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1 tochrome oxidase or stained for cell bodies (Nissl stain).
2 ere depressed with age (PDI and intensity of Nissl staining).
3 tained cellular composition as visualized by Nissl stain.
4 ll brains were stained with Cresyl violet, a Nissl stain.
5 hitecture of the VMH was not recognizable by Nissl stain.
6 ze of mammalian brain regions as revealed by Nissl stains.
7 e defined by histological markers other than Nissl staining.
8 ppaB, protein disulfide isomerase (PDI), and Nissl staining.
9 of neurons in brain sections was examined by Nissl staining.
10 P days) 4 and 6 using cytochrome oxidase and Nissl staining.
11 s determined by MAP2 immunocytochemistry and Nissl staining.
12 eatments were evaluated using cresyl violet (Nissl) staining.
13 orphology were assessed using Cresyl violet (Nissl) staining.
15 MGV) has cellular laminae visible in routine Nissl stains, allowing a direct comparison of the lamina
17 n of a rhesus and 11 cynomolgus monkeys with Nissl staining and immunohistochemistry for neuron-speci
18 rew's brain, we sectioned brains and applied Nissl staining and NeuN (neuronal nuclei) antibody stain
22 ent regions of the ganglion cell layer using Nissl staining and tetramethylrhodamine dextran amine ba
25 of all vocal nuclei, as measured using both Nissl-stained and NADPH-d-stained material, as well as t
27 chitecture of the VMH became recognizable by Nissl stain as GABAergic fibers invaded the nucleus, con
30 ion-level asymmetry in the areal fraction of Nissl-stained cell bodies, a finding that differs from p
31 ations, we performed stereologic analyses of Nissl-stained cells in layer III of area DSCF in mustach
33 e marker tyrosine hydroxylase (TH), 60% more Nissl-stained cells, and 40% less norepinephrine transpo
35 ination of Tbr1 and Lmx1a immunolabeling and Nissl staining confirmed the loss of CN neurons from the
36 ach is demonstrated by (i) development of a "Nissl stain" contrast agent for cellular imaging, (ii) v
41 ions and SMI-32 immunohistochemistry than in Nissl stain, especially for area 2 and subdivisions of a
44 and necrosis to neuronal injury in adjacent Nissl-stained, hematoxylin and eosin-stained, and termin
45 ts, epileptic kainate-treated rats had fewer Nissl-stained hilar neurons and fewer somatostatin-immun
46 At 7 d, traumatic brain lesion volumes on Nissl-stained histological sections were significantly s
47 ranule cell axon reorganization and by using Nissl staining, immunocytochemistry, and the optical fra
49 s of NeuN and DARPP-32 immunoreactivity, and Nissl staining, in the striatum showed no striatal neuro
50 ed SE, bromodeoxyuridine (BrdU) labeling and Nissl staining increased in the rostral forebrain SVZ.
51 D) interneuron numbers without diminution in Nissl-stained interneuron numbers, indicating loss of GA
52 of the cellular architecture as derived from Nissl-stained material and in terms of the dendritic pro
53 al region (STR) in the human was analyzed in Nissl-stained material to see whether auditory cortex is
55 d boutons were seen in close apposition with Nissl-stained motoneurons in the Vmo, VII, XII and Amb.
58 2 oscillations cause significant decrease in NISSL-stained neurons (p < 0.05) and induce inflammation
60 ts, an average of approximately one-third of Nissl-stained neurons and one-third of the GABAergic int
62 Stereology was used to estimate numbers of Nissl-stained neurons per hippocampus in the granule cel
64 a subnuclear cytoarchitectonic parcellation (Nissl stain) of the NST into rostral, intermediate, and
65 gic criteria in coronal and oblique sections Nissl stained or immunoreacted for neuron-specific nucle
66 reticulospinal (RS) neurons were missing in Nissl-stained or neurofilament-immunostained brain whole
67 sections from each tissue block were either Nissl-stained or stained with antibodies against somatos
68 d stereological estimates of cell numbers in Nissl-stained, paraffin-wax sections of brain, we invest
73 d with other histochemical markers including Nissl stains, retrogradely transported fluorescent trace
76 the overall neuronal density was measured on Nissl stained sections and the proportion of CR+ and PV+
77 g (S) than autumn (A), in both the PG21- and Nissl-stained sections (S:A = 1.9 and 1.7, respectively)
78 dopaminergic cell survival were confirmed in Nissl-stained sections and by quantitation of retrograde
79 temporale and Heschl's gyrus was assessed on Nissl-stained sections by semi-automated microscope imag
81 ally toward stereological analyses of thick, Nissl-stained sections for the homotypical cortex of the
83 ually dimorphic male nucleus (MN) is seen in Nissl-stained sections from the dorsal preoptic area/ant
84 d koniocellular (K) layers was calculated in Nissl-stained sections from the LGN of adult marmosets (
85 alling near OD column borders are evident in Nissl-stained sections in all layers and in immunoreacti
87 neuron number and soma size were assessed in Nissl-stained sections of spinal cord segments T17-S1 of
88 differences in DCN organization, we compared Nissl-stained sections of the DCN in five different spec
89 ogical striatal volume analyses performed on Nissl-stained sections revealed that rats transplanted w
91 used to estimate numbers of granule cells in Nissl-stained sections so that numbers of excitatory syn
93 d somatosensory cortices of the mouse, using Nissl-stained sections to define laminar boundaries.
96 logical counting procedures on immunoreacted Nissl-stained sections, CR+ neurons were estimated to co
103 lar layer and granule cell layer and loss of Nissl-stained, somatostatin-immunoreactive, and parvalbu
104 ed by alpha-bungarotoxin binding to numerous Nissl-stained structures in CA1 lacunosum/moleculare in
105 DA cells/section by immunohistochemistry and Nissl staining [TH-labeled cells and thionin-stained cel
106 sparrows (Zonotrichia leucophrys gambelii): Nissl staining, the distribution of acetylcholinesterase
107 re removed after perfusion and processed for Nissl staining, the samples were randomized, and the inv
108 esion was confirmed following examination of Nissl-stained tissue sections at all times post-lesion a
109 ution of NeuN-labeled neurons was similar to Nissl-stained tissue, with the exception of some areas w
110 sed SMI-32 immunohistochemistry, myelin, and Nissl stains to characterize the architecture of the par
111 size of song nuclei have relied primarily on Nissl stains to define the borders of these regions.
114 n after 6 hours of ischemia, as evaluated by Nissl staining, was significantly less in HSP70tg mice c
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