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1 mmunization against both avian influenza and Newcastle disease.
2 d widely to protect village chickens against Newcastle disease, due to their decreased dependence on
4 olytic NDV variant that is unlikely to cause Newcastle disease in its avian host, representing an ess
8 rains of Newcastle disease virus (NDV) cause Newcastle disease (ND), a devastating disease of poultry
9 icken infectious laryngotracheitis (ILT) and Newcastle disease (ND), two of the most economically imp
10 the response of DCs to virus infection with Newcastle disease virus (NDV) after a 24-hour E2 treatme
11 VLP is composed of the NP and M proteins of Newcastle disease virus (NDV) and a chimeric protein con
14 he level of IFN-beta protein induced by both Newcastle disease virus (NDV) and Sendai virus infection
17 linical development of a mesogenic strain of Newcastle disease virus (NDV) as an oncolytic agent for
21 Virus-like particles (VLPs) built on the Newcastle disease virus (NDV) core proteins, NP and M, a
22 h virus-like particles (VLPs) containing the Newcastle disease virus (NDV) core proteins, NP and M, a
23 several paramyxoviruses.IMPORTANCE Oncolytic Newcastle disease virus (NDV) could establish persistent
27 respiratory tract based on recombinant avian Newcastle disease virus (NDV) expressing the hemagglutin
28 such a stimulus, we generated a recombinant Newcastle disease virus (NDV) expressing the MV hemagglu
29 iotin-labeled peptides with sequences of the Newcastle disease virus (NDV) F protein heptad repeat 2
30 and 289 in the structure and function of the Newcastle disease virus (NDV) F protein was explored by
31 the basis of the coordinates of the related Newcastle disease virus (NDV) F protein, Valine-94, a de
32 ith those of the parainfluenza virus SV5 and Newcastle disease virus (NDV) F proteins, the structures
33 has been demonstrated that the V protein of Newcastle disease virus (NDV) functions as an alpha/beta
37 It has been shown that the L289A-mutated Newcastle disease virus (NDV) fusion (F) protein gains t
39 equences in the transmembrane (TM) domain of Newcastle disease virus (NDV) fusion (F) protein in the
40 The role of N-linked glycosylation of the Newcastle disease virus (NDV) fusion (F) protein in vira
43 y viral fusion proteins, the sequence of the Newcastle disease virus (NDV) fusion protein has several
46 onstrate that the F-interactive sites on the Newcastle disease virus (NDV) hemagglutinin-neuraminidas
50 alysis of the three-dimensional structure of Newcastle disease virus (NDV) HN protein revealed the pr
52 tion, mutations in a conserved domain in the Newcastle disease virus (NDV) HN stalk result in a sharp
55 morbidity-mortality event involving virulent Newcastle disease virus (NDV) in wild double-crested cor
58 hemagglutinin-neuraminidase (HN) protein of Newcastle disease virus (NDV) is a multifunctional prote
61 hemagglutinin-neuraminidase (HN) protein of Newcastle disease virus (NDV) is an important determinan
67 hemagglutinin-neuraminidase (HN) protein of Newcastle disease virus (NDV) plays a crucial role in th
71 tinin-neuraminidase (HN) cytoplasmic tail in Newcastle disease virus (NDV) replication and pathogenic
73 Ps) released from avian cells expressing the Newcastle disease virus (NDV) strain AV proteins NP, M,
74 The complete genome sequence of an African Newcastle disease virus (NDV) strain isolated from a chi
76 onomic losses, but little is known about the Newcastle disease virus (NDV) strains circulating in Afr
80 ystallographic structure of an HN dimer from Newcastle disease virus (NDV) suggests that a single sit
84 he present study, we generated a recombinant Newcastle disease virus (NDV) vectoring the fusion (F) p
87 ression of the fusion glycoprotein of RSV by Newcastle disease virus (NDV) would stimulate a more rob
88 xpression, replication, and pathogenicity of Newcastle disease virus (NDV), a green fluorescent prote
90 Pteropus vampyrus bat kidney (PVK) cells to Newcastle disease virus (NDV), an avian paramyxovirus kn
99 the F proteins of SV5 (W3A and WR isolates), Newcastle disease virus (NDV), and human parainfluenza v
100 cular stomatitis virus (VSV), Sindbis virus, Newcastle disease virus (NDV), and Sendai virus (SeV), w
101 of the H5 and H7 hemagglutinin subtypes, and Newcastle disease virus (NDV), are important pathogens i
103 es such as avian metapneumovirus (aMPV), and Newcastle disease virus (NDV), human pathogens such as h
