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1                                        Lesch-Nyhan disease (LND) is an X-linked genetic disorder caus
2                                        Lesch-Nyhan disease (LND) is caused by congenital deficiency o
3                                        Lesch-Nyhan disease (LND) is caused by deficiency of the purin
4                                        Lesch-Nyhan Disease (LND) is the result of mutations in the X-
5                                        Lesch-Nyhan disease and its attenuated variants are caused by
6                                        Lesch-Nyhan disease is a neurogenetic disorder caused by defic
7                                        Lesch-Nyhan disease is a rare, devastating, X-linked recessive
8                                        Lesch-Nyhan disease is an X-linked recessive disorder that is
9                                        Lesch-Nyhan syndrome encompasses a host of neurological sympto
10 rior reports describing a total of 127 Lesch-Nyhan disease variants.
11 ciency states such as Parkinsonism and Lesch-Nyhan disease.
12 ely model Parkinson's disease (PD) and Lesch-Nyhan syndrome (LNS).
13 logical conditions such as cholera and Lesch-Nyhan syndrome.
14  pathway, and mutations in HPRT1 cause Lesch-Nyhan disease.
15 ectly contacts the Mg(2+) ions, causes Lesch-Nyhan syndrome (HGPRT(Kinston), D193N).
16 the spectrum are patients with classic Lesch-Nyhan disease and the full clinical phenotype.
17 (DA) deficiency has been implicated in Lesch-Nyhan disease (LND), a genetic disorder that is characte
18 germ-line HPRT mutations identified in Lesch-Nyhan syndrome or hyperuricemia patients.
19 n-dopaminergic neuronal development in Lesch-Nyhan syndrome.
20  abnormalities in adulthood that mimic Lesch-Nyhan disease, schizophrenia, and other developmental di
21  the use of female hiPSCs for modeling Lesch-Nyhan syndrome.
22 use the intractable neurodevelopmental Lesch-Nyhan disease (LND) associated with aberrant development
23 anifestations of the largest cohort of Lesch-Nyhan disease variants evaluated to date, with 46 patien
24  6-hydroxydopamine (6-OHDA; a model of Lesch-Nyhan syndrome (LNS)) or vehicle (unlesioned rats).
25 transferase (HGPRTase) is the locus of Lesch-Nyhan syndrome, the activator of the prodrugs 6-mercapto
26 )/trisomy 21, and the carrier state of Lesch-Nyhan syndrome.
27 oral abnormalities associated with the Lesch-Nyhan syndrome as previously suggested.
28 T) deficiency in humans results in the Lesch-Nyhan syndrome which is characterized, among other featu
29 order in the HPRT gene (leading to the Lesch-Nyhan syndrome) is unique to human, chimpanzee, and gori
30 ehavioral symptoms associated with the Lesch-Nyhan syndrome.
31        These factors are not unique to Lesch-Nyhan disease, and are relevant to most other genetic di
32 mutations in the human HPRT leading to Lesch-Nyhan syndrome (D193N) or gout (S103R or S109L), as the
33 nlikely to contribute substantially to Lesch-Nyhan syndrome burden.
34 PRT1, the housekeeping gene underlying Lesch-Nyhan syndrome, an X-linked recessive disorder.
35 ions of the brains of 12 patients with Lesch-Nyhan disease (age, 10 to 20 years) and 15 healthy contr
36                          Patients with Lesch-Nyhan disease have abnormally few dopaminergic nerve ter
37 ntum; 57 percent) in the patients with Lesch-Nyhan disease than in the controls.

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