1 Next, we analyzed the oxidative stress in the pancreata.
2 Next, we built a decision tree to predict the presence of EGF
3 Next, we characterized the unique expression of coding, nonco
4 Next, we computed the changes in Ca(2+) affinity for CaM with
5 Next, we conducted a detailed spatiotemporal characterization
6 Next, we confirmed the ability of Plg/Pla to both promote eff
7 Next, we created a logistic regression model, controlling for
8 Next we deliberate on the mechanical, optical, electrical, an
9 Next, we demonstrate convergent NeanderScore-related findings
10 Next, we demonstrated that conditioned medium from HCV-infect
11 Next, we demonstrated that Tfeb and Tfe3 act directly by bind
12 Next, we describe the main modulatory drives of sleep and wak
13 Next, we develop a modeling framework that leverages transfer
14 Next, we discovered through modeling that such projection pat
15 Next, we discuss the molecular mechanisms of phosphatidylseri
16 Next, we discuss the rationale and potential benefits of risk
17 Next, we discuss the use of biomarkers and how clinicians may
18 Next we evaluate known human congenital heart diseases: cardi
19 Next, we examined relations between HMO and maternal anthropo
20 Next, we explore factors that have impeded our understanding
21 Next, we follow the projections of Ir68a neurons to the brain
22 Next, we identified network patterns that distinguish treatme
23 Next, we identified the mechanism by which the engulfment of
24 Next, we map the cell states within each species and also bet
25 Next we microinjected forty-five embryos each with five sgRNA
26 Next, we monitored the lifetime fitness of the terrestrial ad
27 Next, we observed the endothelium structure lost its integrit
28 Next, we provide an overview of data sources available for we
29 Next, we quantified CDH1 promoter methylation levels in CDH1
30 Next, we queried the transcriptomes and inferred dynamic Baye
31 Next, we questioned if these infant USV were also emitted in
32 Next, we review current data regarding the effect of domains
33 Next, we screened a cohort of hereditary spastic paraplegia a
34 Next, we show how the method can be used to simultaneously pr
35 Next, we show that jointly modeling pleiotropic risk regions
36 Next, we show that structure-independent processes, already p
37 Next we showed that brief activation of STN projection neuron
38 Next, we studied how optogenetic stimulation of these project
39 Next, we studied sham- and LHb-lesioned rats in our operant C
40 Next, we test for entrainment of neural oscillations to visua
41 Next, we tested the enzymatic activity of a recombinant mutan
42 Next, we tested the evolutionary specificity of this interact
43 Next, we trained SVM models with polynomial kernel and obtain
44 Next, we transplanted cardiac MSCs from TLR4(-/-) and WT male
45 Next we use optogenetic and chemogenetic approaches to show t
46 Next, we use a biophysically realistic computational model to
47 Next, we used a climate-envelope approach to project how N-fi
48 Next, we used a forward encoding model to construct a channel
49 Next, we used a novel in silico genomic analysis, searchable
50 Next, we will shift our focus to hyperpolarized probe design