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1 eptor site present in the DNA repair protein O6-alkylguanine-DNA alkyltransferase.
2 reversal of the damage by a suicide protein, O(6)-alkylguanine-DNA alkyltransferase.
3 MG and D-245 MG (PR) xenografts displayed no O6-alkylguanine-DNA alkyltransferase activity, and their
4 ical responses proportional to inhibition of O6-alkylguanine-DNA alkyltransferase activity, but a max
5 n of various ethylene crosslinks of DNA with O(6) -alkylguanine-DNA alkyltransferase (AGT, see pictur
6 iviral vector encoding the DNA repair enzyme O(6)-alkylguanine DNA alkyltransferase (AGT) from the O(
8 ansfer-active and -inactive mutants of human O(6)-alkylguanine DNA alkyltransferase (AGT) show that i
10 obacco-specific nitrosamines are repaired by O(6)-alkylguanine DNA alkyltransferase (AGT), which tran
11 l motifs with the cancer chemotherapy target O(6)-alkylguanine-DNA alkyltransferase (AGT) and paradox
23 sion of the C-terminal active site domain of O(6)-alkylguanine-DNA alkyltransferase (AGT) with an end
24 )-BG, an inhibitor of the resistance protein O(6)-alkylguanine-DNA alkyltransferase (AGT), were synth
26 urea therapy involves the DNA repair protein O(6)-alkylguanine-DNA alkyltransferase (AGT), which remo
28 that O(6)-POB-dG can be directly repaired by O(6)-alkylguanine-DNA alkyltransferase (AGT), which tran
36 yl (Bz) guanine in oligonucleotides by human O6-alkylguanine DNA alkyltransferase (AGT) were estimate
37 ere, we describe the involvement of BRCA2 in O6-alkylguanine DNA alkyltransferase (AGT)-mediated repa
40 express low levels of the DNA repair protein O6-alkylguanine-DNA alkyltransferase (AGT) and are sensi
41 ethyltransferase (MGMT), also referred to as O6-alkylguanine-DNA alkyltransferase (AGT) correlate wit
48 sly as a continuous infusion that suppresses O6-alkylguanine-DNA alkyltransferase (AGT) levels in bra
55 nactivation of the human DNA repair protein, O6-alkylguanine-DNA alkyltransferase (AGT), by O6-benzyl
56 potent inhibitor of the DNA repair protein, O6-alkylguanine-DNA alkyltransferase (AGT), has been sho
59 itrosourea therapy is the DNA repair protein O6-alkylguanine-DNA alkyltransferase (AGT), which remove
60 tionship of this resistance to expression of O6-alkylguanine-DNA alkyltransferase (AGT), which repair
64 tion and depletion of the DNA repair protein O6-alkylguanine-DNA-alkyltransferase (AGT) and increases
65 ovel class of DNA repair proteins related to O(6)-alkylguanine-DNA alkyltransferases (AGTs) that tigh
66 than 200-fold less active as inactivators of O(6)-alkylguanine-DNA alkyltransferase (alkyltransferase
68 inactivators of the human DNA repair protein O6-alkylguanine-DNA alkyltransferase (alkyltransferase)
70 h O6-benzylguanine (O6-BeG) on the levels of O6-alkylguanine-DNA alkyltransferase (ATase) in the hema
76 zylguanine is an irreversible inactivator of O(6)-alkylguanine-DNA alkyltransferase currently in clin
77 ed recently that a polymorphism in the human O6-alkylguanine-DNA alkyltransferase gene exists, with a
78 crystallographic study of recombinant human O(6)-alkylguanine-DNA alkyltransferase (hAGT) revealed a
80 s established by expression of human protein O(6)-alkylguanine-DNA alkyltransferase (hAGT), in which
81 sis that the level of the DNA repair protein O6-alkylguanine-DNA alkyltransferase in brain tumors was
82 as been proposed that the DNA repair protein O6-alkylguanine-DNA alkyltransferase increases the mutag
83 was significantly lower in the cells lacking O6-alkylguanine-DNA alkyltransferase, indicating that O6
84 phase II trial of carmustine (BCNU) plus the O(6)-alkylguanine-DNA alkyltransferase inhibitor O(6)-be
86 porter protein (human chorionic gonadotropin-O(6) -alkylguanine-DNA alkyltransferase) led to LAMP-to-
90 -alkylguanine adducts in DNA are repaired by O6-alkylguanine-DNA alkyltransferases (MGMT) by transfer
91 repair defects on stem cell function include O(6)-alkylguanine DNA alkyltransferase, nucleotide excis
92 N-terminal addition of SNAP or CLIP forms of O(6)-alkylguanine-DNA-alkyltransferase plus a peptide ep
94 s observed that the DNA repair protein human O(6)-alkylguanine-DNA alkyltransferase repairs lesions a
95 strain MISU-1.1, differing only in level of O6-alkylguanine-DNA alkyltransferase, were treated with
96 ontain low levels of the DNA repair protein, O6-alkylguanine-DNA alkyltransferase, which may explain
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