genetic and geograp
240 th an MTS derived from S. cerevisiae OXA1 or Neurospora crassa SU9, both coding for hydrophobic mitoc
241 n Mss116 and the related protein Cyt-19 from Neurospora crassa suggest that these proteins form a sub
243 ral potential new PMO families in the fungus Neurospora crassa that are likely to be active on novel
244 tions among the circadian clock mutations of Neurospora crassa that indicate possible physical intera
245 me-mediated TER 3'-end cleavage mechanism in Neurospora crassa that is distinct from that found speci
246 critical component of the circadian clock of Neurospora crassa that regulates the abundance of its co
252 lletotrichum graminicola, the model organism Neurospora crassa, the human pathogen Sporothrix schenck
257 e cloned and characterized the dim-2 gene of Neurospora crassa, the only eukaryotic gene currently kn
260 haromyces pombe, and one filamentous fungus, Neurospora crassa-three species that arguably are not re
261 (Hi-C) with wild-type and mutant strains of Neurospora crassa to gain insight into the role of heter
263 therefore used the model filamentous fungus Neurospora crassa to search for uncharacterized transcri
264 uction of the NPS6 ortholog from the saprobe Neurospora crassa to the Deltanps6 strain of C. heterost
265 acultative and constitutive heterochromatin, Neurospora crassa, to explore possible interactions betw
266 f tigA from Aspergillus niger and erp38 from Neurospora crassa, two novel members of the PDI superfam
267 During meiosis in the filamentous fungus Neurospora crassa, unpaired genes are identified and sil
273 ite-directed mutagenesis at sites throughout Neurospora crassa VDAC (naturally devoid of cysteine) we
276 ntromere region of linkage group (LG) VII of Neurospora crassa was cloned previously from a yeast art
277 y, the ncd-2 gene encoding for the enzyme in Neurospora crassa was cloned, expressed in Escherichia c
280 development) gene of the filamentous fingus Neurospora crassa was identified as a gene expressed pre
281 In this research, the urease-positive fungus Neurospora crassa was investigated for the biomineraliza
282 gene encoding a novel GATA factor, ASD4, of Neurospora crassa was isolated and demonstrated to posse
283 tation, the conidiation rhythm of the fungus Neurospora crassa was monitored in constant darkness dur
285 of the clock in the circadian model organism Neurospora crassa We show that, in a ras2-deficient stra
286 tween H3S10p, H3K9me, and DNA methylation in Neurospora crassa, we built and tested mutants of the pu
287 by allelic differences at the het-c locus of Neurospora crassa, we isolated mutants that suppressed p
288 lear movement in the model ascomycete fungus Neurospora crassa, we show that genetic diversity is mai
290 lopsis (formerly Mortierella) ramanniana and Neurospora crassa were introduced into maize using an em
291 y are acutely active in the meiotic cells of Neurospora crassa, where they evaluate the mutual identi
293 g regulation by a fungal TPP riboswitch from Neurospora crassa, which is mostly located in a large in
294 salinarium Bat, Azotobacter vinelandii NIFL, Neurospora crassa White Collar-1, Escherichia coli Aer,
298 eacetylase 1 (HDA1) mutant (hda-1) strain of Neurospora crassa with inactivated histone deacetylase 1
299 of sequence data from the filamentous fungus Neurospora crassa with the complete genome sequence of S
300 in single cells of the model fungal system, Neurospora crassa, with droplet microfluidics and the us
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