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1 ion (55%) against virus challenge in outbred New Zealand White rabbits.
2 eal keratocytes isolated from the corneas of New Zealand White rabbits.
3 ) was performed on the left eyes of 20 adult New Zealand White rabbits.
4 induced by coronary artery ligation in adult New Zealand White rabbits.
5 ne (n = 5) were given in a volume of 2 ml to New Zealand white rabbits.
6 natured Oms66 (hOms66) were used to immunize New Zealand White rabbits.
7 li was deposited onto the abraded corneas of New Zealand White rabbits.
8 a major organ in catabolism of Lp B-70.5 in New Zealand white rabbits.
9 next to each other in both thighs in 10 male New Zealand white rabbits.
10 by using confocal microscopy in vivo in six New Zealand White rabbits.
11 Seventeen anesthetized adult male New Zealand White rabbits.
12 The coronary ligation model was used in New Zealand white rabbits.
13 nted them into carotid arteries of recipient New Zealand White rabbits.
14 tilated, pump-perfused lungs from euthanized New Zealand White rabbits.
15 ed on anaesthetized, artificially ventilated New Zealand White rabbits.
16 ging experimental atherosclerotic lesions in New Zealand White rabbits.
17 ained from the lacrimal glands of adult male New Zealand White rabbits.
18 ies treated with mitomycin-C were created in New Zealand White rabbits.
19 lly into the vitreous of the right eye of 10 New Zealand White rabbits.
20 DL)-mediated endothelial dysfunction in male New Zealand White rabbits.
21 ted into the vitreous of the right eye of 10 New Zealand White rabbits.
22 e tubercle bacilli in commercially available New Zealand white rabbits.
23 loon-damaged, flow-reduced iliac arteries of New Zealand White rabbits.
24 rmal or previously denuded iliac arteries of New Zealand White rabbits.
25 ameter) were performed using microneedles in New Zealand white rabbits.
26 twelve 3-month-old and ten 12-month-old male New Zealand White rabbits.
27 e created at the root of the right CCA in 16 New Zealand white rabbits.
28 hemia and hemorrhage in large clot embolized New Zealand white rabbits.
29 , obtained from both Sprague-Dawley rats and New Zealand White rabbits.
30 superficial femoral artery was induced in 14 New Zealand white rabbits.
31 Experiments were performed on New Zealand White rabbits.
32 -8 weeks) and aged (approximately 30 months) New Zealand White rabbits.
33 A) to detect atherosclerotic lesions in male New Zealand White rabbits.
34 r samples from normal and dry eyes of female New Zealand White rabbits.
35 ently available methods, of measuring IOP in New Zealand White rabbits.
36 es obtained from naive untreated and treated New Zealand White rabbits.
37 asuring IOP in the range of 0 to 50 mm Hg in New Zealand White rabbits.
38 g growth and invasion inhibitory antisera in New Zealand White rabbits.
39 artery angioplasty was performed in 25 male New Zealand White rabbits.
40 tracellularly in retinas isolated from adult New Zealand White rabbits.
41 al division of the medial geniculate body of New Zealand white rabbits.
46 ed by a 0.5% cholesterol diet for 16 wk in 7 New Zealand White rabbits; 5 unmanipulated rabbits, fed
47 noparticles-47 [MION-47]) in cholesterol-fed New Zealand White rabbits 6 months after balloon injury.
49 endrimer eye drop formulation was studied in New Zealand White rabbits after a single dose or multipl
50 distribution of carboplatin was examined in New Zealand White Rabbits after a single intravenous inf
52 rnal administration of aluminum maltolate to New Zealand white rabbits, an animal system relevant to
55 ed from gluteal biopsies from volunteers and New Zealand White rabbits and mounted on a wire myograph
56 llow microneedle was performed in the eye of New Zealand White rabbits and was found to reduce IOP in
61 A single retinal detachment was produced in New Zealand White rabbits by injecting approximately 50
63 wing large clot embolism-induced ischemia in New Zealand white rabbits by intracisternally administer
66 of detrusor myocytes following PBOO in male New Zealand White rabbits compared to that of controls.
