1 Next, we applied the sensitization hypothesis to our model, w
2 Next, we asked whether monkeys would generalize the middle co
3 Next, we carried out a validation study using paired DBS-whol
4 Next, we characterize the in vivo function of a circRNA gener
5 Next, we characterized the domains of MtBzaC and reconstitute
6 Next, we chemically fuse two clusters to realize a larger clu
7 Next, we compared the gene expression profiles across the thr
8 Next, we conducted deconvolutional analyses to comprehensivel
9 Next, we correlated the afterpotentials with the cells' prope
10 Next, we demonstrate that intrahippocampal infusion of the pr
11 Next, we demonstrate that the same pessimistic model but with
12 Next, we describe the consequences of the presence of long st
13 Next, we determined how many of the patients met the same cri
14 Next, we determined significantly correlated TF-gene/miRNA an
15 Next, we determined the connectivity of individual M1 sites w
16 Next, we determined the network of brain regions functionally
17 Next, we discuss mechanisms by which histone variants influen
18 Next, we discuss the roles of BRs in development and stress r
19 Next, we examine the important role the glycocalyx plays in d
20 Next, we explored the possibility to culture cells and tissue
21 Next, we found that activin A regulates phosphorylation of NM
22 Next, we generated an activity-based small-molecule probe (AB
23 Next, we illustrate applications intended to improve individu
24 Next, we measured functional conservation between mouse and h
25 Next, we measured VDAC in a separate task by presenting these
26 Next, we mimicked BNST G(i)-coupled alpha(2a)-AR heterorecept
27 Next, we pharmacogenetically stimulated PVN -> NAc neurons an
28 Next, we predicted how muscle weakness may cause deviations f
29 Next, we propose a theoretical link between two key decision-
30 Next, we propose R2 measures for the PH and PO models that ca
31 Next, we review different strategies that have been reported
32 Next, we review the evolution of the most complete GEM that h
33 Next, we review the modality and somatotopic arrangements of
34 Next, we screened four large cohorts of infertile men and ide
35 Next, we selected a constitutive SFFV promoter and NFkappaB b
36 Next, we show that chemogenetically inhibiting D1 medium spin
37 Next, we show that human iPSC-derived motor neurons are more
38 Next, we show that the affinity of a single molecule of profi
39 Next, we show that the increased SR load in atrial myocytes p
40 Next, we show that the iron transporter Tsf1 is induced by in
41 Next, we show that the physical mechanism of internalization
42 Next, we studied a separate group of HD patients who were pre
43 Next, we summarize recent anatomical, behavioral, and physiol
44 Next, we summarize the bottom-up and top-down fabrication app
45 Next, we tested these gene sets on their relevance to human t
46 Next, we used an anatomical disconnection procedure to determ
47 Next, we used optogenetic inhibition of VTA TH neurons to sho
48 Next, we used our methods to analyze metagenomics data from 1
49 Next, we used PET/MRI to define uptake in six brain regions.
50 Next, we used three CNN-LSTM models with a different set of s