104 Here we find that intratumoral therapy with Newcastle disease virus (NDV), in addition to the activa
105 yxovirus 1 (APMV-1) RNA, also referred to as Newcastle disease virus (NDV), in clinical samples from
108 Virulent and moderately virulent strains of Newcastle disease virus (NDV), representing avian paramy
109 DCs showed a reduced type I IFN response to Newcastle disease virus (NDV), Sendai virus (SeV), and S
111 ns (UTRs) in replication and pathogenesis of Newcastle disease virus (NDV), we generated recombinant
112 is hypothesis, L929 cells were infected with Newcastle disease virus (NDV), which led to the inductio
116 e effects of lipopolysaccharide (LPS) on the Newcastle disease virus (NDV)-mediated induction of cyto
120 ion with vectored vaccine, using recombinant Newcastle disease virus (rNDV) expressing glycoprotein D
123 me this obstacle, we generated a recombinant Newcastle disease virus (rNDV)-vectored experimental nor
128 We found that matrix proteins purified from Newcastle disease virus adsorb on a phospholipid bilayer
129 d RNA viruses that have dsRNA intermediates, Newcastle disease virus and Sendai virus, and a DNA viru
134 ns for the ability to rescue the growth of a Newcastle disease virus expressing green fluorescent pro
135 mutational analysis of the HR1 domain of the Newcastle disease virus fusion protein, focusing on the
137 on of HN of parainfluenza virus 5 (PIV5) and Newcastle disease virus HN abolishes cell-cell fusion, w
139 embrane fusion, we characterized a series of Newcastle disease virus HN proteins whose surface residu
140 ith functionally active "headless" mumps and Newcastle disease virus HN proteins, provide insights in
144 d receptor binding site of the paramyxovirus Newcastle disease virus in its function and its relation
145 le NF-kappaB activity, both Sendai virus and Newcastle disease virus infection led to robust IFN-beta
150 opathological characterization of a virulent Newcastle disease virus isolate (NDV-Peru/08) obtained f
151 ed to mimic sites that are found in virulent Newcastle disease virus isolates and to contain 4 or 5 b
154 hemagglutinin-neuraminidase (HN) protein of Newcastle disease virus mediates attachment to sialic ac
158 rs by about 2.5-fold, and neuraminidase from Newcastle disease virus typically increased infectivity
159 the growth of a highly IFN-sensitive virus (Newcastle disease virus) in the presence of IFN, suggest
160 aramyxovirus 1 (APMV-1; a group encompassing Newcastle disease virus), which is a highly contagious p
162 omography to show that the matrix protein of Newcastle disease virus, a paramyxovirus and relative of
163 mutation is demonstrated in the F protein of Newcastle disease virus, a paramyxovirus of a different
164 nduced by all IL-33-inducing agonists except Newcastle disease virus, a RIG-I agonist that induced ex
165 from non-target viruses such as H6N2, H9N2, Newcastle disease virus, and infectious bronchitis virus
167 ense RNA viruses (influenza viruses A and B, Newcastle disease virus, and vesicular stomatitis virus)
168 simplex virus type-1 (HSV-1), Sendai virus, Newcastle disease virus, and vesicular stomatitis virus.
169 PV701, a replication-competent strain of Newcastle disease virus, causes regression of tumor xeno
170 paramyxoviruses measles virus, mumps virus, Newcastle disease virus, human parainfluenza virus 3, an
171 DCs) by negative-strand RNA viruses, such as Newcastle disease virus, leads to the induction of the I
172 ed against CPE resulting from infection with Newcastle disease virus, Sendai virus, canine distemper
174 y this network, we studied DCs infected with Newcastle disease virus, which is able to stimulate inna
175 in primary human dendritic cells infected by Newcastle disease virus, with copy numbers varying from
176 nd tumor necrosis factor alpha (TNFalpha) in Newcastle disease virus-infected Irf5(-)(/)(-) mice.
177 not type I interferon gene transcription in Newcastle disease virus-infected peritoneal macrophages.
185 Using reverse genetics, three recombinant Newcastle disease viruses (rNDVs) were engineered, each
186 packaging revealed a majority of infectious Newcastle disease viruses contain one functional genome.
188 ession after infection with either Sendai or Newcastle disease viruses or after engagement of the Tol
189 W proteins) were expressed from recombinant Newcastle disease viruses, and the responses of infected
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