69 onocclusive periarterial carotid collar into New Zealand White rabbits decreased collar-induced endot
70 eated 40 calvarial defects in 20 12-week-old New Zealand White rabbits, divided into four groups: (1)
71 s were taken from the hippocampi of 10 male, New Zealand white rabbits during classical discriminatio
72 ere topically administered to eyes of normal New Zealand White rabbits either as a single dose or thr
73 ts of simvastatin on measures of behavior in New Zealand white rabbits embolized using a suspension o
76 aced into experimental saccular aneurysms in New Zealand White rabbits failed to elicit a fibrotic re
78 d alternative, via sterol 27-hydroxylase) in New Zealand white rabbits fed 3 g cholesterol/per day fo
80 cular injection (2 microg/kg body weight) to New Zealand White rabbits fed with a 0.25% cholesterol d
81 ramagnetic nanoparticles were compared in 30 New Zealand White rabbits (four to six rabbits per group
84 IgG polyclonal antiserum was generated in New Zealand White rabbits immunized with a 16-mer peptid
89 ribe here a comparison of inbred and outbred New Zealand White rabbits infected by aerosol with eithe
90 HTLV-I infection and further establishes the New Zealand White rabbit inoculated with the RH/K34 cell
91 ere performed on 22 human, 29 monkey, and 34 New Zealand White rabbit intact postmortem lenses in sit
92 of radiolabeled arterial LDL was measured in New Zealand White rabbits killed at several different ti
93 Ad.nNOS in atherosclerotic arteries, 10 male New Zealand White rabbits maintained on a 1% cholesterol
94 toradiographic technique, frozen sections of New Zealand white rabbit medulla were incubated with rad
102 lerotic plaques were induced in the aorta of New Zealand White rabbits (n = 7) by a repeated balloon
111 erol diet) and NEP-I-treated cholesterol-fed New Zealand White rabbits (n=8; candoxatril, 30 mg/kg pe
118 lbumin nanoparticles (nPXL) was tested in 38 New Zealand White rabbits receiving bilateral iliac arte
119 mics, and morphology was investigated in the New Zealand White rabbit renal autograft model using nea
120 ree virus from the J94356(PBMC) cell line in New Zealand White rabbits results in seroconversion to t
123 was performed on the iliac arteries of male New Zealand White rabbits that were fed an atherogenic d
124 ed from other locations were fed on dogs and New Zealand White rabbits to assess the competency of th
125 In vivo studies were performed on mice and New Zealand White rabbits to assess the effect of GM-CSF
126 intraarticularly to the knee joints of five New Zealand white rabbits to determine the in vivo diffu
128 s and lactate concentration were measured in New Zealand White rabbits treated with the topical CA in
130 scribe the early response (< or =days) in 52 New Zealand White rabbits undergoing gene transfer (beta
133 a after varying forms of arterial injury, 57 New Zealand White rabbits underwent iliac artery balloon
137 ized, prospective, masked-observer study, 40 New Zealand White rabbits underwent modified glaucoma fi
141 hted imaging were performed on 79% gestation New Zealand white rabbits using a 3-Tesla magnetic reson
143 Experimental periodontitis was induced in 21 New Zealand White rabbits using Porphyromonas gingivalis
144 n this animal care committee-approved study, New Zealand white rabbit VX2 liver tumors were treated t
149 ligation of distal external iliac artery in New Zealand White rabbits, we directly injected 500 micr
161 Male and female Sprague-Dawley rats and New Zealand White rabbits were divided into equal groups
166 dysregulation of DDAH also occurred in vivo, New Zealand White rabbits were fed normal chow or a high
167 enous NO in the regulation of MCP-1 in vivo, New Zealand White rabbits were fed normal chow, normal c
177 Anesthetized, tracheotomized, and ventilated New Zealand White rabbits were injected with freshly pre
186 an globes or eyes obtained from euthanatized New Zealand White rabbits were mounted in a perfusion ch
194 e corneal epithelia in one eye of 8-week-old New Zealand White rabbits were scraped at weekly interva
199 ing sutural mineralization fronts in 8 young New Zealand White rabbits were subjected to nano-indenta
201 rtery from adult male and nonpregnant female New Zealand White rabbits were suspended in organ baths
206 s (HSFs) were used for in vitro studies, and New Zealand White rabbits were used for in vivo studies.
207 CSG) were deployed in the iliac arteries of New Zealand White rabbits, which were killed 28 days aft
209 yclonal antibodies were raised by immunizing New Zealand White rabbits with a mixture of the gel frag
210 Experimental periodontitis was induced in New Zealand white rabbits with silk sutures tied around
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