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1 ere depressed with age (PDI and intensity of Nissl staining).
2 P days) 4 and 6 using cytochrome oxidase and Nissl staining.
3 ppaB, protein disulfide isomerase (PDI), and Nissl staining.
4 s determined by MAP2 immunocytochemistry and Nissl staining.
5 e defined by histological markers other than Nissl staining.
6 of neurons in brain sections was examined by Nissl staining.
7 eatments were evaluated using cresyl violet (Nissl) staining.
8 orphology were assessed using Cresyl violet (Nissl) staining.
9 roke brain in the same rat was determined by Nissl staining 28 days after surgery.
10 n of a rhesus and 11 cynomolgus monkeys with Nissl staining and immunohistochemistry for neuron-speci
11 rew's brain, we sectioned brains and applied Nissl staining and NeuN (neuronal nuclei) antibody stain
12                                              Nissl staining and nicotinamide adenine dinucleotide pho
13  complete (for NF-kappaB) to very minor (for Nissl staining and PDI).
14                                              Nissl staining and terminal deoxynucleotidyl transferase
15 ent regions of the ganglion cell layer using Nissl staining and tetramethylrhodamine dextran amine ba
16                        In this study we used Nissl staining and tyrosine hydroxylase (TH) immunoreact
17          Neuropathological analyses included Nissl-staining and immunohistochemistry to detect APPs,
18 in medium, propidium iodide (PI) uptake, and Nissl staining as markers of cell damage.
19 ination of Tbr1 and Lmx1a immunolabeling and Nissl staining confirmed the loss of CN neurons from the
20                                              Nissl staining, cytochrome oxidase activity, and neurofi
21 /kg i.p.) also protected against neocortical Nissl-staining deficits.
22                                Visualized by Nissl staining, foetal grafts formed clumps of pyramidal
23 ranule cell axon reorganization and by using Nissl staining, immunocytochemistry, and the optical fra
24 s of NeuN and DARPP-32 immunoreactivity, and Nissl staining, in the striatum showed no striatal neuro
25 ed SE, bromodeoxyuridine (BrdU) labeling and Nissl staining increased in the rostral forebrain SVZ.
26  nucleus and are not merely artifacts of the Nissl-staining method.
27                                              Nissl staining, NeuN immunocytochemistry, and in situ hy
28                                              Nissl staining of retinae from normal adult and 5,7-DHT-
29                                              Nissl staining showed pronounced tissue body loss with m
30                  Histological analyses using Nissl staining showed that in NHPs with the aortic arch
31 DA cells/section by immunohistochemistry and Nissl staining [TH-labeled cells and thionin-stained cel
32  sparrows (Zonotrichia leucophrys gambelii): Nissl staining, the distribution of acetylcholinesterase
33 re removed after perfusion and processed for Nissl staining, the samples were randomized, and the inv
34                               In this study, Nissl staining was used to investigate whether the marke
35 n after 6 hours of ischemia, as evaluated by Nissl staining, was significantly less in HSP70tg mice c
36                      NMDA-induced changes in Nissl staining were also different in slices from young
37                              Disturbances of Nissl staining were finally evident at 48 h, with nearly

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