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1 ells infected with a heterologous RNA virus (Newcastle disease virus).
2 ing a control or fusion protein derived from Newcastle disease virus.
3 idase (HN) glycoprotein of the paramyxovirus Newcastle disease virus.
4 onse to HSV-1, but not to Sendai virus or to Newcastle disease virus.
5 d to rescue replication of the IFN-sensitive Newcastle disease virus.
6 nts similar to the one for cells infected by Newcastle disease virus.
7 omography to show that the matrix protein of Newcastle disease virus, a paramyxovirus and relative of
8 mutation is demonstrated in the F protein of Newcastle disease virus, a paramyxovirus of a different
9 nduced by all IL-33-inducing agonists except Newcastle disease virus, a RIG-I agonist that induced ex
10 We found that matrix proteins purified from Newcastle disease virus adsorb on a phospholipid bilayer
11 d RNA viruses that have dsRNA intermediates, Newcastle disease virus and Sendai virus, and a DNA viru
14 from non-target viruses such as H6N2, H9N2, Newcastle disease virus, and infectious bronchitis virus
16 ense RNA viruses (influenza viruses A and B, Newcastle disease virus, and vesicular stomatitis virus)
17 simplex virus type-1 (HSV-1), Sendai virus, Newcastle disease virus, and vesicular stomatitis virus.
18 W proteins) were expressed from recombinant Newcastle disease viruses, and the responses of infected
20 PV701, a replication-competent strain of Newcastle disease virus, causes regression of tumor xeno
21 packaging revealed a majority of infectious Newcastle disease viruses contain one functional genome.
23 ns for the ability to rescue the growth of a Newcastle disease virus expressing green fluorescent pro
24 mutational analysis of the HR1 domain of the Newcastle disease virus fusion protein, focusing on the
26 on of HN of parainfluenza virus 5 (PIV5) and Newcastle disease virus HN abolishes cell-cell fusion, w
28 embrane fusion, we characterized a series of Newcastle disease virus HN proteins whose surface residu
29 ith functionally active "headless" mumps and Newcastle disease virus HN proteins, provide insights in
33 paramyxoviruses measles virus, mumps virus, Newcastle disease virus, human parainfluenza virus 3, an
34 d receptor binding site of the paramyxovirus Newcastle disease virus in its function and its relation
35 the growth of a highly IFN-sensitive virus (Newcastle disease virus) in the presence of IFN, suggest
36 nd tumor necrosis factor alpha (TNFalpha) in Newcastle disease virus-infected Irf5(-)(/)(-) mice.
37 not type I interferon gene transcription in Newcastle disease virus-infected peritoneal macrophages.
39 le NF-kappaB activity, both Sendai virus and Newcastle disease virus infection led to robust IFN-beta
44 opathological characterization of a virulent Newcastle disease virus isolate (NDV-Peru/08) obtained f
46 ed to mimic sites that are found in virulent Newcastle disease virus isolates and to contain 4 or 5 b
49 DCs) by negative-strand RNA viruses, such as Newcastle disease virus, leads to the induction of the I
51 hemagglutinin-neuraminidase (HN) protein of Newcastle disease virus mediates attachment to sialic ac
52 the response of DCs to virus infection with Newcastle disease virus (NDV) after a 24-hour E2 treatme
53 VLP is composed of the NP and M proteins of Newcastle disease virus (NDV) and a chimeric protein con
56 he level of IFN-beta protein induced by both Newcastle disease virus (NDV) and Sendai virus infection
59 linical development of a mesogenic strain of Newcastle disease virus (NDV) as an oncolytic agent for
63 Virus-like particles (VLPs) built on the Newcastle disease virus (NDV) core proteins, NP and M, a
64 h virus-like particles (VLPs) containing the Newcastle disease virus (NDV) core proteins, NP and M, a
65 several paramyxoviruses.IMPORTANCE Oncolytic Newcastle disease virus (NDV) could establish persistent
69 respiratory tract based on recombinant avian Newcastle disease virus (NDV) expressing the hemagglutin
70 such a stimulus, we generated a recombinant Newcastle disease virus (NDV) expressing the MV hemagglu
71 iotin-labeled peptides with sequences of the Newcastle disease virus (NDV) F protein heptad repeat 2
72 and 289 in the structure and function of the Newcastle disease virus (NDV) F protein was explored by
73 the basis of the coordinates of the related Newcastle disease virus (NDV) F protein, Valine-94, a de
74 ith those of the parainfluenza virus SV5 and Newcastle disease virus (NDV) F proteins, the structures
75 has been demonstrated that the V protein of Newcastle disease virus (NDV) functions as an alpha/beta
79 It has been shown that the L289A-mutated Newcastle disease virus (NDV) fusion (F) protein gains t
81 equences in the transmembrane (TM) domain of Newcastle disease virus (NDV) fusion (F) protein in the
82 The role of N-linked glycosylation of the Newcastle disease virus (NDV) fusion (F) protein in vira
85 y viral fusion proteins, the sequence of the Newcastle disease virus (NDV) fusion protein has several
88 onstrate that the F-interactive sites on the Newcastle disease virus (NDV) hemagglutinin-neuraminidas
92 alysis of the three-dimensional structure of Newcastle disease virus (NDV) HN protein revealed the pr
94 tion, mutations in a conserved domain in the Newcastle disease virus (NDV) HN stalk result in a sharp
97 morbidity-mortality event involving virulent Newcastle disease virus (NDV) in wild double-crested cor
100 hemagglutinin-neuraminidase (HN) protein of Newcastle disease virus (NDV) is a multifunctional prote
103 hemagglutinin-neuraminidase (HN) protein of Newcastle disease virus (NDV) is an important determinan
109 hemagglutinin-neuraminidase (HN) protein of Newcastle disease virus (NDV) plays a crucial role in th
113 tinin-neuraminidase (HN) cytoplasmic tail in Newcastle disease virus (NDV) replication and pathogenic
115 Ps) released from avian cells expressing the Newcastle disease virus (NDV) strain AV proteins NP, M,
116 The complete genome sequence of an African Newcastle disease virus (NDV) strain isolated from a chi
118 onomic losses, but little is known about the Newcastle disease virus (NDV) strains circulating in Afr
122 ystallographic structure of an HN dimer from Newcastle disease virus (NDV) suggests that a single sit
126 he present study, we generated a recombinant Newcastle disease virus (NDV) vectoring the fusion (F) p
129 ression of the fusion glycoprotein of RSV by Newcastle disease virus (NDV) would stimulate a more rob
130 xpression, replication, and pathogenicity of Newcastle disease virus (NDV), a green fluorescent prote
132 Pteropus vampyrus bat kidney (PVK) cells to Newcastle disease virus (NDV), an avian paramyxovirus kn
141 the F proteins of SV5 (W3A and WR isolates), Newcastle disease virus (NDV), and human parainfluenza v
142 cular stomatitis virus (VSV), Sindbis virus, Newcastle disease virus (NDV), and Sendai virus (SeV), w
143 of the H5 and H7 hemagglutinin subtypes, and Newcastle disease virus (NDV), are important pathogens i
145 es such as avian metapneumovirus (aMPV), and Newcastle disease virus (NDV), human pathogens such as h
146 Here we find that intratumoral therapy with Newcastle disease virus (NDV), in addition to the activa
147 yxovirus 1 (APMV-1) RNA, also referred to as Newcastle disease virus (NDV), in clinical samples from
150 Virulent and moderately virulent strains of Newcastle disease virus (NDV), representing avian paramy
151 DCs showed a reduced type I IFN response to Newcastle disease virus (NDV), Sendai virus (SeV), and S
153 ns (UTRs) in replication and pathogenesis of Newcastle disease virus (NDV), we generated recombinant
154 is hypothesis, L929 cells were infected with Newcastle disease virus (NDV), which led to the inductio
158 e effects of lipopolysaccharide (LPS) on the Newcastle disease virus (NDV)-mediated induction of cyto
164 ession after infection with either Sendai or Newcastle disease viruses or after engagement of the Tol
166 ion with vectored vaccine, using recombinant Newcastle disease virus (rNDV) expressing glycoprotein D
169 me this obstacle, we generated a recombinant Newcastle disease virus (rNDV)-vectored experimental nor
170 Using reverse genetics, three recombinant Newcastle disease viruses (rNDVs) were engineered, each
171 ed against CPE resulting from infection with Newcastle disease virus, Sendai virus, canine distemper
175 rs by about 2.5-fold, and neuraminidase from Newcastle disease virus typically increased infectivity
178 aramyxovirus 1 (APMV-1; a group encompassing Newcastle disease virus), which is a highly contagious p
179 y this network, we studied DCs infected with Newcastle disease virus, which is able to stimulate inna
180 in primary human dendritic cells infected by Newcastle disease virus, with copy numbers varying from
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